ライフサイエンスコーパス: skin carcinoma

1 n form of skin damage and is associated with skin carcinoma.

2 er, including highly prevalent UV-associated skin carcinomas.

3 various agents such as retinoic acid, and in skin carcinomas.

4 recipients, 2 of whom also had CuV-positive skin carcinomas.

5 oss of Pten are mutually exclusive events in skin carcinomas.

6 ic mice induces the formation of spontaneous skin carcinomas.

7 omas or p53 deficiency to induce spontaneous skin carcinomas.

8 mice, generated from 129P2 ES cells develop skin carcinomas.

9 factor for the increasing incidence of human skin carcinomas.

10 hereas Pold1 mutant mice also developed tail skin carcinomas.

11 dherin that is aberrantly expressed in human skin carcinomas.

12 Similar effects were observed in human skin carcinoma A431 cells expressing mutant p53.

13 non-small cell lung carcinoma, squamous cell skin carcinoma and lymphoma.

14 rentially expressed from the 129P2 allele in skin carcinomas and derived cell lines.

15 Because PACE4 is overexpressed in skin carcinomas and in vitro overexpression of PACE4 res

16 n the epidermis inhibited the development of skin carcinomas and metastases in mice.

17 ss reported for the treatment of superficial skin carcinomas by photodynamic therapy with topical app

18 The excision of Rce1 from a Rce1(flox/flox) skin carcinoma cell line also significantly retarded the

19 to the overexpression of COX-2 in the mouse skin carcinoma cell line JWF2, we investigated cis- and

20 e model of UVB-induced skin cancer and human skin carcinoma cells and show that the mutant p53 confor

21 in-associated NKG2d+ gammadelta cells killed skin carcinoma cells by a mechanism that was sensitive t

22 reduced myofibroblast differentiation of E4 skin carcinoma cells in a tumor allograft.

23 also found that adherent TRAIL-R-expressing skin carcinoma cells were TRAIL resistant in vitro but w

24 ted eicosanoid synthesis that is observed in skin carcinomas contributes to the growth and progressio

25 creased susceptibility to chemically induced skin carcinoma formation, whereas the number of papillom

26 gesting that this pathway is dispensable for skin carcinoma formation.

27 Instead of skin carcinomas, however, the chemical treatment protoco

28 The frequency of squamous cell skin carcinoma in organ transplant patients is around 10

29 Chemically induced skin carcinomas in mice are a paradigm for epithelial ne

30 EV-HPVs cause warts and increase the risk of skin carcinomas in otherwise healthy individuals.

31 tablished two-step protocol of squamous cell skin carcinoma, in which tumorigenesis is initiated by a

32 About 20%-30% of Hras mutant mouse skin carcinomas induced by chemical initiation/promotion

33 Development of UVB-induced skin carcinoma is a multistep and complex process.

34 However, skin carcinoma numbers were very similar, suggesting tha

35 arding the incidence trends of squamous cell skin carcinoma (SCC) and rare skin tumors, including Mer

36 s or nullizygous for p53 develop spontaneous skin carcinomas, which normally are rare in p53-deficien