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1 micked by the tyrosine phosphatase inhibitor sodium vanadate (100 microm).
2                           In the presence of sodium vanadate, a phosphatase inhibitor, phosphorylated
3 th specific inhibitors of p38 MAP kinase and sodium vanadate, a potent protein-tyrosine phosphatase i
4        Hence, treatment of Bcl-xL cells with sodium vanadate, a tyrosine phosphatase inhibitor, resto
5          Preincubation with okadaic acid and sodium vanadate abrogated adhesion-induced dephosphoryla
6                                 In addition, sodium vanadate, also an inhibitor of protein phosphatas
7                                              Sodium vanadate, an inhibitor of phosphotyrosine phospha
8 titive inhibition of alkaline phosphatase by sodium vanadate and sodium arsenate has been examined, a
9 g stem metabolic activity (infiltration with sodium vanadate and sodium cyanide; plant exposure to ca
10  known PTPase inhibitors phenylarsine oxide, sodium vanadate, and iodoacetate also inhibited enzyme a
11 g protein-tyrosine phosphatase inhibition by sodium vanadate both EGFR expressing Chinese hamster ova
12 t of GEO cells with a phosphatase inhibitor (sodium vanadate) caused a dose-dependent increase in ERK
13 phatase, we found that sodium pyrophosphate, sodium vanadate, cyclosporin A, tautomycin, and okadaic
14 e Rho, or the tyrosine phosphatase inhibitor sodium vanadate increased the level of phosphorylation o
15 ion because pretreatment of hepatocytes with sodium vanadate increases (and 25 microM genistein reduc
16 lished by the tyrosine phosphatase inhibitor sodium vanadate, indicating involvement of protein tyros
17                                 Furthermore, sodium vanadate induced primitive endoderm differentiati
18                                              Sodium vanadate is an effective agent for the enrichment
19                                              Sodium vanadate is an effective drug for the enrichment
20 , the phosphotyrosine phosphatase inhibitor, sodium vanadate, or expression of mutationally activated
21 o increased and decreased by the addition of sodium vanadate, respectively, but these changes were el
22 ition of the tyrosine phosphatase inhibitor, sodium vanadate, selectively repressed Nanog transcripti
23  the astrocytes, while both okadaic acid and sodium vanadate significantly reversed these anti-prolif
24         The enzyme activity was inhibited by sodium vanadate, sodium fluoride, N-ethylmaleimide, and
25 c peptides caused a 70-80% inhibition of the sodium vanadate-stimulated MAPK activity, complete inhib
26 ith compounds acting by distinct mechanisms: sodium vanadate (SV), an inhibitor of protein phosphatas
27 ated tyrosine phosphatases were blocked with sodium vanadate, the high-frequency gating remained rela
28          We measured the stiffness of 50 muM sodium vanadate treated bull sperm in the presence of 4
29  We investigated the molecular mechanisms of sodium vanadate (vanadate)-induced nitric oxide (NO) pro
30                                              Sodium vanadate (Vi) is a phosphate analog that inhibits
31  force to 6% of pCa 4.0 controls with 1.0 mM sodium vanadate (Vi).
32 s-bridge binding to intermediate levels with sodium vanadate (Vi).
33                               The effects of sodium vanadate were abrogated by Grb2 deficiency or by
34 minin 5, we found that phosphatase inhibitor sodium vanadate, which blocked the p80 dephosphorylation