ライフサイエンスコーパス: slow-wave sleep

1 ction, P < 0.02), urinary catecholamines, or slow wave sleep.

2 ved in the cortex and the hippocampus during slow wave sleep.

3 ather tended to increase waking and decrease slow wave sleep.

4 ent sleep, whereas targeting D2-MSNs affects slow wave sleep.

5 ave ripples observed in quiet wakefulness or slow wave sleep.

6 share many features with the down states of slow wave sleep.

7 lective memory generalization during REM and slow-wave sleep.

8 , that it also does not arise during natural slow-wave sleep.

9 cting total sleep time, sleep efficiency, or slow-wave sleep.

10 delta oscillations have been associated with slow-wave sleep.

11 derlies the generation of sharp waves during slow-wave sleep.

12 d fast-sigma oscillations, especially during slow-wave sleep.

13 ory gating, thalamocortical rhythmicity, and slow-wave sleep.

14 ith performance improvement were specific to slow-wave sleep.

15 ivation process for specific memories during slow-wave sleep.

16 and its role in sharp-wave generation during slow-wave sleep.

17 EEGs is the K-complex (KC), which occurs in slow-wave sleep.

18 ating cellular and network plasticity during slow-wave sleep.

19 uent in waking and paradoxical sleep than in slow-wave sleep.

20 oscillatory activity (spindle waves) during slow-wave sleep.

21 ay severe sleep loss as a result of unstable slow-wave sleep.

22 role in organizing cortical activity during slow-wave sleep.

23 r are reactivated during rest and subsequent slow-wave sleep.

24 ge II and REM but is preempted by arousal in slow-wave sleep.

25 preempted development of EUCR and 2P during slow-wave sleep.

26 ring behaviours such as alert immobility and slow-wave sleep.

27 ponse to auditory stimuli is greatest during slow-wave sleep.

28 as the slow (0.1-0.5 Hz) oscillation during slow-wave sleep.

29 f hippocampal projections within mPFC during slow-wave sleep.

30 ncreased wakefulness at the expense of deep, slow-wave sleep.

31 timescale during single sharp-wave bursts of slow-wave sleep.

32 ay dilator activity in sleep and/or enhanced slow-wave sleep.

33 al cell spikes in the rat hippocampus during slow-wave sleep.

34 ion and were most frequently observed during slow-wave sleep.

35 sitions across cortical areas during natural slow-wave sleep.

36 aking behavior spontaneously reemerge during slow-wave sleep.

37 al decrease in effective interactions during slow-wave sleep.

38 rousal but to rapidly and selectively induce slow-wave sleep.

39 ot occur with MECIII input inhibition during slow-wave sleep.

40 ity in healthy humans during wakefulness and slow-wave sleep.

41 during wakefulness, to a stable focus during slow-wave sleep.

42 ll brain nodes to best match wakefulness and slow-wave sleep.

43 play of cortical cell spike sequences during slow-wave sleep.

44 s deviations from perfect balance, mostly in slow-wave sleep.

45 ef periods of desynchronization prevalent in slow-wave sleep.

46 f stimulation in memory consolidation during slow-wave sleep.

47 is important for this transformation during slow-wave sleep.

48 during waking and accelerated tenfold during slow-wave sleep.

49 onists induce wakefulness and reduce REM and slow-wave sleep.

50 th one learned sequence were replayed during slow-wave sleep.

51 ast two sleep stages: rapid eye movement and slow wave sleep(1-4), in part characterized by wake-like

52 s both REM sleep and wakefulness and reduces slow-wave sleep 2.

53 ists during subsequent quiet wakefulness and slow-wave sleep, a process that may facilitate the conso

54 in extended sleep paralleling changes in EEG slow-wave sleep activity.

55 These results suggest that enhancing slow-wave sleep acutely after trauma may have a benefici

56 l of the thalamocortical network to test how slow-wave sleep affects performance on an unordered rela

57 tates of diminished consciousness, including slow wave sleep, anaesthesia, generalized epileptic seiz

58 g, i.p.) was found to significantly increase slow wave sleep and decrease REM sleep in rats implanted

59 s thalamic network activity occurring during slow wave sleep and paroxysmal discharges critically dep

60 In men, age-related changes in slow wave sleep and REM sleep occur with markedly differ

61 Hcrt cells are silent in slow wave sleep and tonic periods of REM sleep, with occ

62 Slow wave sleep and total sleep time are indistinguishab

63 amount of delta (1-4 Hz) oscillations during slow-wave sleep and a time-of-day-dependent alteration i

64 Ebselen decreased slow-wave sleep and affected emotional processing by inc

65 spent in rapid eye movement (REM) sleep and slow-wave sleep and an increase in muscle tone during RE

66 ("sharp wave-ripples") occur during rest and slow-wave sleep and are thought to be important for memo

67 During slow-wave sleep and deep anesthesia, the rat hippocampus

68 sleep spindles (6-14 Hz) occur mostly during slow-wave sleep and have been hypothesized to involve th

69 disability and autism which may impact both slow-wave sleep and information processing during waking

70 ave sleep, share commonalities with those of slow-wave sleep and paradoxical or rapid eye movement sl

71 ia-activated neurons (AANs) strongly promote slow-wave sleep and potentiates GA, whereas conditional

72 Slow-wave sleep and rapid eye movement (or paradoxical)

73 robability of an awakening event during both slow-wave sleep and rapid eye movement sleep.

74 xtinguished conditioned fear, increased both slow-wave sleep and rapid-eye movement (REM) sleep.

75 6% more time in a transitional state between slow-wave sleep and REM sleep (tS-R) compared with that

76 natomical substrate for behaviors, including slow-wave sleep and seizure suppression evoked by stimul

77 s showed differentially smaller increases in slow-wave sleep and smaller reductions in stage 2 sleep

78 egative affect stage, there is a decrease in slow-wave sleep and some limited recovery in REM sleep w

79 In old age, the duration of slow-wave sleep and the number of coupling events decrea

80 ane potential oscillations are slower during slow-wave sleep and under anesthesia.

81 neural network oscillations associated with slow-wave sleep and various epilepsies.

82 during quiescent states such as anesthesia, slow-wave sleep, and awake immobility.

83 ty and beta-cell function, for time spent in slow-wave sleep, and for EEG spectral power in the delta

84 patterns of activity associated with waking, slow-wave sleep, and generalized seizures.

85 offline states such as pause in exploration, slow-wave sleep, and quiescent wakefulness.

86 aneous BOLD fluctuations across wakefulness, slow-wave sleep, and rapid-eye-movement sleep.

87 assessed for presence of HEP within stage 2, slow-wave sleep, and REM sleep in 40 children with prima

88 ivity and temperature patterns, increases in slow-wave sleep, and shifts in EEG spectral power, sever

89 0 Hz ripples occur in the hippocampus during slow-wave sleep, and ultrafast (400-600 Hz) oscillations

90 e rat brain ventricles specifically enhances slow wave sleep, apparently by antagonizing the effects

91 and systemic disease and injury (e.g. fever, slow-wave sleep, appetite suppression and neuroendocrine

92 Slow-wave sleep as well as generalized absence seizures

93 nized network oscillations representative of slow-wave sleep, as well as absence seizures, were demon

94 henomenon, which is also termed asymmetrical slow wave sleep (ASWS).

95 water, however, fur seals exhibit asymmetric slow-wave sleep (ASWS), resembling the unihemispheric sl

96 s carried out on five distinct brain states: slow-wave sleep, awake, deep anesthesia-slow waves, ligh

97 and REM sleep than in quiet wakefulness and slow wave sleep, behavioral states that differ with resp

98 During quiescence and slow wave sleep, bouts of synchronized activity represen

99 t patterns of sleep: bilaterally symmetrical slow-wave sleep (BSWS) as seen in terrestrial mammals an

100 e unique in that they display both bilateral slow-wave sleep (BSWS), as seen in all terrestrial mamma

101 involved no further significant decrease in slow wave sleep but an increase in time awake of 28 minu

102 We are unresponsive during slow-wave sleep but continue monitoring external events

103 depression, revealing not only a decrease in Slow Wave Sleep, but also a disinhibition of REM (rapid

104 Such activity emerges not only in slow wave sleep, but also under anesthesia and in brain

105 scillation which occurs predominantly during slow-wave sleep, but may also play a role during awake s

106 ng of HA neurons during wakefulness promotes slow-wave sleep, but not rapid eye movement sleep, durin

107 Slow-wave sleep consists in slowly recurring waves that

108 vity of cholinergic brainstem neurons during slow-wave sleep continues to have a functional impact up

109 We propose that rapid eye movement (REM) and slow-wave sleep contribute differently to the formation

110 During slow-wave sleep, cortical neurons display synchronous fl

111 The mean (SEM) percentage of deep slow wave sleep decreased from 18.9% (1.3%) during early

112 e time awake during the night, a decrease in slow-wave sleep, decreases in delta electroencephalogram

113 Short REM latency and slow wave sleep deficits are familial.

114 Short REM latency was also associated with slow wave sleep deficits.

115 al oscillations that underlie drowsiness and slow-wave sleep depend on rhythmic inhibition of relay c

116 rst half of the night, which is dominated by slow-wave sleep, did not improve recall.

117 eye movement sleep (all P < 0.001), whereas slow-wave sleep duration was preserved (Poverfeeding x s

118 ogic markers of sleep homeostasis, including slow-wave sleep, electroencephalographic slow-wave activ

119 g spontaneous wakefulness as contrasted with slow wave sleep; exhibited progressive increases during

120 ced a transient decrease in REM sleep and in slow-wave sleep followed by a slight improvement of slee

121 ng speech understanding, sensory gating, and slow-wave sleep for a subset of elderly individuals.

122 and is subsequently replayed during rest or slow-wave sleep for consolidation of the encoded experie

123 The decline in slow wave sleep from early adulthood to midlife was para

124 .5 Hz) cerebral cortical oscillations during slow-wave sleep has recently lead to the suggestion that

125 urons that are specifically activated during slow-wave sleep have not previously been described in th

126 During slow-wave sleep, HVC neurons responded preferentially to

127 ry inputs, as in development in utero, or in slow-wave sleep (i.e., throughout the entire lifespan),

128 ce and decreases paradoxical (REM) sleep and slow wave sleep in rats.

129 the production of sharp-wave ripples during slow-wave sleep in a unilateral or bilateral manner, res

130 se supports a potentially beneficial role of slow-wave sleep in neurodegeneration.

131 However, the importance of slow-wave sleep in Parkinson disease is unknown.

132 s probably correlated with the appearance of slow-wave sleep in postnatal animals.

133 oth the visual cortex and hippocampus during slow-wave sleep in rats.

134 The signature of slow-wave sleep in the electroencephalogram (EEG) is lar

135 , decreased delta sleep ratio, and decreased slow wave sleep [in percentage]) were stable, as predict

136 n chronically sleep restricted subjects, low slow-wave sleep intensity over the right prefrontal cort

137 Slow-wave sleep is a marker of sleep need, but its prese

138 Slow-wave sleep is characterized by near-synchronous alt

139 indicate that the control of unihemispheric slow-wave sleep is likely to be a function of interponti

140 In contrast, slow-wave sleep is more directly involved in the consoli

141 synchronization of cortical activity during slow-wave sleep is still controversial, with some studie

142 Slow-wave sleep is thought to be important for retuning

143 sleepwalking, i.e. the partial arousal from slow-wave sleep, is today well-documented, the detailed

144 s bursts occurring in the hippocampus during slow-wave sleep, leading to the selective erasure of inf

145 sleep and anesthesia regimens that induce a slow-wave sleep-like state.

146 icipants, patients had significantly reduced slow-wave sleep-likely due to decreased density of slow

147 rospinal fluid Abeta dynamics, decrements in slow-wave sleep may decrease the clearance of Abeta from

148 first time, a mechanistic explanation of how slow-wave sleep may promote consolidation of recent memo

149 eversible state abnormalities, while reduced slow-wave sleep may represent a more persistent trait ab

150 s showing the mean and standard deviation of slow-wave sleep MI of neighboring non-epileptic channels

151 uring sleep can be related to an increase in slow wave sleep (N3).

152 time, shorter sleep onset latency, and more slow-wave sleep (N3) during sleep opportunities 1-4 but

153 empirical functional changes observed during slow-wave sleep, namely a global shift of the brain's dy

154 sleep (ASWS), resembling the unihemispheric slow-wave sleep of odontocetes (toothed whales).

155 During subsequent slow-wave sleep, one sound was unobtrusively presented t

156 No slow wave sleep or rapid eye movement sleep stages could

157 ity of transition to wakefulness from either slow wave sleep or rapid eye movement sleep.

158 (active) and Down (quiescent) states during slow-wave sleep or anesthesia.

159 These oscillations occur during slow-wave sleep or at rest.

160 During slow-wave sleep or quiet restfulness, SWRs result from t

161 can provide novel insights into neocortical slow-wave sleep oscillations and their relationship to r

162 Auditory stimuli phase-locked to slow-wave sleep oscillations have been shown to augment

163 secretion was significantly associated with slow wave sleep (P<.001).

164 reased sleep efficiency, p = 0.005, enhanced slow-wave sleep, p = 0.0004, and minimized sleep-related

165 SDB during non-REM sleep (stage 2: P = 0.03; slow-wave sleep: P = 0.001).

166 This cohort study found that slow-wave sleep percentage declined with aging and Alzhe

167 expressing glutamatergic neurons to increase slow wave sleep pressure and abundance.

168 o the brain ventricles specifically enhances slow-wave sleep, presumably by antagonizing the effects

169 duration and neurophysiological hallmarks of slow-wave sleep previously linked to sequential neural r

170 ring eating, drinking, awake immobility, and slow-wave sleep, produce a large field excitatory postsy

171 oning, re-exposure to the odorant context in slow-wave sleep promoted stimulus-specific fear extincti

172 ation (TMR) of specific memory traces during slow-wave sleep promotes the emergence of explicit knowl

173 ppocampus resemble those found in nonprimate slow wave sleep, quantitative studies of these oscillati

174 -cell recordings manifesting Up/Down states (slow-wave sleep, quiet wakefulness), probably as a resul

175 s administered to the LH, rats had increased slow-wave sleep, rapid-eye movement (REM) sleep, and sle

176 ocampal local field potentials (LFPs) during slow-wave sleep-related to motor-bursts (micro-arousals)

177 portion = 1.24, 95% CI: 1.14, 1.33) and less slow wave sleep (relative proportion = 0.86, 95% CI: 0.7

178 ced REM sleep, while leaving active wake and slow wave sleep relatively intact.

179 nimal studies support the hypothesis that in slow-wave sleep, replay of waking neocortical activity u

180 tions between the different phases of sleep: Slow-wave sleep requires low ACh concentrations in the b

181 During slow-wave sleep, rhythmic burst firing is prominent, whe

182 ty that underlie rhythmic motor patterns and slow-wave sleep rhythms.

183 e motion stimulation on the fingertip during slow wave sleep selectively enhanced subsequent visual m

184 e motion stimulation on the fingertip during slow wave sleep selectively enhanced subsequent visual m

185 ons are essential for the full expression of slow-wave sleep, show that Down transition is an active

186 sufficient to rapidly and selectively induce slow-wave sleep.SIGNIFICANCE STATEMENT The function of m

187 occur during both behavior (awake SWRs) and slow-wave sleep (sleep SWRs).

188 ults were additionally disadvantaged in %N3 (slow wave sleep), sleepiness, and sleep timing (24-hour

189 Given the reported function of slow-wave sleep states in neocortical and hippocampal me

190 s activity found both in deep anesthesia and slow-wave sleep states, suggesting that slow waves were

191 ule scale relationships disintegrated during slow-wave sleep, suggesting that grid modules function a

192 hierarchy of slow waves and spindles during slow-wave sleep supports memory consolidation.

193 In particular, during episodes of slow wave sleep (SWS) and rapid eye movement (REM) sleep

194 s share specialized forms of sleep including slow wave sleep (SWS) and rapid eye movement sleep (REM)

195 Cortical activity during slow wave sleep (SWS) differs from that during REM sleep

196 equences involving four or more cells during slow wave sleep (SWS) immediately following, but not pre

197 ar activity of cortical neurons while during slow wave sleep (SWS) these neurons show synchronous alt

198 p as a reference, an increase in stage 2 and slow wave sleep (SWS) were protective from hypersomnolen

199 cally exhibit reduced discharge rates during slow wave sleep (SWS), a subpopulation of GABAergic inte

200 tuned for hippocampal ensemble spike data in slow wave sleep (SWS), even in the absence of prior beha

201 that result in reduced interference include slow wave sleep (SWS), NMDA receptor antagonists, benzod

202 In subsequent slow wave sleep (SWS), place cell reactivation was reduc

203 In sleeping zebra finches, we observed slow wave sleep (SWS), rapid eye movement (REM) sleep, a

204 Slow wave sleep (SWS), the deepest sleep stage hallmarke

205 During slow wave sleep (SWS), traces of neuronal activity patte

206 al hyperactivity in exploratory behavior and slow wave sleep (SWS), yet suppressing activity in quiet

207 and humans has suggested the existence of a slow wave sleep (SWS)-promoting/electroencephalogram (EE

208 nists of 5-HT(2A) have been shown to enhance slow wave sleep (SWS).

209 cant increase in wakefulness and decrease in slow wave sleep (SWS).

210 st degree of neuronal synchronization during slow wave sleep (SWS).

211 ly observed in electrophysiogical studies of slow wave sleep (SWS).

212 eep onset; (ii) during Stage 3-4 sleep, i.e. slow wave sleep (SWS); (iii) during rapid eye movement (

213 ally sleeping common marmosets, we show that slow-wave sleep (SWS) alters neural responses in the pri

214 ippocampal sharp-wave/ripple (SWR) bursts in slow-wave sleep (SWS) and are sharply reduced during REM

215 luding several markers of sleep homeostasis: slow-wave sleep (SWS) and electroencephalogram (EEG) slo

216 GABA neuron firing rate decreased 53% during slow-wave sleep (SWS) and increased 79% during REM, rela

217 z EEG slow oscillation (SO) is a hallmark of slow-wave sleep (SWS) and is critically involved in slee

218 s by brief (60- 90 min) naps containing both slow-wave sleep (SWS) and rapid eye movement (REM) sleep

219 s the vigilance states of quiet waking (QW), slow-wave sleep (SWS) and rapid eye movement (REM) sleep

220 erns of EEG activity as a function of awake, slow-wave sleep (SWS) and rapid-eye movement (REM) sleep

221 electrophysiological states observed during slow-wave sleep (SWS) and rapid-eye-movement (REM) sleep

222 two well-characterized physiological states, slow-wave sleep (SWS) and rapid-eye-movement sleep (REM)

223 However, only in slow-wave sleep (SWS) beta- and gamma-oscillations are a

224 respond to 6-h sleep deprivation (SD) with a slow-wave sleep (SWS) EEG delta (1.0 to 4.0 Hz) power re

225 rsistently increased calcium activity during slow-wave sleep (SWS) episodes while spindle-inactive ce

226 During slow-wave sleep (SWS) episodes, mean calcium activity of

227 During natural slow-wave sleep (SWS) in nonanesthetized cats, silent (d

228 Thus, slow-wave sleep (SWS) is characterized by EEG spindles a

229 Slow-wave sleep (SWS) is characterized by synchronized n

230 standing the intricate mechanisms underlying slow-wave sleep (SWS) is crucial for deciphering the bra

231 Slow-wave sleep (SWS) is known to contribute to memory c

232 In the hippocampus, slow-wave sleep (SWS) is marked by high-frequency networ

233 Slow oscillations during slow-wave sleep (SWS) may facilitate memory consolidatio

234 ery similar; all three produced increases in slow-wave sleep (SWS) only in the dark period with no ch

235 th sounds, then replayed these during either slow-wave sleep (SWS) or rapid eye movement (REM) sleep

236 Slow-wave sleep (SWS) supports the aging brain in many w

237 bens (NAc) is a region for the regulation of slow-wave sleep (SWS) through the integration of motivat

238 Slow-wave sleep (SWS) was characterized by low neural fi

239 ep (BSWS) as seen in terrestrial mammals and slow-wave sleep (SWS) with a striking interhemispheric E

240 thening of associated neural circuits during slow-wave sleep (SWS), a process known as "cellular cons

241 tegories: rapid eye movement (REM) sleep and slow-wave sleep (SWS), and accordingly REM and SWS are t

242 to determine wake after sleep onset (WASO), slow-wave sleep (SWS), and rapid eye movement (REM) slee

243 as they cycled normally between waking (W), slow-wave sleep (SWS), and rapid eye movement (REM) slee

244 ENT Convincing evidence supports the role of slow-wave sleep (SWS), and the relevance of close tempor

245 points, including paradoxical sleep (PS) and slow-wave sleep (SWS), as well as the circadian rhythmic

246 Slow-wave sleep (SWS), characterized by slow oscillation

247 rapid eye movement (REM) episode, decreased slow-wave sleep (SWS), disturbed sleep continuity, and d

248 pid eye movement (NREM) sleep, also known as slow-wave sleep (SWS), is thought to be the most "restor

249 traces are spontaneously reactivated during slow-wave sleep (SWS), leading to the consolidation of r

250 e for 48 h in a respiration chamber, whereas slow-wave sleep (SWS), rapid eye movement (REM)-sleep, t

251 of freely moving rats, and the effects on W, slow-wave sleep (SWS), REM sleep, and levels of phosphor

252 d electroencephalogram (EEG) activity during slow-wave sleep (SWS), similar to that observed in all t

253 Recent evidence shows that during slow-wave sleep (SWS), the brain is cleared from potenti

254 s, memory consolidation occurs partially via slow-wave sleep (SWS)-dependent replay of activity patte

255 (50%) in daily wakefulness at the expense of slow-wave sleep (SWS).

256 as positively correlated with the amounts of slow-wave sleep (SWS).

257 lectrophysiological activity observed during slow-wave sleep (SWS).

258 higher during active waking (AW) than during slow-wave sleep (SWS).

259 itory memory cues during human participants' slow-wave sleep (SWS).

260 sleep maintenance and for the regulation of slow-wave sleep (SWS).

261 paradoxical sleep (PS; aka REM) than during slow-wave sleep (SWS).

262 fulness, but in the reverse direction during slow-wave sleep (SWS).

263 the unmarked reward zone to patterns during slow-wave sleep (SWS).

264 motor memory after targeted reactivation in slow-wave sleep (SWS).

265 ial role in the induction and maintenance of slow-wave sleep (SWS).

266 aking or rapid eye movement (REM) sleep from slow-wave sleep (SWS).

267 d 5-HT declined progressively from waking to slow-wave-sleep (SWS) and then to rapid-eye-movement (RE

268 (PZ(Vgat)) neurons in behaving mice produces slow-wave-sleep (SWS), even in the absence of sleep defi

269 During slow-wave sleep, temporal coordination of hippocampal sh

270 ere positively correlated with the amount of slow-wave sleep that patients obtained between training

271 We propose that during slow-wave sleep the tight functional coupling between GA

272 After a period of subsequent slow-wave sleep, the model developed the ability to reca

273 In fact, during slow-wave sleep, the patterns of CA1 pyramidal cell ense

274 and spontaneous ventilation were observed in slow wave sleep time (45 min vs 28 min), rapid eye movem

275 reased nighttime sleep latency and increased slow-wave sleep time in cocaine-dependent participants.

276 ttern A human IgGs on rapid eye movement and slow-wave sleep time parameters in the inactive phase an

277 s bilaterally placed in the preoptic region, slow-wave sleep time was significantly decreased, but RE

278 Comparison of slow-wave sleep time, total sleep time, and sleep latenc

279 Half of the sounds were played during slow-wave sleep to reactivate corresponding memories of

280 hypothesis is that neurons fire less during slow-wave sleep to recover from the "fatigue" accrued du

281 o sleep states, rapid eye movement sleep and slow-wave sleep, to offline memory processing.

282 reactivation of these representations during slow-wave sleep transforms episodic representations into

283 nd suppressed REM sleep time while increased slow wave sleep typifies the inactive phase, findings th

284 ep in an unusual manner, with unihemispheric slow wave sleep (USWS) and suppressed REM sleep, it is u

285 en RR neurons and SPW-R events in subsequent slow wave sleep was diminished.

286 Slow-wave sleep was associated with delta-band sequences

287 Hence, new vocabulary played during slow-wave sleep was stored and influenced decision-makin

288 t bursting occurs only during states such as slow-wave sleep, when little or no information is relaye

289 the first half of the night is dominated by slow-wave sleep, whereas during the second half, rapid e

290 by behavioural state, and was maximal during slow-wave sleep, which may explain the propensity for ne

291 d sounds were re-presented during subsequent slow-wave sleep while participants underwent functional

292 e postreserpine days, sleep was dominated by slow-wave sleep with fast intrusions and reduced hierarc

293 WS), as seen in all terrestrial mammals, and slow-wave sleep with interhemispheric electroencephalogr

294 During subsequent slow-wave sleep within an afternoon nap, we presented ha

295 Slow-wave sleep would thus begin with spindle oscillatio