ライフサイエンスコーパス: sodium-dependent

1 hibition by L-leucine but not by L-lysine is sodium dependent.

2 transport of nucleosides is proton-, but not sodium-, dependent.

3 Similarly, sodium-dependent [3H]taurocholate uptake into membrane v

4 pendent on GLUT transport of DHA rather than sodium-dependent AA uptake.

5 -AP-sensitive channels were activated during sodium-dependent action potentials and mediated a large

6 of CA1 pyramidal neurons and drive bursts of sodium-dependent action potentials at the soma.

7 ed phase advances, suggest that NPY requires sodium-dependent action potentials within GABAergic neur

8 Blockade of sodium-dependent action potentials within the SCN preven

9 vesicle accumulation, and the generation of sodium-dependent action potentials, hallmarks of a neuro

10 that spread to the synaptic terminal without sodium-dependent action potentials.

11 d in part by potassium channels activated by sodium-dependent action potentials.

12 ry across the plasma membrane in response to sodium-dependent action potentials.

13 Larval motor neurons had a long-lasting, sodium-dependent afterhyperpolarization (AHP) following

14 solution of pulmonary edema by up-regulating sodium-dependent alveolar fluid clearance.

15 The sodium-dependent amino acid transport systems responsibl

16 area of mouse chromosome 13 that encodes the sodium-dependent amino acid transporter B(0)AT1.

17 and dissipate the electrical gradient of the sodium-dependent amino acid transporters in the proximal

18 melanoma cells, and ascorbate transport was sodium dependent and inhibited by ouabain.

19 SLC22A15 transport of several substrates was sodium-dependent and exhibited a higher Km for ergothion

20 to assess dapagliflozin's ability to inhibit sodium-dependent and facilitative glucose transport acti

21 ate homeostasis is mediated by high-affinity sodium-dependent and highly hydrophobic plasma membrane

22 The recovery was sodium-dependent and sensitive to Na(+)/H(+)-exchanger (

23 observed in untreated, diabetic rats of both sodium-dependent and sodium-independent glutamate uptake

24 ism and atomic structure in a broad range of sodium-dependent and sodium-independent secondary transp

25 was ATP- and temperature-dependent, but not sodium-dependent, and was inhibited by disulfonic stilbe

26 annel activity in regulating calcium- versus sodium-dependent AP firing.

27 rther strengthened by the comparable loss of sodium-dependent ascorbate transport activity upon the m

28 er mutations of His51 in hSVCT1, significant sodium-dependent ascorbate transport activity was only o

29 tients with correspondingly decreased apical sodium-dependent BA transporter (ASBT) gene expression.

30 Treating mice with an apical sodium-dependent BA transporter inhibitor (SC-435) resul

31 al circulation is primarily dependent upon a sodium-dependent basolateral membrane transporter.

32 ystem substrate-binding protein A)/sbtA (for sodium-dependent bicarbonate transporter A): Delta4 muta

33 sted for their ability to inhibit the apical sodium dependent bile acid transport (ASBT)-mediated upt

34 Apical sodium-dependent bile acid transporter (ASBT) (SLC10A2),

35 Expression of the apical sodium-dependent bile acid transporter (ASBT) and the il

36 rlying the transport of bile acids by apical sodium-dependent bile acid transporter (Asbt) are not we

37 cular mechanisms of regulation of the apical sodium-dependent bile acid transporter (ASBT) by inflamm

38 g transmembrane (TM) segment 7 of the apical sodium-dependent bile acid transporter (ASBT) in substra

39 Ileal expression of the apical sodium-dependent bile acid transporter (ASBT) in the rat

40 The rat ileal sodium-dependent bile acid transporter (Asbt) is a polyt

41 The ileal apical sodium-dependent bile acid transporter (ASBT) is crucial

42 The apical sodium-dependent bile acid transporter (Asbt) is respons

43 er membrane by the well characterized apical sodium-dependent bile acid transporter (Asbt) Slc10a2; h

44 The rat ileal apical sodium-dependent bile acid transporter (Asbt) transports

45 The apical sodium-dependent bile acid transporter (ASBT) transports

46 ), spanning residues V127-T149 of the apical sodium-dependent bile acid transporter (ASBT), a key mem

47 Apical sodium-dependent bile acid transporter (ASBT), an ileal

48 rmacological inhibition of the ileal, apical sodium-dependent bile acid transporter (ASBT), blocks pr

49 tokine-mediated repression of the rat apical sodium-dependent bile acid transporter (ASBT).

50 alts is mediated in large part by the apical sodium-dependent bile acid transporter (ASBT).

51 ng reabsorbed in the intestine by the apical sodium-dependent bile acid transporter (ASBT, also known

52 The apical sodium-dependent bile acid transporter (ASBT, SLC10A2) f

53 The apical sodium-dependent bile acid transporter (ASBT, SLC10A2) m

54 A1) expressed in hepatocytes, and the apical sodium-dependent bile acid transporter (ASBT; also known

55 ulation, is mediated primarily by the apical sodium-dependent bile acid transporter (ASBT=SLC10A2).

56 The human apical sodium-dependent bile acid transporter (hASBT) may serve

57 The membrane topology of the human apical sodium-dependent bile acid transporter (hASBT) remains u

58 in substrate interaction of the human apical sodium-dependent bile acid transporter (hASBT, SLC10A2)

59 Human apical sodium-dependent bile acid transporter (hASBT, SLC10A2)

60 The human apical sodium-dependent bile acid transporter (hASBT, SLC10A2)

61 sed mRNA and protein expression of the ileal sodium-dependent bile acid transporter (ISBT) in the int

62 The recent cloning of a human sodium-dependent bile acid transporter (NTCP) permits an

63 f bile salts via up-regulation of the apical sodium-dependent bile acid transporter and diminished ca

64 cholestasis-1 led to up-regulation of apical sodium-dependent bile acid transporter and down-regulati

65 Transport, apical sodium-dependent bile acid transporter and ileal lipid-b

66 tide 1, and the BA transport systems, apical sodium-dependent bile acid transporter and Na(+) -tauroc

67 al hepatobiliary transporters, including the sodium-dependent bile acid transporter gene, ntcp.

68 The apical sodium-dependent bile acid transporter is critical for i

69 Increased ileal apical sodium-dependent bile acid transporter messenger RNA (mR

70 veloped using 3.0 kilobase of the rat apical sodium-dependent bile acid transporter promoter to drive

71 co-2 cells, the activity of the human apical sodium-dependent bile acid transporter promoter was enha

72 s resulted in activation of the human apical sodium-dependent bile acid transporter promoter.

73 The ileal sodium-dependent bile acid transporter reclaims bile aci

74 The apical sodium-dependent bile acid transporter was up-regulated

75 ning extracellular loop (EL) 1 of the apical sodium-dependent bile acid transporter were determined v

76 rocholate cotransporting polypeptide, apical sodium-dependent bile acid transporter) and an exporter

77 , and transporters, such as the ileal apical sodium-dependent bile acid transporter, appear to affect

78 sion of bile acid signaling machinery apical sodium-dependent bile acid transporter, FXR, and small h

79 ess mature biliary markers, including apical sodium-dependent bile acid transporter, secretin recepto

80 Interactions among the apical sodium-dependent bile acid transporter, the farnesoid X

81 and messenger RNA and protein for the apical sodium-dependent bile acid transporter, the ileal bile a

82 ding involving multiple residues to describe sodium-dependent bile acid transporter-mediated bile aci

83 Mammalian sodium-dependent bile acid transporters (SBATs) responsi

84 deficient, were stably transfected with the sodium-dependent bile acid transporting polypeptide.

85 liver, tissues that also express the apical sodium-dependent bile acid uptake transporter ASBT (SLC1

86 Sodium-dependent bile salt transport was also measured i

87 By that time, the ileal sodium-dependent bile salt transporter messenger RNA and

88 , multidrug resistance protein 3, and apical sodium-dependent bile salt transporter.

89 Apical sodium-dependent BS transporter inhibitors that reduce i

90 During hypoxia, blockade of neurons with sodium-dependent bursting properties abolishes respirato

91 This effect is sodium dependent but not potassium or chloride dependent

92 death of pneumococci specifically required a sodium-dependent calcium influx, as shown using calcium

93 2', 4'-dichlorobenzamil (DCB) inhibits the sodium-dependent calcium transporter (NCX1.1) much more

94 It is caused by mutations in the sodium-dependent carnitine cotransporter OCTN2.

95 dent vitamin C transporter (hSVCT1) mediates sodium-dependent cellular uptake of the essential micron

96 Thus sodium-dependent changes of calcium transport are indire

97 m-bicarbonate cotransporter (NBCn1), and the sodium-dependent chloride-bicarbonate exchanger (NDCBE).

98 eby demonstrate that cells expressing mutant sodium-dependent citrate transporter have a complete los

99 SLC13A5 encodes a high affinity sodium-dependent citrate transporter, which is expressed

100 d by the AE family of Cl-/HCO3- exchangers), sodium-dependent Cl-/HCO3- exchange, and Na+:HCO3- cotra

101 ient to transmembrane solute transfer in the sodium-dependent co-transporter OCTN2.

102 r GDP, suggesting that they were mediated by sodium-dependent conductances in a G-protein-dependent m

103 C in its reduced form, ascorbate, through a sodium-dependent cotransporter.

104 The sodium-dependent current underlying these oscillations,

105 2 (engulfment and cell motility 2), SLC13A3 (sodium-dependent dicarboxylate transporter member 3), an

106 approach, we isolated a novel member of the sodium-dependent dicarboxylate/sulfate transporter calle

107 We further demonstrate sodium-dependent dimerization of PGRP-Ialpha in solution

108 es with largely diminished capacities of (1) sodium-dependent efflux of vacuolar protons and (2) elic

109 nd the nucleus where it colocalized with the sodium-dependent excitatory amino acid transporter, EAAT

110 corbic acid (AA), is taken up into cells via sodium-dependent facilitated transport.

111 ted-glucose transporters (GLUTs) but not for sodium-dependent glucose co-transporters (SGLTs), which

112 e GLUT inhibitor cytochalasin B, but not the sodium-dependent glucose cotransport inhibitor phloridzi

113 Inhibition of the sodium-dependent glucose cotransporter 1 (SGLT1) with ph

114 affinity, Na(+)-coupled, glucose transporter sodium-dependent glucose cotransporter 1, was evaluated

115 al reports of inhibitors directed toward the sodium-dependent glucose cotransporter 2 (SGLT2) as a me

116 osine phosphatase 1B) or by inhibiting renal sodium-dependent glucose cotransporter 2 (SGLT2).

117 The sodium-dependent glucose cotransporter SGLT1 undergoes a

118 Inhibitors of the sodium-dependent glucose cotransporters (SGLT) have appe

119 itated-glucose transporters (GLUTs), not for sodium-dependent glucose cotransporters (SGLTs), which h

120 ia variants at SLC5A2, the gene encoding the sodium-dependent glucose transporter (SGLT2), a protein

121 ChCoT, which was shown to be a member of the sodium-dependent glucose transporter family (SLGT), shou

122 t of restoration of normoglycemia by a novel sodium-dependent glucose transporter inhibitor (T-1095)

123 nal lumen into absorptive enterocytes by the sodium-dependent glucose transporter isoform 1 (SGLT1).

124 the expression of the known facilitative and sodium-dependent glucose transporter isoforms in six dif

125 whose expression, and that of GLUT 2 and the sodium-dependent glucose transporter protein 1 (SGLT1),

126 testinal expression of glucose transporters (sodium-dependent glucose transporter-1 and glucose trans

127 ose is absorbed from the small intestine via sodium-dependent glucose transporter-1 and glucose trans

128 cose absorption and expression of intestinal sodium-dependent glucose transporter-1, glucose transpor

129 critically ill patients, duodenal levels of sodium-dependent glucose transporter-1, glucose transpor

130 jejunum of cecal ligation and puncture mice sodium-dependent glucose transporter-1, glucose transpor

131 sure absolute (human) and relative levels of sodium-dependent glucose transporter-1, glucose transpor

132 rtance of another glucose import system, the sodium-dependent glucose transporters (SGLTs), in pancre

133 and it is removed from the synaptic cleft by sodium-dependent glutamate transport activity.

134 as associated with a progressive decrease in sodium-dependent glutamate transport capacity.

135 kinases C or A modulates the activity of the sodium-dependent glutamate transporter EAAC1.

136 hloride conductance with the properties of a sodium-dependent glutamate transporter has been describe

137 The sodium-dependent glutamate transporter, excitatory amino

138 em and is removed from the synaptic cleft by sodium-dependent glutamate transporters.

139 We found that OLs in culture are capable of sodium-dependent glutamate uptake with a K(m) of 10 +/-

140 and degradation of the EAAC1 transporter, a sodium-dependent glutamate/aspartate transport protein t

141 -dependent neutral amino acid transporter 1, sodium-dependent glutamate/aspartate transporter 3, and

142 It is well documented that the sodium dependent, hemicholinium-3 sensitive, high affini

143 Presynaptic uptake of choline via the sodium-dependent, hemicholinium-3-sensitive choline tran

144 Using [3H]L-glutamate as the tracer, sodium-dependent high affinity glutamate transport was d

145 mutations in SLC5A7 encoding the presynaptic sodium-dependent high-affinity choline transporter 1 (CH

146 The sodium-dependent, high affinity serotonin [5-hydroxytryp

147 tosomes from pleural-pedal ganglia exhibited sodium-dependent, high-affinity Glu transport.

148 we investigated some mechanisms involved in sodium-dependent hypertension of rats exposed to chronic

149 ly validated, showing that proton efflux was sodium-dependent, inhibited by amiloride analogs, and ac

150 Transport was sodium-dependent, inhibited by excess ascorbate, and sim

151 C34A1, SLC34A2 and SLC34A3, which encode the sodium-dependent inorganic phosphate (P(i)) cotransport

152 s associated with expression of the type III sodium-dependent inorganic phosphate (Pi) cotransporter

153 ar in sequence to a mammalian brain-specific sodium-dependent inorganic phosphate cotransporter I (BN

154 A CreER(T2) cassette was knocked into the sodium-dependent inorganic phosphate transporter SLC34a1

155 a virus, this receptor is the human type III sodium-dependent inorganic phosphate transporter, SLC20A

156 ivery across the blood-retina barrier by the sodium-dependent lipid transporter, Mfsd2a.

157 Mfsd2a is a newly described sodium-dependent lysophosphatidylcholine (LPC) symporter

158 In this study, we discovered that the sodium-dependent lysophosphatidylcholine (LPC) transport

159 atty acids from the circulation is through a sodium-dependent lysophosphatidylcholine (LPC) transport

160 2A (MFSD2A) was recently characterized as a sodium-dependent lysophosphatidylcholine transporter exp

161 perfused IBDUs absorbed TCA in a saturable, sodium-dependent manner; in addition, TCA absorption was

162 ocycline through a saturable, concentrative, sodium-dependent mechanism with a Michaelis constant (K(

163 nsports ascorbic acid (ASC) inward through a sodium-dependent mechanism.

164 lytoxin by regulating JNK and SEK1 through a sodium-dependent mechanism.

165 There are two sodium-dependent membrane transporters encoded by SLC23A

166 The nucleotide sequence of the cDNA (sodium-dependent multivitamin transporter (SMVT)) predic

167 an cells accumulate biotin by using both the sodium-dependent multivitamin transporter and monocarbox

168 Three transporters, SMVT (sodium-dependent multivitamin transporter for biotin and

169 ched in L-fucose, a competitive inhibitor of sodium-dependent myo-inositol transport.

170 Expression of the M813 receptor murine sodium-dependent myo-inositol transporter 1 (mSMIT1) all

171 sitol, an osmolyte transported into cells by sodium-dependent myo-inositol transporters (SMITs).

172 The sodium-dependent NADH dehydrogenase (Na(+)-NQR) is a key

173 The sodium-dependent NADH dehydrogenase (Na(+)-NQR) is the m

174 T-1 is coded by snf-11 gene, a member of the sodium-dependent neurotransmitter symporter gene family

175 Sodium-dependent neurotransmitter transporters participa

176 and the norepinephrine transporter (NET) are sodium-dependent neurotransmitter transporters responsib

177 thermophilum encodes a protein homologous to sodium-dependent neurotransmitter transporters.

178 System A, the sodium-dependent neutral amino acid transport activity,

179 rter 1 (GLUT-1), taurine transporter (TAUT), sodium-dependent neutral amino acid transporter (SNAT),

180 ees (,+))-type amino acid transport protein, sodium-dependent neutral amino acid transporter 1, sodiu

181 An ATF4 target gene, SNAT2 (system A sodium-dependent neutral amino acid transporter 2), cont

182 tium formation by interacting with the human sodium-dependent neutral amino acid transporter type 2 (

183 The sodium-dependent neutral amino acid transporter type 2 (

184 these viruses and identified it as the human sodium-dependent neutral amino acid transporter type 2 (

185 lating arsenite-induced ER stress, including sodium-dependent neutral amino acid transporter, SNAT2.

186 g (Kir) potassium channels and activation of sodium-dependent, nonselective cationic channels (NSCCs)

187 the reduction in membrane expression of the sodium-dependent P(i) co-transporters, NPT2a and NPT2c,

188 and for increased expression of the type III sodium-dependent P(i) cotransporter Pit-1 and certain os

189 H 3T3 cells resulted in a marked increase in sodium-dependent P(i) uptake.

190 The sodium-dependent phosphate (Na/P(i)) transporters NaPi-2

191 orbol 12-myristate 13-acetate (PMA) enhanced sodium-dependent phosphate (Na/Pi) uptake.

192 than the adenylyl cyclase, pathway mediates sodium-dependent phosphate co-transport in LLC-PK1 cells

193 , where both PTH-stimulated PLC activity and sodium-dependent phosphate co-transport were essentially

194 hosphate levels and seem to be mediated by a sodium-dependent phosphate co-transporter, Pit-1 (Glvr-1

195 evated phosphate on HSMCs were mediated by a sodium-dependent phosphate cotransporter (NPC), as indic

196 parathyroid hormone receptor (PTHR), type II sodium-dependent phosphate cotransporter (Npt2a), and be

197 pyrophosphatase/phosphodiesterase 1 enzyme, sodium-dependent phosphate cotransporter 1 (encoded by t

198 ranscellular mechanism involving the type II sodium-dependent phosphate cotransporter NPT2b (SLC34a2)

199 e have investigated the role of the type III sodium-dependent phosphate cotransporter, Pit-1, in SMC

200 sodium-glucose cotransporter 2, and type IIa sodium-dependent phosphate cotransporter, suggesting api

201 ion and mineralization via the activity of a sodium-dependent phosphate cotransporter.

202 phosphate-induced calcification, implicating sodium-dependent phosphate cotransporters in this proces

203 However, FeLV-B and A-MLV use different sodium-dependent phosphate symporters, Pit1 and Pit2, re

204 decreased Pit-1 mRNA and protein levels and sodium-dependent phosphate transport activity compared w

205 a resulted in a 36.0 +/- 6.3% higher rate of sodium-dependent phosphate transport and a significant i

206 s a phosphaturic factor that suppresses both sodium-dependent phosphate transport and production of 1

207 ibroblast growth factor-23 (FGF-23) inhibits sodium-dependent phosphate transport in brush border mem

208 Basal sodium-dependent phosphate transport was lower in cells

209 le cells, neither PTH nor dopamine inhibited sodium-dependent phosphate transport.

210 thelial cells, sFRP-4 specifically inhibited sodium-dependent phosphate transport.

211 The sodium-dependent phosphate transporter (HOST5/SLC34A2) e

212 protein (LP), and HOST5 codes for a type II sodium-dependent phosphate transporter (SLC34A2).

213 Whereas KoRV-A uses the sodium-dependent phosphate transporter 1 (PiT1) as a rec

214 domain decreased the binding affinity to the sodium-dependent phosphate transporter 2a (Npt2a) as com

215 ing of cellular targets including the Npt2a (sodium-dependent phosphate transporter 2a).

216 e leukemia virus (A-MuLV) utilizes the Pit-2 sodium-dependent phosphate transporter as a cell surface

217 ne leukemia virus (A-MuLV) utilizes the PiT2 sodium-dependent phosphate transporter as its cell surfa

218 and 1,25(OH)(2)D, reduced expression of the sodium-dependent phosphate transporter NPT2a in the prox

219 acilitating the internalization of the major sodium-dependent phosphate transporter, Npt2a.

220 mal tubule of the kidney by retrieval of the sodium-dependent phosphate transporters (Npt2a and Npt2c

221 etrovirus serve normal cellular functions as sodium-dependent phosphate transporters (Pit-1 and Pit-2

222 the renal tubule by the action of the apical sodium-dependent phosphate transporters, NaPi-IIa/NaPi-I

223 cells, on the other hand, had lower rates of sodium-dependent phosphate uptake and low phosphate medi

224 Mammalian sodium-dependent Pi cotransporters have been grouped int

225 Interestingly, in the absence of sodium-dependent Pi transport activity, the PiT1-PiT2 he

226 diet-induced hypophosphatemia as well as in sodium-dependent Pi transporter solute carrier family 34

227 igated the involvement of the high-affinity, sodium-dependent Pi transporters PiT1 and PiT2 in mediat

228 lpha, induced PiT-1 expression and increased sodium-dependent Pi uptake by >40% in chondrocytes.

229 ase progression through PiT-1 expression and sodium-dependent Pi uptake mediated by CXCR1 signaling.

230 modulating chondrocyte PiT-1 expression and sodium-dependent Pi uptake, and to assess differential r

231 NTT4 was previously thought to function as a sodium-dependent plasma membrane transporter, recent stu

232 ises from an intrinsic cellular mechanism: a sodium-dependent potassium conductance that causes prolo

233 PSC)-derived neurons, we have now found that sodium-dependent potassium currents are increased severa

234 es the direct activation of an electrogenic, sodium-dependent presynaptic transporter, which supplies

235 ockouts identified 15% and 40% reductions in sodium-dependent proline and leucine transport, respecti

236 Sodium-dependent proton secretion in NHE3(-/-) mice was

237 Here we examined apical membrane PCT sodium-dependent proton secretion of wild-type (NHE3(+/+

238 The residual sodium-dependent proton secretion was inhibited by 100 m

239 Luminal sodium-dependent proton secretion was the same in NHE3(-

240 ) and wild-type mice had comparable rates of sodium-dependent proton secretion.

241 to isolate a 2653 bp cDNA encoding the mouse sodium-dependent, purine nucleoside selective, concentra

242 ntity to the previously cloned rat and human sodium-dependent, purine nucleoside selective, nucleosid

243 demonstrated that NHE3 activity, measured as sodium-dependent recovery of the intracellular pH after

244 tegral membrane proteins responsible for the sodium-dependent reuptake of small-molecule neurotransmi

245 o these facilitated urea transporters, three sodium-dependent, secondary active urea transport mechan

246 more potent and less reversible at blocking sodium-dependent short-circuit current than amiloride.

247 ies demonstrated that palytoxin stimulates a sodium-dependent signaling pathway that activates the c-

248 ults demonstrate that palytoxin stimulates a sodium-dependent signaling pathway that activates the SE

249 ls functionally and physically interact with sodium-dependent solute transporters, including myo-inos

250 ses in PIP2, SMIT1, and likely other related sodium-dependent solute transporters, regulates KCNQ cha

251 (2019) identified the location of the sodium-dependent spike initiation zone (Nav-SIZ) in noci

252 sometimes abolished by TTX, suggesting that sodium-dependent spikes play an important role in the tr

253 tions of tetrodotoxin citrate (TTX) to block sodium-dependent spiking; TTX+N-methyl-D-aspartic acid (

254 oposed S2 binding site, respectively, retain sodium-dependent substrate binding in the S1 site simila

255 talline LeuT samples and identify one set of sodium-dependent substrate-specific chemical shifts.

256 The endogenous sodium-dependent succinate transport in Caco-2 cells is

257 ice exhibited increased renal and intestinal sodium-dependent succinate uptake, as well as urinary hy

258 the role of a triad of aromatic residues in sodium-dependent sugar cotransporters (SGLTs).

259 y metabolism that seems to be coupled to the sodium-dependent synthesis of ATP.

260 ene expression of the hepatocyte basolateral sodium-dependent taurocholate cotransporter (Ntcp) to de

261 for the role of the farsenoid X receptor and sodium-dependent taurocholate cotransporting polypeptide

262 nion transporting polypeptide (OATP) 1B1 and sodium-dependent taurocholate cotransporting polypeptide

263 ition 120, epitope mutants displayed active, sodium-dependent taurocholate uptake.

264 Furthermore, sodium-dependent taurocholic acid uptake was inhibited b

265 y Slc10a1(-/-) hepatocytes showed absence of sodium-dependent taurocholic acid uptake, whereas sodium

266 The voltage dependence of the sodium-dependent transient currents of the Phe-294 mutan

267 D451E exhibited robust sodium-dependent transient currents with a voltage-depen

268 Sodium-dependent transmitter exchange and a transporter-

269 In basolateral plasma membrane vesicles, sodium-dependent transport for bile acids was reduced by

270 Sodium-dependent transport into astrocytes is critical f

271 We find: (a) sodium-dependent transport of ascorbate in mixed neurona

272 l epoxide hydrolase (mEH) is able to mediate sodium-dependent transport of bile acids such as tauroch

273 The transfected MDCK cells also exhibited sodium-dependent transport of cholate at levels 150% of

274 SDCT2 expressed in Xenopus oocytes mediated sodium-dependent transport of di- and tricarboxylates wi

275 enopus oocytes, SDCT1 mediated electrogenic, sodium-dependent transport of most Krebs cycle intermedi

276 Sodium-dependent transport of taurocholate was shown to

277 ikely presence of a NBMPR-insensitive and/or sodium-dependent transport system of the N2 (cit) type a

278 across the brush border membrane (BBM) via a sodium-dependent transporter, SGLT, and exit across the

279 omologies to an amino acid transporter and a sodium-dependent transporter.

280 quisqualate reach nuclear receptors via both sodium-dependent transporters and cystine glutamate exch

281 vo, resulting in a reduced driving force for sodium-dependent transporters and subsequently lower mus

282 Neuronal and glial sodium-dependent transporters are crucial for the contro

283 Sodium-dependent transporters clear extracellular glutam

284 nic nitrogen and phosphorus sources and more sodium-dependent transporters than a model freshwater cy

285 ntified apical targeting motifs in two other sodium-dependent transporters, and we suggest this conse

286 y regulation of transport through at least 2 sodium-dependent transporters: Slc23a1 and Slc23a2 (also

287 Sodium-dependent uptake of (3)H-taurocholate in renal br

288 transport function was assessed by examining sodium-dependent uptake of [3H]-taurocholate (TC) into b

289 Sodium-dependent uptake of bile acids across the hepatic

290 nobiotic metabolism as well as mediating the sodium-dependent uptake of bile acids into the liver, wh

291 ent an important class of proteins mediating sodium-dependent uptake of neurotransmitters from the ex

292 Polymorphisms in the genes encoding sodium-dependent vitamin C transport proteins are strong

293 The human sodium-dependent vitamin C transporter (hSVCT1) mediates

294 as well as the expression of transcripts for sodium-dependent vitamin C transporter (SVCT)-1 and SVCT

295 In embryos lacking the sodium-dependent vitamin C transporter 2 (SVCT2), very l

296 were shown to have two variants of the human sodium-dependent vitamin C transporter, hSVCT1; one is a

297 n UVA-irradiated lenses from human IDO/human sodium-dependent vitamin C transporter-2 mice, which con

298 ry active transport of ascorbate through the sodium-dependent vitamin C transporters SVCT1 and SVCT2

299 Human sodium-dependent vitamin C transporters, SVCT1 and SVCT2

300 Sodium-dependent vitamin C transporters, SVCT1 and SVCT2