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1 sm [1, 2] and can change size and shape upon sexual arousal.
2 he first IN, thus indicating a high level of sexual arousal.
3 rginase plays a role in both male and female sexual arousal.
4 tructures whose activation is time-locked to sexual arousal.
5 ch as regulating K+ channels and stimulating sexual arousal.
6 10) effect on subjective ratings of sexual arousal.
7 tion was linearly dependent on self-reported sexual arousal.
8 y altered vaginal immune responses following sexual arousal.
9 such as maternal behavior, reproduction, and sexual arousal.
10 ors, such as sexual behaviors that depend on sexual arousal?
11 tter volume of brain regions associated with sexual arousal (3,723 adults) did not show any causal re
12 d responses in brain regions associated with sexual arousal (amygdala, hypothalamus, and ventral stri
14 increases in "activation" symptoms (energy, sexual arousal, and diminished sleep) (r = 0.55; P =.02)
15 t the brain, including areas associated with sexual arousal, and in diverse non-neural and reproducti
17 edial temporal lobe is centrally involved in sexual arousal, and this circuit may be implicated in or
18 e associations between alterations of female sexual arousal as well as vaginal lubrication and the si
19 nt results regarding the motor correlates of sexual arousal demonstrating an early freezing response
22 at hypoactive sexual desire disorder, female sexual arousal disorder, female orgasmic disorder and fe
27 To develop efficacious treatments for female sexual arousal dysfunction, researchers need to differen
29 ivation can be counteracted by raising their sexual arousal, either by engaging the flies with prolon
30 esire (EST1 = .48 and EST2 = .72; P < .001), sexual arousal (EST2 = .50; P = .008), and vaginal lubri
31 ted.Genes and circuits involved in sleep and sexual arousal have been extensively studied in Drosophi
32 e relationships between brain activation and sexual arousal in a group of young, healthy, heterosexua
36 impact of various neurological disorders on sexual arousal in women and to develop appropriate manag
40 bed nucleus of the stria terminalis (BST) in sexual arousal, inferred from noncontact erection (NCE)
45 er sexual desire (MnD, -0.3; P < .01), lower sexual arousal (MnD, -0.3; P < .01), lower sexual satisf
47 pretation that sexual conditioning increases sexual arousal or receptivity in both sexes but the incr
49 a symptoms were largely associated with poor sexual arousal, orgasmic dysfunction, sexual distress, a
50 (P = .003) and orgasm (P = .006) in men and sexual arousal (P = .05) and sexual satisfaction (P = .0
51 ion compared with placebo, (2) physiological sexual arousal (penile tumescence), and (3) behavioral m
53 ies have shown that not all women who report sexual arousal problems have decreased genital arousal,
55 muli revealed large networks correlated with sexual arousal, spanning multiple cortical and subcortic
56 sexual motivation and ratings of subjective sexual arousal to and enjoyment of an auditory stimulus.
57 ile viewing the films, participants' vaginal sexual arousal was recorded using vaginal photoplethysmo
58 Sexual sadists show increased peripheral sexual arousal when observing other individuals in pain.
59 ETH affects courtship behavior by increasing sexual arousal while decreasing successful sexual perfor
61 ignificant increase in vaginal IgA following sexual arousal, while women with CSA histories had a dec