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1 d and reversed loss of saliva production and sialadenitis.
2 th Idd3 and Idd5) have been shown to control sialadenitis.
3 F-alpha; which replicates conditions seen in sialadenitis, an inflammation of the salivary glands res
4 eously develop autoimmune dacryoadenitis and sialadenitis and are a model for the human disorder Sjog
8 ferent diseases, including Sjogren syndrome, sialadenitis, and iatrogenic disease, due to radiotherap
9 he HCV patients had low saliva flow, chronic sialadenitis, and SG fibrosis and lacked Sjogren syndrom
10 xperienced observers, nonspecific changes of sialadenitis are frequently confused with the focal lymp
11 lpha-transgenic mice develop autoantibodies, sialadenitis, as in Sjogren's syndrome, and immune compl
14 pecimens included 61% with focal lymphocytic sialadenitis (FLS; 69% of which had focus scores of >/=1
15 development of salivary gland inflammation (sialadenitis), indicating that the effect was tissue-sel
18 "atypical" manifestations were noted: fever, sialadenitis, lymphadenopathy, erythema nodosum, leukocy
25 ary glands from 37 SS patients and 9 chronic sialadenitis patients were analyzed by immunohistochemis
26 gren's disease (SjD) is a chronic autoimmune sialadenitis resulting in salivary gland hypofunction wi
27 nces between the groups in the prevalence of sialadenitis, stomatitis, xerostomia, or dysgeusia over
29 ,726 LSG specimens exhibiting any pattern of sialadenitis, we compared biopsy diagnoses against concu