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1 d and reversed loss of saliva production and sialadenitis.
2 th Idd3 and Idd5) have been shown to control sialadenitis.
3 F-alpha; which replicates conditions seen in sialadenitis, an inflammation of the salivary glands res
4 eously develop autoimmune dacryoadenitis and sialadenitis and are a model for the human disorder Sjog
5 ed at different time points and examined for sialadenitis and salivary gland-infiltrating cells.
6                          Chronic lymphocytic sialadenitis and sicca syndrome have been reported in ch
7                  At 7 weeks, the severity of sialadenitis and the number of T and B cells infiltratin
8 ferent diseases, including Sjogren syndrome, sialadenitis, and iatrogenic disease, due to radiotherap
9 he HCV patients had low saliva flow, chronic sialadenitis, and SG fibrosis and lacked Sjogren syndrom
10 xperienced observers, nonspecific changes of sialadenitis are frequently confused with the focal lymp
11 lpha-transgenic mice develop autoantibodies, sialadenitis, as in Sjogren's syndrome, and immune compl
12                                 IgG4-related sialadenitis belongs to the IgG4-related systemic diseas
13 port the evolving concept of IgG4-associated sialadenitis/dacroadenitis.
14 pecimens included 61% with focal lymphocytic sialadenitis (FLS; 69% of which had focus scores of >/=1
15  development of salivary gland inflammation (sialadenitis), indicating that the effect was tissue-sel
16                 The course of iodine-induced sialadenitis is extremely benign, and rapid resolution o
17 t, and the pathophysiology of iodine-induced sialadenitis is not yet fully known.
18 "atypical" manifestations were noted: fever, sialadenitis, lymphadenopathy, erythema nodosum, leukocy
19  autopsy survey study, indicating that focal sialadenitis may occur in the absence of SS.
20                                Myoepithelial sialadenitis (MESA) is the reactive salivary gland lymph
21                 Elimination of IL-17 reduced sialadenitis more drastically in females than males.
22 d 37% with nonspecific or sclerosing chronic sialadenitis (NS/SCS).
23 oints did not correlate with the severity of sialadenitis or levels of autoantibodies.
24 e did not reduce the occurrence of radiation sialadenitis or stomatitis.
25 ary glands from 37 SS patients and 9 chronic sialadenitis patients were analyzed by immunohistochemis
26 gren's disease (SjD) is a chronic autoimmune sialadenitis resulting in salivary gland hypofunction wi
27 nces between the groups in the prevalence of sialadenitis, stomatitis, xerostomia, or dysgeusia over
28                A diagnosis of iodine-related sialadenitis was made.
29 ,726 LSG specimens exhibiting any pattern of sialadenitis, we compared biopsy diagnoses against concu