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1 tors (GPCRs) play a vital role in regulating skin aging.
2 lar lentigines (SLs) are a hallmark of human skin aging.
3 enes that contribute to different aspects of skin aging.
4 at pigmentation is a critical determinant of skin aging.
5 rinkling is one of the most notable signs of skin aging.
6 nt studies expand our understanding of human skin aging.
7 enes to study the molecular basis of healthy skin aging.
8 beta), which is a known gene associated with skin aging.
9 n performed in women with varying degrees of skin aging.
10 gen production stimulators, were examined in skin aging.
11  this self perpetuating cycle promotes human skin aging.
12 ar target for antioxidant protection against skin aging.
13  attenuate solar UVB light-induced premature skin aging.
14 s such as dermal elastin and collagen during skin aging.
15 ous effects and may thereby retard premature skin aging.
16 y a key role in the pathophysiology of human skin aging.
17 creen group showed no detectable increase in skin aging after 4.5 years.
18 ene supplementation had no overall effect on skin aging, although contrasting associations were seen
19  measured by antioxidant capacity may retard skin aging among healthy men and women aged >45 years.
20 understanding and studying the mechanisms of skin aging and age-related skin disorders.
21 ajor external insults to cells and can cause skin aging and cancer.
22 UVR) from sunlight is the major effector for skin aging and carcinogenesis.
23 Chronic exposure to sunlight plays a role in skin aging and carcinogenesis.
24  work has uncovered genetic contributions to skin aging and confirmed previous findings, showing that
25 tal processes, such as those associated with skin aging and disease, and the development and delivery
26 quent loss of skin hydration is a feature of skin aging and eczematous dermatitis.
27                            We also show that skin aging and reported experience of sunburns was propo
28  characterized by skin blistering, premature skin aging and skin cancer of unknown etiology.
29 tion damages human skin and causes premature skin aging and skin cancer.
30 sms of skin aging, the role of stem cells in skin aging and the implications of skin aging for the mi
31 nctions of senescent cells in the context of skin aging and wound healing.
32 whether DNA methylation is altered following skin aging and/or chronic sun exposure in humans.
33 d intrinsic skin aging (collectively termed "skin aging"), and the impact of broadband light (BBL) tr
34 y SCINEXA (SCore for INtrinsic and EXtrinsic skin Aging) and airflow obstruction by spirometry, using
35 olet light can cause inflammation, premature skin aging, and cancer.
36  between 25-hydroxyvitamin D and features of skin aging are causal.
37 on numbers of nevi and atypical nevi, and on skin aging as measured by microtopography.
38 therapies against pigmentary skin disorders, skin aging, as well as skin cancers.
39 ate the expression of proteins implicated in skin aging, but also affect cells at a genetic level, br
40  the use of fossil fuels was associated with skin aging, but no direct link between indoor air pollut
41                It is considered a feature of skin aging, but risk factors other than aging are largel
42 antioxidants are advocated as preventives of skin aging, but supporting evidence is lacking.
43 ith alphaVbeta3 integrin contribute to human skin aging by stimulating MMP-1-mediated collagen fibril
44               Photoaging, which is premature skin aging caused by long-term UV exposure, is of aesthe
45 rom foods may mitigate the main component of skin aging caused by sun exposure.
46 sociated with human photoaging and intrinsic skin aging (collectively termed "skin aging"), and the i
47                     Similar to that in human skin aging, expression levels of dermal collagens are de
48  detrimental effects such as cancerogenesis, skin aging, eye damage, and autoimmune disorders.
49  D concentration was not associated with any skin aging feature (P-values > 0.05).
50          Facial pigmented spots are a common skin aging feature, but genetic predisposition has yet t
51  50.5% women, median age 63.1 years), facial skin aging features (perceived age, wrinkling, pigmented
52 Associations between 25-hydroxyvitamin D and skin aging features were tested by multivariable linear
53  cells in skin aging and the implications of skin aging for the microbiome and for the development of
54 n the dermal matrix may serve as markers for skin aging, for photoaging, and for immediate assessment
55                                              Skin aging from baseline to the end of the trial was 24%
56 roxyvitamin D concentrations and features of skin aging in a bidirectional Mendelian randomization st
57                Regular sunscreen use retards skin aging in healthy, middle-aged men and women.
58       We show that similar to aging in human skin, aging in NMRs is accompanied by a decrease in epid
59 echanisms underlying intrinsic and extrinsic skin aging, including reactive oxygen species formation,
60 rs provide a review of current insights into skin aging, including the mechanisms of skin aging, the
61                                              Skin aging is a complex biological process affected by a
62                                              Skin aging is a complex process involving the additive e
63  considered both physiologic and inevitable, skin aging is a degenerative phenomenon whereby both int
64                              The hallmark of skin aging is fragmentation of collagen fibrils.
65              The long-term effect of diet on skin aging is largely unknown, but evidence suggests tha
66                                              Skin aging is linked to reduced epidermal proliferation
67                                              Skin aging is not associated with systematic changes in
68                                       In the skin, aging is associated with overall epidermal thinnin
69                                              Skin aging leads to decreased elasticity and the loss of
70 natural aging, we determined whether natural skin aging, like photoaging, gives rise to increased mat
71 the indoor PM2.5 exposure is associated with skin aging manifestation in a Chinese population.
72 osure levels were positively associated with skin aging manifestation, including score of pigment spo
73 e association between the PM2.5 exposure and skin aging manifestations by linear regression.
74 irect link between indoor air pollutants and skin aging manifestations has ever been shown.
75 air pollution is known to be associated with skin aging manifestations.
76 ents, there is neither increased photodamage/skin aging nor a higher incidence for sun-induced nonmel
77 O(2)-induced oxidative stresses that lead to skin aging or skin cancer has not been fully studied.
78 associated with facial SL but not with other skin aging or skin pigmentation traits.
79           These findings suggest that during skin aging, PANX3 is critical in the maintenance of dors
80 t irradiation from sunlight causes premature skin aging (photoaging), characterized in part by wrinkl
81 ar matrix (ECM), thereby promoting premature skin aging (photoaging).
82 diation, is a major determinant of premature skin aging (photoaging).
83  feature of the pathophysiology of premature skin aging (photoaging).
84 ctive tissue and may contribute to premature skin aging (photoaging).
85 ociated with age may offer insights into the skin aging process.
86 cepted as an effective approach to delay the skin aging process.
87 that may lead to new insights into the human skin aging process.
88                                              Skin aging processes are modeled by treating keratinocyt
89                               In conclusion, skin aging processes may involve ROS-induced protein dys
90 idated, and relevant models for the study of skin aging progression are lacking.
91 e model to study the key factors involved in skin-aging progression, we demonstrate that transgenic m
92 of the cellular and molecular intricacies of skin aging provide a foundation for future approaches de
93 e a single-cell molecular framework of human skin aging, providing a rich resource for developing the
94 rapeutic target for skin disorders including skin aging, psoriasis, and atopic dermatitis.
95                                              Skin aging-reducing strategies should incorporate the se
96 ulates rather than enhance the expression of skin aging-related biomarkers ex vivo.
97  role of telomerase reverse transcriptase in skin aging-related lentigines formation.
98                  The pathobiologic basis for skin aging remains poorly understood.
99                          Understanding human skin aging requires in-depth knowledge of the molecular
100 nd controls were identified by global facial skin aging severity including intrinsic and extrinsic pa
101    Conquest of the oft overlooked disease of skin aging should have broad implications that transcend
102  clock; and 80% of them were associated with skin aging showing similarities between glucocorticoid-t
103 egulated epidermal homeostasis and premature skin aging, such as photoaging.
104 ssed in the skin and reviewed their roles in skin aging, such as pigmentation, loss of elasticity, wr
105       Oxidative stress is the major cause of skin aging that includes wrinkles, pigmentation, and wea
106        UVR is a major etiology for premature skin aging that leads to photoaging and UV-induced skin
107 into skin aging, including the mechanisms of skin aging, the role of stem cells in skin aging and the
108 es reveal that in contrast to human or mouse skin aging, the transcript levels of several longevity-a
109 dermal pathology in radiation dermatitis and skin aging through IKK modulation and motivate the explo
110 gen peroxide (H2O2) elicits ROS that induces skin aging through oxidation of proteins, forming disulf
111 clinical inflammation are concomitant during skin aging, thus raising the question of a causal link.
112 t rs185146 near SLC45A2 is associated with a skin aging trait at genome-wide significance (P = 4.1 x
113 ta suggest that specific features in the NMR skin aging transcriptome might contribute to the resista
114                                 We find that skin aging was associated with a significantly altered e
115        No overall effect of beta-carotene on skin aging was identified, and further study is required
116                                              Skin aging was not associated with residence or holidays
117  To investigate the role of elevated MMP1 in skin aging, we generated a conditional bitransgenic mous
118 s suggest that vitamin D reduces the rate of skin aging, whereas population studies suggest the oppos
119 fect skin homeostasis, skin differentiation, skin aging, wound healing, and, potentially, skin cancer

 
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