ライフサイエンスコーパス: sodium sulfate

1 sis and epithelial injury induced by dextran sodium sulfate.

2 itis in mice after administration of dextran sodium sulfate.

3 its stability at different concentrations of sodium sulfate.

4 ration with diatomaceous earth and anhydrous sodium sulfate.

5 r administration of azoxymethane and dextran sodium sulfate.

6 e after exposure to azoxymethane and dextran sodium sulfate.

7 susceptibility to colitis induced by dextran sodium sulfate.

8 y 3 cycles of oral administration of dextran sodium sulfate.

9 xymethane followed by treatment with dextran sodium sulfate.

10 aused by chemical irritants, such as dextran sodium sulfate.

11 hesis was measured by incorporation of [35S]-sodium sulfate.

12 e to wounding of the epithelium with dextran sodium sulfate.

13 ments are repeated in the presence of 400 mM sodium sulfate.

14 absence and presence of a stabilizing salt, sodium sulfate.

15 uced by exposure to azoxymethane and dextran sodium sulfate.

16 given oral Salmonella Typhimurium or dextran sodium sulfate.

17 as induced by oral administration of dextran sodium sulfate.

18 signaling during colitis induced by dextran sodium sulfate.

19 ed by the high sodium content of the dextran sodium sulfate.

20 loys treatment with azoxymethane and dextran sodium sulfate.

21 inflammation induced by azoxymethane/dextran sodium sulfate.

22 a mouse model of colitis induced by dextran sodium sulfate.

23 stration of azoxymethane followed by dextran sodium sulfate.

24 l injury following administration of dextran sodium sulfate.

25 colitis induced by a single cycle of dextran sodium sulfate administration.

26 presence of the stabilizing additives 0.23 M sodium sulfate and 1 M sodium chloride.

27 ults were found when 0.5g mussel shell, 0.5g sodium sulfate and 5mL ethanol were used.

28 entium infection and exacerbation of dextran sodium sulfate and anti-IL10R-triggered colitis.

29 al tumorigenesis in the azoxymethane-dextran sodium sulfate and Apc(Min/+) mouse models and in human

30 c epithelium after administration of dextran sodium sulfate and azoxymethane than control mice.

31 cer was induced by administration of dextran sodium sulfate and azoxymethane.

32 Here we report that the dextran sodium sulfate and CD4+CD45RBhigh T-cell transfer models

33 We found that in both the dextran sodium sulfate and Citrobacter rodentium models of colit

34 was not observed after ingestion of dextran sodium sulfate and correlated with exacerbation of the m

35 ator (Cftr) knockout mice exposed to dextran sodium sulfate and in vitro in primary cholangiocytes is

36 tis was induced by administration of dextran sodium sulfate, and colitis-associated cancer was induce

37 hobiont challenge, administration of dextran sodium sulfate, and high-fat diets.

38 in C57/Bl6 mice by administration of dextran sodium sulfate, and mice were given 10(8) bacteria for 1

39 litis was induced in mice using 2.5% dextran sodium sulfate, and severity was assessed with histology

40 ry (ATOFMS) revealed the presence of halite, sodium sulfates, and sodium carbonates that were strongl

41 Similarly, Spns2 deletion reduced dextran sodium sulfate- and oxazolone-induced colitis.

42 nogenesis protocol [azoxymethane and dextran sodium sulfate (AOM-DSS) administration] exhibited a two

43 on tumorigenesis in the azoxymethane-dextran sodium sulfate (AOM-DSS) model of colitis-associated car

44 c-deficient mice in the azoxymethane/dextran sodium sulfate (AOM/DSS) model of colorectal cancer.

45 arcinogenesis using the azoxymethane/dextran sodium sulfate (AOM/DSS) model.

46 Using the azoxymethane and dextran sodium sulfate (AOM/DSS) murine model of colitis-associa

47 nfected ApcP (Min/+) or azoxymethane/dextran sodium sulfate (AOM/DSS)-treated mice had high LRP5/6 or

48 ecreased in both the presence and absence of sodium sulfate, as previously reported for a variety of

49 n of a 3.41 mol/kg water aqueous solution of sodium sulfate at 1.54 GPa in a diamond-anvil cell resul

50 (PAA) and Amplon (blend of sulfuric acid and sodium sulfate) at a poultry processing pilot plant scal

51 formation following an azoxymethane/dextran sodium sulfate challenge.

52 enomas formed following azoxymethane/dextran sodium sulfate challenge.

53 We identified and characterized the sodium/sulfate co-transporter (NaS-1; Slc13a1) as an Fxr

54 signaling protected mice from acute dextran sodium sulfate colitis because DR3(-/-) mice showed more

55 (DeltaIEC)) mice develop more severe dextran sodium sulfate colitis due to delayed ulcer healing and

56 issue from Ier3(-/-) mice subject of dextran sodium sulfate colitis exhibit greater Nrf2 activity tha

57 /2(-/-) BM chimera mice with chronic dextran sodium sulfate colitis exhibited delayed ulcer healing,

58 Studies of dextran sodium sulfate colitis in intestinal epithelial-specific

59 Dextran sodium sulfate colitis led to increased levels of inflam

60 e were similarly more susceptible to dextran sodium sulfate colitis, although without mortality and w

61 Using the azoxymethane and dextran sodium sulfate colitis-associated colorectal cancer mode

62 esults in enhanced susceptibility to dextran sodium sulfate colitis-induced systemic inflammation and

63 lity and increased susceptibility to dextran sodium sulfate colitis.

64 ins in macrophages protect mice from dextran sodium sulfate-colitis by enhancing 15-PGDH-dependent ox

65 In response to dextran sodium sulfate, colonic infiltration of neutrophils and

66 himurium and after administration of dextran sodium sulfate compared with wild-type mice.

67 nt gas, the extract was dried with anhydrous sodium sulfate, concentrated through evaporation, and th

68 of one-dimensional bonded chains observed in sodium sulfate decahydrate (mirabilite).

69 stinal inflammation upon exposure to dextran sodium sulfate, demonstrating a previously unrecognized

70 e mice treated with azoxymethane and dextran sodium sulfate developed approximately 7-10 tumors per m

71 burdens following azoxymethane (AOM)/dextran sodium sulfate (DSS) administration compared with wild-t

72 We administered 2 exogenous agents, dextran sodium sulfate (DSS) and acetic acid, to assess the susc

73 In the dextran sodium sulfate (DSS) and adoptive T-cell transfer models

74 tis and colitis-associated CRC using dextran sodium sulfate (DSS) and azoxymethane (AOM)-DSS experime

75 Dextran sodium sulfate (DSS) and Citrobacter rodentium colitis (

76 mely sensitive to colitis induced by dextran sodium sulfate (DSS) and developed spontaneous ileitis a

77 using two models, administration of dextran sodium sulfate (DSS) and Salmonella enterica subsp. sero

78 nitrobenzene sulfonic acid (TNBS) or dextran sodium sulfate (DSS) and the inflammatory responses were

79 The dextran sodium sulfate (DSS) and trinitrobenzene sulfonic acid (

80 ucosal injury and colitis induced by dextran sodium sulfate (DSS) are ameliorated in epimorphin-/- mi

81 nted diets was assayed after a 7-day dextran sodium sulfate (DSS) challenge by quantitative real-time

82 sessment of mucosal damage in murine dextran sodium sulfate (DSS) colitis and human IBD.

83 DeltaIEC) mice) were subjected to 2% dextran sodium sulfate (DSS) colitis for 7 days.

84 eficient mice and chemically induced dextran sodium sulfate (DSS) colitis have led to inconsistent re

85 he development of azoxymethane (AOM)/dextran sodium sulfate (DSS) colitis-associated cancer (CAC).

86 eoplasia, we compared differences in dextran sodium sulfate (DSS) colitis-associated neoplasia betwee

87 nistering the specific inhibitors in dextran sodium sulfate (DSS) colitis.

88 e (10 mg/kg) and two treatments with dextran sodium sulfate (DSS) during a 20-week assay period.

89 Dextran sodium sulfate (DSS) exposure induced STAT-6 and NF-kapp

90 Mice given dextran sodium sulfate (DSS) followed by NP-KPV were protected a

91 type littermates by administering 3% dextran sodium sulfate (DSS) for 7 days followed by 2-week recov

92 Mice (C57BL/6) were exposed to 3% dextran sodium sulfate (DSS) for 7 days or 4% DSS for 5 days fol

93 tis was induced by administration of dextran sodium sulfate (DSS) in distilled water.

94 and wild-type mice by administering dextran sodium sulfate (DSS) in drinking water.

95 litis based on the administration of dextran sodium sulfate (DSS) in the drinking water, we sought to

96 extracellular osmolarity induced by dextran sodium sulfate (DSS) in vivo Collectively, these finding

97 gammadelta IEL was evaluated in the dextran sodium sulfate (DSS) induced mouse colitis model system.

98 duces the severity of colitis in the dextran sodium sulfate (DSS) model of murine colonic injury.

99 evels in colon contents, we used the dextran sodium sulfate (DSS) model to test the enzyme's ability

100 itrobenzene sulfonic acid (TNBS) and dextran sodium sulfate (DSS) models of colitis.

101 Administration of dextran sodium sulfate (DSS) significantly increased intestinal

102 thane followed by multiple rounds of dextran sodium sulfate (DSS) to induce colitis and tumorigenesis

103 Using dextran sodium sulfate (DSS) to induce colitis in mice, we ident

104 ymethane followed by three cycles of dextran sodium sulfate (DSS) to induce colitis-associated cancer

105 Some mice were given dextran sodium sulfate (DSS) to induce colitis.

106 Mice were fed 3% dextran sodium sulfate (DSS) to induce colonic inflammation, wit

107 beta(-/-) mice, and, when exposed to dextran sodium sulfate (DSS) to induce inflammatory bowel diseas

108 tis was induced by administration of dextran sodium sulfate (DSS) to mice or transfer of T cells to l

109 X-2(-/-), and heterozygous mice with dextran sodium sulfate (DSS) to provoke acute colonic inflammati

110 We show that dextran sodium sulfate (DSS) treatment promotes the recruitment

111 Chronic Dextran sodium sulfate (DSS) treatment, an inflammatory inducer

112 microbiota was achieved via low-dose dextran sodium sulfate (DSS) treatment.

113 Dextran sodium sulfate (DSS) was used to induce colitis in C57BL

114 lowed by three 1-week cycles of 2.5% dextran sodium sulfate (DSS) water, each cycle separated by 2 we

115 Dextran sodium sulfate (DSS), 2,4,6-trinitrobenzene sulfonic aci

116 intestinal inflammation elicited by dextran sodium sulfate (DSS), a model of experimental colitis.

117 exposure to the oral innate trigger dextran sodium sulfate (DSS), a nonredundant proinflammatory rol

118 induced in mice by administration of dextran sodium sulfate (DSS), and carcinogenesis was induced by

119 Dextran sodium sulfate (DSS), at doses that resulted in little e

120 ice, or given azoxymethane (AOM) and dextran sodium sulfate (DSS), or 1,2-dimethylhydrazine and DSS,

121 nvironmental epithelial injury using dextran sodium sulfate (DSS), Tfeb (DeltaIEC) mice exhibited exa

122 In acute colitis induced by dextran sodium sulfate (DSS), Vim KO mice develop significantly

123 epithelium with low-molecular-weight dextran sodium sulfate (DSS), which is a well-studied model of m

124 is were induced by administration of dextran sodium sulfate (DSS), with or without azoxymethane (AOM)

125 pecifically in mice with infectious, dextran sodium sulfate (DSS)-, and T-cell-induced colitis.

126 of Gal2 in colitis, we employed the dextran sodium sulfate (DSS)-induced acute colitis model in mice

127 taIEC) mice were less susceptible to dextran sodium sulfate (DSS)-induced acute colitis.

128 We used dextran sodium sulfate (DSS)-induced chronic colitis to investig

129 In this article, we show that dextran sodium sulfate (DSS)-induced clinical disease and histol

130 Mucosal repair was assessed after dextran sodium sulfate (DSS)-induced colitis in mice receiving i

131 antioxidants on gut permeability and dextran sodium sulfate (DSS)-induced colitis in mice was tested.

132 ions that increase susceptibility to dextran sodium sulfate (DSS)-induced colitis in mice, we identif

133 l thrombosis that is associated with dextran sodium sulfate (DSS)-induced colitis in mice.

134 ve component of the host response to dextran sodium sulfate (DSS)-induced colitis in the mouse is med

135 Although dextran sodium sulfate (DSS)-induced colitis is a commonly used

136 i in male mice, in formalin test and dextran sodium sulfate (DSS)-induced colitis model, respectively

137 estinal tract, significantly reduced dextran sodium sulfate (DSS)-induced colitis severity, whereas d

138 alactosylceramide (alpha-GalCer), on dextran sodium sulfate (DSS)-induced colitis were examined.

139 IL-6-deficient (IL-6(-/-)) mice with dextran sodium sulfate (DSS)-induced colitis.

140 caspase-1 were highly susceptible to dextran sodium sulfate (DSS)-induced colitis.

141 f Pirb-/- and wild-type (WT) mice to dextran sodium sulfate (DSS)-induced colitis.

142 at causes enhanced susceptibility to dextran sodium sulfate (DSS)-induced colitis.

143 inflammation or crypt damage during dextran sodium sulfate (DSS)-induced colitis.

144 protect intestinal epithelium during dextran sodium sulfate (DSS)-induced colitis.

145 or 50 mg/kg), in an animal model of dextran sodium sulfate (DSS)-induced colitis.

146 in vivo, and enhances recovery from dextran sodium sulfate (DSS)-induced colonic damage.

147 Here we address this issue using a dextran sodium sulfate (DSS)-induced colonic regeneration model.

148 han WT mice using Azoxymethane (AOM)/dextran sodium sulfate (DSS)-induced colorectal cancer model.

149 to induce IL-36gamma in response to dextran sodium sulfate (DSS)-induced damage, suggesting that gut

150 pared with wild-type (WT) mice after dextran sodium sulfate (DSS)-induced injury.

151 Using the dextran sodium sulfate (DSS)-induced intestinal epithelial injur

152 Colon was examined after acute dextran sodium sulfate (DSS)-induced mucosal injury or after azo

153 oethylene-o,o') tellurate (AS101) on dextran sodium sulfate (DSS)-induced murine colitis.

154 ell repopulation of steady-state and dextran sodium sulfate (DSS)-inflamed small intestine/colon and

155 )Apc(Min/+) mice, azoxymethane (AOM)/dextran sodium sulfate (DSS)-treated mice and de-identified huma

156 ring TNFalpha-siRNA-loaded NPs to 3% dextran sodium sulfate (DSS)-treated mice and investigated the t

157 In comparison with wild-type mice, Dextran Sodium Sulfate (DSS)-treated TRPM8 knockout mice showed

158 Colitis was chemically induced with dextran sodium sulfate (DSS).

159 ion and tissue repair in response to dextran sodium sulfate (DSS).

160 equently exposed to colonic irritant dextran sodium sulfate (DSS).

161 d IL-8Tg mice given azoxymethane and dextran sodium sulfate (DSS).

162 mical colitis induced by exposure to dextran sodium sulfate (DSS).

163 then subjected to repeated cycles of dextran sodium sulfate (DSS).

164 ollowed by induction of colitis with dextran sodium sulfate (DSS).

165 d the severity of colitis induced by dextran sodium sulfate (DSS).

166 We induced colitis in mice using Dextran Sodium Sulfate (DSS).

167 he mouse model of colitis induced by dextran sodium sulfate (DSS).

168 mipenem before or after receiving 5% dextran sodium sulfate (DSS).

169 by continuous oral administration of dextran sodium sulfate (DSS).

170 treatment in severe IBD induced via dextran sodium sulfate (DSS).

171 alities after oral administration of dextran sodium sulfate (DSS).

172 he mouse, which are augmented during dextran sodium sulfate (DSS)/azoxymethane (AOM)-induced CAC.

173 as induced by oral administration of dextran sodium sulfate (DSS, 5 g/dL) to knockout mice, their gen

174 of IFN-gamma to G2A(-/-) mice during dextran sodium sulfate exposure abolished the excess colitic inf

175 methane alone or in combination with dextran sodium sulfate; formation of aberrant crypt foci and col

176 s of sodium sulfate is the highly metastable sodium sulfate heptahydrate (Na(2)SO(4).7H(2)O).

177 ted in the formation of a previously unknown sodium sulfate hydrate, which we have determined by sing

178 is of proteoglycans via incorporation of 35S-sodium sulfate in the culture medium.

179 Acute colitis was induced using 4% dextran sodium sulfate in wild-type mice maintained on Se-defici

180 e models (Salmonella typhimurium and dextran sodium sulfate) in PHB transgenic mice and wild-type lit

181 naturation were observed; for example, 0.4 M sodium sulfate increased the free energy of wild-type SN

182 in altering the onset and relapse of dextran sodium sulfate induced murine colitis.

183 ccordingly, knockdown of ORMDL3 in a dextran sodium sulfate -induced colitis mouse model showed reduc

184 is under normal conditions; however, dextran sodium sulfate-induced (DSS-induced) colitis promoted th

185 In the dextran sodium sulfate-induced acute colitis model, systemic tre

186 In a dextran sodium sulfate-induced acute colitis model, WT mice lost

187 wound healing was corroborated in a dextran sodium sulfate-induced acute colitis mouse model.

188 tes, and increases susceptibility to dextran sodium sulfate-induced bowel injury.

189 in mice inhibited azoxymethane- and dextran sodium sulfate-induced CAC, IL-6 expression, STAT3 phosp

190 CDDO-Me suppressed azoxymethane plus dextran sodium sulfate-induced carcinogenesis in wild-type anima

191 d neutrophil infiltration in a mouse dextran sodium sulfate-induced chronic inflammatory bowel diseas

192 in epithelial cells during both the dextran sodium sulfate-induced colitic and the recovery phase.

193 Further, administration of EGCG to dextran sodium sulfate-induced colitic mice significantly reduce

194 Mouse models of dextran sodium sulfate-induced colitis and a T cell transfer mod

195 ts were observed in a mouse model of dextran sodium sulfate-induced colitis and in Caco2-BBE cells tr

196 th trinitrobenzene sulfonic acid- or dextran sodium sulfate-induced colitis and in Il10(-/-) mice.

197 Dextran sodium sulfate-induced colitis and Salmonella typhimuriu

198 that the presence of GIV ameliorates dextran sodium sulfate-induced colitis and sepsis-induced death.

199 bsence of villin predisposes mice to dextran sodium sulfate-induced colitis by promoting apoptosis.

200 od2 results in higher sensitivity to dextran sodium sulfate-induced colitis compared with a single de

201 ve effects of engineered EcN against dextran sodium sulfate-induced colitis in mice, associated with

202 ignificantly reduced the severity of dextran sodium sulfate-induced colitis in mice.

203 Increased sensitivity to dextran sodium sulfate-induced colitis in Pglyrp3(-/-)Nod2(-/-)

204 ministration of MDP does not prevent dextran sodium sulfate-induced colitis in SAMP mice and that the

205 ed lymphoid follicles and attenuates dextran sodium sulfate-induced colitis independent of endothelia

206 d ameliorating disease phenotypes in dextran sodium sulfate-induced colitis mice.

207 repair during recovery in the acute dextran sodium sulfate-induced colitis model in GH-overexpressin

208 Treatment of mice with HDACi in a dextran sodium sulfate-induced colitis model resulted in a stron

209 Dextran sodium sulfate-induced colitis model was established in

210 etermined in vitro as well as in the dextran sodium sulfate-induced colitis mouse model.

211 nt model of allergic airway disease, dextran sodium sulfate-induced colitis was significantly reduced

212 1(-/-) mice and CRHR2(-/-) mice with dextran sodium sulfate-induced colitis were analyzed in comparis

213 R (NCM460-NK-1R cells) and mice with dextran sodium sulfate-induced colitis were used.

214 rom TNF exposure, and exhibit severe dextran sodium sulfate-induced colitis, ameliorated by TNF inhib

215 led to impaired resolution of acute dextran sodium sulfate-induced colitis, which was characterized

216 elevated in the colons of mice with dextran sodium sulfate-induced colitis, which was reduced by tre

217 re transferred to CD1 nude mice with dextran sodium sulfate-induced colitis, with or without oral adm

218 epithelial cell-derived IL-33 during dextran sodium sulfate-induced colitis.

219 lfonic acid (DNBS)-, oxazolone-, and dextran-sodium sulfate-induced colitis.

220 en associated with susceptibility to dextran sodium sulfate-induced colitis.

221 ver, the IGF2BP1 deletion aggravated dextran sodium sulfate-induced colitis.

222 while promoting Treg development in dextran sodium sulfate-induced colitis.

223 , it exerted protective functions in dextran sodium sulfate-induced colitis.

224 cking colonic RelA were sensitive to dextran sodium sulfate-induced colitis.

225 oth autoimmune encephalomyelitis and dextran sodium sulfate-induced colitis.

226 enhance or attenuate the severity of dextran sodium sulfate-induced colitis.

227 rfering (si)RNA to C57BL/6 mice with dextran sodium sulfate-induced colitis.

228 , TNF-alpha, and IL-17) in mice with dextran sodium sulfate-induced colitis.

229 comparing healthy mice and mice with dextran sodium sulfate-induced colitis.

230 ing are transmissible and exacerbate dextran sodium sulfate-induced colitis.

231 and exaggerated inflammation during dextran sodium sulfate-induced colitis.

232 reater incidence of azoxymethane and dextran sodium sulfate-induced colon carcinoma.

233 rotected the colonic epithelium from dextran sodium sulfate-induced damage.

234 We show that this antibody blocks dextran sodium sulfate-induced HK cleavage and bradykinin produc

235 e highly susceptible to azoxymethane/dextran sodium sulfate-induced inflammation and suffered from dr

236 than Rag(-/-) mice to development of dextran sodium sulfate-induced intestinal inflammation, indicati

237 tenance of intestinal homeostasis in dextran sodium sulfate-induced intestinal injury.

238 Using a dextran sodium sulfate-induced mouse model of acute colitis, we

239 n in Cftr-knockout mice exposed to a dextran sodium sulfate-induced portal endotoxemia.

240 revention and recovery rat models of dextran-sodium-sulfate-induced colitis.

241 portantly, PPARdelta is required for dextran sodium sulfate induction of proinflammatory mediators, i

242 in TNBS-inflamed guinea pigs, and in dextran sodium sulfate-inflamed mice, treated with a free radica

243 yl dipeptide (to stimulate NOD2), or dextran sodium sulfate; intestinal lamina propria cells were col

244 sis was measured as the incorporation of 35S-sodium sulfate into macromolecules separated from uninco

245 the crystallization of aqueous solutions of sodium sulfate is the highly metastable sodium sulfate h

246 ption of the epithelial barrier with dextran sodium sulfate leads to increased IL-19 expression.

247 served for sulfate in some samples with high sodium/sulfate mass ratios.

248 colon into the lymphatic system in a dextran sodium sulfate mediated model of inflammatory bowel dise

249 letion failed to modify azoxymethane/dextran sodium sulfate-mediated tumorigenesis.

250 models of colonic inflammation: the dextran sodium sulfate model and multidrug resistance gene 1a-de

251 Here, studies utilizing the murine dextran sodium sulfate model of colitis revealed the crucial rol

252 ENT FINDINGS: Using the azoxymethane-dextran sodium sulfate model, wound healing pathways seem to be

253 ion and colitis-associated cancer in dextran sodium sulfate model.

254 he trinitrobenzene sulfonic acid and dextran sodium sulfate models of colitis, we show the importance

255 Separate sets of mice were given dextran sodium sulfate or 2,4,6-trinitrobenzenesulfonic acid to

256 d ulcers following administration of dextran sodium sulfate or 2,4,6-trinitrobenzenesulfonic acid.

257 litis was induced in mice by oral 5% dextran sodium sulfate or rectal 5% acetic acid, followed by ene

258 OD, and B6.AKR) by administration of dextran sodium sulfate or rectal application of trinitrobenzene

259 MMP-9-/- mice exposed to dextran sodium sulfate or salmonella had a significantly reduced

260 vere colitis after administration of dextran sodium sulfate or trinitrobenzene sulfonate than mice wi

261 Colitis was induced using dextran sodium sulfate or trinitrobenzene sulfonic acid (TNBS).

262 olitis, induced by administration of dextran sodium sulfate or trinitrobenzene sulfonic acid.

263 e signal response was found to be linear for sodium sulfate over the concentration ranges of 0.2-100

264 05% of the total protein population in 20 mM sodium sulfate, respectively.

265 response, induction of colitis with dextran sodium sulfate resulted in a MyD88-dependent serum Ab re

266 s following initiation of refolding in 0.4 M sodium sulfate revealed weak protection in the first bet

267 Addition of sodium sulfate shifts the rate profile to higher denatur

268 in the rectum following injury with dextran sodium sulfate, similarly treated Myd88(-/-) (TLR signal

269 studied the influence of a number of salts (sodium sulfate, sodium fluoride, sodium acetate, and sod

270 mmonium sulfate (AS), ammonium nitrate (AN), sodium sulfate (SS), or sodium nitrate (SN) solutions wi

271 ic (Step 5A) and, when combined with dextran sodium sulfate (Step 5B), inflammation-associated tumor

272 ablated all but the first exon of SLC13A1, a sodium/sulfate symporter responsible for regulating seru

273 and four saline water treatments dominant in sodium-sulfate (T2), sodium-chloride (T3), sodium-chlori

274 all HPP starches and was generally lower in sodium sulfate than in water.

275 vere colitis after administration of dextran sodium sulfate than mice infected with LF82-DeltachiA or

276 oncentrations, especially in the presence of sodium sulfate), the kinetics of folding shows evidence

277 This resembles anhydrous sodium sulfate (thenardite) but contrasts with the hepta

278 o Rag(-/-) mice or administration of dextran sodium sulfate to C57BL/6 mice.

279 ced in some mice by addition of 2.5% dextran sodium sulfate to drinking water for 5-9 consecutive day

280 ice were exposed to azoxymethane and dextran sodium sulfate to induce colitis and tumorigenesis.

281 n was measured; some mice were given dextran sodium sulfate to induce colitis and/or gavage with an a

282 ice (controls); some mice were given dextran sodium sulfate to induce colitis, with or without a FFAR

283 l polyinosinic:polycytidylic acid or dextran sodium sulfate to induce colitis.

284 t-free (control) diet and then given dextran sodium sulfate to induce colitis; we also studied Il10(-

285 n of sodium chloride, potassium chloride, or sodium sulfate to leptospiral medium to physiological os

286 tis was induced by administration of dextran sodium sulfate to wild-type and Cav-1(-/-) mice, as well

287 e, or given azoxymethane followed by dextran sodium sulfate, to assess intestinal tumor formation.

288 Dextran sodium sulfate-treated SLC15A4-deficient mice exhibit de

289 g azoxymethane injection followed by dextran sodium sulfate treatment in TLR4-deficient or wild-type

290 nuated colon inflammation induced by dextran sodium sulfate treatment or Citrobacter rodentium infect

291 rcinogenesis induced by azoxymethane/dextran sodium sulfate treatment.

292 e gut in response to indomethacin or dextran sodium sulfate treatment.

293 uced tumor burden after azoxymethane/dextran sodium sulfate treatment.

294 ation compared with WT animals after dextran sodium sulfate treatment.

295 ion-driven tumor model (azoxymethane/dextran sodium sulfate), VS28 mice developed a significantly hig

296 njury induced by the toxic substance dextran sodium sulfate was fundamentally altered to include path

297 mon colitis model, administration of dextran sodium sulfate, was hopelessly confounded by the high so

298 alpha-d-glucose and citric acid, along with sodium sulfate, were produced using established and newl

299 human FN promoter, given water or 3% dextran sodium sulfate, were used as animal models of colitis.

300 For example, a sodium sulfate with single, oligomeric vinyl acetate (VA