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1 seeking out salt or consuming salt following sodium depletion.
2 are typically attractive, particularly after sodium depletion.
3 r FoxP2 and become c-Fos activated following sodium depletion.
4  on the consumption of salt itself following sodium depletion.
5 downstream targets that are activated during sodium depletion.
6 ange mRNA abundance was increased by dietary sodium depletion.
7 by potassium depletion but only minimally by sodium depletion.
8 stimuli transform into appetitive ones under sodium depletion.
9 ve similar but lower efficacies and increase sodium depletion.
10                                       During sodium depletion, 7-NI (partially) and -NAME (completely
11                                       During sodium depletion, 7-NI decreased RIF cGMP, but the reduc
12     Except in instances of severe, prolonged sodium depletion, a sodium-specific appetite has not bee
13 ation after furosemide diuresis and 22 hr of sodium depletion affects the amount of water or 0.3 M Na
14                                       During sodium depletion and angiotensin II coinfusion, (Pyr(1))
15 re indicate that a strong natural motivator, sodium depletion and associated salt appetite, also lead
16                                              Sodium depletion and captopril led to activation and dif
17 nal abnormalities are also common, including sodium depletion and hypovolemia, hypophosphatemia and h
18 sults indicate that repeated experience with sodium depletion and ingestion affects both behavioral a
19 ant (albeit reduced) salt appetite following sodium depletion, and a normal conditioned taste aversio
20 anscription level in SONs and PVNs following sodium depletion, and as a consequence of diurnal rhythm
21  neurons, which are selectively activated by sodium depletion, and by the A2 noradrenergic neurons, w
22                                    Following sodium depletion, and in the absence of either sucrose o
23 ent as WT mice, even during profound dietary sodium depletion, as a result of the upregulation of tra
24                                       During sodium depletion, both PD and -NAME caused a similar dec
25  DNA and protein content were not altered by sodium depletion but were significantly lowered by losar
26 ading, volume contraction, and potassium and sodium depletion completely corrects alkalosis by a rena
27 that provoke a robust sodium appetite (e.g., sodium depletion, deoxycorticosterone acetate) decrease
28                              Experience with sodium depletion enhances its ingestion and may have a d
29 cleus accumbens of rats that had experienced sodium depletions had significantly more dendritic branc
30 ced by various experimental procedures (e.g. sodium depletion; hypovolaemia).
31                   Animals receiving repeated sodium depletions increased sodium ingestion across init
32 tion of the renin- angiotensin system during sodium depletion increases renal nitric oxide production
33  food intake, decreases salt ingestion after sodium depletion, induces pica, and produces robust cond
34                              Because dietary sodium depletion markedly increases ouabain-insensitive
35 he upregulation of renal AT1 mRNA induced by sodium depletion occurs conjointly with an elevation of
36 ance index (P<0.001 for all) irrespective of sodium depletion or angiotensin II (0.5 ng/kg per minute
37  of renin-angiotensin system activation with sodium depletion or angiotensin II coinfusion.
38 s in sodium appetite induced by either acute sodium depletion or mineralocorticoid administration but
39 T1 receptor binding in the rats subjected to sodium depletion plus losartan did not differ from that
40                                        Also, sodium depletion produced increased heart rate and reduc
41                                   Thus, mild sodium depletion produces hypohedonia and cardiovascular
42                  Potassium depletion but not sodium depletion resulted in an increase in KCC1 protein
43                                              Sodium depletion significantly increased RIF cGMP.
44       Yet, upon first reencounter in a novel sodium-depletion state to promote mesocorticolimbic reac
45                                              Sodium depletion substantially increased ouabain-insensi
46 t intakes after their second experience with sodium depletion than their first, and microstructural a
47 tes transmit information related to systemic sodium depletion to forebrain sites and are part of the
48                                Here, we used sodium depletion to reversibly manipulate the appetitive
49               We have recently reported that sodium depletion up-regulates H,K-ATPase alpha-subunit m
50  Dopamine responses to the NaCl US following sodium depletion updated independent of prior experience
51                    In addition, a history of sodium depletions was found to have cross-sensitization
52 ctivation of the renin-angiotensin system by sodium depletion will enhance the growth of cysts in ani
53                           Following 48 hr of sodium depletion with a diuretic (furosemide) and a sodi