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1 -2 (H-2k) (bm1) or F1 (B6 x bm1) mice before skin transplantation.
2 with an emphasis on cell-based therapies and skin transplantation.
3 ited number of lymphatic capillaries through skin transplantation.
4 ent inflammatory process in a mouse model of skin transplantation.
5 CD154 antibody following allogeneic islet or skin transplantation.
6 important in initiating rejection in murine skin transplantation.
7 reg expansion by mIL-2 in 2 murine models of skin transplantation.
8 to prolong allograft survival in a model of skin transplantation.
9 in rhesus macaques sensitized by sequential skin transplantations.
10 with thromboembolism risk) in the context of skin transplantation 2 years before the angiography stud
11 ntation with LN excision (n = 6), allogeneic skin transplantation alone (n = 6), or syngeneic skin tr
12 ishing mixed chimerism was tested in vivo by skin transplantation and in vitro by mixed leukocyte rea
14 F rats were presensitized to ACI antigens by skin transplantation and received heterotopic osteomyocu
15 These T cells proliferated modestly after skin transplantation and underwent relatively weak funct
19 of WF spleen cells (2x10[7]) at the time of skin transplantation (day -7) abrogated <24-hr rejection
20 clinically relevant humanised mouse model of skin transplantation, expBreg prolongs human allograft s
21 stinct antigenic hierarchy between heart and skin transplantation: H60-specific CD8(+) T cells were t
22 ases allograft survival in a murine model of skin transplantation in fully mismatched strain combinat
27 espite this difference in outcome, heart and skin transplantation induced antidonor T cell responses
28 phase of allograft rejection in a humanized skin transplantation model in mice reconstituted with hu
39 solution to these problems, but traditional skin transplantation techniques cause substantial morbid
40 The authors demonstrated that, following skin transplantation, the donor mast cell-mediated senes
41 keletal muscle combined with glabrous dermal skin transplantation, thus forming a bi-directional comm
42 used a mouse model of MHC-class I mismatched skin transplantation to investigate the contribution of
44 compounds, and a murine model of allogeneic skin transplantation was adopted to assess the in vivo e
45 lantation, C57BL/6 mice underwent allogeneic skin transplantation with LN excision (n = 6), allogenei