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1 hroughout stratum radiatum of the CA1 field (sharp wave).
2 tate interrupted by transient active states (sharp waves).
3 e related to the occurrence of an entorhinal sharp wave.
4 in the generation of gamma oscillations and sharp waves.
5 ubsequently enhance, the local generation of sharp waves.
6 in anticipation of the run, coinciding with sharp waves.
7 t local field potential (LFP) events, termed sharp waves.
8 ng theta states and feature detection during sharp waves.
9 repotentials) in some pyramidal cells during sharp waves.
10 phasic or polyphasic slow waves, or slow and sharp waves.
11 ges of CA3-CA1 neurons concurrent with field sharp waves.
12 ncy activity follow population bursts called sharp waves.
13 bserved in the hippocampus in vivo following sharp waves.
14 ll types during large network events such as sharp waves.
15 ssociated gamma oscillations and hippocampal sharp waves.
16 iming of imposed replay relative to cortical sharp-waves.
18 ; 95% CI, 1.67-8.02; P = .001), and sporadic sharp waves and periodic discharges (OR, 2.59; 95% CI, 1
21 ds of intense pyramidal cell firing, such as sharp waves, and lead to their altered activation during
23 ories of hippocampal processing suggest that sharp waves arise from strengthened plasticity, and that
28 e, or fire infrequently, superimposed on the sharp wave-associated depolarization, whereas interneuro
33 city, expressed as long-lasting increases in sharp wave-associated synaptic currents, exhibits enhanc
34 -locked to theta oscillations (4-8 Hz) or to sharp-wave-associated ripple oscillations (120-200 Hz),
36 ctances by the backpropagating spikes during sharp wave bursts may be critical for synaptic plasticit
38 ion bursts including epileptiform spikes and sharp waves can be generated in and propagate through th
41 RI hyperintensity (56% vs 19%), EEG periodic sharp-waves complexes (41% vs 6%), and CSF RT-QuIC posit
45 charge during the time (50-100 msec) of each sharp wave, each wave of a ripple (approximately 5 msec)
46 dies in CA3 and CA1 found that the number of sharp waves emitted also increased in CA3 recordings as
48 e mammalian hippocampus exhibits spontaneous sharp wave events (1-30 Hz) with an often-present superi
53 ients, and advance for many millimeters as a sharp wave front perpendicular to the pial surface, at s
58 G oscillations superimposed on physiological sharp waves in a number of limbic regions of the rat, ei
59 ed intracellularly from distal dendrites and sharp waves in the electrocorticogram were accompanied b
60 istic encephalogram abnormality of spikes or sharp waves in the pericentral region (centroparietal, c
61 ome from investigation of large deflections (sharp-waves) in the hippocampal local field potential th
63 interictal EEG may show asymmetric spikes or sharp waves, indicating the presence of epileptic activi
64 gion of the hippocampus is the major site of sharp wave initiation, and a brain region crucially invo
65 The approximately 200 Hz activity of the sharp wave itself may serve to enhance synaptic connecti
66 e slower negative transients, referred to as sharp waves, occur simultaneously in the stratum radiatu
67 nts; and (ii) 'delayed responses', spikes or sharp waves occurring between 100 ms and 1 s after stimu
68 For example, hippocampal theta, gamma, and sharp wave oscillations are critical for memory formatio
69 for example, caused a matching modulation of sharp-wave production there and in the neighbouring DVR.
71 Inhibitory GABAergic neurons participate in sharp wave ripple (SPW-R) oscillations in the hippocampa
72 activated during high frequency (100-250 Hz) sharp wave ripple (SWR) activity in a manner that likely
74 consolidation is correlated with hippocampal sharp wave ripple (SWR) density, cortical delta waves (D
75 record synaptic and spiking activity during sharp wave ripple (SWR) events in early amyloid patholog
76 ation but not during navigation, (2) reduced sharp wave ripple (SWR) incidence at goal location, and
78 havioral sequences in the hippocampus during sharp wave ripple complexes (SWRs) provides a potential
79 in the hippocampal-entorhinal circuit during sharp wave ripple events (SWRs) that occur during sleep
81 cortical alpha oscillations and hippocampal sharp wave ripple oscillations (~150 Hz) during immobili
83 ty that encodes current location, as well as sharp-wave ripple (SWR) activity during which representa
84 activated during high frequency (100-250 Hz) sharp-wave ripple (SWR) activity in a manner that probab
85 ited a 2.5-fold increase in the abundance of sharp-wave ripple (SWR) events during awake resting peri
88 resses these types of representations during sharp-wave ripple (SWR) events, and previous work identi
92 increase then predicts a drop in hippocampal sharp-wave ripple (SWR)-M1 slow oscillation coupling-sug
94 ased FFI in DG - CA3 identified enhanced CA1 sharp-wave ripple - ACC spindle coupling as a potential
95 mpus, whereas non-REM-associated hippocampal sharp-wave ripple activity evoked discrete LFP modulatio
96 ovement sleep and were highly reminiscent of sharp-wave ripple activity observed in the rodent hippoc
97 Taste-responsive units exhibited increased sharp-wave ripple co-activation during the taste deliver
98 ctrical activity in CA1 hippocampal neurons [sharp-wave ripple complexes (SPW-Rs)] functionally coupl
100 g activity of hippocampal CA1 neurons during sharp-wave ripple events encode the position of mice.
102 l field and single-unit activity surrounding sharp-wave ripple events were examined in the CA1 region
103 prior observations of their distinct role in sharp-wave ripple firing, suggest a key role of this neu
104 cn1a mutation in DS mice reduces hippocampal sharp-wave ripple occurrence and slows internal ripple f
109 play of spatial sequences during hippocampal sharp wave-ripple (SPW-R) events of quiet wakefulness an
112 activity of the hippocampus is dominated by sharp wave-ripple events (SWRs), which have been shown t
113 tween hippocampal input, such as mediated by sharp wave-ripple events, cortical slow oscillations, an
114 findings demonstrate that a function of the sharp wave-ripple is to modulate peripheral glucose home
116 ocampus and the neocortex during hippocampal sharp wave-ripple oscillations is believed to be critica
117 Pattern reinstatement was strongest during sharp wave-ripple oscillations, suggesting that these ev
125 al-related assembly patterns associated with sharp wave/ripple network oscillations, during both lear
126 During phases of rest and sleep, it exhibits sharp-wave/ripple (SPW/R) complexes, which are short epi
127 te dependent: they are driven by hippocampal sharp-wave/ripple (SWR) bursts in slow-wave sleep (SWS)
128 might contribute to the organization of the sharp-wave/ripple episodes by decreased firing during an
131 Bouts of high frequency activity known as sharp wave ripples (SPW-Rs) facilitate communication bet
134 rch have established the role of hippocampal sharp wave ripples (SPW-Rs) in consolidating and guiding
135 ain state changed during reward consumption, sharp wave ripples (SPW-Rs) occurred on some trials, and
137 NRG1-ErbB4 signaling.SIGNIFICANCE STATEMENT Sharp wave ripples (SW-Rs) are a hippocampal event, impo
140 reviously stored memories during hippocampal sharp wave ripples (SWRs) is thought to support both mem
141 mouse models of AD, there are disruptions to sharp wave ripples (SWRs), hippocampal population events
144 arning increased the coupling of hippocampal sharp wave ripples and cortical spindles, and these lear
145 Cholinergic stimulation completely blocked sharp wave ripples and strongly suppressed the power of
147 activity by reducing gamma oscillations and sharp wave ripples, changes associated with a decrease i
148 activity, including theta-gamma coupling and sharp wave ripples, in response to novel versus familiar
149 epolarizations in CA1 pyramidal cells during sharp wave ripples, which are associated with ripple fre
152 neuronal assemblies during hippocampal awake Sharp-Wave Ripples (aSWRs) changed within individual ses
155 ischarges (IEDs) in mice with TLE as well as sharp-wave ripples (SPW-Rs) in healthy mice, and find th
160 s reactivation is coordinated by hippocampal sharp-wave ripples (SWRs) and is believed to support the
161 n relies on coordination between hippocampal sharp-wave ripples (SWRs) and neocortical UP/DOWN states
162 thought to occur primarily when hippocampal sharp-wave ripples (SWRs) and thalamocortical spindles a
163 lay of waking experiences during hippocampal sharp-wave ripples (SWRs) are considered to be crucial f
171 olidation.SIGNIFICANCE STATEMENT Hippocampal sharp-wave ripples (SWRs) occur both in the awake state
172 offline place cell activity associated with sharp-wave ripples (SWRs) reflects predominantly station
173 tive spatial task and focused on hippocampal sharp-wave ripples (SWRs) to identify times of memory re
174 cilitated by the coordination of hippocampal sharp-wave ripples (SWRs) with cortical slow and spindle
175 The classic LFP events of the CA1 region, sharp-wave ripples (SWRs), are induced by CA3 activity a
176 ught to play a key role in the generation of sharp-wave ripples (SWRs), electrophysiological oscillat
177 two functional apoE4-KI phenotypes involving sharp-wave ripples (SWRs), hippocampal network events cr
178 lections and oscillations define hippocampal sharp-wave ripples (SWRs), one of the most synchronous e
179 al oscillations in the hippocampus, known as sharp-wave ripples (SWRs), synchronise the firing behavi
180 tingly, the incidences of sleep spindles and sharp-wave ripples (SWRs), typically associated with cor
182 Memory consolidation crucially depends on sharp-wave ripples (SWRs), which are local field potenti
189 onal ensembles, organized by the hippocampal sharp-wave ripples (SWRs, 80 to 150 Hz), subcortical/cor
190 st intimate interactions between hippocampal sharp-wave ripples and ACC neurons in a state-dependent
191 ivity in the titmouse hippocampus, including sharp-wave ripples and anatomically organized place cell
195 cusing on hippocampal theta oscillations and sharp-wave ripples and how they coordinate with cortical
196 ent hippocampal place cell activities during sharp-wave ripples and spatial contents of hippocampal r
199 te the underlying communication: Hippocampal sharp-wave ripples coalescing with thalamic spindles med
202 ry replay of recent experiences dominated in sharp-wave ripples during contracted pupil substates of
203 of the claustrum suppress the production of sharp-wave ripples during slow-wave sleep in a unilatera
204 in rodent CA1 occurs during exploration and sharp-wave ripples emerge in quiescence, it is less clea
205 , thalamo-cortical spindles, and hippocampal sharp-wave ripples has convincingly been shown to be a k
208 during synchronous population events called sharp-wave ripples in the hippocampus while mice are in
210 pal neurons called replays, concomitant with sharp-wave ripples in the local field potential, are cri
211 h experimental evidence supports the role of sharp-wave ripples in transferring hippocampal informati
212 eactivated in specific temporal order during sharp-wave ripples observed in quiet wakefulness or slow
214 d spatial representations within hippocampal sharp-wave ripples that were stable for hours during sle
216 emerged during theta-oscillations and awake sharp-wave ripples while on the maze, revealing the cont
217 ulation event that frequently occurs between sharp-wave ripples(12), may underlie such a mechanism.
218 memory.SIGNIFICANCE STATEMENT Disruption of sharp-wave ripples, a characteristic hippocampal rhythm
219 and altered the properties of physiological sharp-wave ripples, altered their physiological properti
220 field potential oscillations associated with sharp-wave ripples, and controlled the phase of action p
221 ents, hippocampal replay occurs during local sharp-wave ripples, and the associated neocortical repla
223 sentations also accumulate inhibition during sharp-wave ripples, experimentally validating a major pr
224 were markedly suppressed during hippocampal sharp-wave ripples, had a low burst incidence, and sever
225 alamic contribution to non-REM oscillations (sharp-wave ripples, SWRs; slow/delta; spindles), we reco
248 e show that interactions between hippocampal sharp wave-ripples (SPW-R), cortical spindles (SPI), and
250 hese reactivations peaked during hippocampal sharp wave-ripples (SPW-Rs) and involved a subgroup of B
251 g "fast gamma" or "epsilon" oscillations and sharp wave-ripples (SPW-Rs), are one exception, showing
254 play of neuronal activity during hippocampal sharp wave-ripples (SWRs) is essential in memory formati
255 gional coupling of slow waves, spindles, and sharp wave-ripples (SWRs), across the cortex, thalamus,
257 r reversed order, during bursts of activity (sharp wave-ripples [SWRs]) that occur in sleep and awake
258 sleep, temporal coordination of hippocampal sharp wave-ripples and medial prefrontal cortex spindles
259 rmore, we found that CB1R activation reduces sharp wave-ripples by impairing the innate SPW-R-generat
260 ell ensembles during active behaviors, while sharp wave-ripples coordinate place cell sequences durin
261 of place cell sequences by theta rhythms and sharp wave-ripples develops as a reward location is lear
262 ed the slow oscillation in the neocortex and sharp wave-ripples in the hippocampus, these alternation
265 (SO) power, the coordination of hippocampal sharp wave-ripples with both the SO and thalamocortical
267 In the hippocampus, replay occurs within sharp wave-ripples: short bouts of high-frequency activi
273 havior, coactivation of hippocampal cells in sharp-wave/ripples represent inferred relationships that
274 ivity of deep and superficial neurons within sharp-wave/ripples was bidirectionally changed across ex
280 projection, we cross-correlated hippocampal sharp wave (SPW) ripples or theta activity and extracell
281 a naturally occurring event, the hippocampal sharp wave (SPW), for the assessment of synaptic strengt
282 among pyramidal neurons was observed during sharp wave (SPW)-related population bursts, with stronge
287 ce rates, ripple frequencies, and ripple and sharp wave (SW) amplitudes were increased in both, while
288 of memory formation, prompting us to cluster sharp waves (SWs) in the DG [dentate SWs (DSWs)] during
289 spikes were observed in the dendrites during sharp waves than in the soma, suggesting that local dend
291 scillations, and increased firing during CA1 sharp waves, thus supporting the role of CA3 networks in
293 automatisms that were coupled with polyspike/sharp-wave trains with increasing amplitude and slowing
296 dominant rhythm, and presence of generalized sharp waves were statistically significantly associated
298 The voltammetry of P(C) displays relatively sharp waves with minimal history or relaxation effects.