ライフサイエンスコーパス: skeletal muscle cell

1 he proton-motive force, throughout the mouse skeletal muscle cell.

2 determining the metabolic health of the aged skeletal muscle cell.

3 ting the multifunctional role of Staufen1 in skeletal muscle cells.

4 the PGC-1alpha-mediated hypoxic response in skeletal muscle cells.

5 vascular endothelial growth factor (VEGF) in skeletal muscle cells.

6 ose uptake by target tissues such as fat and skeletal muscle cells.

7 ed rates of complete fatty acid oxidation in skeletal muscle cells.

8 2 affected lipid metabolism in human primary skeletal muscle cells.

9 ylcholine receptors (AChRs) to the center of skeletal muscle cells.

10 en species that promote damage in dystrophic skeletal muscle cells.

11 ptional activation during differentiation of skeletal muscle cells.

12 a mechanism that was independent of mTOR in skeletal muscle cells.

13 ically, the inhibition of mTOR signalling in skeletal muscle cells.

14 ully dependent on mTOR signalling within the skeletal muscle cells.

15 tatively regulated by PGC-1alpha in cultured skeletal muscle cells.

16 cal for maintenance of metabolic function in skeletal muscle cells.

17 hancer that supports Igf2 gene activation in skeletal muscle cells.

18 orm as the mediator of NO-induced effects in skeletal muscle cells.

19 eta, localized at the Golgi complex in mouse skeletal muscle cells.

20 s a negative modulator of agrin signaling in skeletal muscle cells.

21 sponsive Glut4-storage vesicles from fat and skeletal muscle cells.

22 , insulin resistance, and protein wasting in skeletal muscle cells.

23 creased insulin-stimulated glucose uptake in skeletal muscle cells.

24 ilization, and fatty acid oxidation (FAO) in skeletal muscle cells.

25 lecules that induce PGC-1alpha expression in skeletal muscle cells.

26 -fos expression by junctional epithelial and skeletal muscle cells.

27 in zebrafish embryos, and in vitro in murine skeletal muscle cells.

28 el to study the differentiation of fetal rat skeletal muscle cells.

29 A) to promote inappropriate proliferation of skeletal muscle cells.

30 ved in TNF-alpha-induced MMP-9 production in skeletal muscle cells.

31 I-induced inhibition of insulin signaling in skeletal muscle cells.

32 retain expression of some proteins common to skeletal muscle cells.

33 giform papillae in the tongue, as well as in skeletal muscle cells.

34 blunted SRF-dependent transcription in C2C12 skeletal muscle cells.

35 ain flotillin-1, a marker of lipid rafts, in skeletal muscle cells.

36 y physiological form of Ca(V)1.1 channels in skeletal muscle cells.

37 se did not determine reovirus replication in skeletal muscle cells.

38 K(b) induced ER stress response in C(2)C(12) skeletal muscle cells.

39 without altering the mRNA level in cultured skeletal muscle cells.

40 ed for peptide selection against C2C12 mouse skeletal muscle cells.

41 ative multipotent etv2 progenitor cells into skeletal muscle cells.

42 ed reduction in IRS-1 expression in cultured skeletal muscle cells.

43 by insulin and hyperosmotic stress in L6 rat skeletal muscle cells.

44 human endothelial cells and also apparently skeletal muscle cells.

45 sulin-independent basal glucose uptake in L6 skeletal muscle cells.

46 enchymal stem cells and dedifferentiation in skeletal muscle cells.

47 pstream of genes differentially expressed in skeletal muscle cells.

48 ipant in the agrin-MuSK signaling pathway of skeletal muscle cells.

49 for tissue-specific expression in liver and skeletal muscle cells.

50 (nNOS), which is expressed constitutively in skeletal muscle cells.

51 tive calcium entry (CCE) in Jurkat and in L6 skeletal muscle cells.

52 hat is expressed specifically in cardiac and skeletal muscle cells.

53 ompartment to the plasma membrane in fat and skeletal muscle cells.

54 yte chemoattractant protein 1) production by skeletal muscle cells.

55 abolism and gene expression in primary human skeletal muscle cells.

56 nts with type 2 diabetes, into primary human skeletal muscle cells.

57 is critical for terminal differentiation of skeletal muscle cells.

58 nt potentiation of L-type Ca(2+) channels in skeletal muscle cells.

59 +)](i) in type I astrocytes, neurons, and in skeletal muscle cells.

60 ptional control seen in ordinary cardiac and skeletal muscle cells.

61 t the z-line of the sarcomere of cardiac and skeletal muscle cells.

62 s of myositis patients and in cultured human skeletal muscle cells.

63 ultrastructural changes in both cardiac and skeletal muscle cells.

64 increased basal glycogen synthesis in human skeletal muscle cells.

65 Two pools of IRE1alpha in cardiac and skeletal muscle cells.

66 panded AR causes damage to motor neurons and skeletal muscle cells.

67 llular oxygen consumption, and glycolysis in skeletal muscle cells.

68 ARP2/3 subunit p41ARC is a PAK1 substrate in skeletal muscle cells.

69 that lncRNA AK017368 is highly expressed in skeletal muscle cells.

70 n secretion and enhances glucose uptake into skeletal muscle cells.

71 esicle translocation and glucose uptake into skeletal muscle cells.

72 us genome inserted into the genome of murine skeletal muscle cells.

73 anslocation in both 3T3-L1 adipocytes and L6 skeletal muscle cells.

74 drial morphology and fission protein Drp1 in skeletal muscle cells.

75 K by causing Ser(485/491) phosphorylation in skeletal muscle cells.

76 and insulin signaling is validated in human skeletal muscle cells.

77 ble-homeodomain transcription factor DUX4 in skeletal muscle cells.

78 egulate proliferation and differentiation of skeletal muscle cells.

79 ogical functions of these receptors in mouse skeletal muscle cells.

80 hat SMAD3 suppresses FNDC5 and PGC-1alpha in skeletal muscle cells.

81 ogical functions of these receptors in mouse skeletal muscle cells.

82 ncode proteins of the contractile complex of skeletal muscle cells.

83 or-derived microvesicles induce apoptosis of skeletal muscle cells.

84 gehog (Hh) signaling, and differentiation of skeletal muscle cells.

85 e molecules that are up-regulated on injured skeletal-muscle cells.

86 uscle cells (96-well-plate format) and human skeletal muscle cells (24-well-plate format).

87 NE and play a role in nuclear positioning in skeletal muscle cells [8-12].

88 -glutamyltransferase (GT) activity in rat L6 skeletal muscle cells (96-well-plate format) and human s

89 ation enhanced insulin signaling in cultured skeletal muscle cells, adipocytes, and hepatocytes; this

90 Migrating muscle cells, but not all skeletal muscle cells, also expressed MMP-13.

91 m activated macrophages is critical for both skeletal muscle cell and hBD-MSCs death in PIRI-CLI.

92 ffect of mediating insulin on SREBP-1 in L-6 skeletal muscle cells and 3T3 L1 adipocytes, using wortm

93 d Akt and ERK1/2 phosphorylation in cultured skeletal muscle cells and C2C12 myotubes.

94 reduced SRF activity in differentiated C2C12 skeletal muscle cells and cardiac myocytes.

95 small heat shock protein (HspB8) in ischemic skeletal muscle cells and enhanced ischemic muscle autop

96 ia-induced apoptosis in both endothelial and skeletal muscle cells and enhanced proliferation in both

97 d Mlx associate with mitochondria in primary skeletal muscle cells and erythroblast K562 cells.

98 interstitial space, thus reducing binding to skeletal muscle cells and glucose uptake.

99 strate that simvastatin induces mitophagy in skeletal muscle cells and hypothesized that attenuating

100 response to electrical pulse stimulation of skeletal muscle cells and in exercized mice and healthy

101 e very low in undifferentiated human primary skeletal muscle cells and myoblasts (HSMM) but increased

102 in the fetal tissue and in undifferentiated skeletal muscle cells and myoblasts.

103 The overly active RyRs in cardiac and skeletal muscle cells and neuronal cells would result in

104 elial cells, HeLa cells, and human embryonic skeletal muscle cells and optic nerve head (ONH) astrocy

105 stricting the expression of foreign genes to skeletal muscle cells and presumably to other cells that

106 and the biological activity of Pip6a-PMO in skeletal muscle cells and primary cardiomyocytes.

107 IF1 stimulates glucose uptake via AMPK in skeletal muscle cells and primary cultured myoblasts.

108 These data implicate replication in skeletal muscle cells and release of IL-6 as important m

109 red the contractile properties of individual skeletal muscle cells and the activation and relaxation

110 se findings indicate that prion infection of skeletal muscle cells and the epithelial layer in the to

111 The role of skeletal muscle cells and their contribution to the immu

112 d is sufficient to convert a fibroblast to a skeletal muscle cell, and, as such, is a model system in

113 metabolism and its underlying mechanisms in skeletal muscle cells, and evaluated whether the observe

114 trite was abolished in SIRT3-deficient human skeletal muscle cells, and in SIRT3 knockout mice fed a

115 Ca(V)1 family Ca(2+) channels in cardiac and skeletal muscle cells, and reveal a unique ion channel r

116 ow fiber-specific gene promoters in cultured skeletal muscle cells, and the calcineurin inhibitor, cy

117 n at S307 in endothelial cells, hepatocytes, skeletal muscle cells, and vascular smooth muscle cells.

118 trate for the first time that normal primary skeletal muscle cells are capable of secreting IL-1beta

119 Results show that CFS skeletal muscle cells are unable to utilise glucose to t

120 f the C-terminal lobe of troponin C (TnC) in skeletal muscle cells as a step toward elucidating the m

121 INK4A) to promote malignant proliferation of skeletal muscle cells as an early step in ARMS tumorigen

122 muscle (VSM) cells, but also in cardiac and skeletal muscle cells as well as in kidney.

123 factor hypoxia-inducible factor-1 (HIF-1) in skeletal muscle cells, as well as invading myeloid cells

124 e developed a hybrid muscle powered by C2C12 skeletal muscle cells based on the functionalized multi-

125 e relevant cells, specifically primary human skeletal muscle cells because these cells can be convert

126 ulation, a previously-developed model of the skeletal muscle cell bioenergetic system was used to sim

127 slocation to the cell surface in cardiac and skeletal muscle cells by activating a PI3K dependent pat

128 s indicate that Pip6a-PMO is taken up in the skeletal muscle cells by an energy- and caveolae-mediate

129 nctional sarcoplasmic reticulum in heart and skeletal muscle cells by an undefined mechanism.

130 regulate many genes expressed in cardiac and skeletal muscle cells by binding to myocyte-specific chl

131 odel of insulin-stimulated glucose uptake in skeletal muscle cells by implicating p41ARC as a new com

132 e have shown 'robust' production of lung and skeletal muscle cells by marrow cells in the presence of

133 rin receptor-1 and ferritin-H in hepatic and skeletal muscle cells by promoting the binding of iron r

134 Infection of mammalian skeletal muscle cells by Trichinella spiralis causes hos

135 A co-culture of skeletal muscle cells (C2C12) and cholinergic neurons, g

136 tivated under hypoxic conditions in cultured skeletal muscle cells (C2C12).

137 6 inhibitor paxillin interacts with HDAC6 in skeletal muscle cells, colocalizes with AChR aggregates,

138 ure and mouse nerve-muscle ex-vivo) that the skeletal muscle cell constantly senses, through two iden

139 Skeletal muscle cells contain hundreds of myonuclei with

140 additional defect, namely impaired smooth-to-skeletal muscle cell conversion in the abdominal segment

141 ccur in islet beta cells, adipocytes, and/or skeletal muscle cells correlate with increased susceptib

142 We have developed a primary skeletal muscle cell culture model derived from normal p

143 Exposure of human skeletal muscle cell culture to type 1 interferons produ

144 ication of glutamate to embryonic vertebrate skeletal muscle cells cultured before innervation is nec

145 ndoplasmic reticulum stress and autophagy in skeletal muscle cell death and dysfunction in myositis.

146 e and secrete AGE-albumin, which induced the skeletal muscle cell death and injected hBD-MSCs in PIRI

147 Here, overexpression of XIAP in cultured skeletal muscle cells decreased protein degradation indu

148 pared to adult myoblast cultures, children's skeletal muscle cells demonstrated higher basal and day

149 appearance of Ca(2+) sparks in permeabilized skeletal muscle cells depends on the fibre's oxidative s

150 al during the myoblast fusion stage of early skeletal muscle cell development.

151 RK downstream kinase, is a novel mediator of skeletal muscle cell differentiation through its regulat

152 However, Panxs functions in skeletal muscle cell differentiation, and proliferation

153 to the inhibitory role of Notch signaling on skeletal muscle cell differentiation, the Notch pathway

154 Oncogenic Ha-Ras is a potent inhibitor of skeletal muscle cell differentiation, yet the Ras effect

155 t TAK1 is an important upstream regulator of skeletal muscle cell differentiation.

156 essential myosin heavy chain cluster during skeletal muscle cell differentiation.

157 ndocannabinoids and cannabinoid receptors in skeletal muscle cell differentiation.

158 At all stages of skeletal muscle cells differentiation, we show a permane

159 noblotting and kinase assays) in cultured L6 skeletal muscle cells during 30 min of cyclic stretch an

160 xpression in developing smooth, cardiac, and skeletal muscle cells during early embryogenesis.

161 and inhibited protein degradation in L6 rat skeletal muscle cells (EC(50) 4 muM) mediated in part by

162 Endothelial cells, macrophages, and skeletal muscle cells expressed TG throughout the healin

163 In summary, our data suggest that raised skeletal muscle cell expression of GRalpha and 11beta -H

164 ene regulatory network determines cardiac or skeletal muscle cell fates.

165 t the Notch pathway promotes vascular versus skeletal muscle cell fates.

166 nes encoding mitochondrial proteins in human skeletal muscle cells following treatment with 1alpha,25

167 domyosarcoma (aRMS), an aggressive cancer of skeletal muscle cells for which patient outcomes remain

168 derived conditionally immortalized precursor skeletal muscle cells from caveolin-3 transgenic and nul

169 t the utilisation of different substrates by skeletal muscle cells from CFS patients (n = 9) and heal

170 Skeletal muscle cells from DM patients fail to induce cy

171 tly been made toward the production of human skeletal muscle cells from induced pluripotent stem (iPS

172 imals, induced-pluripotent-stem-cell-derived skeletal muscle cells from patients with Becker MD and m

173 In summary, skeletal muscle cells from type 2 diabetic patients disp

174 ament arrays, is an essential contributor to skeletal muscle-cell fusion in developing mouse embryos.

175 In extracts of fat and skeletal muscle cells, Glut4 is predominantly found in s

176 These data indicate that skeletal muscle cell grafting gives rise to a subpopulat

177 Skeletal muscle cells grown on vertically aligned CNTs i

178 Given this novel role for PGF2alpha in skeletal muscle cell growth, these studies raise caution

179 It has previously been shown that CFS skeletal muscle cells have lower levels of ATP and have

180 anscriptional activities in undifferentiated skeletal muscle cells have not yet been determined.

181 Skeletal muscle cells have served as a paradigm for unde

182 Although previous studies in skeletal muscle cells have shown that HDAC4 lacking seri

183 pletion from mdx mice prevented compensatory skeletal muscle cell hypertrophy, decreased myofiber cen

184 rounded the role of calcineurin in mediating skeletal muscle cell hypertrophy.

185 terization of the underlying SOCE current in skeletal muscle cells (I(SkCRAC)) has not been reported.

186 Fast and accurate automatic segmentation of skeletal muscle cell image is crucial for the diagnosis

187 erm SKE, displayed diminished replication in skeletal muscle cells in a mouse model of CHIKV disease.

188 Furthermore, treatment of skeletal muscle cells in culture (C2C12 myotubes) with a

189 tochondrial network in cardiomyocytes and L6 skeletal muscle cells in culture.

190 gs between identified spinal motoneurons and skeletal muscle cells in larval zebrafish.

191 the mitochondrial morphology of mouse C2C12 skeletal muscle cells in response to heat acclimation an

192 rincipal structural component of caveolae in skeletal muscle cells in vivo.

193 entin expression was up-regulated on injured skeletal-muscle cells in vitro and was expressed in musc

194 e prion protein, PrP(Sc), accumulates within skeletal muscle cells, in addition to axons, in the tong

195 of stem cell myogenesis (transformation into skeletal muscle cells) includes several stages character

196 on of three alpha-dystroglycan glycoforms in skeletal muscle cells, including two minor glycoforms ma

197 Stimulation by IFN-gamma in skeletal muscle cells induces CIITA expression as well a

198 In primary human skeletal muscle cells, inhibition and overexpression str

199 etv2(ci32Gt); UAS:GFP cells differentiate as skeletal muscle cells instead of contributing to vascula

200 In fat and skeletal muscle cells, insulin-responsive amino peptidas

201 In fat and skeletal muscle cells, insulin-responsive vesicles, or I

202 The differentiation and maturation of skeletal muscle cells into functional fibers is coordina

203 nsulin resistance induced by high glucose in skeletal muscle cells is a consequence of Nox2 activatio

204 nic stem cells (hESCs) to differentiate into skeletal muscle cells is an important criterion in using

205 Contraction of skeletal muscle cells is initiated by a well-known signa

206 se during excitation-contraction coupling of skeletal muscle cells is initiated by the functional int

207 that oncogenic Ras-induced proliferation of skeletal muscle cells is mediated via a unique and novel

208 A hallmark of Nrg-1 signaling in skeletal muscle cells is the activation of extracellular

209 enabled us to demonstrate that mTOR, within skeletal muscle cells, is the rapamycin-sensitive elemen

210 s, namely myelomonocytic cells, osteoblasts, skeletal muscle cells, keratinocytes, and T lymphocytes.

211 Transient depletion of JARID2 in skeletal muscle cells leads to a transient up-regulation

212 Differentiation of skeletal muscle cells, like most other cell types, requi

213 Overexpression of ZnT7 in a rat skeletal muscle cell line (L6) increased Irs2 mRNA expre

214 In the L6E9 skeletal muscle cell line and in 10T1/2 fibroblasts, a p

215 ork of differentiating cultures of the mouse skeletal muscle cell line C2.

216 An L6 rat skeletal muscle cell line expressing ss-galactosidase (s

217 1B5s are a mouse skeletal muscle cell line that carries a null mutation f

218 toskeletal linkage protein dystrophin, and a skeletal muscle cell line, 129 CB3.

219 ature IGF-I were performed in C2C12 cells, a skeletal muscle cell line.

220 tinguish the differentiated state of a mouse skeletal muscle cell line.

221 regulation of gene expression in cardiac and skeletal muscle cell lineages.

222 ase reporter gene analysis using cardiac and skeletal muscle cell lines demonstrated a pattern of dis

223 We treated skeletal muscle cell lines with ryanodine, at concentrat

224 iomyopathy development observed in blood and skeletal muscle cells may have prognostic utility.

225 nderstanding the immunologic capabilities of skeletal muscle cells may provide important clues not on

226 4 (Glut4) to the plasma membrane in fat and skeletal muscle cells may represent a primary defect in

227 that is thought to stabilize the cardiac and skeletal muscle cell membranes during contraction.

228 mine whether strain-dependent differences in skeletal muscle cells might account for the differential

229 ertrophy and failure as well as in the C2C12 skeletal muscle cell model of differentiation; (3) the a

230 sults in loss of maternal Igf2 repression in skeletal muscle cells, most strikingly in the tongue, la

231 o-5N loaded into the SR of single, mammalian skeletal muscle cells (murine flexor digitorum brevis my

232 ts the hypothesis that activation of RyR3 in skeletal muscle cells must be indirect and provides the

233 of protein phosphatase-1 (PP-1(G)) in L6 rat skeletal muscle cell myogenesis.

234 In mouse C(2)C(12) cells, similarly to human skeletal muscle cells, myotube formation increased the e

235 te the existence of SOCE in freshly isolated skeletal muscle cells obtained from embryonic days 15 an

236 same panel of genetic changes, altering the skeletal muscle cell of origin led to different tumor mo

237 In addition, primary skeletal muscle cells of Cyp-D KO mice subjected to elec

238 Skeletal muscle cells of skalpha2(-/-) mice completely l

239 In skeletal muscle cells, oleic acid treatment increased in

240 2 distinct pools of IRE1alpha in cardiac and skeletal muscle cells, one localized at the perinuclear

241 In primary skeletal muscle cells, PGC-1beta induction of endogenous

242 4 (Glut4) to the plasma membrane of fat and skeletal muscle cells plays the key role in postprandial

243 coma by converting less differentiated human skeletal muscle cell precursors (SkMC) and committed hum

244 moted both fetal and postnatal primary human skeletal muscle cell precursors to bypass the senescence

245 o, activation of PPARdelta in adipocytes and skeletal muscle cells promotes fatty acid oxidation and

246 t that SIRT6 depletion in cardiac as well as skeletal muscle cells promotes myostatin (Mstn) expressi

247 icated that the addition of motor neurons to skeletal muscle cells reduced the secretion of GDNF by s

248 GLUT4 recruitment to the plasma membrane of skeletal muscle cells requires F-actin remodeling.

249 Taken together, our data show that skeletal muscle cells respond to defective myosin chaper

250 shown that lack of expression of triadins in skeletal muscle cells results in significant increase of

251 (CHO) and energy availability (EA) on potent skeletal muscle cell signalling pathways (regulating mit

252 mediator of adipose tissue inflammation and skeletal muscle cell (SkMC) insulin sensitivity and to q

253 unctional validation in human adipocytes and skeletal muscle cells (SKMCs) confirmed the relevance of

254 ls and in committed cell lineages, including skeletal muscle cells (SMC).

255 ls via incorporation of target sequences for skeletal muscle cell-specific miR-206.

256 However, differentiation of hESCs into skeletal muscle cells still remains a challenge, often r

257 ut not Bcl-2, is expressed in cultured human skeletal muscle cells stimulated with proinflammatory cy

258 to be expressed in neuronal, pancreatic, and skeletal muscle cells, suggesting a widespread role in r

259 Skeletal muscle cell survival and differentiation into m

260 tion-competent vesicular carriers in fat and skeletal muscle cells that deliver Glut4 to the plasma m

261 Myoblasts are precursor skeletal muscle cells that differentiate into fused, mul

262 mechanism triggered by mechanical stretch of skeletal muscle cells that leads to an EGR1-dependent tr

263 diffraction of frog (Rana temporaria) single skeletal muscle cells that, although the well-known thin

264 Here we show using differentiated skeletal muscle cells, that tumor necrosis factor (TNF)

265 f the expression of the genes is confined to skeletal muscle cells, the CD8(+) T-cell response is muc

266 (MURF) expressed specifically in cardiac and skeletal muscle cells throughout pre- and postnatal mous

267 kappaB activity, but the contribution of the skeletal muscle cell to this process has been unclear.

268 the PRC2 complex regulate the cell cycle in skeletal muscle cells to control proliferation and mitot

269 We conclude that commitment of skeletal muscle cells to differentiation is calcium and

270 tein that connects the actin cytoskeleton in skeletal muscle cells to extracellular matrix.

271 the cross talk between human adipocytes and skeletal muscle cells to identify mechanisms linking adi

272 ess the contribution of CHIKV replication in skeletal muscle cells to pathogenesis, we engineered a C

273 f a functional signaling pathway that allows skeletal muscle cells to sense and react to nutrient ava

274 recapitulated by simulating lipotoxicity in skeletal muscle cells treated with saturated FA, palmita

275 Our single-cell expression map of skeletal muscle cell types will further the understandin

276 ntify genome-wide binding of MyoD in several skeletal muscle cell types.

277 In vitro (macrophages, endothelial cells, skeletal muscle cells under normal and hypoxia serum sta

278 Skeletal muscle cells undergo in vitro maturation result

279 erstood, but may be related to the fact that skeletal muscle cells, unlike heart cells, are electrica

280 cells to generate both the motor neurons and skeletal muscle cells used.

281 investigated the gene expression patterns of skeletal muscle cells using RNA-seq of subtype-pooled si

282 cell culture-based model of damage to C2C12 skeletal muscle cells using the calcium ionophore, A2318

283 ults showed that DGAT1 was dominant in human skeletal muscle cells utilizing fatty acids (FAs) derive

284 erent in undifferentiated and differentiated skeletal muscle cells (vesicular versus nuclear).

285 ide (NO) induces mitochondrial biogenesis in skeletal muscle cells via upregulation of the peroxisome

286 In skeletal muscle cells, voltage-dependent potentiation of

287 ormin (Met) action on glucose uptake (GU) in skeletal muscle cells was investigated.

288 ion demonstrated activation of LacZ when the skeletal muscle cells were implanted into hearts of -MHC

289 -derived neural stem cells in coculture with skeletal muscle cells were induced to become neurons exp

290 CFS skeletal muscle cells were shown to oxidise galactose an

291 in C57BL/6 and BALB/c mice, endothelial and skeletal muscle cells were subjected to hypoxia and nutr

292 AAV6, which demonstrate robust infection in skeletal muscle cells, were less effective in crossing t

293 ll studies demonstrate on-target activity in skeletal muscle cells, whereas their mouse results sugge

294 This is especially true in skeletal muscle cells, which contain hundreds of myonucl

295 steroid drugs is comparable in rat and human skeletal muscle cells, which emphasizes the potential of

296 In contrast to brown preadipocytes or skeletal muscle cells, white preadipocytes express Tcf21

297 Acute treatment (30 min) of cultured human skeletal muscle cells with either INH resulted in a dose

298 Transfecting skeletal muscle cells with shRNAs specific for PUM2 up-r

299 During differentiation, skeletal muscle cells withdraw from the cell cycle and f

300 a parallel loss of BRCA1 function in patient skeletal muscle cells would potentially result in implic