ライフサイエンスコーパス: small intestinal mucosa

1 mediated inhibition of TLR4 signaling in the small intestinal mucosa.

2 elial or crypt cell regions of healthy human small intestinal mucosa.

3 in human intestinal Caco-2 cells and rodent small intestinal mucosa.

4 s in splenocytes and epithelial cells of the small intestinal mucosa.

5 orizin hydrolase (LPH) was purified from rat small intestinal mucosa.

6 the fractalkine receptor CX3CR1 in the human small intestinal mucosa.

7 hesion within the healthy and diseased human small intestinal mucosa.

8 ance of normal rates of protein synthesis in small intestinal mucosa.

9 ions are composed of heterotopic stomach and small intestinal mucosa.

10 d interleukin 12 (IL12) were investigated in small intestinal mucosa.

11 me closely mirrored the transcriptome of the small intestinal mucosa.

12 ogically mediated inflammatory damage to the small-intestinal mucosa.

13 compared these data to RNA-Seq from both the small intestinal mucosa and colonic mucosa of healthy co

14 d by the difficulty in directly sampling the small intestinal mucosa and microbial community (microbi

15 impact on the structure and function of the small intestinal mucosa and suggest signaling through th

16 orption, morphology and DNA proliferation of small intestinal mucosa, and gastrointestinal transit we

17 homeobox genes are expressed in human adult small intestinal mucosa, and some are found predominantl

18 tandard for successful treatment is a healed small intestinal mucosa, and therefore, the outcome meas

19 n the luminal aspect of the bacteria-exposed small-intestinal mucosa, and was followed by a decrease

20 The major fuels for the small intestinal mucosa are amino acids (glutamine, glut

21 Growth inhibition of the small intestinal mucosa by fasting in mice was associate

22 is a syndrome characterized by damage of the small intestinal mucosa caused by the gliadin fraction o

23 is a syndrome characterized by damage of the small intestinal mucosa caused by the gluten fraction of

24 The small intestinal mucosa constitutes a physical barrier s

25 tissue, the decline in the synthesis rate of small intestinal mucosa during insulin deprivation may b

26 in (NT) stimulates Cl(-) secretion in animal small intestinal mucosa in vitro.

27 29b-reduced endogenous miR-29b levels in the small intestinal mucosa increases cyclin-dependent kinas

28 Morphological development of the small intestinal mucosa involves the stepwise remodeling

29 The human small intestinal mucosa is an unlikely reservoir for thi

30 studies reveal that growth inhibition of the small intestinal mucosa is associated with increased exp

31 earch into the structure and function of the small intestinal mucosa is becoming increasingly focused

32 Although the small intestinal mucosa is designed to transport large q

33 The small intestinal mucosa is highly specialized for termin

34 of the molecular and cellular biology of the small-intestinal mucosa is providing insights into the r

35 imin-alpha) restored the ability to colonize small intestinal mucosa like the parent strain.

36 tered polymer film designed to adhere to the small intestinal mucosa, maximizing contact between ther

37 Extracts of small intestinal mucosa obtained from healthy cows have

38 of DQ2-mediated antigen presentation in the small intestinal mucosa of Celiac Sprue patients therefo

39 G2 can be activated by dietary gluten in the small intestinal mucosa of celiac sprue patients, our fi

40 formed in vitro AMG uptake measurements into small intestinal mucosa of mice and human.

41 n (GFP)-tagged gammadelta T cells within the small intestinal mucosa of mice infected with DsRed-labe

42 the gammadelta TCR are more abundant in the small intestinal mucosa of patients with celiac disease

43 (P = 0.38) from the gastrointestinal lumen, small intestinal mucosa, or lymph.

44 messenger RNA targeting relationships in the small intestinal mucosa provides insight into the molecu

45 Vagal afferents innervating the small intestinal mucosa regulate feeding, gastrointestin

46 M d-glucose, hSGLT1-mediated AMG uptake into small intestinal mucosa was decreased by 40% to 50%.

47 an villous height/crypt depth (Vh/Cd) in the small-intestinal mucosa was significantly lower and the

48 on's terminals and their distribution in the small intestinal mucosa were examined by quantitatively

49 , dramatic, and reversible remodeling of the small intestinal mucosa with significant villus shorteni