ライフサイエンスコーパス: skin cancer

1 carcinoma (MCC), an extremely lethal form of skin cancer.

2 be lagging strand specific in patients with skin cancer.

3 e strategies that promote early detection of skin cancer.

4 nt recipients are at high risk of developing skin cancer.

5 skin aging, wound healing, and, potentially, skin cancer.

6 role for HPVs in driving the development of skin cancer.

7 predict risk of, and time to posttransplant skin cancer.

8 emains a challenging and often fatal form of skin cancer.

9 nd genetic mutation can subsequently lead to skin cancer.

10 ies, melanoma is still the deadliest form of skin cancer.

11 hnique for non-Hodgkin lymphoma and melanoma skin cancer.

12 biquitous carcinogen in sunlight that causes skin cancer.

13 melanoma (MM) is the most aggressive form of skin cancer.

14 l proportion can progress into squamous cell skin cancer.

15 Melanoma is a lethal form of skin cancer.

16 ad to strategies for preventing and treating skin cancer.

17 lts aged 18 to 60 years without a history of skin cancer.

18 hat tomato consumption would protect against skin cancer.

19 NER protects against skin cancer.

20 Cutaneous melanoma (CM) is the most lethal skin cancer.

21 presents the identification of the deadliest skin cancer.

22 are highly expressed in human patients with skin cancer.

23 are considered potential early precursors of skin cancer.

24 ase in incidence of melanoma and nonmelanoma skin cancer.

25 g adulthood, increase the risk of developing skin cancer.

26 metastatic melanoma, the most lethal form of skin cancer.

27 ntial therapeutic targets in this metastatic skin cancer.

28 and sunburn and thus prevent future cases of skin cancer.

29 rmatology clinic underwent biopsy to exclude skin cancer.

30 the sun's UV radiation is a leading cause of skin cancer.

31 ard, kappa, 0.41-0.63), for the diagnosis of skin cancer.

32 to most effectively predict the incidence of skin cancer.

33 for solar ultraviolet (UV) radiation-induced skin cancer.

34 Melanoma is the most aggressive type of skin cancer.

35 rcinoma (cSCC) is the most common metastatic skin cancer.

36 physical appearance and risk of keratinocyte skin cancer.

37 rare and aggressive, yet highly immunogenic skin cancer.

38 lation of mutations and an increased risk of skin cancer.

39 , p=0.0042), owing primarily to non-melanoma skin cancer.

40 checkpoint inhibitor therapy for metastatic skin cancer.

41 to measure cumulative sun damage and risk of skin cancer.

42 time noninvasive preoperative delineation of skin cancer.

43 rcinoma (MCC), a rare and aggressive form of skin cancer.

44 s, with major complications of infection and skin cancer.

45 In addition, 26.4% of patients developed skin cancer.

46 e discovery of additional treatments against skin cancer.

47 iseases, including arthritis, cataracts, and skin cancer.

48 t development of actinic keratoses (AKs) and skin cancers.

49 siologic events including the development of skin cancers.

50 Overall, 119 patients had 221 skin cancers.

51 bedrock and tool of choice for the study of skin cancers.

52 rs, including melanoma and other nonmelanoma skin cancers.

53 gher risk of all-site, urothelial, lung, and skin cancers.

54 5B levels were unchanged in betaHPV positive skin cancers.

55 s for prevention or treatment of UVR-induced skin cancers.

56 hair development, hair growth disorders, and skin cancers.

57 233 benign meningiomas, and 1856 nonmelanoma skin cancers.

58 improving the chemotherapeutic efficiency of skin cancers.

59 t malignancies, meningiomas, and nonmelanoma skin cancers.

60 nt lesions will permit us to prevent or cure skin cancers.

61 y of the biodegradable nanogels for treating skin cancers.

62 nocyte carcinomas, also known as nonmelanoma skin cancers.

63 gy in clinical practice for the diagnosis of skin cancer?

64 1.4%] vs 8 [36.4%]) and knowing the signs of skin cancer (11 [25.0%] vs 10 [45.4%]).

65 ] vs 19 [6.1%] answered yes), and history of skin cancer (76 [33.3%] vs 15 [4.8%] answered yes) (all

66 elated phenotypes, and increase the risk for skin cancer-a phenomenon defined as photoaging.

67 The high risk of skin cancer after organ transplantation is a major clini

68 cell polyomavirus (MCV) causes an aggressive skin cancer after prolonged infection and requires an ac

69 The course of skin cancer after retransplantation in organ-transplant

70 f specific treatments to prevent nonmelanoma skin cancers among solid organ transplant recipients.

71 ary, and rectal cancer and three nonmelanoma skin cancers) among 825 patients who received ustekinuma

72 nts in the genesis of many cancers including skin cancer and are often implicated in tumor progressio

73 carcinoma (cuSCC) is the second most common skin cancer and commonly arises in chronically UV-expose

74 enhance neoplastic progression in models of skin cancer and cutaneous T-cell lymphoma (CTCL).

75 calculated bidirectionally for any SPC after skin cancer and for skin cancer as SPC.

76 an ultraviolet radiation (UV)-induced human skin cancer and from a mouse model of urethane-induced c

77 th small to moderate elevations in risks for skin cancer and gastrointestinal, infectious, pulmonary,

78 iation is fundamental to pathologies such as skin cancer and mucosal inflammatory diseases.

79 ability of solar ultraviolet (UV) to induce skin cancer and photoaging is well recognized.

80 une diseases, and detrimental in the case of skin cancer and the response to several infectious agent

81 is the most common cancer after nonmelanoma skin cancer and the second leading cause of cancer death

82 rmatologist after 33 (53.2%) for presumptive skin cancers and 15 (24.2%) for precancers.

83 .2%; 4 of 27 patients [14.8%] diagnosed with skin cancers and 5 of 11 patients [45.5%] diagnosed with

84 ure is a major environmental risk factor for skin cancers and is also an important source of vitamin

85 idirectional risks between immune responsive skin cancers and most other cancers suggest that immune

86 for age, sex, educational level, history of skin cancer, and history of AK.

87 ried MC1R variants known to increase risk of skin cancer, and there was diversity in the observed var

88 e tumors are similar to those found in human skin cancers, and PMA promotes proliferation of human sk

89 Because rates of skin cancer are greater among adult survivors of childho

90 n estimated 5.4 million cases of nonmelanoma skin cancer are reported in the United States at an asso

91 ther skin phenotypes such as acne, color and skin cancers are also being investigated with GWAS.

92 Mucosal and skin cancers are associated with infections by human pap

93 onally for any SPC after skin cancer and for skin cancer as SPC.

94 relative risks were for invasive and in situ skin cancer as SPCs (14.59 and 16.71, respectively).

95 To assess incidence and risk factors for skin cancer associated with allogeneic hematopoietic ste

96 Merkel cell carcinoma is a rare skin cancer associated with Merkel cell polyomavirus in

97 Cases were identified among subjects free of skin cancer at baseline but who later reported a physici

98 evelopment of prostate cancer, colon cancer, skin cancer, breast cancer, lung cancer and pancreas can

99 d lung; malignant skin melanoma; nonmelanoma skin cancer; breast; cervical; uterine; ovarian; prostat

100 Surgeon General's Call to Action to Prevent Skin Cancer broadly identified research gaps, but specif

101 ncer prevention and, therefore, decrease the skin cancer burden.

102 ows the opposite asymmetry in NER-proficient skin cancers, but not in NER-deficient cancers, indicati

103 proaches that could block the development of skin cancer by boosting immunity against the commensal H

104 on may increase the risk of solar UV-induced skin cancer by promoting photochemical damage to the NER

105 of the (6-4) photoadduct in the induction of skin cancer by sunlight, crucial mechanistic details abo

106 From 1993 to 2008, a total of 2003 skin cancer cases were ascertained among 67,332 women, i

107 rt that NFAT3 is highly expressed in various skin cancer cell lines and tumor tissues.

108 chitosan for ionic interaction with anionic skin cancer cell membrane.

109 We employed this device to detect melanoma skin cancer cells through specific immunogenic binding o

110 d colony formation and cell proliferation of skin cancer cells.

111 roven melanomas in 134 patients treated in 9 skin cancer centers in Spain, France, Italy, and Austria

112 Prespecified outcomes were nonmelanoma skin cancer, clearance and prevention of keratotic skin

113 l cell carcinoma (MCC) is a rare, aggressive skin cancer commonly driven by the Merkel cell polyomavi

114 mTOR inhibitors probably reduced skin cancer compared to calcineurin inhibitors (12 trial

115 ylated NFAT3-Ser259 were highly expressed in skin cancer compared with normal skin tissues.

116 eduction in viral activity and load in human skin cancer compared with the adjacent healthy skin, sug

117 Keratinocyte skin cancer, comprising cutaneous squamous (cSCC) and ba

118 e of sunscreen and clothing, annual cases of skin cancer continue to rise.

119 vioral research addressing all points of the skin cancer control continuum, measuring interventions t

120 eceiving treatment of a condition other than skin cancer (controls) at the dermatology clinics at the

121 ystem, we found that brain, lung, colon, and skin cancers could be detected in situ during surgery wi

122 Calcipotriol suppressed skin cancer development in mice in a TSLP-dependent mann

123 ially represent founding driver mutations in skin cancer development.

124 ons: (1) How accurate is teledermatology for skin cancer diagnosis compared with usual care (face-to-

125 ements in the accuracy of image-based AI for skin cancer diagnosis to address the effects of varied r

126 tive and specific for non-invasively guiding skin cancer diagnosis.

127 The influence of the screening program on skin cancer epidemiological findings and the cost per qu

128 ess their budget effect and the influence on skin cancer epidemiological findings.

129 effective imaging techniques in diagnosis of skin cancer, especially for pigmented lesions.

130 und no associations between vitamin D intake skin cancers, except positive associations with BCC risk

131 pid, and global assessment of margins during skin cancer excision, allowing same-day reexcision when

132 who at baseline reported no past history of skin cancer excisions and no more than five destructivel

133 ed information on skin, hair, and eye color; skin cancer family history; and sun exposure history, su

134 carcinoma (MCC) is a rare, highly aggressive skin cancer for which immune modulation by immune checkp

135 and 1.71 (95% CI, 0.88-3.33) for nonmelanoma skin cancers for survivors with reference characteristic

136 To address the role of TLS in skin cancer formation, we determined which DNA polymeras

137 n of positive section margins in nonmelanoma skin cancer from 8.4% to 12.8%.

138 erapeutic target for the prevention of human skin cancer given that it is a major negative regulator

139 Point-of-care fluorescent detection of skin cancer had a clinically relevant sensitivity of 0.7

140 Melanocytes located adjacent to a skin cancer had higher mutation burdens than melanocytes

141 Recipients with pretransplant skin cancer had increased risk of PTM (sub-HR [SHR], 2.6

142 The risk for skin cancer has been well characterized in white organ t

143 ts who have already developed posttransplant skin cancer has not been assessed.

144 ted 647 patients with carcinoma, nonmelanoma skin cancer, hematological second cancer, and melanoma d

145 in patients with and without a pretransplant skin cancer history was 31.6% and 7.4%, respectively (P

146 ractices that further increase their risk of skin cancer; however, gaps in the literature exist in yo

147 With the exception of increased risk for skin cancer (HR, 2.05 [CI, 1.28 to 3.28]), the treatment

148 ransplant skin cancer included pretransplant skin cancer (HR, 4.69; 95% CI, 3.26-6.73), male sex (HR,

149 to the MD was associated with lower risk of skin cancer (HR: 0.83; 95% CI: 0.73, 0.93 for high compa

150 as a foundation enabling faster diagnosis of skin cancer, identification of cases for specialist revi

151 ning sign of progression toward non-melanoma skin cancer, if ignored.

152 burdens than melanocytes from donors without skin cancer, implying that the mutation burden of normal

153 erefore, discoid lupus inflammation promotes skin cancer in high-risk DLE patients, and blocking the

154 gainst commensal papillomaviruses suppresses skin cancer in immunocompetent hosts, and the loss of th

155 f HPVs-causes the markedly increased risk of skin cancer in immunosuppressed patients.

156 anding and controlling the high incidence of skin cancer in OTRs.

157 orbol 12-myristate 13-acetate (PMA) promotes skin cancer in rodents.

158 s research has reported an increased risk of skin cancer in solid organ transplant recipients (OTRs),

159 arms of interventions to prevent nonmelanoma skin cancer in solid organ transplant recipients have no

160 There was an increased risk of skin cancer in white vs nonwhite OTRs (HR 4.4, 95% CI 3.

161 ty, and examined the incidence of UV-induced skin cancers in Poltheta(-/-), Poleta(-/-), and Poltheta

162 Nonmelanoma skin cancers, in particular cutaneous squamous cell carc

163 significant risk factors for posttransplant skin cancer included pretransplant skin cancer (HR, 4.69

164 between adherence to the MD and the risk of skin cancer, including melanomas, basal cell carcinomas

165 evaluate the risk factors for posttransplant skin cancer, including squamous cell carcinoma (SCC), me

166 olonged exposure, but the incidence of other skin cancers increases.

167 , and KMT2C, which are frequently mutated in skin cancers, indicating their potential role as foundin

168 al role in UV-induced immune suppression and skin cancer induction.

169 or are also sufficient to delay prostate and skin cancer initiation of Pten-deficient mice.

170 SPCs associated with known immune responsive skin cancers, invasive and in situ squamous cell carcino

171 High-risk skin cancer is a rare, but severe, complication associat

172 Posttransplant skin cancer is common, with elevated risk imparted by in

173 he risk factors and trends in posttransplant skin cancer is fundamental to targeted screening and pre

174 d of care diagnostic procedure for suspected skin cancer is microscopic examination of hematoxylin &

175 Skin cancer is the most common malignancy occurring afte

176 Skin cancer is the most frequent kind of cancer in white

177 Skin cancer is the most frequently diagnosed cancer in t

178 ions in South Asia, but its association with skin cancers is as yet unknown.

179 ignant melanoma, the most aggressive form of skin cancer, is characterized by high prevalence of BRAF

180 of sun/UV exposure-related illness, such as skin cancer, is seriously concerning public health autho

181 hough melanoma is the least frequent type of skin cancer, it accounts for the majority of skin cancer

182 Nonmelanoma skin cancer, Kaposi sarcoma, and posttransplant lymphopr

183 virus associated with an aggressive form of skin cancer, Merkel cell carcinoma (MCC).

184 To tackle this problem, we designed a skin cancer model for squamous cell carcinoma (SCC) that

185 This papillomavirus-induced skin cancer model opens future investigations into viral

186 Herein, we established and characterized a skin cancer model, in which Mus musculus papillomavirus

187 , adults, and parents, with an aim to reduce skin cancer morbidity and mortality.

188 and is predicted to result in a reduction of skin cancer mortality over 20 years and 50 years.

189 inoma (SCC) in QSkin, a prospective study of skin cancer (N = 43,794).

190 s across the United States in the Transplant Skin Cancer Network during 1 of 2 calendar years (either

191 ffects of UV-B radiation against nonmelanoma skin cancer (NMSC) are exerted via signaling mechanisms

192 ients have an increased risk of non-melanoma skin cancer (NMSC) compared to in the general population

193 Nonmelanoma skin cancer (NMSC) such as cutaneous squamous cell carci

194 k than HIV-uninfected persons of nonmelanoma skin cancer (NMSC), defined as basal cell carcinoma (BCC

195 billing codes and categorized as nonmelanoma skin cancer (NMSC), viral-linked and "other" cancers.

196 V infection and a high risk for non-melanoma skin cancer (NMSC).

197 de novo malignancies (excluding non-melanoma skin cancers [NMSCs]), post-transplantation lymphoprolif

198 ss all three trials, adjudicated nonmelanoma skin cancer occurred in five patients who received tofac

199 Merkel cell carcinoma (MCC) is an aggressive skin cancer often caused by the Merkel cell polyomavirus

200 referrals overall and those for presumptive skin cancer or actinic keratoses, skin biopsies, or PCP

201 in biopsies, and PCP diagnostic accuracy for skin cancer or precancer compared with dermatologist dia

202 ignant melanoma is one of the most dangerous skin cancer originating from melanocytes.

203 tions, herpes zoster infection, non-melanoma skin cancer, other malignancies, major cardiovascular ev

204 Secondary endpoints included the course of skin cancers over 3 periods (first transplantation, retu

205 The incidence rates for posttransplant skin cancer overall and for SCC, MM, and MCC were calcul

206 To study temporal trends for the risk of skin cancer, particularly SCC, after organ transplantati

207 ost disease as a risk factor for nonmelanoma skin cancer, particularly squamous cell carcinoma.

208 ts into UVR-induced immune tolerance against skin cancers, particularly cutaneous melanoma, have been

209 iations with white race and history of prior skin cancer point to an important role for ultraviolet r

210 r tanning frequency and behaviors related to skin cancer prevention and to investigate whether these

211 importance of sun protection and facilitate skin cancer prevention and, therefore, decrease the skin

212 Therefore, beta HPV represents a target for skin cancer prevention, especially in high-risk populati

213 findings may have important implications in skin cancer prevention.

214 Sun-protective behavior affects skin cancer prevention.

215 ed XP-V cells is highly similar to the human skin cancer profile, revealing how studies involving cel

216 against loss of bone mass, chronic diseases, skin cancer, prostate cancer, and cardiovascular disease

217 ted with an increased risk of posttransplant skin cancer, PTLD, solid organ cancer, death and graft f

218 tudied the association between pretransplant skin cancer, PTM, death, and graft failure.

219 en compared with the number of biopsy-proven skin cancers recorded over a similar period before the f

220 Grant success rate in skin cancer-related behavioral science compares favorabl

221 l Institute of Health (NIH) grants targeting skin cancer-related behaviors and relevant outcomes.

222 ant applications from 2000 to 2014 targeting skin cancer-related behaviors or testing behavioral inte

223 skin cancer, it accounts for the majority of skin cancer-related deaths.

224 Melanoma, the deadliest skin cancer, remains largely incurable at advanced stage

225 morbidities and mortalities associated with skin cancers requires sustained research with the goal o

226 Our findings that the incidence of skin cancers rises in Poltheta(-/-) mice and is further

227 D (25(OH)D) level and/or vitamin D intake on skin cancer risk are conflicting.

228 and 95% CIs adjusted for age and main known skin cancer risk factors.

229 as a dosimeter of exposure and predictor of skin cancer risk has been proposed by multiple groups.

230 ate that SP may increase UVB mutagenesis and skin cancer risk in certain individuals.

231 Little is known about cutaneous disease and skin cancer risk in this OTR population.

232 herence to the MD is associated with a lower skin cancer risk in women, particularly melanoma and BCC

233 Skin cancer risk information based on melanocortin-1 rec

234 ts carrying MC1R variants imparting elevated skin cancer risk was consistent across quartiles of Euro

235 at vitamin D may have a protective effect on skin cancer risk, epidemiologic studies investigating th

236 nd repeated shows a causal relationship with skin cancer risk.

237 re proposed explanations for this heightened skin cancer risk; however, the exact mechanism driving s

238 mplementation of a campaign promoting annual skin cancer screening by FBSE, including training of PCP

239 ined from 370 individuals undergoing routine skin cancer screening examinations.

240 ve assessment of survivors' adherence to the skin cancer screening guidelines associated with skin se

241 is critical to emphasize sun protection and skin cancer screening in individuals who tan indoors.

242 The cost-effectiveness for skin cancer screening is higher in women than in men.

243 Skin cancer screening may improve melanoma outcomes and

244 the cost-effectiveness of 2 population-based skin cancer screening methods and to assess their budget

245 ation, cancer treatment characteristics, and skin cancer screening practice.

246 are payer perspective) of 2 population-based skin cancer screening programs in Belgium compared with

247 To describe a skin cancer screening quality initiative in a large heal

248 ive Services Task Force for population-based skin cancer screening.

249 to sunburn, avoid sun protection, and avoid skin cancer screening.

250 .g., concealed weapon detection in airports, skin cancer screenings) and communication technologies.

251 L-33 were protected from chronic ACD and its skin cancer sequela compared with wild-type controls (P

252 2.60; 95% CI, 2.27-2.98), and posttransplant skin cancer (SHR, 2.92; 95% CI, 2.52-3.39), PTLD (SHR, 1

253 the highest and moderate risk of developing skin cancer (skin types I, II, III, and IV) than in skin

254 osttransplant malignancy was classified into skin cancer, solid tumor, and posttransplant lymphoproli

255 nscriptional repressor, is down-modulated in skin cancer stromal cells, and Atf3 knockout mice develo

256 a (MCC), a rare but aggressive form of human skin cancer, strongly suggesting that this virus is tumo

257 Nonmelanoma skin cancer such as cutaneous squamous cell carcinoma (c

258 6 outcomes (mean difference of 0.5 or more): skin cancer, surgical complications, cognition, blood pr

259 ociated only with increased adherence to COG skin cancer surveillance (36.9% v 23.2%; PR, 1.24; CI, 1

260 ls, and a higher incidence of non-basal-cell skin cancers than placebo.

261 Merkel cell carcinoma (MCC) is a lethal skin cancer that metastasizes rapidly.

262 Skin cancer, the most common human malignancy, is primar

263 Although highly expressed in normal skin and skin cancer, the role of the atypical E2Fs, E2F7 and E2F

264 rved for 26 cancers associated with invasive skin cancer; the Spearman rank correlation was 0.72 (P =

265 tial as a novel target for the prevention of skin cancer through its role in the regulation of STAT3

266 UVR promotes skin cancer through multiple mechanisms, including induc

267 f video-mosaics of melanoma and non-melanoma skin cancers, to demonstrate potential clinical utility.

268 but little is known about the feasibility of skin cancer training and clinical skin examination (CSE)

269 This pilot study suggests that PCP-based skin cancer training and screening are feasible and have

270 To assess the association of skin cancer training and screening by PCPs with dermatol

271 CC risk, albeit with no heterogeneity across skin cancer type.

272 0.68), although with no heterogeneity across skin cancer types (Pheterogeneity = 0.23).

273 ed based on age, sex, history of nonmelanoma skin cancer, US geographic region, and population densit

274 The incidence rates for posttransplant skin cancer was 1437 per 100000 person-years.

275 ated protective response against UVB-induced skin cancer was accompanied by enhanced DNA damage repai

276 Risk of skin cancer was analyzed using standardized incidence ra

277 Pretransplant skin cancer was associated with an increased risk of pos

278 Incident skin cancer was determined through detailed medical reco

279 Skin cancer was diagnosed in 64 (41.6%) white OTRs and 1

280 No secondary skin cancer was observed.

281 t commonly affected gene in human UV-induced skin cancer, was applied as an electrochemical DNA senso

282 fects of papillomavirus on carcinogen-driven skin cancer, we colonized several strains of immunocompe

283 nalysis, age, sex, and previous diagnosis of skin cancer were not significantly associated with the p

284 Potential risk factors for posttransplant skin cancer were tested using multivariate Cox regressio

285 Effects on nonmelanoma skin cancer were uncertain for photodynamic therapy (3 t

286 Biopsy-proven skin cancers were recorded for 16 months (for patient 1)

287 years from diagnosis, excluding nonmelanoma skin cancers, were evaluated in survivors diagnosed when

288 ce of all-type cancer (excluding nonmelanoma skin cancers), which was evaluated using Kaplan-Meier su

289 Melanoma is the most aggressive type of skin cancer, which readily metastasizes through lymph no

290 or head and neck, genitalia, hands, and feet skin cancers, which may represent an additional financia

291 association between eczema and non-melanoma skin cancer, while the remaining study failed to identif

292 imers (CPDs) are DNA photoproducts linked to skin cancer, whose mutagenicity depends in part on their

293 tificial intelligence capable of classifying skin cancer with a level of competence comparable to der

294 sosquamous carcinoma (BSC) is a rare form of skin cancer with both basaloid and squamous morphology.

295 Melanoma is the deadliest type of skin cancer with one of the fastest increasing incidence

296 Malignant melanoma is an aggressive skin cancer with poor survival outcomes for patients dia

297 (MCC) is a highly aggressive neuroendocrine skin cancer with steadily increasing incidence and poor

298 inct co-localization of multiple independent skin cancers with areas of active inflammation in two DL

299 (cSCC) is one of the most common metastatic skin cancers with increasing incidence.

300 e counseled more effectively on the signs of skin cancer, with focused discussion points contingent o