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1 sgene developed AML with 100% penetrance and short latency.
2 e stimulation depresses Golgi cell firing at short latency.
3 leus processes error in trial performance at short latency.
4  CIN3 or invasive cervical carcinoma after a short latency.
5 ol into the NPO elicited active sleep with a short latency.
6 ersion to malignancy with tumor formation of short latency.
7 duces RMS with extremely high penetrance and short latency.
8 ress the excitability of the motor cortex at short latency.
9 s the commonest occupational lung disease of short latency.
10 put can reach the motor cortex at relatively short latency.
11  cells induces ERMS with high penetrance and short latency.
12 an respond to the stimulation with extremely short latencies.
13 ystematically stimulus-driven, and with very short latencies.
14 tically affects stomach homoeostasis at very short latencies.
15 ve and process visual sensory information at short latencies?
16  evoked dopamine release (67 +/- 20 nm) with short latency (0.2 +/- 0.1 sec onset).
17                                              Short-latency (0.7 msec) evoked crossed phrenic potentia
18 instance, their brains perceptually suppress short latency (1-10 ms) echoes by constructing a represe
19 tes of the right prefrontal cortex, we found short-latency (120-160 ms) responses selective for avers
20 caused reliable decreases in HVC activity at short latencies (20-80 ms).
21  subjects in imaginary flexion tasks at very short latencies (26.4 +/- 3.7 msec), again similar to th
22 tified by electrical stimulation, produced a short-latency (35 +/- 3.5 s, n = 11) decrease of rigid h
23 ts during motor imagery, were activated at a short latency (38.6 +/- 10.6 msec).
24 ubgroups, neuronal excitations occurred with short latencies (4-8 s), peaked at 10-20 s (30-40% incre
25 ocial stress passively, assuming defeat with short latencies (48%), or actively, with proactive behav
26 ked HP EPSPs were greatly attenuated after a short latency (50 ms) following burst-like PFC electrica
27                                              Short-latency (80-140 ms) striatal responses to a first
28  progress to invasive carcinomas with a very short latency, a process that is dampened by treatment w
29            Spinal neurons were classified as short latency abrupt (SL-A) or short latency sustained (
30 hree types of neurones were characterized as short-latency abrupt (SLA, n = 24), short latency sustai
31 c responses were observed in VTA DA neurons: short latency activation (<25 msec; 55.1% of cells), lon
32                                   We studied short-latency activations of dopamine neurons to unrewar
33 No wind-up of either long latency C-fibre or short latency Adelta responses was seen during trains of
34                                              Short latency afferent inhibition (SAI) and long latency
35 ng effect argues against the hypothesis that short-latency afferent activity from V1 is the stage of
36 d 72% of variability in gait speed with only short-latency afferent inhibition and attention emerging
37                                              Short-latency afferent inhibition is a surrogate measure
38 egression model explored the contribution of short-latency afferent inhibition to gait speed, control
39                                              Short-latency afferent inhibition was also significantly
40 with Parkinson's disease showed that reduced short-latency afferent inhibition was an independent pre
41                                              Short-latency afferent inhibition was determined as the
42                                              Short-latency afferent inhibition was measured by condit
43 tion (LICI), intracortical facilitation, and short-latency afferent inhibition were measured before a
44                         We hypothesized that short-latency afferent inhibition would be an independen
45  associations were found between gait speed, short-latency afferent inhibition, age and postural inst
46 hibition, and sensorimotor interaction, i.e. short-latency afferent inhibition, were recorded from 15
47 No other gait variables were associated with short-latency afferent inhibition.
48 extual cueing effect involves changes in the short-latency afferent visual signal from V1 that have a
49    Ascending interneurons are not excited at short latency after skin stimulation but are strongly ac
50 igenicity, 10 formed malignant tumors with a short latency, all 10 lacked wild-type p53.
51 f conditioning and extinction disclosed that short latency, alpha(1) components of the CRs were acqui
52 a virus oncogene (MPL) efficiently induced a short-latency AMKL that recapitulated all the features o
53                    BCT depolarisation evoked short-latency, AMPA/kainate receptor-mediated EPSCs in c
54                                          The short latencies and incomplete cue invariance suggest th
55        MGBv neurons were identified by their short latencies and sharp tuning curves.
56 under voltage-clamp conditions had identical short latencies and similar amplitudes, but were kinetic
57 s into the ipsilesional field had abnormally short latencies and tended to overshoot their mark.
58  mice generated high-grade astrocytomas with short latency and 100% penetrance.
59  animals, a transplantable AML of relatively short latency and frequent granulocytic sarcoma was note
60 unpigmented and nonmetastatic melanomas with short latency and high penetrance.
61 -CreER; R26-SmoM2 mice developed tumors with short latency and high penetrance.
62 trical stimulation in CeA evoked consistent, short latency and intensity-dependent vlPAG neuronal fir
63 ing direct input from the LGN, identified by short latency and low jitter of LGN-evoked PSPs, showed
64                                          The short latency and posterior scalp location of the effect
65 ers of homogeneous pathophysiology: DES with short latency and spastic achalasia.
66 evelopment of melanoma with 100% penetrance, short latency and with metastases observed in lymph node
67                              We recorded the short-latency and delayed responses of STN units and fro
68 responses allowed for discrimination between short-latency and long-latency C-starts (SLCs vs. LLCs)
69 responses allowed for discrimination between short-latency and long-latency C-starts (SLCs vs. LLCs)
70     Photoinhibition of the thalamus caused a short-latency and near-complete collapse of ALM activity
71  brainstem pathway characterized by low CFs, short latencies, and high-fidelity transmission of perio
72              Based on their high penetrance, short latency, and histologic fidelity, these models of
73 rm fibrosarcomas in athymic mice with a very short latency, and the cells from the tumors express the
74 om PML/RARalpha transgenic mice results in a short latency APL-like disease with complete penetrance.
75                    All effects appeared with short latency ( approximately 5 min) and were reversible
76 at neighboring off-center alpha-GCs maintain short-latency (approximately 2.5 msec) synchronous spiki
77    Category-selective responses can occur at short latency (as early as 130 ms) in middle cortical la
78 ically stereotyped responses with relatively short latencies, as well as bilateral recruitment of vSP
79 d fraction of the trials, it appeared with a short latency at one location; on the complementary frac
80 y thalamic nuclei may modulate activity in a short-latency auditory CS pathway or serve as part of a
81                          These disconjugate, short-latency axis perturbations appear intrinsic to the
82 areas of cerebral cortex in man can activate short latency bilateral cortical projections to the phar
83 cruciate cortex prevented the development of short latency blink CRs produced by associative pairing
84 anglion cells that encode light onset with a short latency burst of spikes.
85 itive resources when these events occur (via short latency bursting).
86 tual responses are automatically prepared at short latency but subsequently replaced by goal-directed
87    The network can be switched on and off at short latency by brief synaptic excitation and inhibitio
88                                              Short-latency Ca(2)(+) channel opening coupled to multiv
89                                              Short-latency CCEPs were observed when stimulating MMCx
90 nstrated that phrenic nerve afferents have a short-latency central projection to the SI somatosensory
91  signals to smooth pursuit and observed very short-latency changes in smooth eye movements to minimiz
92 rge visual stimuli, however, they respond at short latencies coincident with their target cells and s
93                                          Its short-latency component was abolished within the first h
94 cal coupling via gap junctions underlies the short-latency concerted spike activity of neighboring al
95 ing the IN with GABA(A) antagonists produces short-latency conditioned responses (SLRs).
96 mulation protocol to repeatedly activate the short-latency connection between the posterior parietal
97 s, we propose that the insula, via its known short-latency connections with the tectal system, mediat
98 monstrated that the neurons with stereotyped short latencies constitute an effective temporal referen
99              Electrical stimulation produced short-latency coordinated activity in the laryngeal nerv
100                              It follows that short latencies correlated significantly with low CFs, o
101 urthermore, demonstration of such transient, short-latency correlated firing between similar CA3 and
102 imilar (or identical), the short CS produced short-latency CRs in the left eye, whereas the long CS p
103               Despite its aggressiveness and short latency, current progress on its etiology, pathoge
104 ht EMGs and abnormal sharing of long but not short-latency cutaneomuscular reflexes.
105  was demonstrated by the presence of graded, short latency depolarising potentials following ventral
106 uency electrical stimulation in Uva elicited short-latency depolarizing postsynaptic potentials in HV
107 ow-intensity stimulation of the VLF evoked a short-latency depolarizing potential in the ventral root
108 tile and electrical stimuli showed different short-latency dipoles between the two age groups, and th
109  defining criterion, ignoring the concept of short latency distal contractions as an important featur
110 keep track of the target, on top of inducing short latency disturbance of grip force, single-pulse TM
111 ropose a different functional role for this 'short-latency dopamine response' in the mechanisms that
112 nd TRP to photoreceptor terminals produces a short-latency, dose-dependent hyperpolarization with a d
113 Ps were also present and superimposed on the short-latency DRPs on nearby roots.
114  distention and potential loss of stool, (2) short-latency EAS contraction when perceiving rectal dis
115  signalling that triggers a kinase-dependent short latency effect and a delayed longer latency effect
116 nable a behaviorally selective engagement of short-latency effector pathways.
117 latency EMG response was abolished while the short-latency EMG response was maintained.
118     Electrical stimulation of HG resulted in short-latency EPs in an area that overlaps PLST, indicat
119        A trial-by-trial analysis showed that short-latency ERP activity localized to auditory cortex
120                 Fish are elusive prey with a short-latency escape behavior--the C-start--initiated to
121 ponse to aversive stimuli is correlated with short-latency escapes [1-3].
122 etic activation increases the probability of short-latency escapes, supporting the notion that spiral
123 ns of spiral fiber neurons largely eliminate short-latency escapes.
124 s evoked by subthalamic stimulation revealed short latency events indicative of monosynaptic connecti
125                                   Widespread short latency excitation, compatible with monosynaptic t
126       Single stimulation of the STN evoked a short-latency excitation followed by a weak inhibition i
127 minals at low frequencies (</=1 Hz) evoked a short-latency excitation of BA interneurons (INs) that w
128 reward contexts, dopamine neurons acquired a short-latency excitation to aversive events that masked
129                                       Brief, short-latency excitations and reductions in firing were
130 STN stimulation pulses at 2.4-3.0 V revealed short-latency excitations at 2.5-4.5 and 5.5-7.0 msec af
131                         Large cells received short-latency excitatory inputs and had short first-spik
132 nctional classes of CT cells: those having a short-latency excitatory response to whisker deflection,
133                                      Maximal short-latency excitatory responses originated from stimu
134 lenged as a result of a lack of evidence for short-latency fear-related responses in primate amygdala
135  glomerular activation with highly reliable, short-latency firing consistent with tufted cell-mediate
136                                    We evoked short-latency fixed vector saccades at low currents (<50
137 , non-pyramidal neurons started spiking with short latency, followed by a decrease in firing frequenc
138 ch excited fast-spiking (FS) interneurons at short latency, followed by activation of E neurons and s
139               Some are also depolarised with short latencies following stimulation of a mesothoracic
140 s in the midbrain exterolateral nucleus at a short latency following receptor synchronization.
141                                          The short latency for activation of these eager sites sugges
142 with different skill levels, we found that a short latency for the first saccade distinguished good f
143 year after the end of treatment, with a very short latency from molecular to morphologic relapse, nec
144            ST shocks evoked large-amplitude, short-latency, glutamatergic EPSCs (ST-EPSCs) in 90% of
145           Killing all three cells eliminated short-latency, high-performance escape responses to both
146 tivated cation current (I(h)) contributes to short-latency, high-precision post-hyperpolarisation spi
147 us promoter with PML-RAR alpha to generate a short-latency, highly penetrant mouse model of APL.
148                Neurotrophin-3 normalized the short latency Hoffmann reflex to a treated hand muscle a
149 in vivo to efficiently trigger leukemia with short latency in the mouse.
150 tency measures show more events occur with a short latency in the presence of 5-HT.
151 opsins in the cerebral cortex evoked robust, short-latency increases in firing of cortical neurons.
152 ously we reported that vlPAG neurons showing short-latency increases in firing to a danger cue - the
153            Such beta-bursts were followed by short-latency increases of bilateral sensorimotor beta-b
154 in the pons of the rat is a locus supporting short-latency induction of a REM sleep-like state follow
155  that convey complementary, unambiguous, and short-latency information about antennal movement to tho
156                    All subjects demonstrated short latency inhibition and excitation between agonist/
157 hort-pass duration-selective cells exhibited short-latency inhibition and delayed excitation.
158 nsitive to GABA (0-40 nA, 20 s); most showed short-latency inhibitions during GABA diffusion from the
159  muscles to protract the vibrissae receive a short latency inhibitory input, followed by synaptic exc
160  two-component optical response containing a short latency initial-spike and a longer latency after-d
161 -potentials "input-output (IO) curve" and of short-latency intracortical inhibition (SICI curve), and
162 al excitability and GABA-A-receptor mediated short-latency intracortical inhibition (SICI) at rest du
163 of motor-evoked potentials and decreases the short-latency intracortical inhibition (SICI) in the vib
164 voked potentials, input-output (IOcurve) and short-latency intracortical inhibition (SICI) recruitmen
165 ctions [input-output curve (IOcurve)] and of short-latency intracortical inhibition (SICIcurve).
166                                              Short-latency intracortical inhibition can be considered
167  and first-episode patients showed a reduced short-latency intracortical inhibition compared with hea
168 t primary motor cortex was used to determine short-latency intracortical inhibition, intracortical fa
169 ies between 4 and 10 ms, and high amplitude [short-latency IPSCs (slIPSCs)].
170 loid progenitor immortalization in vitro and short latency leukemogenesis in vivo.
171                                 The ratio of short latency/long-lasting responses of cuneate neurons
172 m intact GCs, generating full-height APs and short-latency low-threshold Ca(2+) spikes.
173 mber of times that cells fired together with short latencies (&lt;50 ms) during exploration, and was str
174  of ipsilateral frontal cortex led to robust short-latency (&lt;20 ms) interneuron spiking, indicating m
175 d an even higher incidence than DeltaLR-9 of short-latency lymphomas with viral integrations into c-m
176  the MA protein was responsible for inducing short-latency lymphomas, we generated viruses with NRS p
177 f normal bursa and partially shared with the short-latency lymphomas.
178 t cause a high frequency of c-myb-associated short-latency lymphomas.
179 red mouse model in which all animals develop short latency malignant osteosarcoma.
180 also resulted in mammary tumors after only a short latency, many of which were positive for estrogen
181 at they respond to vibrissa stimulation with short latency (median = 7 ms) and large magnitude respon
182 nt background produced acute leukemia with a short latency (median survival, 67 days).
183              Typical clinical features were: short latency (median, 10 days) and abrupt onset of feve
184 eived sensory input from the hand, showing a short-latency modulation in their discharge following a
185                              We found robust short-latency modulations in eye velocity and accelerati
186                            The occurrence of short-latency mossy fiber-evoked granule cell epileptifo
187 ics, or predators, antennal contacts trigger short-latency motor responses.
188 th effective spike propagation, an extremely short-latency neuronal output is produced for greatly re
189                                              Short-latency neurons had a mean onset latency of 10.4 +
190 abbits performed fewer adaptive CRs and more short-latency non-adaptive responses than sham-lesioned
191                      Thyroid hormones induce short-latency nongenomic effects in adult brain tissue,
192                                          The short latency of attacks to water movements suggests shr
193                                 The similar, short latency of both the behavioral and neural effects
194                    Professional exposure and short latency of symptoms onset were risk factors for a
195                                 Based on the short latency of the onset of muscle activity (typically
196 ric acid), an mGluR6 agonist, blocks normal, short-latency ON responses but unmasks longer-latency on
197 or environmental influences are required for short latency oncogenic transformation.
198 nputs that fire the postsynaptic neuron with short latency or that act in correlated groups are able
199         Under physiological conditions, this short latency pathway was capable of facilitating optima
200 ng and electrical stimulation indicated that short-latency pathways linking motor cortex with spinal
201 is temporally stereotyped, consisting of two short latency peaks caused by convergent trigeminal syna
202 ngly implicated on the basis of significant, short-latency peaks in cross-correlogram plots.
203 idence for connectivity can be inferred from short-latency peaks in the correlogram between two neuro
204                          It is unlikely that short-latency perceptually correlated activity is inheri
205 otic twins is thought to be 100% with a very short latency period, suggesting that either the MLL fus
206  agreement with previous studies, we found a short-latency phase-locked current sink, thought to corr
207                      Many DRN neurons showed short-latency phasic activations in response to the cues
208 ected, biologically salient stimuli elicit a short-latency, phasic response in midbrain dopaminergic
209          Dopaminergic (DA) neurons exhibit a short-latency, phasic response to unexpected, biological
210                   Dopamine neurons display a short-latency, phasic reward signal indicating the diffe
211                                              Short-latency plasticity developed in the thalamus durin
212 ciated modulation of DCN activity included a short-latency post-CS inhibition and long-latency excita
213  using spike-triggered averages representing short-latency postspike facilitations to multiple motor
214 loproliferative phenotype (35%) or a lethal, short-latency pre-B-cell leukemia (20%).
215 as the strength of hippocampal bursts rises, short-latency prefrontal responses are augmented by incr
216 te can be bypassed if needed, but there is a short-latency preparatory step that is performed prior t
217                                              Short-latency, primary cortical evoked potentials (1 deg
218  base neurons (high frequency coding) showed short latency, rapidly adapting responses to the same st
219                                              Short-latency reciprocal inhibition from ankle flexor to
220                                              Short latency reflexes were unaffected.
221                                              Short-latency reflexes (SRs) in ventral roots, presumabl
222  human subjects, these stimuli induce robust short-latency reflexive vergence eye movements, initiall
223 actory sensory neuron (OSN) stimulation with short latencies regardless of stimulation intensity, MC
224                                 In contrast, short-latency response direction measured from ground-re
225 om 64 to 91%), and the proportion exhibiting short-latency response increased from 41 to 61%.
226 ne, and the progressive loss of tone-evoked, short-latency response over an initially large, very bro
227 ted by eye opening, leaving only the mature, short-latency response.
228 rigeminal nucleus interpolaris (SpVi) evoked short latency responses (median = 3.8 ms) in vibrissa-re
229                            Across all types, short latency responses are mostly evoked by L/M-opponen
230                                              Short latency responses were observed consistent with ac
231  located in the dorsal tip of LAd, exhibited short-latency responses (<20 ms) that were only transien
232 ut spares the expression of abnormally timed short-latency responses (SLRs).
233 -latency synaptic responses in lamina II and short-latency responses in lamina III.
234 uced high-frequency (70-150 Hz) activity and short-latency responses that phase-lock to rapid transie
235 learning: these neurons produced very brief, short-latency responses to rewarding stimuli; when the r
236 subthalamic nucleus, which may explain their short-latency responses to salient events; and the latte
237 sponses ranged from 40 to 60 Hz, whereas the short-latency responses were consistent from 5 to 130 Hz
238                                        These short-latency responses were present with 136 Hz stimula
239                                        These short-latency responses were used as an indication that
240 lity in the target thalamic neurons, evoking short-latency responses.
241 ined through conditional facilitation of the short-latency (Rl) component of the rat eyeblink reflex.
242                                              Short-latency saccades are determined mainly by salience
243  Stimulation of trigeminal afferents induced short-latency (SAI) but not long-latency (LAI) afferent
244  key brain area responsible for transmitting short-latency salience signals to thalamus and midbrain
245             These data suggest that although short-latency sensory input does not increase as symptom
246 l and quantitative assessments revealed that short-latency SEPs and somatosensory-induced gamma-oscil
247 atterns of neural activity was recorded: (1) short-latency, short-duration activation of neurons and
248 ly significant have the capacity to elicit a short-latency, short-duration burst of firing in mesence
249           In addition to the well documented short-latency side-band suppression elicited by masking
250 ssive or active coping strategies based on a short latency (SL) or longer latency (LL) to assume a de
251                                         Both short-latency (SL; 3-5 ms) and long-latency (LL; >/=9 ms
252 heir magnitude, progressive recruitment, and short latency, slIPSCs are a effective mechanism of regu
253 ns exhibited context-dependent spike firing; short-latency spike firing was greater to both CSs when
254            SGNs were phasic, firing a single short-latency spike for sustained currents of sufficient
255           This response was characterized by short latency spikes and spike adaptation for the durati
256 itionally, DA receptor activation attenuated short-latency spikes evoked by electrical stimulation of
257 ingle-pulse thalamic stimulation led to weak short-latency spiking, but firing probability increased
258  of gamma-motoneurone during walking through short latency spinal inhibitory pathways.
259 ivity in m. soleus and m. gastrocnemius is a short-latency spinal reflex triggered by ankle joint rot
260                            Here, we examined short-latency startle responses (SLCs) in larval zebrafi
261 hold of the network in response to a second, short-latency stimulus.
262                      We measured the size of short-latency, stimulus-induced visual bursts in superio
263 ells in the cerebellar cortex caused robust, short-latency suppression of preparatory activity in ant
264 classified as short latency abrupt (SL-A) or short latency sustained (SL-S).
265 rized as short-latency abrupt (SLA, n = 24), short latency sustained (SLS, n = 12), and long-latency
266 duration stimuli results from integration of short-latency, sustained inhibition with delayed, phasic
267 n the axon of a POMC neuron with autapses, a short-latency synaptic current was recorded in the same
268 ous synaptic excitation and highly reliable, short-latency synaptic inhibition onto granule cells via
269 x, spiny stellate cells predominate, receive short-latency synaptic inputs, and project to supergranu
270 with extrinsic current injection resulted in short-latency synchronized spiking in neighboring off-ce
271 ulation [5-7] and ablating M-cells abolishes short-latency tail-elicited startles [8, 9], we hypothes
272 livary gland tumors with 100% penetrance and short latency that showed a remarkable morphologic simil
273 pikes in inhibitory interneurons followed at short latency the onset of excitatory monosynaptic respo
274                                              Short-latency throughput from a recorded neuron to muscl
275  are selective for attack action and exhibit short-latency, time-locked spiking relative to the activ
276 processing into motor commands, responded at short latencies to the target stimulus whether or not th
277 lso appeared more anxious, as indicated by a short latency to vocalize when faced with a novel object
278                              Local DRPs with short latency-to-onset were evoked on roots close to the
279                     Surprisingly, changes in short-latency tone-evoked excitatory input cannot explai
280 timuli delivered to the first layer evoked a short-latency transient response followed by sustained a
281            Caudal STN units did not show the short-latency triphasic response but often displayed a p
282 al cortex, but not temporal cortex, evoked a short-latency triphasic response, followed by a sustaine
283 (robust nucleus of the arcopallium) caused a short-latency truncation of ongoing song syllables, whic
284 lightly truncated Myb protein, which induces short-latency tumors.
285 blockade substantially reduced spikes in all short-latency units (< 12 ms) but never in long-latency
286       NMDAR blockade had variable effects on short-latency units but reduced spikes substantially for
287                                          The short-latency units were recorded at an average depth of
288 what more effectively than NMDAR blockade in short-latency units, but NMDAR blockade reduced onset sp
289 ing later elements of sustained responses in short-latency units, whereas NMDAR blockade was much mor
290 ed by a weak inhibition in GPe neurons and a short-latency, very short-duration excitation followed b
291           A small proportion of cells showed short latency visual modulation that persisted during th
292 s from humans and found that monkeys exhibit short-latency visual components sensitive to sensory pro
293  the SC is a critical relay for transmitting short-latency visual information to DA neurons.
294 igral projection is ideally located to relay short-latency visual information to dopamine-containing
295 entary electrophysiological data reveal that short-latency visual responses in the SNc are abolished
296 culomotor periphery has privileged access to short-latency visual signals.
297 ectothalamic projection neurons can generate short-latency, well-timed, feed-forward inhibition, whic
298 ficial half of the GCL and were activated at short latencies, whereas those driven synaptically by AF
299 uced papillary carcinomas developing after a short latency, whereas BRAF point mutations were absent
300 uce the formation of adenocarcinomas after a short latency without additional genetic manipulation of

 
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