ライフサイエンスコーパス: skeletal muscle

1 2) , especially within fast-twitch oxidative skeletal muscle.

2 d ventricles that are distinct from those of skeletal muscle.

3 glucose transporter 4 translocation in mouse skeletal muscle.

4 d uptake and protein synthesis in IUGR fetal skeletal muscle.

5 ochondrial density and ETS proteins in fetal skeletal muscle.

6 1, in exercise-induced activation of AMPK in skeletal muscle.

7 ize, and the limits of adaptability in adult skeletal muscle.

8 me, as a hallmark of aged tissues, including skeletal muscle.

9 peripheral nervous system, liver, kidney and skeletal muscle.

10 mes play distinct roles in TAG metabolism in skeletal muscle.

11 ional role of DOCK3 in normal and dystrophic skeletal muscle.

12 ing inflammatory stimulation by TNF-alpha in skeletal muscle.

13 ne signaling and mitochondrial physiology in skeletal muscle.

14 ate and nitrite on mitochondrial function in skeletal muscle.

15 re preferentially associated with obesity in skeletal muscle.

16 not affect NAD metabolite concentrations in skeletal muscle.

17 measure the PCr concentrations in exercised skeletal muscle.

18 tes repair of acutely or chronically injured skeletal muscle.

19 also by a versatile circadian system within skeletal muscle.

20 B) phosphorylation in endothelial cells and skeletal muscle.

21 A-catabolizing enzyme expression only in the skeletal muscle.

22 se is a bona fide environmental time cue for skeletal muscle.

23 ntly increased ATP and NAD(+) levels in mice skeletal muscle.

24 ely to negatively regulate both processes in skeletal muscle.

25 in progressive weakness and degeneration of skeletal muscle.

26 ee of redundance for the two transporters in skeletal muscle.

27 drial respiration, content and morphology in skeletal muscle.

28 peripheral dysfunction, particularly within skeletal muscle.

29 arterioles in the heart, adipose tissue and skeletal muscle.

30 oint of connection between motor neurons and skeletal muscle.

31 anes isolated from control and IUGR hindlimb skeletal muscle.

32 nce for reduced pH-buffering capacity in the skeletal muscle.

33 sies of affected tissues, such as kidney and skeletal muscle.

34 xcitation-contraction coupling in vertebrate skeletal muscles.

35 fl2 mRNA levels in various tissues including skeletal muscles.

36 es, but expression was considerably lower in skeletal muscles.

37 res of sophisticated biological devices like skeletal muscles.

38 FSHD) is caused by the expression of DUX4 in skeletal muscles.

39 A transgenic mouse model with elevated skeletal muscle 2-deoxy-ATP (dATP) was used to study how

40 beclin 1, is required for AMPK activation in skeletal muscle(3).

41 Sarcopenia is characterized by low skeletal muscle, a complex trait with high heritability.

42 etes mellitus (DM2) and DPN.PurposeTo assess skeletal muscle abnormalities in participants with DM2 w

43 d skeletal muscle NAD+ metabolites, affected skeletal muscle acetylcarnitine metabolism, and induced

44 njamini-Hochberg correction, actin, alpha 1, skeletal muscle (ACTA1) was found to be significantly in

45 pyrene)iodoacetamide was first used to label skeletal muscle actin in 1981, the pyrene-labeled actin

46 Bcl2 mediates exercise-induced autophagy and skeletal muscle adaptions to training during high-fat di

47 ide and methyl nicotinamide-were elevated in skeletal muscle after NR compared with placebo.

48 activation or suppression is beneficial for skeletal muscle aging.

49 Here we use skin epithelium and skeletal muscle-among the most highly-stressed tissues i

50 Aging is associated with skeletal muscle anabolic resistance (i.e., reduced muscl

51 ion based on their physical location between skeletal muscle and bone, tendon is a surprisingly genet

52 GPRC6A's unique regulation of beta-cell, skeletal muscle and hepatic function may represent a new

53 and unsaturated long-chain FAs (LCFAs) into skeletal muscle and knockdown (Kd) of a subset of RabGAP

54 nervous, cardiovascular and immune systems, skeletal muscle and metabolic regulation as well as agei

55 Expression in skeletal muscle and organs across animal species remains

56 athies (IIM) involve chronic inflammation of skeletal muscle and subsequent muscle degeneration due t

57 both biochemical and biomechanical roles in skeletal muscle and that mitochondrial dynamics can be m

58 - 4.4 years) and aged (83 +/- 4 years) human skeletal muscle and that of young/aged heterogenous musc

59 x7 expression marks stem cells in developing skeletal muscles and adult satellite cells during homeos

60 id nutrients for vital tissues (e.g., heart, skeletal muscle, and adipose tissue).

61 se and lipid metabolism in the liver, heart, skeletal muscle, and adipose tissue.

62 es suppress sarcolemmal resealing in healthy skeletal muscle, and depletion of TRIM72 antibodies from

63 mitochondrial coupling efficiency in murine skeletal muscle, and expression of UCP3, AAC1, or AAC2,

64 d mice alters aging phenotypes in the brain, skeletal muscle, and heart.

65 xpression of which was recently confirmed in skeletal muscle, and its down-regulation is linked to re

66 abolic organs, including the adipose tissue, skeletal muscle, and liver by 9 weeks post-infection.

67 as a negative regulator of glucose uptake by skeletal muscle, and of pancreatic beta-cell phenotype i

68 iral-mediated gene transfer to liver, heart, skeletal muscle, and the central nervous system, its use

69 cretome that targets distant adipose depots, skeletal muscle, and the nervous system.

70 total force-producing capacity of exercising skeletal muscle are altered during OCC.

71 total force-producing capacity of exercising skeletal muscle are significantly altered during blood f

72 o data on the usefulness of these markers in skeletal muscle are very limited and inconsistent.

73 Skeletal muscle area z scores were significantly predict

74 High-quality PCr mapping of human skeletal muscle, as well as the information of exchange

75 odel are consistent with a Zip14 function in skeletal muscle at steady state that supports myogenesis

76 Skeletal muscle atrophy is a highly-prevalent and debili

77 1) day(-1) ) induces similar improvements in skeletal muscle autophagic flux and contractility proper

78 The miRNome profiles of skeletal muscle biopsies acquired from four different MD

79 is the first study to use cells derived from skeletal muscle biopsies in CFS patients and healthy con

80 Skeletal muscle biopsies were collected before and after

81 mparison between NDD-CKD and HC populations, skeletal muscle biopsies were collected from the vastus

82 ession is an inconsistent biomarker for FSHD skeletal muscle biopsies, displaying efficacy only on pa

83 lization of the GLUT4 glucose transporter in skeletal muscle, but are not deficient in autophagy.

84 lentivirus achieved comparable expression in skeletal muscle, but did not ameliorate the disease phen

85 uced expression of the ryanodine receptor in skeletal muscle, but its observed content is even lower.

86 le: Sleep deprivation can alter endurance of skeletal muscles, but its impact on respiratory command

87 e provide evidence that loss of lamin A/C in skeletal muscles, but not osteoblast (OB)-lineage cells,

88 es the dystrophic phenotype of DMD-afflicted skeletal muscle by dysregulating muscle stem cells invol

89 f low SMN in one relevant peripheral organ - skeletal muscle - by selectively depleting the protein i

90 re mitophagy directly, we generated a stable skeletal muscle C2C12 cell line, expressing a mitophagy

91 he liver and lactate derived from exercising skeletal muscle can also become important energy substra

92 papers coalesced anatomical observations of skeletal muscle capillary numbers with O(2) diffusion th

93 ls via incorporation of target sequences for skeletal muscle cell-specific miR-206.

94 unctional validation in human adipocytes and skeletal muscle cells (SKMCs) confirmed the relevance of

95 At all stages of skeletal muscle cells differentiation, we show a permane

96 It has previously been shown that CFS skeletal muscle cells have lower levels of ATP and have

97 erm SKE, displayed diminished replication in skeletal muscle cells in a mouse model of CHIKV disease.

98 ess the contribution of CHIKV replication in skeletal muscle cells to pathogenesis, we engineered a C

99 recapitulated by simulating lipotoxicity in skeletal muscle cells treated with saturated FA, palmita

100 cells to generate both the motor neurons and skeletal muscle cells used.

101 investigated the gene expression patterns of skeletal muscle cells using RNA-seq of subtype-pooled si

102 ults showed that DGAT1 was dominant in human skeletal muscle cells utilizing fatty acids (FAs) derive

103 metabolism and its underlying mechanisms in skeletal muscle cells, and evaluated whether the observe

104 llular oxygen consumption, and glycolysis in skeletal muscle cells.

105 2 affected lipid metabolism in human primary skeletal muscle cells.

106 d small-molecule compounds that modulate the skeletal muscle channel isoform (RyR1) interaction with

107 ey role of the metabolic-sensing function of skeletal muscle clock in partitioning nutrient flux betw

108 effect of one bout of treadmill exercise on skeletal muscle clock phase changes.

109 sured the effect of 7 days' HFHC diet on (1) skeletal muscle concentration of lipid metabolites, and

110 Neural input into this bioprinted skeletal muscle construct shows the improvement of myofi

111 A bioengineered skeletal muscle construct that mimics structural and fun

112 neural cell integration into the bioprinted skeletal muscle construct to accelerate functional muscl

113 suggest that the 3D bioprinted human neural-skeletal muscle constructs can be rapidly integrated wit

114 We previously showed that bioprinted human skeletal muscle constructs were able to form multi-layer

115 SPEG-deficient skeletal muscles contained fewer myogenic cells, which o

116 The study of skeletal muscle continues to support the accurate diagno

117 ip, the effects of mitochondrial dynamics on skeletal muscle contractility are poorly understood.

118 iggered Ca(2+) release and its influences on skeletal muscle contractility are widely used as experim

119 Skeletal muscle contraction in these mice, however, was

120 al cellular functions, including cardiac and skeletal muscle contraction.

121 phosphate-activated protein kinase (AMPK) in skeletal muscle coordinates systemic metabolic responses

122 f the cytotoxic protein levels and increased skeletal muscle cross-sectional area and contractility p

123 , an essential NAD(+) biosynthetic enzyme in skeletal muscle, decreased by 14% with NR.

124 of skeletal muscle mass, and to evaluate the skeletal muscle density (SMD).

125 ifferentiated myocytes is a critical step in skeletal muscle development and repair.

126 Mechanical stimulation can regulate skeletal muscle differentiation, growth and metabolism;

127 PE caused robust vasoconstriction in resting skeletal muscle during control vasodilator infusions (De

128 KC3-C2)-which contains beclin 1 and UVRAG-in skeletal muscle during exercise, and knockout of beclin

129 ues (ectopic fat deposition in liver, heart, skeletal muscle, etc).

130 EV biology and what is currently known about skeletal muscle EVs and their potential role in the resp

131 tive correlation between NOX4 expression and skeletal muscle fiber cross-sectional area in pancreatic

132 himeras protected them from both cardiac and skeletal muscle fiber damage.

133 Furthermore, repletion of vitamin D improved skeletal muscle fiber size and in vivo muscle function,

134 Transport distances in skeletal muscle fibers are mitigated by these cells havi

135 In skeletal muscle fibers, mitochondria are densely packed

136 K(+) released from active skeletal muscle fibres could facilitate vasodilatation i

137 ivation in intact loose-patch clamped murine skeletal muscle fibres subject to a double pulse procedu

138 evelopment of cachexia, as well as liver and skeletal muscle fibrosis, is dependent on intact signali

139 3019 for therapeutic treatment of persistent skeletal muscle fibrosis, such as those induced with chr

140 perfusion (IR) injury results in devastating skeletal muscle fibrosis.

141 e model of hindlimb IR injury which leads to skeletal muscle fibrosis.

142 m pathways and molecular networks of Nrf2 in skeletal muscle following Nrf2 or Keap1 deletion.

143 with reported kinetics from bulk studies of skeletal muscle for the relaxed and SRX subpopulations,

144 he effects of augmented nitric oxide (NO) on skeletal muscle force production and oxygen consumption

145 illion deletions (~ 470,000 unique spans) in skeletal muscle from 22 individuals with and 19 individu

146 We compared metabolic parameters of skeletal muscle from global Zip14 knockout (KO) and wild

147 ng small RNA sequencing of brain, heart, and skeletal muscle from individuals in late hibernation and

148 activation and stimulation of AMP kinase in skeletal muscle from smPit1(-/-); smPit2(-/-) mice consi

149 rf2 or Keap1 separately impaired or improved skeletal muscle function.

150 in adipose tissue, which, in turn, supports skeletal muscle function.

151 ents reveal that CASZ1 directly up-regulates skeletal muscle genes and represses non-muscle genes thr

152 The effect of such treatments on juvenile skeletal muscle growth has yet to be investigated.

153 y of growth factors and negatively regulates skeletal muscle growth.

154 th muscle of resistance arterioles supplying skeletal muscle, heart and adipose tissue.

155 However, only the KI/KO mice have clear skeletal muscle histologic changes in MFM.

156 cin pathway were similar in control and IUGR skeletal muscle homogenate.

157 hermia (MH) is characterized by induction of skeletal muscle hyperthermia in response to a dysregulat

158 affected child revealed complete absence of skeletal muscle (i.e., segmental amyoplasia).

159 is associated with molecular adaptations in skeletal muscle, improving glucose uptake and metabolism

160 n of thermogenic genes in adipose tissue and skeletal muscle in CKD mice.

161 very of drug-loaded liposomes to an inflamed skeletal muscle in mice.

162 tudies and suggest an active contribution of skeletal muscle in NMJ dysfunction.

163 hat has been shown to be produced acutely by skeletal muscle in response to exercise, yet chronically

164 d the impact of VDR knockdown (KD) on mature skeletal muscle in vivo, and myogenic regulation in vitr

165 ng from ~ 25% in heart muscle to ~ 30-35% in skeletal muscles in vivo.

166 animal models also indicates involvement of skeletal muscle including loss of fast-twitch type 2 fib

167 During exercise, blood flow to working skeletal muscle increases in parallel with contractile a

168 , CT derived body composition as measured by skeletal muscle index (SMI) and skeletal muscle radioden

169 third lumbar vertebra (L3), to estimate the skeletal muscle index (SMI), a surrogate of skeletal mus

170 Despite the low skeletal muscle index and significant muscle fiber atrop

171 Denervation of skeletal muscles induces severe muscle atrophy, which is

172 Using a model of skeletal muscle injury and repair, herein we identified

173 Regenerative response to skeletal muscle injury in Speg-KO mice was compared with

174 A1/FPR2/AMPK axis as an important pathway in skeletal muscle injury regeneration.

175 c program elicited in mSCs early after acute skeletal muscle injury.

176 an energy-matched control on whole-body and skeletal muscle insulin and anabolic sensitivity.

177 These included miRNAs with functions in skeletal muscle insulin metabolism (miR-106b and miR-20b

178 iR-20b-5p) and miRNAs with functions in both skeletal muscle insulin metabolism and cell cycle regula

179 oplets (LDs) does not directly contribute to skeletal muscle insulin resistance.

180 of lipid metabolites known to contribute to skeletal muscle insulin resistance.

181 nd improved whole-body glucose clearance and skeletal muscle insulin sensitivity along with enhanced

182 Skeletal muscle insulin sensitivity was determined using

183 accumulation of lipid metabolites to protect skeletal muscle insulin signalling following 7 days' HFH

184 lation of lipid metabolites known to disrupt skeletal muscle insulin signalling in sedentary and obes

185 ole body glucose clearance without impairing skeletal muscle insulin signalling, in healthy lean indi

186 model consisting of motor neurons coupled to skeletal muscles interacting via the neuromuscular junct

187 s across four murine muscular organs: heart, skeletal muscle, intestine and bladder.

188 ssing insulin receptors (IR) specifically in skeletal muscle (IRMOE).

189 Although skeletal muscle is a key peripheral tissue, it remains u

190 Skeletal muscle is a key site of shivering and non-shive

191 ral and functional characteristics of native skeletal muscle is a promising therapeutic option to tre

192 Maintenance of skeletal muscle is beneficial in obesity and Type 2 diab

193 ue that the normally low MHC I expression in skeletal muscle is host protective by allowing for patho

194 and cachexia, suggesting that denervation of skeletal muscle is not a major driver of pathogenesis.

195 ese results suggest that the loss of ARNT in skeletal muscle is partially responsible for diminished

196 Accumulation of lipid in skeletal muscle is thought to be related to the developm

197 Secondary mitochondrial damage in skeletal muscles is a common feature of different neurom

198 r, these results suggest a critical role for skeletal muscle lamin A/C to prevent cellular senescence

199 demonstrate that RabGAP-mediated control of skeletal muscle lipid metabolism converges with glucose

200 we present evidence for a novel mechanism of skeletal muscle lipid utilization involving the two RabG

201 Three established sarcopenia definitions - %Skeletal Muscle Mass (%SMM), Skeletal Muscle Mass Index

202 Percentages of body fat (BF) and skeletal muscle mass (SM) were calculated using validate

203 definitions - %Skeletal Muscle Mass (%SMM), Skeletal Muscle Mass Index (SMI) and European Working Gr

204 we calculated body fat percentage (%BF) and skeletal muscle mass index (SMI).

205 light the autonomous role the VDR has within skeletal muscle mass regulation.

206 was assessed with a handgrip dynamometer and skeletal muscle mass was estimated using bioelectrical i

207 skeletal muscle index (SMI), a surrogate of skeletal muscle mass, and to evaluate the skeletal muscl

208 cytoskeleton and the extracellular matrix in skeletal muscle may contribute to reduced amino acid met

209 cytoskeleton and the extracellular matrix in skeletal muscle may contribute to reduced amino acid met

210 Future studies on the effects of NR on human skeletal muscle may include both sexes and potentially p

211 y strong and systemically dominant effect of skeletal muscle MHC expression on maintaining T cell fun

212 Inducible enhancement of skeletal muscle MHC I in mice during the first 20 d of T

213 h convective arterial oxygen delivery to the skeletal muscle microvasculature and subsequent diffusiv

214 and respiratory systems to supply oxygen to skeletal muscle mitochondria for energy production neede

215 ong the O(2) transport pathway from lungs to skeletal muscle mitochondria.

216 NDD-CKD patients exhibit reduced skeletal muscle mitochondrial mass and gene expression o

217 To this end, phenotypic ALS skeletal muscle models were developed from induced pluri

218 We identified 11 human skeletal muscle mononuclear cell types, including two fi

219 variants can be increased in human and mouse skeletal muscle myoblast cell lines using a single-guide

220 function of regulating enhancer activity in skeletal muscle myoblasts cells, further confirming the

221 mRNA transcript variants in human and mouse skeletal muscle myoblasts promoted myotube differentiati

222 s been accepted that the force produced by a skeletal muscle myofibril depends on its cross-sectional

223 ydrate on whole-body protein metabolism, and skeletal muscle myofibrillar (MyoPS) and mitochondrial (

224 Targeting skeletal muscle myosin by MPH-220 enabled muscle relaxat

225 is and the atomic structure of MPH-220-bound skeletal muscle myosin confirmed the mechanism of specif

226 ture of an insect myosin: the D melanogaster skeletal muscle myosin II embryonic isoform (EMB).

227 , which represents the CaM binding domain of skeletal muscle myosin light chain kinase, forms a compl

228 overweight or obese men and women increased skeletal muscle NAD+ metabolites, affected skeletal musc

229 leading to instability of the sarcolemma and skeletal muscle necrosis and atrophy.

230 h we demonstrated that selective deletion of skeletal muscle Nrf2 or Keap1 separately impaired or imp

231 mitochondria-enriched proteome of heart and skeletal muscle of aged mutator mice.

232 tectable in the primary olfactory system and skeletal muscle of Carns1-deficient mice.

233 ng intensity and improvements in VO(2max) In skeletal muscle of CON but not PCOS, training increased

234 mmatory cytokines was increased in liver and skeletal muscle of CysC KO mice.

235 The skeletal muscle of fruit flies communicates with other o

236 abundant peptides in the nervous system and skeletal muscle of many vertebrates.

237 he most highly expressed zinc transporter in skeletal muscle of mice in response to LPS-induced infla

238 performed single-nucleus transcriptomics of skeletal muscle of mice with dystrophin exon 51 deletion

239 lerance, and insulin resistance in liver and skeletal muscle of obese mice, and such effects were ass

240 oxia signaling pathway, was less abundant in skeletal muscle of old (23-25 months old) mice.

241 of the lower esophageal sphincter (LES) and skeletal muscle of the crural diaphragm (esophagus hiatu

242 such as VEGFA and CDH5 which were blunted in skeletal muscles of 28 week old mice were found to be up

243 -resistant glycogen in as little as 30 mg of skeletal muscle or a single hippocampus from Lafora dise

244 t analysis, at three months post excision in skeletal muscles or by 6 months post gene excision in he

245 Knocking out Nox4 in skeletal muscles or pharmacological blockade of Nox4 act

246 ction force, microvascular O(2) delivery and skeletal muscle oxidative metabolism.

247 f acute nitrite infusion on muscle force and skeletal muscle oxidative metabolism.

248 l and histological progression of the D2.mdx skeletal muscle pathology was evaluated to determine the

249 The conversion of proliferating skeletal muscle precursors (myoblasts) to terminally-dif

250 pCHi-C), and other genome-wide approaches in skeletal muscle progenitors that inducibly express a mas

251 ults showed that PLV-LMCs do not derive from skeletal muscle progenitors.

252 Aging appears to attenuate the response of skeletal muscle protein synthesis (MPS) to anabolic stim

253 s of beta(2) -adrenoceptor activation on the skeletal muscle proteostasis and contractility propertie

254 neficial effects of beta(2) -adrenoceptor on skeletal muscle proteostasis and contractility propertie

255 measured by skeletal muscle index (SMI) and skeletal muscle radiodensity (SMD), the systemic inflamm

256 easured biomechanical changes that accompany skeletal muscle regeneration and determined the implicat

257 miR-206 is required for skeletal muscle regeneration in vivo.

258 findings contribute to the understanding of skeletal muscle regeneration through the identification

259 e linked to the inflammatory response during skeletal muscle regeneration, suppressed Fbxl2 mRNA expr

260 In mice and humans, exercising skeletal muscle releases the mitochondrial metabolite su

261 patiotemporal control of self-renewal during skeletal muscle repair.

262 l-based transcriptome analyses revealed that skeletal muscle-resident macrophages are distinctive fro

263 pical glibenclamide superfused onto hindlimb skeletal muscle) resolved a decreased blood flow and int

264 letion of Gprc6a in pancreatic beta-cell and skeletal muscle respectively impair insulin secretion an

265 ue of the JCI, Lentscher et al. engineered a skeletal muscle-restricted CHIKV to show that while musc

266 anics in contracting intact fibres from frog skeletal muscle reveal an I-band spring with an undamped

267 function, was significantly reduced in IUGR skeletal muscle sarcolemma compared to control.

268 yrromethene-labeled ATP molecules in relaxed skeletal muscle sarcomeres from rat soleus.

269 High levels of skeletal muscle sensory feedback related to peripheral f

270 We observe a cell population with a skeletal muscle signature in etv2-deficient embryos.

271 use-keeping Pi transporters Pit1 and Pit2 in skeletal muscle (sm), using the postnatally expressed hu

272 Myosin heavy chain-embryonic (MyHC-emb) is a skeletal muscle-specific contractile protein expressed d

273 in part mediated by the release of myokines, skeletal muscle-specific cytokines, in response to exerc

274 hanism for beta(2) -adrenoceptor activation, skeletal muscle-specific deletion of ATG7 blunts the ben

275 Employing skeletal muscle-specific transgenic mouse models with un

276 ain/spinal cord and assemble them with human skeletal muscle spheroids to generate 3D cortico-motor a

277 Skeletal muscle-targeted Lrrc8a KO mice have smaller myo

278 SLN has also been implicated in skeletal muscle thermogenesis.

279 ndrial dysfunction and structural changes in skeletal muscle tissue remains to be discovered.

280 single-cell RNA sequencing to profile human skeletal muscle tissues from embryonic, fetal, and postn

281 The ability of contracting skeletal muscle to attenuate sympathetic vasoconstrictio

282 The ability of skeletal muscle to regenerate declines significantly wit

283 GCG (sarcoglycan gamma), highly expressed in skeletal muscle, to concordantly associate with weight l

284 Northern blots from skeletal muscle total RNA showed severe reduction in abu

285 utaneous WAT and can be greater than that in skeletal muscle, underscoring the potential of BMAT to i

286 89 (0.18, 3.60); P = 0.03; eta2p = 0.29] and skeletal muscle uptake of glucose [between-group differe

287 ous quantification of perfusion and T(2)* in skeletal muscle using the developed technique.

288 Altogether, our data demonstrate that skeletal muscles utilize miR-133b to mitigate the delete

289 Skeletal muscle wasting is also common in COPD, but less

290 g for sarcopenia, a debilitating age-related skeletal muscle wasting syndrome.

291 s compared to cytoplasmic CELF1 functions in skeletal muscle wasting.

292 To assess miR-133b function in DMD-affected skeletal muscles, we genetically ablated miR-133b in the

293 Skeletal muscle weakness and eventual muscle degradation

294 myopathy characterized by slowly progressive skeletal muscle weakness and wasting.

295 The mice demonstrated skeletal muscle weakness but did not experience early mo

296 ecapitulates hypertrophic cardiomyopathy and skeletal muscle weakness of human IOPD, indicating its u

297 ty, transgenic mice expressing human BDNF in skeletal muscle were crossed with '97Q' KD mice.

298 stprandial glycogen storage in the liver and skeletal muscle were not altered.

299 ism, and muscle differentiation in recruited skeletal muscles, which were confirmed by increased expr

300 , but not entirely, positive for aging mouse skeletal muscle, while genetic, muscle fiber-specific ac