ライフサイエンスコーパス: sleeping

1 SPW-R was more pronounced during waking than sleeping.

2 r in the brain waves that occur while we are sleeping.

3 infants devote the majority of their time to sleeping.

4 e time with screens than any activity except sleeping.

5 allowing (2.1, 1.1-3.9; p=0.017), difficulty sleeping (1.9, 1.3-2.8; p=0.001), arthralgias (2.0, 1.1-

6 ing, 33 (30%); concurrent illness, 13 (12%); sleeping, 10 (9%); drugs/medication, 9 (8%); and emotion

7 DBP ranged from 16.8% to 40.4% for daytime, sleeping, 24-hour and office beat-to-beat measurements.

8 -71% for BBzP), cooking (+27% for DEHP), and sleeping (-26% for DEHP).

9 Compared with sleeping 6 to 9 h/night, short sleepers had a 20% higher

10 fer alternative to benzodiazepines and other sleeping aids(8,9), and is one of the most popular suppl

11 Waking and sleeping also affected lower- and higher-firing neurons

12 illations were once thought to only occur in sleeping and anesthetized states.

13 Infants spend most of their time sleeping and are likely to be exposed to elevated concen

14 s with KCN should inquire about breathing or sleeping and, when appropriate, refer patients for evalu

15 ssess sleep patterns - with focus on daytime sleeping - and alertness in a Latin American crew overwi

16 ayers of the frontoparietal cortex in awake, sleeping, and anesthetized macaques.

17 tial (LFP) recordings on flies spontaneously sleeping, and compare their brain activity to flies indu

18 , circuits, and behaviors including feeding, sleeping, and pain perception.

19 Sleeping animals are vulnerable to predation.

20 Sleeping animals do not move or feed and are less respon

21 als who sleep >7 hours/night with no trouble sleeping are 40% less likely to have severe periodontal

22 the new design, especially the second-storey sleeping area because of the privacy and security of ups

23 choretic insect that hides within its host's sleeping area manages to travel long distances is not ye

24 t time, how leaving worn clothing exposed in sleeping areas when travelling can be exploited by bed b

25 intense physical labour and less comfortable sleeping arrangements.

26 Sleeping at least 9 hours per 24-hour period was reporte

27 incidence of botulism in shoreline birds at Sleeping Bear Dunes National Lakeshore (SLBE) in Lake Mi

28 ation operatorOPE) ranged from 93 pg/m(3) at Sleeping Bear Dunes to 1046 pg/m(3) at Chicago.

29 bling times of 5-10 years at Sturgeon Point, Sleeping Bear Dunes, and Eagle Harbor, but these concent

30 ased sequencing and bioinformatics pipeline, Sleeping Beauty (SB) capture hybridization sequencing (S

31 son mutagenesis strategy based on a two-step Sleeping Beauty (SB) forward genetic screen to identify

32 A Sleeping Beauty (SB) in science refers to a paper whose

33 ystem actively suppresses tumorigenesis in a Sleeping Beauty (SB) mouse model of cancer.

34 e have advanced DNA vectors derived from the Sleeping Beauty (SB) system to avoid the expense and man

35 -derived CD19 CAR T cells generated with the Sleeping Beauty (SB) transposon and differentiated into

36 a hybrid genetic screening system where the Sleeping Beauty (SB) transposon and single guide RNA cas

37 strate biased genome-wide integration of the Sleeping Beauty (SB) transposon by combining it with com

38 The Sleeping Beauty (SB) transposon mutagenesis screen is a

39 ectal cancer (CRC) progression, we performed Sleeping Beauty (SB) transposon mutagenesis screens in m

40 We used Sleeping Beauty (SB) transposon mutagenesis to identify

41 driving leukemia, we targeted cre-dependent Sleeping Beauty (SB) transposon mutagenesis to the blood

42 Using Sleeping Beauty (SB) transposon mutagenesis, we identifi

43 sion (GEC(HO-1) rats) were generated using a Sleeping Beauty (SB) transposon system and extent of les

44 The Sleeping Beauty (SB) transposon system was used to eluci

45 ed transgene expression in pigs by using the Sleeping Beauty (SB) transposon system.

46 transgene expression in rabbits by using the Sleeping Beauty (SB) transposon system.

47 The latest generation of Sleeping Beauty (SB) transposon vectors fulfills these r

48 a development and metastasis, we performed a Sleeping Beauty (SB) transposon-based forward genetic sc

49 Here we show that Sleeping Beauty (SB) transposon-mediated mutagenesis dri

50 lls that were genetically modified using the Sleeping Beauty (SB) transposon/transposase system to ex

51 We used the Sleeping Beauty (SB) transposon/transposase system to id

52 ite profiling of the piggyBac (PB), Tol2 and Sleeping Beauty (SB) transposons and the murine leukemia

53 Forward genetic screens using Sleeping Beauty (SB)-mobilized T2/Onc transposons have b

54 eted copies in the IR/DR subgroup, including Sleeping Beauty (SB).

55 ON) and compare them with the preferences of Sleeping Beauty and piggyBac, showing that each superfam

56 lated when delivered into the genome via the Sleeping Beauty DNA transposon, suggesting that the obse

57 gene for colorectal cancer (CRC) in several Sleeping Beauty DNA transposon-based forward genetic scr

58 tract cancer susceptibility gene in multiple Sleeping Beauty DNA transposon-based forward genetic scr

59 Sleeping Beauty insertional mutagenesis in this model le

60 A forward genetic screen utilizing Sleeping Beauty mutagenesis in mice previously identifie

61 ells with preserved TSCM potential using the Sleeping Beauty platform.

62 T cells genetically modified with the Sleeping Beauty system to express D-CAR stably were prop

63 5 (Stat5b-CA) with mice in which a mutagenic Sleeping Beauty transposon (T2/Onc) was mobilized only i

64 For example, the Sleeping Beauty transposon has become highly instrumenta

65 We used it to perform a Sleeping Beauty transposon mutagenesis screen to identif

66 A new study uses Sleeping Beauty transposon mutagenesis to drive osteosar

67 An unbiased Sleeping Beauty transposon screen revealed MyoD as a put

68 an insertional mutagenesis screen using the Sleeping Beauty transposon system in mice with mammary-s

69 Recently in Nature, Sun et al. (2014) used a sleeping beauty transposon system to demonstrate that na

70 models, the mouse and the rat, by using the Sleeping Beauty transposon system.

71 d mobilization of a single-copy inactivating Sleeping Beauty transposon to Pten disruption within the

72 in the biliary tract was accomplished by the Sleeping Beauty transposon transfection system with tran

73 ovirus, lentivirus, PiggyBac transposon, and Sleeping Beauty transposon, in aggregate >108,000 vector

74 Exploiting the local hopping activity of the Sleeping Beauty transposon, the lacZ reporter gene was d

75 formed genetic screen in Emu-TCL1 mice using Sleeping Beauty transposon-mediated mutagenesis.

76 in cultured human PAX7+ satellite cells with Sleeping Beauty transposon-mediated nonviral gene transf

77 Kras was expressed in p53fl/fl mice using a sleeping beauty transposon.

78 approach using ex vivo mutagenesis with the Sleeping Beauty transposon.

79 ession of hMet and mutant-beta-catenin using sleeping beauty transposon/transposase leads to hepatoce

80 essing mice and forward genetic screens with Sleeping Beauty transposons implicate additional signali

81 ion elements, which were further deployed by Sleeping Beauty transposons throughout the genome of hum

82 Just like a sleeping beauty, alpha-oxocarboxylic acids were awakened

83 le with two transposon systems, PiggyBac and Sleeping Beauty, and give guidance on the use of differe

84 y hydrodynamic injection in combination with Sleeping Beauty-mediated somatic integration.

85 ve been associated with infants' feeding and sleeping behavior.

86 , attractiveness to mosquitoes, and modified sleeping behaviour.

87 rons to the presentation of song variants in sleeping birds, and by examining HVC activity in singing

88 There is a complex relation between the sleeping brain and its waking function.

89 the brain to force an awakening of the human sleeping brain and vice versa.

90 The sleeping brain exhibits characteristic slow-wave activit

91 These results demonstrate that the sleeping brain exhibits waves of CSF flow on a macroscop

92 Our results suggest a model of the human sleeping brain in which rapid bidirectional interactions

93 However, recent research has shown that the sleeping brain is not completely disconnected from its e

94 But how does the sleeping brain know which information to preserve and wh

95 definition, but it is still unclear how the sleeping brain responds differently to sensory stimuli.

96 a flexible and task-dependent manner by the sleeping brain, all the way up to the preparation of rel

97 hatic function is primarily a feature of the sleeping brain, rather than the waking brain, and is slo

98 an transiently "wake up" [8] in an otherwise sleeping brain.

99 their large-scale coherence in the awake and sleeping brain.

100 a common feature of both the waking and the sleeping brain.

101 responses triggered by spoken stimuli in the sleeping brain.

102 ns that support systems consolidation in the sleeping brain.

103 s it continue to unfold automatically in the sleeping brain?

104 Here we used bioinformatics to identify a "sleeping carboxylase function" in the superfamily of med

105 aging in close-to-perfect conditions, these sleeping champagne bottles awoke to tell us a chapter of

106 adiposity measures is exacerbated by adverse sleeping characteristics.

107 Exposure levels estimated for sleeping child/infant indicate that SME can be a signifi

108 Under sleeping conditions VOC emissions increased significantl

109 CSR) is believed to only occur in supine and sleeping conditions, and thus, CSR treatment is applied

110 f age, warm season, male gender, and crowded sleeping conditions.

111 commercial sleep technologies during in-home sleeping conditions.

112 homeostatic adjustment is disrupted in short-sleeping cv-c mutants.

113 mance measures compared to sleeping on LR or sleeping directly on spring mattresses without a topper.

114 as modafinil, are used for the treatment of sleeping disorders and investigated as potential therape

115 Sleeping disorders are one of the underrecognized sequal

116 of specific tryptamines for the treatment of sleeping disorders, bupropion for substance abuse disord

117 ts and anxiety phenotype, without detectable sleeping disorders.

118 ms of hypomania, agitation, impulsivity, and sleeping disorders.

119 lay in the development and severity of these sleeping disorders.

120 improves sleep efficiency in multiple short-sleeping Drosophila mutants.

121 d to increased insomnia symptoms and trouble sleeping due to bad dreams.

122 alance between maintaining the wake state or sleeping during the day has important health implication

123 ambers to measure changes in EE (EEchamber), sleeping EE (SEE), and RQ.

124 Twenty-four-hour EE (24EE) and sleeping EE (SleepEE) were measured on days 1, 14, and 5

125 No difference was found in resting or sleeping energy expenditure, normalised to lean tissue w

126 As such, measures of light in the sleeping environment are critical for evaluating sleep h

127 stions addressing perception of light in the sleeping environment may provide a crude yet affordable

128 ly indicating poorer sleep with light in the sleeping environment.

129 nt stimuli are larger in awake flies than in sleeping flies.

130 uable compounds to be discovered when these 'sleeping' genes are activated.

131 hereas regular physical activity and healthy sleeping habits help prevent it.

132 sed 2 additional contacts for guidance about sleeping habits.

133 rt system administered questions on life and sleeping habits; health; and sleep, mental, and organic

134 igh frequency distal body temperature (DBT), sleeping heart rate (HR), sleeping heart rate variabilit

135 esting (P = 0.001), stressed (P = 0.037) and sleeping heart rate (P = 0.038) were increased compared

136 temperature (DBT), sleeping heart rate (HR), sleeping heart rate variability (HRV), and sleep timing,

137 rt devices were fitted for 7 days to measure sleeping heart rate, activity levels, and resting, activ

138 eviant external stimuli detected by the less-sleeping hemisphere caused more arousals and faster beha

139 The number of observed sleeping hours at night was assessed by the bedside nurs

140 me in bed during weekdays, and later weekend sleeping hours correlate with smaller brain grey matter

141 ttachment locations for 24 hours, waking and sleeping hours, and to test comparability of PA cut poin

142 rgy the eye must withstand during waking and sleeping hours, respectively.

143 Bed bugs are attracted to the odour of sleeping humans and we suggest that soiled clothing may

144 ative upper airway hydraulic diameter (D) in sleeping humans with narrowed upper airways.

145 se pharyngeal hypotonia pharmacologically in sleeping humans.

146 n in vitro model and in the upper airways of sleeping humans.

147 le, which has similarities to flying west or sleeping in the daytime after night shifts.

148 It is not known if, as a result, daytime sleeping in the form of naps increases.

149 Household contacts (individuals sleeping in the home >=four nights per week) and indoor

150 It now appears that humans sleeping in unfamiliar environments do something quite s

151 in one hundred sixty-two 2- to 25-month-old sleeping infants (60 epsilon4 carriers and 102 noncarrie

152 s in a cohort of obese individuals reporting sleeping less than 6.5 hours per night.

153 uction of solid foods), and early childhood (sleeping <12 h/d, presence of a television set in the

154 recordings along the dorsal CA1-DG axis from sleeping male mice, we detected and classified two types

155 egulation and control found in bihemispheric sleeping mammals and the harbor porpoise were present in

156 egulation and control found in bihemispheric sleeping mammals were present in the harbor porpoise, wi

157 igher than other large-brained bihemispheric sleeping mammals.

158 er than in other large-brained bihemispheric sleeping mammals.

159 conditions, we analyzed changes in 24 EE and sleeping metabolic rate (SLEEP) in a whole-room indirect

160 r baseline energy requirements for 8 wk, and sleeping metabolic rate (SMR) and 24-h sedentary energy

161 stage for the whole night and separately for sleeping metabolic rate (SMR; ie, 3-h period during the

162 nduced minor changes in body composition and sleeping metabolic rate.

163 ge: 1.34% +/- 0.50%, P = 0.02) and increased sleeping metabolic rate.

164 Local cortical activity of sleeping mice could be readily converted to the stereoty

165 cooling of lightly anesthetized or naturally sleeping mice disrupts thalamocortical slow oscillation

166 Sleeping microenvironment (SME), is characterized by hig

167 as protein levels are up-regulated in short-sleeping mutants and also in wild type animals following

168 emically mutagenized flies to identify short-sleeping mutants and found one, redeye (rye) that shows

169 uth irritation (n = 4 and n = 0), difficulty sleeping (n = 3 and n = 2), and vivid dreams (n = 3 and

170 We found that chimpanzees left their sleeping nests earlier (often before sunrise when the fo

171 Moreover, the females positioned their sleeping nests more in the direction of the next day's b

172 ng data were obtained on a 3T scanner in 138 sleeping nonsedated neonates: 55 full-term neonates (ges

173 We recently demonstrated that sleeping on high rebound [HR] mattress toppers induced a

174 Sleeping on HR mattress toppers for four to six weeks im

175 or only 0.69 h d(-1) (7.4% of the time spent sleeping on land), indicating that ecological demands fo

176 me athletic performance measures compared to sleeping on LR or sleeping directly on spring mattresses

177 ation sessions did not prevent patients from sleeping; on the contrary, they seem to aid sleep when c

178 ce an exogenous stressor, such as prone/side sleeping or soft bedding, during a critical developmenta

179 For instance, sleeping participants can create novel sensory associati

180 roves after interventions restoring a normal sleeping pattern.

181 and ambient temperature on human resting or sleeping patterns using mobile phone data of a large num

182 Behavioral arousal in the sleeping period phase shifts the master clock in the sup

183 by sleep deprivation or caffeine during the sleeping period potentiates light resetting of the maste

184 study demonstrates the strong dependency of sleeping person exposure on air exchange rate between hi

185 , a kinase partner of CycA, rescue the short-sleeping phenotype of tara and CycA mutants, while incre

186 brain injury when they are treated with the sleeping pill zolpidem.

187 idem (Ambien) is the most widely prescribed "sleeping pill." It makes the inhibitory neurotransmitter

188 This knowledge could help in the design of sleeping pills that induce a more natural sleep.

189 position), which mimics the natural resting/sleeping position of rodents.

190 es, addition of hydrocortisone reduced total sleeping problems and disorders of initiating and mainta

191 blems, quality of life, parenting stress and sleeping problems were compared between groups.

192 e SCORAD), symptoms (POEM, VAS pruritus, VAS sleeping problems) and previous treatment of AD were ass

193 e for previous AD severity and/or associated sleeping problems.

194 vity from the primary motor cortex of 4 male sleeping rats to investigate how SO and spindles interac

195 aring experimental observations of naturally-sleeping rats with a mean field model of an adapting, re

196 cillations in the spindle range in naturally sleeping rodents.

197 re time in screen activities (RR = 0.98) and sleeping (RR = 0.96) had the lowest compliance.

198 Women (>=41 years) reported sleeping shorter times or slightly less efficiently than

199 Trypanosoma brucei, the causative agent of sleeping sickness (Human African Trypanosomiasis, HAT),

200 Chagas disease, leishmaniasis and sleeping sickness affect 20 million people worldwide and

201 cei, which is the parasite that causes human sleeping sickness and is also partially responsible for

202 oma brucei is the causative agent of African sleeping sickness and is known for its unique RNA proces

203 vities against diseases that include African sleeping sickness and malaria.

204 anosoma brucei is a protist parasite causing sleeping sickness and nagana in sub-Saharan Africa.

205 very that may undermine efforts to eliminate sleeping sickness by 2020.

206 Trypanosoma brucei causes African sleeping sickness for which no vaccine exists and availa

207 cei causes African trypanosomiasis, known as sleeping sickness in humans and nagana in domestic anima

208 African trypanosomiasis, sleeping sickness in humans or nagana in animals, is a p

209 When they infect mammals, they cause African sleeping sickness in humans.

210 lated protozoan parasite that causes African sleeping sickness in humans.

211 oms, in this study, we used a mouse model of sleeping sickness in which the acute infection was treat

212 Sleeping sickness is a fatal disease caused by the proto

213 c adenosine signaling.SIGNIFICANCE STATEMENT Sleeping sickness is a fatal disease that disrupts the c

214 d, our findings suggest that the symptoms of sleeping sickness may be because of alterations in homeo

215 In the sleeping sickness parasite Trypanosoma brucei, the kinet

216 this model, we evaluated the effects of the sleeping sickness parasite, Trypanosoma brucei, on sleep

217 The sleeping sickness parasite, Trypanosoma brucei, uses quo

218 The strategy that sleeping sickness parasites use to evade the mammalian i

219 ction of rare cell types in blood (including sleeping sickness parasites), and has the potential to e

220 Protozoan parasites such as the sleeping sickness pathogen Trypanosoma brucei adapt to d

221 me infection in both murine models and human sleeping sickness patients.

222 s the only example of cross-resistance among sleeping sickness therapies.

223 biense human African trypanosomiasis ([gHAT] sleeping sickness) is a vector-borne disease that is typ

224 ambiense Human African Trypanosomiasis (HAT, sleeping sickness), a disease that has often been consid

225 en argued that the risk for trypanosomiasis (sleeping sickness), carried by tsetse flies in bushy env

226 copeia for human African trypanosomiasis (or sleeping sickness), orally dosed fexinidazole stands poi

227 tive agent of Human African Trypanosomiasis (sleeping sickness), yet little is known about which PKs

228 iasis, visceral leishmaniasis, and gambiense sleeping sickness).

229 of early-stage East African trypanosomiasis (sleeping sickness).

230 oma brucei is the causative agent of African sleeping sickness, a devastating disease endemic to sub-

231 ing target to develop new treatments against sleeping sickness, a fatal disease caused by this protoz

232 t and Southern Africa are foci for Rhodesian sleeping sickness, a fatal zoonotic disease caused by tr

233 animal trypanosomiases, including Rhodesian sleeping sickness, a zoonosis associated with wilderness

234 or such human pathologies as Chagas disease, sleeping sickness, and leishmaniasis.

235 ovide different levels of protection against sleeping sickness, but this comes with an increased risk

236 Patients with sleeping sickness, caused by the parasite Trypanosoma br

237 African sleeping sickness, caused by the protozoan parasite Tryp

238 anosoma brucei, a causative agent of African Sleeping Sickness, constantly changes its dense variant

239 e Trypanosoma brucei, the causative agent of sleeping sickness, cycles between an insect and a mammal

240 osoma brucei, the etiologic agent of African Sleeping Sickness, deploys an RNA pol II that contains a

241 osoma brucei, the causative agent of African sleeping sickness, differs from its human host in severa

242 anosoma brucei, the causative agent of human sleeping sickness, has an intrinsic circadian clock that

243 an African trypanosomiasis (HAT), or African sleeping sickness, is a fatal disease found throughout s

244 Human African trypanosomiasis, or sleeping sickness, is caused by infection with parasites

245 Human African trypanosomiasis (HAT), or sleeping sickness, is caused by the protozoan parasite T

246 osoma brucei, the causative agent of African sleeping sickness, is transmitted to its mammalian host

247 Trypanosoma brucei, the etiological agent of sleeping sickness, localized its replication origins to

248 he parasitic trypanosomes that cause African sleeping sickness, mating occurs during transmission by

249 ite Trypanosoma brucei, which causes African sleeping sickness, TbISWI down-regulates RNA polymerase

250 s typified by the causative agent of African sleeping sickness, Trypanosoma brucei In mitochondria of

251 energy metabolism in the causative agent of sleeping sickness, Trypanosoma brucei, with that of huma

252 e the causative agents of Chagas disease and sleeping sickness, two neglected tropical diseases where

253 can trypanosomes, parasites that cause human sleeping sickness, undergo a density-dependent different

254 compounds to treat Chagas disease and human sleeping sickness.

255 t for human African trypanosomiasis (HAT) or sleeping sickness.

256 t pathogenic trypanosomes that cause African sleeping sickness.

257 cei is an extracellular parasite that causes sleeping sickness.

258 ally explain the weight loss associated with sleeping sickness.

259 n disease called African trypanosomiasis, or sleeping sickness.

260 asite that causes the deadly disease African sleeping sickness.

261 seases, including malaria, dengue fever, and sleeping sickness.

262 ucei rhodesiense, the cause of acute African sleeping sickness.

263 n trypanosomiasis (HAT), also called African sleeping sickness.

264 panosoma brucei, the causal agent of African sleeping sickness.

265 fforts to develop new treatments for African sleeping sickness.

266 ed by the tsetse fly and that causes African sleeping sickness.

267 the future therapeutic treatment of African sleeping sickness.

268 an African trypanosomiasis (HAT), or African sleeping sickness.

269 lyses trypanosomes, protecting against human sleeping sickness.

270 osoma brucei, the causative agent of African sleeping sickness.

271 idin is a promising lead to treat late-stage sleeping sickness.

272 osoma brucei, the causative agent of African sleeping sickness; and Plasmodium spp., the causative ag

273 ge attraction and repulsion from the troop's sleeping site, to relatively local influences including

274 k factors for infants include prone and side sleeping, soft bedding, bed sharing, inappropriate sleep

275 During resting and sleeping states, slow fluctuations (<1 Hz) of HFB activi

276 cond (recovery) night of sleep revealed that sleeping the first half of the night, which is dominated

277 Conversely, sleeping the second half of the night, which is dominate

278 glycemia associated with different levels of sleeping time (<11.0, 11.0-11.9, and >/=12.0 hours/day)

279 (SWS), rapid eye movement (REM)-sleep, total sleeping time (TST), sleep stage 2 (S2), and QS [(SWS +

280 based on arbitrary threshold parameters for sleeping time and number of citations, applied to small

281 rse association of either indoor activity or sleeping time with the risk of hyperglycemia among offsp

282 tion of physical activity, TV watching time, sleeping time with the risks of obesity and hyperglycemi

283 activity, outdoor activity, TV watching, and sleeping time.

284 Lower sleeping times and fragmented sleep are independently as

285 ld that owned a bed net, only 36.6% reported sleeping under a long-lasting insecticide-treated net (L

286 difference 0.02, 95% CI - 0.02 to 0.05) and sleeping under an LLIN (0.01; - 0.02 to 0.04) were not p

287 of 1371; 1.10, 1.04-1.16; p=0.001), and baby sleeping under bednet for 8-56 days (4548 [79%] of 5756

288 ill at high altitude and whether training or sleeping under normobaric hypoxic conditions in the week

289 ) and (2) protective efficacy for volunteers sleeping under the LLINs (bite reduction and mosquitoes

290 ion usually exceed the attention afforded by sleeping unihemispherically.

291 an array of fundamental behaviors, including sleeping, waking, feeding, stress and motivated behavior

292 ng non-REM sleep in freely moving, naturally sleeping-waking rats.

293 cellular recording and labeling in naturally sleeping-waking, head-fixed rats, we investigated the di

294 Sometimes while sleeping we find ourselves thrown amidst an authentic, a

295 The symptoms of edema and trouble sleeping were variable among the countries and fell into

296 r orthokeratology (OR, undefined; P = 0.02), sleeping while wearing lenses (OR, 8.00; P = 0.04), stor

297 5% CI, 1.2-10.3) compared to 5-14 years, and sleeping with index case (OR 2.7; 95% CI, 1.3-5.5).

298 nmental awareness and aerodynamic control by sleeping with only one eye closed and one cerebral hemis

299 ever, the benefit of conserving energy while sleeping with the head tucked was countered by reduced a

300 In addition, rather than sleeping with their head facing forward, birds in poor c