戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 he enzymatic function of TET proteins during somatic cell reprogramming.
2 ns and predicts experimental observations of somatic cell reprogramming.
3 olecular rules necessary to achieve complete somatic cell reprogramming.
4 4 - Sox2 binding lncRNA that is activated in somatic cell reprogramming.
5 argets of ectopically induced factors during somatic cell reprogramming.
6 ation along with active transcription during somatic cell reprogramming.
7  oxidation steps, examining their effects on somatic cell reprogramming.
8 ferentiation (TD) is a recent advancement in somatic cell reprogramming.
9 ripotency of embryonic stem cells (ESCs) and somatic cell reprogramming.
10 ternative splicing regulatory network during somatic cell reprogramming.
11 he DKC1 complex in stem cell maintenance and somatic cell reprogramming.
12 t signaling has been implicated in promoting somatic cell reprogramming.
13 sed translation of p21, a known inhibitor of somatic cell reprogramming.
14 hway, which may be responsible for promoting somatic cell reprogramming.
15 epair-related molecular mechanisms affecting somatic cell reprogramming.
16  understand the molecular pathways governing somatic cell reprogramming.
17 s of modeling of dementia disorders based on somatic cell reprogramming.
18 r embryonic stem cell (ESC) self-renewal and somatic cell reprogramming.
19 nisms controlling stem cell pluripotency and somatic cell reprogramming.
20 al derivation of embryonic stem cells or via somatic cell reprogramming.
21 stablishing ground state pluripotency during somatic cell reprogramming.
22  an epigenetic barrier during the process of somatic cell reprogramming.
23 chondrial Akt1 signaling was required during somatic cell reprogramming.
24 repression presents a roadblock to efficient somatic cell reprogramming.
25 no p53 activity favors the entire process of somatic cell reprogramming.
26  for the maintenance of ESC self-renewal and somatic cell reprogramming.
27 issecting mechanisms of ESC pluripotency and somatic cell reprogramming.
28 tional induction at pluripotency loci during somatic cell reprogramming.
29 enerate them from embryonic stem cells or by somatic cell reprogramming.
30 SETSIP) was indentified to be induced during somatic cell reprogramming.
31 owed the examination of mechanisms governing somatic cell reprogramming.
32 lation of self-renewal, differentiation, and somatic cell reprogramming.
33 s hsa-miR-302b and hsa-miR-372 promote human somatic cell reprogramming.
34 s pluripotency factors with the capacity for somatic cell reprogramming.
35  the maintenance of ES cell self-renewal and somatic cell reprogramming.
36  govern self-renewal and differentiation and somatic cell reprogramming.
37 ruppel-like factor 4 (Klf4) is essential for somatic cell reprogramming.
38             Pluripotency can be recreated by somatic cell reprogramming.
39 ction is recapitulated in the culmination of somatic cell reprogramming.
40 iation and, conversely, acts as a barrier to somatic-cell reprogramming.
41                                              Somatic cell reprogramming also has been achieved more r
42 pluripotency, because its depletion inhibits somatic cell reprogramming and blastocyst development.
43  enhancers in biological processes including somatic cell reprogramming and guided differentiation.
44 e isolation of true hiPSCs immediately after somatic cell reprogramming and involves column-based pos
45 mental genes, cell differentiation, stem and somatic cell reprogramming and response to environmental
46  as FBXL10) controls stem cell self-renewal, somatic cell reprogramming and senescence, and tumorigen
47  drive cellular plasticity in the context of somatic cell reprogramming and tumorigenesis.
48 ly chromatin remodeler, in ESC self-renewal, somatic cell reprogramming, and blastocyst development.
49 onal regulator in embryonic stem (ES) cells, somatic cell reprogramming, and cancer.
50       Finally, Rif1 acts as a barrier during somatic cell reprogramming, and its depletion significan
51 nscriptome, although its functional roles in somatic cell reprogramming are largely unexplored.
52         The signaling mechanisms controlling somatic cell reprogramming are not fully understood.
53 ects of RNA metabolism, the roles of RBPs in somatic cell reprogramming are poorly understood.
54 t that C35 is an important tool for inducing somatic cell reprogramming, as well as for dissecting th
55 ctivation of the pluripotency network during somatic cell reprogramming by exogenous transcription fa
56        The MKL1-actin pathway weakens during somatic cell reprogramming by pluripotency transcription
57 evelopment in vitro and, increasingly due to somatic cell reprogramming, cellular and molecular mecha
58                                              Somatic cell reprogramming, directed differentiation of
59                                      Because somatic cell reprogramming during induced pluripotent st
60 r mitochondrial Akt1 signaling that enhanced somatic cell reprogramming efficiency.
61 ers of nine transcription factors, including somatic cell reprogramming factors (Oct4, Sox2, Klf4, an
62 nces in stem and progenitor cell biology and somatic cell reprogramming for applications directed to
63 7 depletion compromises ESC self-renewal and somatic cell reprogramming, globally increases m(6)A RNA
64                                              Somatic cell reprogramming has generated enormous intere
65 NAs and rsRNAs that are downregulated during somatic cell reprogramming impact cellular translation i
66 ming block of cell intermediates and enables somatic cell reprogramming in absence of otherwise essen
67 enhanced the ability of ES cells to initiate somatic cell reprogramming in heterokaryons.
68                                Nanog enables somatic cell reprogramming in serum-free medium suppleme
69                            In the context of somatic cell reprogramming, inhibition of ZNF398 abolish
70           MicroRNAs (miRNAs) are critical to somatic cell reprogramming into induced pluripotent stem
71 ctor ASCL1 regulates neurogenesis and drives somatic cell reprogramming into neurons.
72 ently discovered an unexpected phenomenon of somatic cell reprogramming into pluripotent cells by exp
73                                              Somatic cell reprogramming involves epigenomic reconfigu
74 of induced pluripotent stem cells (iPSCs) by somatic cell reprogramming involves global epigenetic re
75                 These findings indicate that somatic cell reprogramming is associated with marked inc
76                             Patient-specific somatic cell reprogramming is likely to have a large imp
77 tors modulate this process and contribute to somatic cell reprogramming is not clear.
78 hromatin architecture is reconfigured during somatic cell reprogramming is poorly understood.
79  how Klf4 regulates ES cell self-renewal and somatic cell reprogramming is still poorly understood.
80                                              Somatic cell reprogramming is the process that allows di
81 newal and acquisition of pluripotency during somatic cell reprogramming is well-documented.
82 licated in bypass of cellular senescence and somatic cell reprogramming, is markedly overexpressed in
83  the comprehension of the complex process of somatic cell reprogramming, many questions regarding the
84                                              Somatic cell reprogramming may afford models of nonfamil
85                              However, during somatic cell reprogramming, mesenchymal-epithelial trans
86 ompromised both pluripotency in ES cells and somatic cell reprogramming of fibroblasts to induced plu
87  whether such abnormalities are intrinsic to somatic cell reprogramming or secondary to the reprogram
88 tablishment of early epigenetic marks during somatic cell reprogramming: Parp1 functions in the regul
89  we describe an early and essential stage of somatic cell reprogramming, preceding the induction of t
90                 Transdifferentiation (TD), a somatic cell reprogramming process that eliminates pluri
91 or cytoskeletal remodeling in modulating the somatic cell reprogramming process.
92 in-specific protease 26 negatively regulates somatic cell-reprogramming process by stabilizing chromo
93             Even though different methods of somatic cell reprogramming result in stem cell lines tha
94 further enhanced global demethylation during somatic cell reprogramming (SCR) of hHFCs.
95                                The advent of somatic cell reprogramming technologies-which enables th
96  temporarily arrested in mitosis can support somatic cell reprogramming, the production of embryonic
97                                              Somatic cell reprogramming to a pluripotent state contin
98                    The recent description of somatic cell reprogramming to an embryonic stem (ES) cel
99                                     Although somatic cell reprogramming to generate inducible pluripo
100 of DNA double-strand breaks, is required for somatic cell reprogramming to induced pluripotent stem c
101 d analysis to identify kinases that regulate somatic cell reprogramming to iPSCs.
102                                              Somatic cell reprogramming to pluripotency requires an i
103 otent stem cells, including the promotion of somatic cell reprogramming to pluripotency, the regulati
104 s role in self-renewal, differentiation, and somatic cell reprogramming to pluripotency.
105 luripotency of embryonic stem cells, and for somatic cell reprogramming to the pluripotent state.
106 udy we found that during the early stages of somatic cell reprogramming toward a pluripotent state, s
107            The role of mitochondrial Akt1 in somatic cell reprogramming was investigated by transduci
108                                     Directed somatic cell reprogramming, which does not pass through
109  we show extensive TAD reorganization during somatic cell reprogramming, which is correlated with gen
110 ready rejuvenated by the maturation phase of somatic cell reprogramming, which suggests full reprogra
111  critical importance of the NHEJ pathway for somatic cell reprogramming, with a major role for LIG4 a
112 r identity and constitute a barrier to human somatic cell reprogramming; yet a comprehensive understa

 
Page Top