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1 wn direct nitration of a benzimidazole using sodium nitrite.
2 ant extracts as an alternative to commercial sodium nitrite.
3 s were assessed for intentional exposures to sodium nitrite.
4 n center reports of intentional exposures to sodium nitrite.
5 ized in 4 steps from methyl vinyl ketone and sodium nitrite.
6 oked sausages produced with reduced level of sodium nitrite.
7 ity phase 1 dose-escalation trial of topical sodium nitrite.
8 s was completely inhibited by a low level of sodium nitrite.
9 rite and reacts with hemoglobin similarly to sodium nitrite.
10 s highly susceptible to killing by acidified sodium nitrite.
11 dium nitrite and 0.150 uL/g TP; T4: 50 mg of sodium nitrite, 0.075 uL/g TP and 0.075 uL/g PM; T5: 50
12 randomized to receive intracoronary (10 mL) sodium nitrite (1.8 mumol) or NaCl (placebo) before ball
15 Active treatment was divided as low dose (sodium nitrite, 3%, with citric acid, 4.5%, creams appli
16 of reperfusion in the absence or presence of sodium nitrite (30 nmol) administered topically 1 min be
17 ry bypass graft patients treated with either sodium nitrite (30-min infusion of 10 mumol/min) or vehi
19 system by mixing a solution of KY jelly and sodium nitrite (5% weight/volume), with a solution of KY
20 %, creams applied twice daily), middle dose (sodium nitrite, 6%, with citric acid, 9%, creams applied
21 cebo applied in the morning), and high dose (sodium nitrite, 6%, with citric acid, 9%, creams applied
28 rein, is investigated the effects of topical sodium nitrite administration in a rat model of renal is
29 were given inhaled NO gas (20 ppm), inhaled sodium nitrite aerosol (0.87 mol/L), or an intravascular
31 ts of decedents where the cause of death was sodium nitrite and (2) poison center reports of intentio
33 e; T2: 50 mg of sodium nitrite; T3: 50 mg of sodium nitrite and 0.150 uL/g TP; T4: 50 mg of sodium ni
35 th metabolic syndrome after 12 weeks of oral sodium nitrite and nitrate treatment (IND#115926) displa
36 n spectra of wastewater solutions containing sodium nitrite and nitrate were measured in the concentr
37 unctionality of TPS/PBAT (50/50) blends with sodium nitrite and sodium erythorbate (1-5%) via blown f
40 es, followed by their alpha-nitrosation with sodium nitrite and subsequent base mediated intramolecul
41 ch as potassium ferricyanide, hydroxylamine, sodium nitrite, and 4-dimethylaminophenol (DMAP), added
45 of most strains exposed to either acidified sodium nitrite (ASN) or hydrogen peroxide (H(2)O(2)) was
47 ro than other clinical isolates to acidified sodium nitrite (ASN), a generator of reactive nitrogen a
48 at higher rates in the presence of acidified sodium nitrite (ASN), a model system used to generate ni
50 sis of electrochemical sensing platforms for sodium nitrite, B-nicotinamide adenine dinucleotide (NAD
54 f-hospital cardiac arrest, administration of sodium nitrite, compared with placebo, did not significa
56 oncentration, sulfanilic acid concentration, sodium nitrite concentration, reaction time, sulfanilic
59 as treated with a different concentration of sodium nitrite cream (cohort 1: 0.5%, cohort 2: 1.0%, co
61 n 2 New York medical examiner jurisdictions, sodium nitrite deaths increased yearly, and the medical
63 scavenger carboxy PTIO completely abolished sodium nitrite-dependent ischemic tissue blood flow and
64 These findings do not support the use of sodium nitrite during resuscitation from out-of-hospital
67 TICIPANTS: This retrospective case series of sodium nitrite exposures and deaths was conducted in 2 u
69 Overall, 205 patients (41%) in the 45 mg of sodium nitrite group and 212 patients (43%) in the 60 mg
70 arge for 66 patients (13.2%) in the 45 mg of sodium nitrite group and 72 patients (14.5%) in the 60 m
71 group and 212 patients (43%) in the 60 mg of sodium nitrite group compared with 218 patients (44%) in
72 roup and 72 patients (14.5%) in the 60 mg of sodium nitrite group compared with 74 patients (14.9%) i
74 lly converted to 4 in 45% yield by employing sodium nitrite in aqueous tartaric acid at 0-5 degrees C
75 olerability, and pharmacokinetics of topical sodium nitrite in patients with sickle cell disease and
76 catalyse nitric oxide generation from benign sodium nitrite in the presence of modest electric fields
80 ver; combined infusion of acetazolamide with sodium nitrite inhalation did not further increase exhal
83 ntervention, including inhaled nitric oxide, sodium nitrite, L-arginine, phosphodiesterase-5 inhibito
84 45 mg of sodium nitrite (n = 500), 60 mg of sodium nitrite (n = 498), or placebo (n = 499), which wa
85 were randomized (1:1:1) to receive 45 mg of sodium nitrite (n = 500), 60 mg of sodium nitrite (n = 4
86 ble myocardial ischemia, saline and low-dose sodium nitrite (NaNO(2)) (1.5 mumol/min for 20 min) were
88 only marginally dilating resistance vessels, sodium nitrite (NaNO2) infusion would increase cardiac o
92 ects of local and systemic administration of sodium nitrite on the radial artery (a muscular conduit
97 A subset of HFpEF subjects (n = 52) received sodium nitrite or placebo therapy in a 1:1 double-blind,
99 2 wk, then supplemented with sodium nitrate, sodium nitrite, or sodium chloride (1 g/L) in drinking w
101 ings demonstrate that topically administered sodium nitrite protects the rat kidney against I/R injur
102 illance underestimates confirmed deaths from sodium nitrite; public health authorities should rely on
104 ence of any NO generation, whereas nebulized sodium nitrite reduces HPV by NO formation; however; com
107 and their nano-entities was compared against sodium nitrite (SN)-added and without antioxidants-added
108 rototype toxic bait (HOGGONE 2 containing 5% sodium nitrite [SN]), though, revealed that wild pigs sp
109 , naloxone, pralidoxime, sodium bicarbonate, sodium nitrite, sodium thiosulfate, vasodilators, and va
111 Five batches were produced: T1: 100 mg/kg of sodium nitrite; T2: 50 mg of sodium nitrite; T3: 50 mg o
112 1: 100 mg/kg of sodium nitrite; T2: 50 mg of sodium nitrite; T3: 50 mg of sodium nitrite and 0.150 uL
117 Here, we test the hypothesis that chronic sodium nitrite therapy can selectively augment angiogeni
126 o a cerebral nitric oxide donor (intravenous sodium nitrite) to explore whether this correlates with
127 , reaction time, sulfanilic acid volume, and sodium nitrite volume were identified using a Plackett-B
133 vinyl alcohol (PVA) films, incorporated with sodium nitrite were developed, characterized and applied
134 ed by exhaled gas concentration from inhaled sodium nitrite were not increased by acetazolamide durin