1 ent site, respectively, had minimal areas of skin necrosis.

2 explaining the tendency for warfarin-induced skin necrosis.

3 nse pain, alteration of vascularization, and skin necrosis.

4 e complications (32.8% vs 11.4%; P < 0.001), skin necrosis (10.3% vs 4.0%; P = 0.043), wound breakdow

5                                       Finger skin necrosis and histologic signs of severe chronic all

6                                        Local skin necrosis and tissue neutrophil infiltration were se

7 rgery, systemic complications, postoperative skin necrosis, and reported skin and nipple areolar comp

8 T also reduces the risk of wound dehiscence, skin necrosis, and seroma.

9                           Smoking aggravates skin necrosis as a complication of random-pattern flap i

10                                              Skin necrosis, at threshold fluences in photodynamic the

11 aneous injection of p75TNF did not result in skin necrosis but did result in a modest dermal infiltra

12 ults show that haptoglobin release following skin necrosis contributes to accelerated transplant reje

13 a fever, who developed extensive acute nasal skin necrosis early in the course of a life-threatening

14 s with reversible superficial distal digital skin necrosis in the absence of overt evidence of system

15 ications (thromboembolic events, amputation, skin necrosis) occurred in 11.7% of patients treated wit

16                      The threshold doses for skin necrosis on rabbits were equal at both wavelengths.

17 GPER expression is associated with increased skin necrosis, reduced flap perfusion and altered vessel

18 l evidence indicated that iNPWT also reduced skin necrosis (RR 0.49, 95% CI: 0.33-0.74), seroma (RR 0

19 hat iNPWT may also prevent wound dehiscence, skin necrosis, seroma, and hematoma.

20           Skin breaks, ulcers, blisters, and skin necrosis were significantly more common in patients

21 n infected haematoma and an area of proximal skin necrosis were surgically treated.