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1 es after intravenous infusions of hypertonic sodium lactate.
2 racellular solutions that contained 20-80 mm sodium lactate.
3 lular TG storage was induced in the cells by sodium lactate.
4      Continuous intraventricular infusion of sodium lactate (1.0 or 10.0 microM/h) or vehicle was ini
5 ptake and this inhibition was potentiated by sodium lactate (25 mM).
6 erents to myocardial ischaemia, lactic acid, sodium lactate, acidic phosphate buffer and hypercapnia
7                      In CD4+ T helper cells, sodium lactate also induces a switch towards the Th17 su
8                                Extracellular sodium lactate and lactic acid inhibit the motility of C
9 xytocin-induced contractility in response to sodium lactate and other weak acids (1-20 mM) were studi
10                     A detailed comparison of sodium, lactate and cortisol from saliva is reported, de
11 e metabolites: 1-(13)C-pyruvic acid, 1-(13)C-sodium lactate, and 1-(13)C-acetic acid.
12 ces exposed to 0 mM glucose were given 20 mM sodium lactate as a metabolic substrate.
13  density by >80% (0.95-1.76 muA/cm(2)) using sodium lactate as the sole carbon source.
14               Subcutaneous administration of sodium lactate but not glucose to mice bearing transplan
15          In this study, we confirm that 2 mM sodium lactate, but not sodium pyruvate, increases respi
16  the effects of NaCl, KCl, CaCl(2), MgCl(2), sodium lactate, calcium lactate, and magnesium lactate o
17 f 10-100 microg ml-1 of lactic acid, but not sodium lactate, concentration-dependently stimulated 18
18 enous administration of the anxiogenic agent sodium lactate elicited robust increases in blood pressu
19 ics were significantly improved in the molar sodium lactate group compared with the control group (hi
20  between the groups (10/12, 83% in the molar sodium lactate group vs nine of 12, 75% in the control g
21 roup, n = 12) or molar sodium lactate (molar sodium lactate group, n = 12) at a rate of 5 mL/kg/hr du
22 nt activities in pulmonary C fibres, whereas sodium lactate had no effect.
23 y-like states and a similar vulnerability to sodium lactate-induced cardioexcitatory responses.
24 ole of plasma cortisol levels in determining sodium lactate-induced panic by reporting psychological,
25  the hypothalamus as putative substrates for sodium lactate-induced panic-like responses in vulnerabl
26                                              Sodium lactate induces flashbacks in persons with PTSD w
27                 This study addressed whether sodium lactate induces flashbacks or other intrusive PTS
28 les) completed protocols of: (1) intravenous sodium lactate infusion (exogenous hyperlactataemia); an
29  Asn administered either alone or prior to a sodium lactate infusion had no effect on the primed rat'
30                                              Sodium lactate infusion has induced flashbacks accompani
31 roduced during 'priming' as well as during a sodium lactate infusion into 'primed' rats.
32 hysiologically distinct states of exogenous (sodium lactate infusion) and endogenous (exercise-induce
33 s: a 20-minute baseline, 20-minute 0.5-mol/L sodium lactate infusion, and 15-minute postinfusion peri
34 r experience panic attacks after intravenous sodium lactate infusions by an as yet unexplained mechan
35 has no effect on anxiogenic responses due to sodium lactate infusions in 'primed' rats
36                                  Intravenous sodium lactate infusions or the noradrenergic agent yohi
37 acid decarboxylase inhibitor L-allylglycine, sodium lactate infusions or yohimbine elicits panic-like
38 is 'priming' effect induces a sensitivity to sodium lactate infusions that results in a panic-like re
39 ial interaction (SI) test, after intravenous sodium lactate infusions, a phenomenon similar to patien
40 ther show both in vitro and in vivo that the sodium lactate-mediated inhibition of CD4+ T cell motili
41 rmal saline (control group, n = 12) or molar sodium lactate (molar sodium lactate group, n = 12) at a
42 dy investigates the effect of treatment with sodium lactate (NaLac) on the changes in brain energy st
43 ing panic-prone rats with mGluR2 PAM blocked sodium lactate (NaLac)-induced panic responses and norma
44 on was MCT-1 dependent and reproducible with sodium lactate or formic acid.
45 between saline or Ringer's lactate (compound sodium lactate or Hartmann's solution).
46 ice were given intraperitoneal injections of sodium lactate or small interfering RNA against Gpr81.
47                                        Molar sodium lactate reduced brain injury, with a higher propo
48 late transporters (MCT) or replacing LA with sodium lactate revealed that LA effects are MCT-1- and p
49                                        Molar sodium lactate significantly prevented cardiac arrest-in
50 y was tested every 24 h by administering 50% sodium lactate to achieve perfusate concentrations of 10
51 LDHA [lactate dehydrogenase A] inhibitor) or sodium lactate to modulate histone lactylation.
52  animals (11 +/- 1 mL/kg) than that in molar sodium lactate-treated rabbits (1 +/- 3 mL/kg; p < 0.01)
53                                        Molar sodium lactate was effective in limiting the severity of
54                                              Sodium lactate was substituted iso-osmotically for sodiu
55  and cortisol responses to infusion of 0.5 M sodium lactate were compared among seven subjects with P
56                            Administration of sodium lactate, which bypasses the block in the glycolyt