ライフサイエンスコーパス: snoring

1 ratio = 1.43, 95% CI: 1.33, 1.5 for regular snoring).

2 e back were directly associated with regular snoring.

3 ren with mild OSA from children with primary snoring.

4 , long sleep duration (>=9 hours/night), and snoring.

5 greater odds of mild OSA rather than primary snoring.

6 sleep traits and obesity and are largest for snoring.

7 , sleep disturbance, daytime sleepiness, and snoring.

8 eatment options were found to reduce primary snoring.

9 al congestion is a risk factor for apnea and snoring.

10 the basis of either sleep apnea or habitual snoring.

11 ren had obstructive apnea and 21 had primary snoring.

12 ess and standardized questionnaires assessed snoring.

13 nd 23% of the variability in self-reports of snoring.

14 l index or self-reported history of habitual snoring.

15 ng and 1.55 (95% CI: 1.42, 1.70) for regular snoring.

16 rted in 810 children (6.8%), and nonhabitual snoring (1-2 nights per week) was reported in 4058 child

17 Overall, 9.7% of patients endorsed frequent snoring, 12.2% sleep problems, and 34.4% insufficient sl

18 ted global sleep disturbance, 30.6% endorsed snoring, 24.3% sleep <6 hrs/night, and over half screene

19 per week; OR = 2.06 (95% CI: 1.01, 4.31) for snoring 3 or more times per week), particularly for term

20 Of those, habitual snoring ( 3 nights per week) was reported in 810 childre

21 sus 75 +/- 16 kg) and prevalence of habitual snoring (78% versus 28%).

22 reports distinguished children with primary snoring (AHI <1; 311 patients [67.8%]) from children wit

23 obstructive sleep apnea, and also that even snoring alone affects neurocognitive function.

24 Mild OSA vs snoring alone did not identify a clinical group of child

25 ere 1.29 (95% CI: 1.22, 1.37) for occasional snoring and 1.55 (95% CI: 1.42, 1.70) for regular snorin

26 scriminating between 13 subjects with no EDS/snoring and 21 patients with EDS and snoring were identi

27 hful waiting or adenotonsillectomy for mSDB (snoring and an obstructive apnea-hypopnea index <3) betw

28 lling 459 children aged 3 to 12.9 years with snoring and an obstructive apnea-hypopnea index (AHI) le

29 cant genetic correlation between obesity and snoring and between obesity and excessive daytime sleepi

30 Patients with mild snoring and body mass index lower than 26 are unlikely t

31 It is unknown whether children with primary snoring and children with mild obstructive sleep apnea (

32 association between parent-reported habitual snoring and cognitive performance was substantially atte

33 The association between habitual snoring and cognitive performance was substantially atte

34 Associations between habitual snoring and cognitive performance were assessed using th

35 covariates, the positive association between snoring and CVD was attenuated but remained statisticall

36 om these data that self-reported symptoms of snoring and daytime sleepiness in older men have a genet

37 higher BMI and, conversely, of higher BMI on snoring and daytime sleepiness.

38 he variation and covariation of obesity with snoring and daytime sleepiness.

39 etes showed a consistent association between snoring and diabetes within the categories of these vari

40 athing (SDB) in children is characterized by snoring and difficulty breathing during sleep.

41 opulation were similar to those for frequent snoring and included: male sex, higher Body Mass Index,

42 e identified an association between habitual snoring and lower cognitive performance in children.

43 the PATS randomized clinical trial, primary snoring and mild OSA were difficult to distinguish witho

44 reports did not distinguish between primary snoring and mild OSA.

45 ial where children aged 3 to 12.9 years with snoring and mild oSDB (Obstructive Apnea-Hypopnea Index

46 A total of 1,010 snoring and nonsnoring children ages 5-7 years were pros

47 AUP) has been used as a treatment option for snoring and obstructive sleep apnea for almost three dec

48 Position therapy plays a role in treating snoring and obstructive sleep apnea syndrome (OSAS).

49 nce interval (CI): 1.16, 1.27 for occasional snoring and odds ratio = 1.43, 95% CI: 1.33, 1.5 for reg

50 We conclude that both snoring and RDI are independently associated with excess

51 The effects of snoring and RDI on sleepiness were little affected by ad

52 To examine the association between snoring and risk of developing type II diabetes mellitus

53 The authors examined the association between snoring and risk of hypertension in a cohort of 73,231 U

54 o younger children and children with primary snoring and severe OSA.

55 ty (U3P) has been advocated for treatment of snoring and sleep apnea/hypopnea syndrome (SAHS), but of

56 napping, ease of getting up in the morning, snoring and sleep apnoea) among 450,000 participants fro

57 gh for the most part the genetic variance in snoring and sleepiness was nonoverlapping with the genet

58 subjects may disavow knowledge of their own snoring and suggest that future studies consider the "do

59 tion was very strong in persons who reported snoring and weak in those who did not.

60 I motoneurons is common during sleep causing snoring and, in serious cases, airway obstruction that i

61 sed surrogate information to define SDB (eg, snoring) and were based on small clinic populations, or

62 of sleep per day, low insomnia symptoms, no snoring, and no frequent daytime dysfunction.

63 g and maintaining sleep, unrefreshing sleep, snoring, and the presence of physician-diagnosed sleep d

64 time below 90% oxygen saturation; history of snoring; and presence of hypertension, defined as restin

65 eating soil (ARR, 1.45; 95% CI, 1.13-1.85), snoring (ARR, 1.50; 95% CI, 1.27-1.76), and being landle

66 the "don't know" response to questions about snoring as a response of potential interest.

67 tems addressed the presence and frequency of snoring behavior, waketime sleepiness or fatigue, and hi

68 lly sweetened beverages), physical activity, snoring, binge eating, and earlier puberty were positive

69 p-disordered breathing (SDB) (i.e., habitual snoring but not frequent obstructive breathing events) r

70 absence of obstructive sleep apnea (primary snoring), but few studies have examined the efficacy of

71 Parent-reported snoring categorized as none, nonhabitual (<3 nights/week

72 useful clinical adjunct to the evaluation of snoring children, with more accurate identification of t

73 inical evaluation and referral of habitually snoring children.

74 try may contribute to diagnostic accuracy in snoring children.

75 en and in a subsequent post hoc sample of 94 snoring children.

76 sighs, severe dysphonia, severe dysarthria, snoring, cold hands and feet, pathological laughter and

77 osed on the basis of characteristic history (snoring, daytime sleepiness) and physical examination (i

78 rity for menopausal women with complaints of snoring, daytime sleepiness, or unsatisfactory sleep.

79 sleep apnea or >/= 2 hallmarks of OSA: loud snoring, daytime sleepiness, witnessed apnea, and hypert

80 based on the ABCD dataset, the prevalence of snoring declined over time; snoring in adolescents was a

81 ; additional covariates: vasomotor symptoms, snoring, depression).

82 noring index (SI, expressed as the number of snoring events per hour measured on an acoustic analytic

83 in, discomfort to breathe, and cough or loud snoring), excessive daytime sleepiness, and reduced vigo

84 roarousal index (P = 0.008); and symptoms of snoring, fatigue, and sleepiness (P < 0.001).

85 Snoring frequency was collected from parents.

86 core was also seen across five categories of snoring frequency, from 6.4 (4.2) in current nonsnorers

87 Measurements and Main Results: The habitual snoring group had significantly smaller airway volumes t

88 eener assessing infant bed sharing, frequent snoring (>=3 nights/week), perceived sleep problems, ins

89 Snoring history was obtained via a self-completion quest

90 he prevalence of snoring declined over time; snoring in adolescents was associated with problem behav

91 Parent-reported habitual snoring in children that occurs 3 or more nights per wee

92 Results suggest that snoring in early gestation may be a significant risk fac

93 Compared with nonsnorers, snoring in early gestation was independently associated

94 in the generation of apneas, hypopneas, and snoring in healthy subjects.

95 ments for snoring or evaluated the effect of snoring in sleeping partners.

96 Whether snoring increases risk of CVD remains unclear; most prev

97 Whether snoring increases the risk of hypertension remains uncle

98 cale ranging from 0 to 10) and the objective snoring index (SI, expressed as the number of snoring ev

99 Snoring is also associated with excess sleepiness, altho

100 These data suggested that snoring is associated with a modest but significantly in

101 Habitual snoring is associated with cognitive and behavioral prob

102 Snoring is central to sleep-disordered breathing (SDB),

103 Snoring is common in sleep apnea patients but is not use

104 These results suggest that snoring is independently associated with elevated risk o

105 ndicator of obstructive sleep apnea, whereas snoring is not very specific.

106 an independent effect of snoring or whether snoring is simply a marker for obstructive sleep apnea.

107 hool-aged children and children with primary snoring limited the generalizability of findings.

108 gical mechanism underlying this association, snoring may help clinicians identify individuals at high

109 These data suggest that snoring may increase risk of hypertension in women, inde

110 Symptoms include loud snoring, nocturnal awakening, and daytime sleepiness.

111 ning 103 subjects (14 nonsnoring non-EDS, 21 snoring non-EDS, 68 snoring with EDS).

112 clampsia (OR = 1.72 (95% CI: 1.09, 2.73) for snoring once or twice per week; OR = 2.06 (95% CI: 1.01,

113 have examined the efficacy of treatments for snoring or evaluated the effect of snoring in sleeping p

114 ether this reflects an independent effect of snoring or whether snoring is simply a marker for obstru

115 isted when adjusting for vasomotor symptoms, snoring, or depression.

116 m the Pediatric Adenotonsillectomy Trial for Snoring (PATS) study, a multicenter, single-blind, rando

117 m the Pediatric Adenotonsillectomy Trial for Snoring (PATS), a multicenter, randomized, single-blinde

118 f the Pediatric Adenotonsillectomy Trial for Snoring (PATS), a randomized clinical trial where childr

119 Snoring patterns were ascertained by questionnaire.

120 e waves in the UA, similar to those found in snoring, produce reflex inhibition of inspiratory motor

121 h OSAS, compared to 26 children with primary snoring (PS).

122 ildren with OSA and 19 children with primary snoring (PS).

123 slightly attenuated the risk (for occasional snoring, RR = 1.41 (95% CI: 1.22, 1.63); for regular sno

124 RR = 1.41 (95% CI: 1.22, 1.63); for regular snoring, RR = 2.03 (95% CI: 1.71, 2.40); p for trend < 0

125 ary outcome measures included the subjective snoring severity (SS, measured on a visual analogue scal

126 Snoring & sleep apnea (27.8%), and III.

127 ose with anxiety had higher odds of being in Snoring & sleep apnea cluster and Poor sleep quality and

128 d HIV duration were associated with being in Snoring & sleep apnea cluster, while age (OR = 1.027, 95

129 pregnancy was related to poor sleep quality, snoring, sleep aids, and short sleep.

130 ng and support the large-scale evaluation of snoring sound characters as markers of surveillance and

131 Our findings suggest that snoring sound detection may be helpful in determining ob

132 p, CCA-IMT was significantly correlated with snoring sound energies of 0-20 Hz (r = 0.608, p = 0.036)

133 obstruction detected by either DISE or mean snoring sound intensity (301-850 Hz), and AHI could sign

134 ignificantly, inversely correlated with mean snoring sound intensity (301-850 Hz; OR, 0.84, 95% CI 0.

135 ficantly, positively associated with maximal snoring sound intensity (40-300 Hz; odds ratio [OR], 1.2

136 study recruited 36 OSAHS patients for 6-hour snoring sound recordings during in-lab full-night polyso

137 Snoring sounds generated by different vibrators of the u

138 Good-quality signals of full-night snoring sounds in an ordinary sleep condition obtained f

139 artery intima-media thickness (CCA-IMT) with snoring sounds in OSA patients.

140 a head-positioning pillow (HPP) could reduce snoring sounds in patients with mild-to-moderate positio

141 Our findings suggest that underlying snoring sounds may cause carotid wall thickening and sup

142 dy aimed to investigate associations between snoring sounds, obstruction sites, and surgical response

143 Patients with simple snoring (SS) often complain of poor sleep quality despit

144 hysical activity, other sleep disorders, and snoring status.

145 ered breathing (SDB), which includes primary snoring through to obstructive sleep apnea syndrome (OSA

146 ith no significant change in the relation of snoring to ESS score after adjustment for RDI in multiva

147 The relation of snoring to sleepiness was seen at all levels of RDI, wit

148 To further explore the relation of snoring to sleepiness, we conducted a cross-sectional co

149 ir sleeping partner who underwent 4 weeks of snoring treatment was conducted at an academic medical c

150 Ten symptomatic patients (snoring, upper airway resistance syndrome [UARS], or OSA

151 ciated with risk of diabetes (for occasional snoring vs. nonsnoring, relative risk (RR) = 1.48 (95% c

152 ence interval (CI): 1.29, 1.70); for regular snoring vs. nonsnoring, RR = 2.25 (95% CI: 1.91, 2.66);

153 Frequency of snoring was assessed using mailed questionnaires at base

154 , waist circumference, and other covariates, snoring was associated with a significantly higher preva

155 alyses adjusted for age and body mass index, snoring was associated with risk of diabetes (for occasi

156 In addition, snoring was associated with significantly higher systoli

157 g partners who reported that their partner's snoring was either very much improved or much improved (

158 A statistically significant association of snoring was identified with all CBCL scales.

159 Snoring was not associated with any of the NIH-TB scores

160 ggregation parameter, but parental report of snoring was positively associated with TRAP aggregation

161 uestionnaire (gender, obesity, age, and loud snoring), was developed and subsequently validated, with

162 ytime napping (PLEKHM1, ANKRD12 and ZBTB21), snoring (WDR59) and sleep apnoea (13 genes).

163 is best for individual patients with primary snoring, weighing convenience, adverse effects, and cost

164 no EDS/snoring and 21 patients with EDS and snoring were identified by receiver operator curve analy

165 was accompanied by a virtual elimination of snoring, which fell from 16.5 +/- 3.0% of total sleep ti

166 enter Pediatric Adenotonsillectomy Trial for Snoring, which included children aged 3 to 12 years rand

167 ore effective than CAPT for treating primary snoring, while both treatment options were found to redu

168 d 44% of the cohort answered questions about snoring with a "don't know" response.

169 this cohort study analyzing associations of snoring with cognitive test scores and problem behaviors

170 4 nonsnoring non-EDS, 21 snoring non-EDS, 68 snoring with EDS).

171 The largest-magnitude association was of snoring with the CBCL Total Problems scale among adolesc

172 Longitudinal associations of snoring with these assessment measures were analyzed usi

173 otonsillectomy in children who have habitual snoring without frequent obstructive breathing events (m

174 vere sleep apnea, and self-reported habitual snoring without PDSA (HS), a surrogate for mild sleep ap

175 Features include snoring, witnessed apnoeas, and sleepiness.