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1 (ACe)] and control animals (that received a sham operation).
2 ely; P < .05 for IRE vs both RF ablation and sham operation).
3 with livers preserved for 30 hr (P<0.01 vs. sham operation).
4 rfused myocardial infarction to induce HF or sham operation.
5 ongus muscles 24 hrs after CLP compared with sham operation.
6 fter the lesion compared with only 11% after sham operation.
7 fter the ventral rhizotomy but not after the sham operation.
8 into the lungs of rats subjected to 2CLP or sham operation.
9 35 +/- 5 mm Hg for 90 mins, resuscitated) or sham operation.
10 mice were killed after 3 and 14 d of UUO or sham operation.
11 cine or vehicle 2 weeks after ovariectomy or sham operation.
12 mm Hg for 90 minutes, then resuscitated) or sham operation.
13 larly treated AM from animals subjected to a sham operation.
14 central mechanisms following nerve injury or sham operation.
15 subjected to PNX than in mice subjected to a sham operation.
16 ts were made tolerant by conditioning SLH or sham operation.
17 5 mm Hg for 90 minutes and resuscitation) or sham operation.
18 rd on days 14, 35 and 55 after either CCI or sham operation.
19 c dose of glucocorticoid (dexamethasone), or sham operation.
20 either aspiration lesions of the AP/NTS or a sham operation.
21 ffering mechanisms following nerve injury or sham operation.
22 controls examined at the same interval after sham operation.
23 after unilateral CCI to the sciatic nerve or sham operation.
24 pared subsequent metabolic remodeling with a sham operation.
25 teral (VLFX) funiculus, and with rats with a sham operation.
26 The control animals received a sham operation.
27 ury of moderate severity (1.9 to 2.0 atm) or sham operation.
28 rague-Dawley rats underwent two-thirds PH or sham operation.
29 p implantation compared with the value after sham operation.
30 onary dilation catheter (second injury) or a sham operation.
31 , with livers preserved for 30 hr, and after sham operation.
32 Control animals underwent sham operation.
33 Hepatic ischemia-reperfusion injury or sham operation.
34 in rats 1 and 6 weeks after transmural MI or sham operation.
35 artery occlusion, and one was subjected to a sham operation.
36 of the locus coeruleus (LC) and rats with a sham operation.
37 ifferent ages were assessed following ADX or sham operation.
38 s were subjected to myocardial infarction or sham operation.
39 duce peritonitis, while control groups had a sham operation.
40 a as well as T3-T5 DRG neurons compared with sham operation.
41 al ligation and puncture (a sepsis model) or sham operation.
42 llowing vertical sleeve gastrectomy (VSG) or sham operation.
43 sue samples obtained 6- and 24 hr post MI or sham operation.
44 in mice with cervical unilateral vagotomy or sham operation.
45 /6J mice underwent uninephrectomy (UniNx) or sham operation.
46 left unilateral ureteral obstruction versus sham operation.
47 randomly subjected to either ovariectomy or sham operation.
48 hours after 30 minutes of renal ischemia or sham operation.
49 arts first undergo ventricular amputation or sham operation.
50 mpared with control subject that underwent a sham operation.
51 tion (left ventricular hypertrophy (LVH)) or sham operation.
52 ollowing spinal nerve ligation (SNL) but not sham operation.
53 ed 4 weeks after bile duct ligation (BDL) or sham operation.
54 1 mo beginning 6 wk after 5/6 nephrectomy or sham operation.
55 derwent cecal ligation and puncture (CLP) or sham operation.
56 s underwent pMCAO by electro-coagulation and sham operation.
57 egenerating lobe was similar to that after a sham operation.
58 were then subjected to trauma-hemorrhage or sham operation.
59 erwent 30 min of bilateral renal clamping or sham operation.
60 gastrointestinal transit (GIT) compared with sham operation.
61 ouse intestinal crypt cells following SBR or sham operation.
62 ditions after a 50% small-bowel resection or sham operation.
63 h LVD 28 days after myocardial infarction or sham operation.
64 nd 4 weeks after bile duct ligation (BDL) or sham operation.
65 and 14 days after femoral artery ligation or sham operation.
66 fter RSLT and twofold after full-size versus sham operation.
67 and in five rats who underwent a unilateral sham operation.
68 ns of a 5-mm segment of the cord below T8 or sham-operation.
69 ask after either unilateral FLsmc lesions or sham operations.
70 Control rats underwent sham operations.
71 32-min bilateral renal ischemia or identical sham operations.
72 eptal lesions as compared to those receiving sham operations.
73 r) mice underwent 50% or 75% proximal SBR or sham operations.
74 t mice underwent bile duct ligation (BDL) or sham operations.
75 tical impact (CCI) injury (2.7 mm; 4 m/s) or sham operations.
76 eived neonatal neurotoxic amygdala damage or sham-operations.
77 l electrolytic lesions of insular cortex or 'sham' operations.
78 ral electrolytic lesions of the amygdala or "sham' operations.
80 ) was ~ 1.3-fold higher than at 30-min after sham-operation (120 [77.5-170]), and twofold higher than
81 ent unilateral ureteral obstruction (UUO) or sham operation; 28 days later, renal interstitial fibros
82 more infiltrating lymphocytes compared with sham operation (7.6 cells per frame +/- 1.9 vs 11.2 +/-
84 Foxm1(flox/flox)) were subjected to PPx or a sham operation, after which islet expression of Foxm1 an
85 with U74389F or vehicle were subjected to a sham operation and 30 mins of ischemia by occlusion of t
87 AM were harvested from mice subjected to a sham operation and CLP 24 h after laparotomy, adherence
89 ilateral common carotid artery stenosis or a sham operation and fed normal or cilostazol diet for thr
90 Our results demonstrate that, compared with sham operation and ischemia alone, I/R significantly inc
91 at 1 hr after cecal ligation and puncture or sham operation and repeated at 12-hr intervals thereafte
92 blation, partial surgical hepatectomy, and a sham operation and to inhibit HCC recurrence after RF ab
93 s: (1) sham-operation controls (n = 10); (2) sham-operation and ligature-induced bone loss (n = 10);
95 erwent surgical coronary artery ligation (or sham operation) and were treated with either anakinra 1
104 tion with freshly harvested livers and after sham operation, but rose in animals transplanted with li
105 ce were used either as control, subjected to sham operation (cannulation or laparotomy only or cannul
112 ndomly assigned to the following groups: (1) sham-operation controls (n = 10); (2) sham-operation and
114 rtial hepatectomy (ie, left lobectomy), or a sham operation (controls) by using Kaplan-Meier survival
115 In another set of mice following TSE or sham operation, corneas were harvested for ELISA (VEGFR3
119 nd 0.001, t-test, n = 8 rats/group), whereas sham-operation eyes had no significant loss (mean differ
121 sepsis by cecal ligation and puncture or to sham operation followed by the administration of normal
122 ial sepsis by cecal ligation and puncture or sham operation followed by the administration of normal
123 psis by cecal ligation and puncture (CLP) or sham operation followed by the administration of normal
124 ial sepsis by cecal ligation and puncture or sham operation followed by the administration of normal
125 s by cecal ligation and puncture (CLP) or to sham operation followed by the administration of normal
126 sepsis by cecal ligation and puncture or to sham operation followed by the administration of normal
127 s by cecal ligation and puncture (CLP) or to sham operation followed by the administration of normal
131 sed in WT mice compared to mice undergoing a sham operation, however leukocyte attachment was reduced
132 yrus granule cells 2 weeks after a stroke or sham operation in DCX/DsRed transgenic mice of either se
133 performed either subtotal nephrectomy (N) or sham operations in WT, leptin receptor-deficient (db/db)
135 larger than mice treated with RF ablation or sham operation (mean, 3.6 +/- 1.3 [standard deviation] v
139 er CCI of the sciatic nerve (n = 12) or else sham operation (n = 8) which involved exposure but no li
143 In Model 2 Sprague-Dawley rats underwent sham operation (n=9), or 45 min of warm ischemia and 10
144 on carotid artery (CCA) ligation (n = 24) or sham-operation (n = 24) was performed with a 1-week inte
146 eCl(3)-mediated carotid thrombosis (n = 72), sham-operation (n = 79), or non-operation (n = 26).
147 20 animals (aorto-pulmonary shunt [n=10] or sham operation [n=10]) 3 months after the intervention.
148 ion, 12 pigs (aorto-pulmonary shunt [n=6] or sham operation [n=6]) were evaluated monthly with right
157 ermanent middle cerebral artery occlusion or sham operations on multiple measures of sensory, motor a
159 +/+ mice or iNOS-/- mice were subjected to a sham operation or 30 mins of superior mesenteric artery
160 Male Sprague-Dawley rats were subjected to sham operation or bilateral adrenal demedullation fitted
163 th placebo, UDCA, or CDCA for 2 weeks before sham operation or induction of AC by bile duct ligation
165 t with grafts preserved for 12 hr than after sham operation or orthotopic liver transplant with nonpr
166 ut ovalbumin immunization) were subjected to sham operation or T-H and sacrificed after 24 hours.
169 ere isolated from adult male rats 24 h after sham operation or two-thirds PHX and treated with catech
171 0-12-d-old monkeys (Macaca mulatta) received sham operations or neurotoxic hippocampal lesions and we
172 y and 5 mg/kg daily afterward) and underwent sham-operation or approximately 90% partial hepatectomy.
175 nderwent cecal ligation and double puncture, sham operation, or no operation and were evaluated 48 hr
176 rats 4 weeks after bile duct ligation (BDL)/Sham-operation, or naive rats fed a hyperammonemic diet
177 cture at 24 hours after trauma-hemorrhage or sham operation, ovariectomized mice had a significantly
178 Twenty-five female sheep were randomized to sham operation, ovariectomy, or ovariectomy plus 17beta-
181 819 mm(3) +/- 327 vs 2241 mm(3) +/- 548 for sham operation; P < .05) that was accompanied by more in
184 mmon bile duct ligation-induced cirrhosis or sham operation received prophylactic or therapeutic caff
186 ee groups were studied: time controls with a sham operation, saline-treated ischemia-reperfusion, and
187 Aortocaval fistula creation (AVF group) or a sham operation (sham group) was performed in C57BL/6 mic
188 ary artery ligation (placebo-MI or E2-MI) or sham operation (sham) and hearts were harvested 6, 24, a
189 demic mice underwent uninephrectomy (UNx) or sham operation (sham) followed by treatment with Ang II
191 of pressure overload (hypertrophied; n=9) or sham operation (sham; n=8) using dynamic 13C-nuclear mag
196 rdU) incorporation increased from 0.2% after sham operation to 2%, 18%, and 1.2% in full-size, half-s
197 after CLP (i.e., a late stage of sepsis) or sham operation to measure plasma levels of corticosteron
198 igeminal stereotactic electrolysis (TSE), or sham operation, to ablate the ophthalmic branch of the t
201 daily in rats after portal vein ligation or sham operation until a hyperdynamic circulatory state de
206 atm) lateral fluid percussion head injury or sham operation was produced in rats 4 or 48 h prior to h
208 d and tumor frequency in mice that underwent sham operation were further compared with those of mice
211 derwent cecal ligation and puncture (CLP) or sham-operation with or without soluble complement recept