ライフサイエンスコーパス: sham

1 was treated similarly except without e-stim (sham).

2 reased new dentate gyrus neurons relative to Sham.

3 of active iTBS, or 2-weeks if randomized to sham.

4 nd lower in the Disruptive condition, versus Sham.

5 nal biomechanical properties were similar to sham.

6 MS (F(1,13) = 14.108, P = 0.009) compared to sham.

7 nal forearm sNVT were similar between IH and sham.

8 omary resuscitation strategies (hippocampus: sham, 0.4 +/- 0.2; cardiopulmonary resuscitation, 1.7 +/

9 /- 26 pg/mL; p < 0.05) and heme oxygenase-1 (sham, 1 +/- 0.1; cardiopulmonary resuscitation, 2.5 +/-

10 ng, 35.6+/-8.7, P=0.71; phosphocreatine/ATP: sham, 1.22+/-0.23 debanding, 1.11+/-0.24, P=0.59; ATP/AD

11 dative stress (dihydroethidium fluorescence: sham, 1.6x10(8)+/-6.1x10(7), banding, 2.6x10(8)+/-4.5x10

12 arly-diastolic annular velocity ratio, E/E': sham, 13.6+/-2.1, banding, 18.5+/-4.1, P=0.014) accompan

13 r compared to those originally randomized to sham (2-weeks active iTBS); log-rank ChiSq = 5.871, df =

14 .4% and 9.4 kg/8.3% loss, respectively) than sham (3.0 kg/2.8% and 1.9 kg/1.8%, respectively; p = 0.0

15 anding towards sham values (phosphocreatine: sham, 38.4+/-7.4, debanding, 35.6+/-8.7, P=0.71; phospho

16 .23 debanding, 1.11+/-0.24, P=0.59; ATP/ADP: sham, 6.2+/-0.9, debanding, 5.6+/-1.6, P=0.66).

17 significantly reduced with FAi compared with sham (65.5% vs. 97.6%, respectively; P < 0.001); time to

18 e was significantly greater than that for P7 shams (691.6 kPa vs 429.2 kPa, p = 0.0194).

19 rol group, but less (P < 0.01) than those in Sham-A and VH groups on day 22.

20 Goats in the Sham-A group were inserted with needles for 0.5 h at the

21 hibited lower (P < 0.01) VH as compared with Sham-A or VH group.

22 ts were allocated into VH, Sham acupuncture (Sham-A) and EA groups, while goats treated with saline i

23 Goats of Sham-A, VH or EA group showed higher (P < 0.01) VH at da

24 ders the potential value and challenges of a sham ablation randomized trial.

25 mly assigned to either acupuncture (n=42) or sham acupuncture (n=42) groups.

26 TNBS-injected goats were allocated into VH, Sham acupuncture (Sham-A) and EA groups, while goats tre

27 ted with significantly lower scores than the sham acupuncture group at week 12 and during the 20-week

28 nd of treatment between real acupuncture and sham acupuncture group was -2.38 (95% CI, -3.46 to -1.30

29 tively) in the acupuncture group than in the sham acupuncture group.

30 ntrol group A, or Streitberger non-insertion sham acupuncture in the control group B.

31 Both CDH-sham and CDH-TO fetuses treated with placebo had an incr

32 C57BL/6 mice were implanted with GL261/Sham and given TMZ/Saline.

33 compared to pre-stimulation, and compared to sham and left frontopolar stimulation.

34 Implants remained in place in SHAM and OVX ewes but were lost in all ZOL ewes.

35 P < 0.05), and showed no differences between SHAM and OVX.

36 e volume were measured prior to sacrifice of sham and REL+veh or REL+rapa, and PBO.

37 Four groups were studied: sham and SCI plus phosphate-buffered saline (SCI + PBS),

38 nsity, and trabeculae number were similar in SHAM and ZOL, and lower in OVX (P < 0.05).

39 Sham animals served as controls.

40 free gingival grafts (FGG) harvest, received sham application of electrotherapy.

41 fferences were found between lampalizumab or sham arms for changes from baseline in functional assess

42 Sham arms were pooled for analysis.

43 Mice infected with RV-A1B at day 6 and sham at day 13 showed an increased number of bronchoalve

44 ) and cathodal tDCS (ctDCS), CT, sham CT and sham brain stimulation.

45 in a reduction of gastric volume relative to sham, but none of the interventions were as effective as

46 rtical proteins, many of which differed from sham by 8 hours post-mTBI, particularly GAS-1 and VEGF-B

47 On Day 20, sham calves avoided the lidocaine-paired stimulus, consi

48 Day 0 sham calves did not avoid the lidocaine-paired stimulus,

49 dy-weight ratio was significantly reduced in sham-CDH fetuses either (1.2 +/- 0.3% vs 2.3 +/- 0.3% in

50 asured before and during a low-stress event (sham clipping) and compared with heart rate variability

51 mg cohort compared with their corresponding sham cohorts.

52 TIDieR to assist the reporting of placebo or sham components and the trials in which they are used.

53 ither nocturnal oxygen or ambient air from a sham concentrator (placebo).

54 Current Stimulation to the rATL (active and sham condition) or the left frontopolar region while par

55 fter real neurofeedback as compared with the sham control (p < .05).

56 chondrial function as in the spinal cords of sham control animals, it significantly attenuated mitoch

57 and from 24.8 +/- 8.1 to 23.3 +/- 8.5 in the sham control condition.

58 ced clinical allergy scores to 1 from 3.5 in sham control mice (P < .001) after 6 treatments accompan

59 with Ad26 vectors that encoded S variants or sham control, and then challenged with SARS-CoV-2 by the

60 ely to engage in feeding and grooming than a sham control.

61 ither bleomycin to induce ALI or saline as a sham control.

62 The lack of a formal sham-control renal denervation procedure should be consi

63 ignificantly change (AAA SUV=0.86+/-0.17 and sham-control SUV=0.46+/-0.10), independent of variations

64 In a patient-assessor-blind, randomized and sham controlled trial, 90 depression patients with insom

65 Here, we performed a randomized, sham-controlled clinical trial and measured electrophysi

66 Using a within-participants, double-blinded, sham-controlled crossover design, we recorded EEG while

67 ranial magnetic stimulation in a randomized, sham-controlled design.

68 We conducted a double-blind sham-controlled study on 20 human volunteers (10 females

69 ion.SIGNIFICANCE STATEMENT This double-blind sham-controlled study suggested that neurofeedback train

70 12-21 years) were enrolled in a randomized, sham-controlled trial of TMS across 13 sites.

71 In the present double-blind, randomized, sham-controlled trial, 105 patients with recurrent depre

72 international, prospective, single-blinded, sham-controlled trial, done at 44 study sites in Austral

73 Double-blinded sham-controlled trials are needed to confirm the remissi

74 ed therapy for hypertension, and randomized, sham-controlled trials have provided proof-of-principle

75 be confirmed in larger, adequately powered, sham-controlled trials.

76 were enrolled in a double-blind, randomized, sham-controlled, crossover study involving two sessions

77 This double-blind, sham-controlled, randomised controlled trial provides cl

78 We conducted a double-blind, sham-controlled, randomised controlled trial to assess s

79 shown to affect study outcomes, placebo and sham controls are rarely reported and often not matched

80 Placebo or sham controls are the standard against which the benefit

81 of both active interventions and placebo or sham controls to be concisely summarised by researchers.

82 Rats were divided into sham controls, and two groups surviving post-lesion for

83 Compared to sham controls, at one week post-SAC systolic blood press

84 Without adequate descriptions of placebo or sham controls, it is difficult to interpret results abou

85 as2 mutant females and confirmed that unlike sham controls, ovariectomized mutant mice showed no incr

86 espectively, as compared with viral loads in sham controls.

87 ory responses after injury without impacting sham controls.

88 d over the same recording period in surgical sham controls.

89 ntricular ejection fraction (LVEF), versus 5 sham controls.

90 0.2) of PVC-CM and recovered animals versus sham controls.

91 0.91+/-0.25) was approximately twice that of sham-controls (SUV=0.47+/-0.10; P<0.01).

92 tDCS (atDCS) and cathodal tDCS (ctDCS), CT, sham CT and sham brain stimulation.

93 We show that, relative to sham, cTBS targeting this network impairs reward-related

94 Kohn-Sham density functional theory (DFT) is a standard tool

95 In Kohn-Sham DFT simulations, the balance between accuracy and c

96 ence for analgesia in 44 calves disbudded or sham-disbudded 6 hours (Day 0) or 20 days (Day 20) befor

97 Relative to sham, e-stim training increased DG volume by approximate

98 We compared short (3.4 s) trials of tVNS to sham electrical stimulation at the earlobe (far from the

99 apid left ventricular dilation compared with sham (end-systolic volume, day 2: 40.6 +/- 10.2 muL vs.

100 mized 2:1:2:1 to lampalizumab every 4 weeks, sham every 4 weeks, lampalizumab every 6 weeks, or sham

101 very 4 weeks, lampalizumab every 6 weeks, or sham every 6 weeks.

102 rom EHS and as late as 30 days compared with sham exercise controls.

103 ges were significantly greater than those in SHAM + FD animals (-5.5 D and 40 mum of elongation after

104 nderwent either sham surgery followed by FD (SHAM + FD, n = 7); or ONS followed by FD (ONS + FD, n =

105 still retained -3 D of relative myopia when SHAM+FD animals had returned to normal).

106 Data were analyzed and compared with sham-fed controls.

107 cortex in healthy motor control better than sham feedback.

108 o receive 12 sessions of rTMS (25Hz, 1Hz, or sham) followed by treadmill training.

109 - 3 in the active group and 60% +/- 3 in the sham group (difference, -3; 95% CI, -10 to 3; p = 0.30).

110 ctive group versus 22 out of 76 (29%) in the sham group (p = 0.006).

111 e 41.7% in the active group and 36.4% in the sham group (P = 0.6).

112 ays of BCCAO, but was not different from the sham group after 7 and 14 days.

113 Subjects in the sham group appropriately avoided Pavlovian cues associat

114 less frequently in the EE group than in the sham group at 3-day follow-up (P = 0.008).

115 ater decreases in depression scores than the sham group at week 2 (-40.0% vs. -13.9%; p < .001 after

116 nt in the real feedback group but not in the sham group, confirming the effect of neurofeedback train

117 Compared to the sham group, DO(2) and MO(2) were reduced after all BCCAO

118 Compared to the sham group, there were more serious adverse events repor

119 14 days after harvest when compared with the sham group.

120 1 day and 3 months postintervention than the sham group.

121 In the control (sham) group (n = 27), palatal wounds, after free gingiva

122 imilar between the active (-11.1 [2.03]) and sham groups (-10.6 [2.00]; P = 0.8; difference [95% CI],

123 nificantly increased in TBI compared to both sham groups and TBI RIC.

124 into acute and chronic treatment groups and sham groups with day-matched periods.

125 tentials in VMs and ex vivo optically mapped Sham hearts.

126 tion to the challenged animals compared with sham immunized mice, although none of our candidates wer

127 f ~1:4 to ~1:10 relative to non-infected and sham-infected flies.

128 e 3, double-masked, multicenter, randomized, sham injection-controlled clinical trials that evaluated

129 Participants were randomized 2:1 to FAi or sham (injection plus standard of care) treatment.

130 ministered as intravenous saline followed by sham injections.

131 fuse TBI by midline fluid percussion or were sham-injured.

132 ups: Unilateral RLN Transection (n = 11) and Sham Injury (n = 10).

133 on in the RLN transection group, but not the sham injury group.

134 subject to fluid percussion brain injury or sham injury.

135 checklist and guide for reporting placebo or sham interventions.

136 coplan monthly or every other month (EOM) or sham intravitreal injections monthly or EOM for 12 month

137 nderwent three separate sessions of HD-tDCS (sham, left DLPFC and right DLPFC) for 20 min.

138 L augmented CLOCK and NPAS2 expression above sham levels, rapamycin treatment during release signific

139 RIC intervention returned metabolites to sham levels.

140 ual volumes and micturition fractions toward sham levels.

141 Compared to sham-MA control without manual stimulation, we find that

142 stration was without effect in either ORX or sham males.

143 formed 4 weeks post-MI showed that P1 MI and sham mice (n = 22, each) had similar LV wall thickness,

144 r (18)F-FDG accumulation in TAC mice than in sham mice on day 3 (10.5 +/- 4.1 percentage injected dos

145 As negative control, sham mice received patches containing excipient.

146 While LV tissue modulus for P1 MI and sham mice were similar (341.9 kPa vs 363.4 kPa, p = 0.61

147 ure overload (6-fold in decompensated versus sham mice).

148 n our model, in comparisons with nondiabetic sham mice, neither diabetes nor MI resulted in monocytos

149 et and reduced disease severity, relative to sham mice.

150 before, CLP led to EAE disease equivalent to sham mice.

151 Furthermore, spectroscopic evidence, Kohn-Sham molecular orbital compositions and natural bond orb

152 l using the activation strain model and Kohn-Sham molecular orbital theory at ZORA-OLYP/QZ4P.

153 d-treated group (ZOL) (n = 6): OVX plus ZOL; SHAM (n = 4): SHAM plus saline solution.

154 Compared to P7 shams (n = 20), P7 MI mice (n = 20) had significant LV w

155 teral GC lesions and sham-operated controls (SHAM, n = 16) were trained to discriminate a tastant fro

156 D28, fetuses were randomly assigned to TO or sham neck dissection.

157 bituation night (night 1) and an intervening Sham night (night 3).

158 ons in SCI-nociceptors to a level similar to sham-nociceptors.

159 d as follows: (1) sham on day 6 of life plus sham on day 13 of life, (2) RV-A1B on day 6 plus sham on

160 on day 13 of life, (2) RV-A1B on day 6 plus sham on day 13, (3) sham on day 6 plus RV-A2 on day 13,

161 immune response), whereas mice infected with sham on day 6 and RV-A2 on day 13 of life demonstrated i

162 e lacking ILC2s were treated as follows: (1) sham on day 6 of life plus sham on day 13 of life, (2) R

163 (2) RV-A1B on day 6 plus sham on day 13, (3) sham on day 6 plus RV-A2 on day 13, and (4) RV-A1B on da

164 Hepatocyte-specific p53 ablation in sham-operated ad libitum-fed mice impaired glucose homeo

165 was induced by cecal ligation and puncture; sham-operated animals were used as control.

166 left (LGCX, n = 9) unilateral GC lesions and sham-operated controls (SHAM, n = 16) were trained to di

167 Compared with sham-operated controls, Px-UNx induced albuminuria and i

168 ed glycated hemoglobin (HbA1c) compared with sham-operated controls.

169 G-CSFR treatment were indistinguishable from sham-operated kidneys without IRI.

170 Sham-operated mice exhibited similar distributions of vi

171 Sham-operated mice were fed ad libitum or food restricte

172 trol), subdiaphragmatic vagotomy (VAGX), and sham-operated mice.

173 levels were increased in UniNx compared with sham-operated mice.

174 , and decreased I(p) relative to that for 10 sham-operated rabbits.

175 (p) significantly to levels of myocytes from sham-operated rabbits.

176 g electron microscope) BD rats (40% +/- 6%), sham-operated rats (75% +/- 8%), and BD-E2 rats (67% +/-

177 = 10/group): rats that were trepanned only (sham-operated), rats subjected to rapid-onset BD, and br

178 Heat-inactivated PPE was used to generate a sham operative control.

179 S) diet for a period of 6 weeks, followed by sham or active stimulation (LLTS) for 30 min daily for 4

180 ression increased in debanding compared with sham or banding.

181 t 1, male Long-Evans rats received bilateral sham or neurotoxic NAcc lesions, recovered, and underwen

182 th Tamoxifen to activate the reporter before sham or reperfusion (I/R) MI surgeries.

183 d-type or NRLP3-/- mice were inoculated with sham or RV-A1B.

184 3.8 kg/m(2)) were randomized to undergo the sham or TBE procedure with no device-related complicatio

185 to 55 kg/m(2)) were randomized 1:1 to either sham or TBE targeting the left gastric artery using an o

186 encing across the chromosomal coordinates of sham- or transverse aortic constriction-operated mouse h

187 in bronchoalveolar lavage (BAL) compared to sham-OVA mice.

188 first molars were extracted at 21 days after sham/ovariectomy surgery.

189 % +/- 2 in the active group and 65% +/- 2 in sham patients for calories (difference, -1; 95% CI, -8 t

190 combined or not with exercise but not after sham-PCMS combined with exercise.

191 n the group receiving exercise combined with sham-PCMS, suggesting that the stimulation contributed t

192 ospinal-motor neuronal stimulation (PCMS) or sham-PCMS.

193 p (ZOL) (n = 6): OVX plus ZOL; SHAM (n = 4): SHAM plus saline solution.

194 treatment group, superficial acupuncture at sham points in the control group A, or Streitberger non-

195 per millimeter of mercury) compared with the sham procedure (-0.08+/-0.05 mul/minute per millimeter o

196 thalamotomy (active treatment) and 13 to the sham procedure (control).

197 ned to either renal denervation (n=166) or a sham procedure (n=165).

198 Mice (CD-1, 30-40 g) received a sham procedure or 30 g weight-drop and were euthanized 8

199 study was to examine TBE in a single-blind, sham procedure randomized trial.

200 eter-based renal denervation compared with a sham procedure to safely lower blood pressure in the abs

201 piglets were randomized to cardiac arrest or sham procedure with continuous monitoring of cerebral bl

202 In the MED group, a sham procedure with mapping but no ablation was performe

203 ficant improvement in vision relative to the sham procedure, demonstrating the efficacy of suprachoro

204 Six rats underwent a sham procedure.

205 he side opposite their main motor signs or a sham procedure.

206 ntation of an SC glaucoma drainage device or sham procedure.

207 ssigned 1:1 to either a renal denervation or sham procedure.

208 es clinically significant weight loss over a sham procedure.(The Lowering Weight in Severe Obesity by

209 Compared with the ventilatory pattern of the sham rat pups, injured rat pups had increased fR and pre

210 This did not affect sham rats, but mitigated changes in fR and VPV in ALI ra

211 Compared with sham rats, rats a week after acute lung injury (ALI) exp

212 eus tractus solitarii (nTS) from ALI but not sham rats.

213 minidase levels in serum and BAL compared to sham-rDer p2 mice.

214 schemic preconditioning in nonresponders) or sham-remote ischemic preconditioning (control).

215 mple, N=30; active replication sample, N=81; sham replication sample, N=87).

216 l, 100 nmol L(-1) ) evoked I(SK) in VMs from Shams, resulting in shortening of action potentials in V

217 fter recovery of righting reflex: sham, TBI, sham RIC, TBI RIC.

218 y at 24 days post-rmTBI compared to repeated sham (rSham) injury.

219 We found that active, but not sham rTMS elicited (1) an increase in dlPFC global conne

220 ated during attentive tracking compared with sham rTMS or rTMS over PPC.

221 were randomized to receive either active or sham rTMS to the left dorsolateral prefrontal cortex (dl

222 e tracking was less pronounced compared with sham rTMS.

223 potentiated startle following active but not sham rTMS.

224 st to no mortality in S. pneumoniae-infected sham (Sham + Sp) animals.

225 d reactive oxygen species when compared with Sham + Sp mice.

226 60% mortality compared with 5% mortality in Sham + Sp mice.

227 no mortality in S. pneumoniae-infected sham (Sham + Sp) animals.

228 agnetic stimulation monotherapy (n = 35), or sham stimulation (n = 35).

229 Although tVNS and sham stimulation did not differ in subjective intensity

230 rve stimulation (taVNS, Cerbomed Nemos) with sham stimulation during an oral glucose tolerance test i

231 educed classification accuracy compared with Sham stimulation when it was applied to 2 spots in the v

232 cranial direct current stimulation (tDCS) or sham stimulation, guided by functional mapping of the pr

233 We found that tDCS, compared to sham stimulation, significantly decreased reports of dre

234 of active prefrontal stimulation at 2 mA or sham stimulation.

235 that was significantly higher than following sham stimulation.

236 s, and were specific to active compared with sham stimulation.

237 uation of alpha oscillations by tVNS than by sham stimulation.

238 ulation and 454 patients received control or sham stimulation.

239 overing the left premotor cortex (PMC) and 2 Sham stimulations.

240 To mask both groups, sham suprachoroidal and intravitreal injections were uti

241 ansverse aortic constriction (TAC) (n = 22), sham surgery (n = 12), or coronary ligation as an inflam

242 omized (OVX) ewes and four ewes subjected to sham surgery (SHAM) were treated as follows: OVX (n = 5)

243 , and CD36, were stratified to either VSG or sham surgery before body weight, body composition, diet

244 Young guinea pigs underwent either sham surgery followed by FD (SHAM + FD, n = 7); or ONS f

245 coronary artery to induce experimental MI or sham surgery in nondiabetic and diabetic mice with preex

246 ht groups of rats were studied 4-weeks after sham surgery or bile duct ligation and were injected wit

247 C57BL/6J mice underwent sham surgery or left anterior descending coronary artery

248 C57BL/6J mice were subjected to sham surgery or moderate-level controlled cortical impac

249 nistrated from the 14th day post-ovariectomy/sham surgery until euthanasia.

250 VSG or sham surgery was performed in high-fat diet-fed male hep

251 ar (LV) tissue from mice subjected to I/R or sham surgery were used to assess PKARIalpha disulfide fo

252 n to bilateral renal ischemia-reperfusion or sham surgery, either in the absence or presence of gonad

253 Compared with Sham surgery, the I/R MI group had increased single and

254 by ovariectomy (OVX), orchiectomy (ORX), or sham surgery.

255 d to 1-h middle cerebral artery occlusion or sham surgery.

256 se aortic constriction, or vehicle injection/sham surgery.

257 rague-Dawley rats had lumbar IVD puncture or sham surgery.

258 nt); ovariectomy+water (estrogen-deficient), sham-surgery+strontium ranelate (625 mg/kg/d) (strontium

259 allocated into one of the following groups: sham-surgery+water (estrogen-sufficient); ovariectomy+wa

260 iTBS-sham tACS facilitated single-pulse MEPs in HSs, but not

261 induced a larger MEP facilitation than iTBS-sham tACS in both groups, with similar values in patient

262 ICI was similar to that obtained in the iTBS-sham tACS session.

263 (iTBS-gamma tACS) and during sham-tACS (iTBS-sham tACS) in two sessions.

264 ring gamma-tACS (iTBS-gamma tACS) and during sham-tACS (iTBS-sham tACS) in two sessions.

265 roups 1 h after recovery of righting reflex: sham, TBI, sham RIC, TBI RIC.

266 z pulses with a 5 kHz carrier frequency) and sham-TESS applied between C5 and C6 spinous processes in

267 l arm muscles for 75 min after TESS, but not sham-TESS, in control subjects and SCI participants, sug

268 In shams, the urethra was exposed, but no suture tied.

269 treated with 20 Gy of external beam therapy; sham therapy was given to the other side.

270 active NeuroStar TMS monotherapy (n = 48) or sham TMS (n = 55) for 30 daily treatments over 6 weeks.

271 grafts were treated with (177)Lu-DOTATATE or sham-treated and coinjected with (111)In-anti-gammaH2AX-

272 lls in the periphery of the mice compared to sham-treated CD34(+) cells.

273 onthly and EOM groups, respectively) than in sham-treated eyes (1/81 eyes [1.2%]).

274 ined macular edema at month 36 compared with sham-treated eyes (33.3% vs. 14.7% and 13.0% vs. 27.3% f

275 acceptable side-effect profile compared with sham-treated eyes.

276 months in the FAi-treated compared with the sham-treated group (73.8% vs. 23.8% of eyes, respectivel

277 y lower in the FAi-treated compared with the sham-treated group (mean 1.7 vs. 5.3, respectively, P <

278 the FAi-treated group when compared with the sham-treated group underwent IOP-lowering surgery (5.7%

279 ology in PBM-treated animals compared to the sham-treated group.

280 Fewer FAi compared with sham-treated participants required adjunctive treatments

281 ce between the proportion of FAi-treated and sham-treated patients who had a uveitis recurrence.

282 Sham-treated rats were restrained, but not treated with

283 ine participants (n = 87 FAi-treated; n = 42 sham-treated) were enrolled.

284 (95% CI, 0-40; P = 0.067) compared with the sham treatment group.

285 in the active treatment group and 82 in the sham treatment group.

286 Relative to Sham treatment, (56)Fe irradiation did not overtly chang

287 d 3:2 to suprachoroidally injected CLS-TA or sham treatment, with administrations at day 0 and week 1

288 e symptom severity, this did not differ from sham treatment.

289 Tube; E-Motion Medical, Tel Aviv, Israel) or sham treatment.

290 reductions in the growth of GA compared with sham treatment.

291 of the diverse contexts in which placebo or sham treatments are used in clinical research, we consul

292 inal mucosal mast cells and eosinophils over sham treatments.

293 yed a ramified morphology similar to that of Sham uninjured mice, whereas microglia in vehicle-treate

294 While all sham-vaccinated animals developed viraemia, high tissue

295 s were significantly lower than those of the sham-vaccinated group (P <= 0.05).

296 nd ATP/ADP to normalize in debanding towards sham values (phosphocreatine: sham, 38.4+/-7.4, debandin

297 th normalization of myocardial ATP levels to sham values.

298 wes and four ewes subjected to sham surgery (SHAM) were treated as follows: OVX (n = 5): OVX plus sal

299 itated SK2 channels in VMs from TAB rats vs. Shams, which was reversible by incubation of the VMs wit

300 njury have greater Mouse Grimace Scores than sham within 24 hours of injury, suggestive of spontaneou