ライフサイエンスコーパス: small-vessel vasculitis

1 arthritis, systemic lupus erythematosus, and small vessel vasculitis.

2 ic antibodies (ANCAs) can result in systemic small vessel vasculitis.

3 antineutrophil cytoplasm antibody-associated small vessel vasculitis.

4 he PR3 ANCA immune response in patients with small vessel vasculitis.

5 Strauss Syndrome is an eosinophil-associated small vessel vasculitis.

6 ealing, and open lung biopsy revealed active small vessel vasculitis.

7 ischemic infarctions, presumed secondary to small vessel vasculitis.

8 (ANCA) causes vascular injury that leads to small-vessel vasculitis.

9 A large number of entities can mimic small-vessel vasculitis.

10 ophil cytoplasmic antibody (ANCA)-associated small-vessel vasculitis.

11 ten than other patients with ANCA-associated small-vessel vasculitis.

12 th polyarteritis nodosa and one patient with small-vessel vasculitis.

13 patients with clinical manifestations due to small-vessel vasculitis.

14 rysms of renal and hepatic arteries, whereas small vessel vasculitis affected the skin and glomeruli,

15 NOTCH activation did not occur in small-vessel vasculitis affecting branches of the vasa v

16 ophil cytoplasmic antibody (ANCA)-associated small-vessel vasculitis (ANCA-SVV) and to gather evidenc

17 nase 3 are detected in sera of patients with small vessel vasculitis and participate in the pathogene

18 ic or soft plaques of the coronary arteries, small vessel vasculitis and small aneurysm.

19 th perinatal-onset of neutrophilic cutaneous small vessel vasculitis and systemic inflammation.

20 oup of disorders involving severe, systemic, small-vessel vasculitis and are characterized by the dev

21 (EGPA), that share features of pauci-immune small-vessel vasculitis and the positivity of ANCA targe

22 The BVAS is a validated tool for small-vessel vasculitis and used to assess the level of

23 een consistently detected in ANCA-associated small-vessel vasculitis, and this association prompted u

24 angiitis (Churg-Strauss, EGPA) is a systemic small-vessel vasculitis associated with asthma and eosin

25 neutrophil cytoplasm autoantibody-associated small vessel vasculitis based on antineutrophil cytoplas

26 strointestinal disease, febrile attacks, and small-vessel vasculitis characteristic of Behcet disease

27 entic glomerulonephritis (NCGN) and systemic small vessel vasculitis in humans.

28 i-neutrophil cytoplasmic antibody-associated small-vessel vasculitis, large-vessel vasculitis and Beh

29 vascular lesions associated with autoimmune small-vessel vasculitis may arise from activation of cir

30 th polyangiitis (GPA) is a potentially fatal small vessel vasculitis of unknown etiology, characteriz

31 e IgG (ANCA IgG), derived from patients with small-vessel vasculitis, on neutrophil adhesion.

32 due to secondary ischemia from BD-associated small-vessel vasculitis, potentially exacerbated by CGRP

33 omerular lesions with crescents, mimicking a small vessel vasculitis such as ANCA-associated GN, are

34 d crescentic glomerulonephritis and systemic small vessel vasculitis, such as microscopic polyangiiti

35 microdissected glomeruli from patients with small vessel vasculitis (SVV) had markedly higher levels

36 l cytoplasmic autoantibody (ANCA)-associated small-vessel vasculitis (SVV) and systemic lupus erythem

37 Microscopic polyangiitis is an autoimmune small-vessel vasculitis that often manifests as focal an

38 three patients with polyarteritis nodosa or small-vessel vasculitis were homozygous for the p.Gly47A

39 granulomatosis with polyangiitis (eGPA) is a small-vessel vasculitis where 40% of patients present wi