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1 0 mg or 40 mg of hyoscine butylbromide or no spasmolytic.
2 ly, the five tested ITCs exerted significant spasmolytic activity (on rat distal colon), with PhPeITC
3 lalkyl isothiocyanates were tested for their spasmolytic, cytotoxic and antimicrobial activities.
4      Prior to the examination, 20-40 mg of a spasmolytic drug, hioscine-N-butylobromide (Buscolysin((
5 ared with control subjects, patients given a spasmolytic had odds of 6.49 for clinically adequate dis
6 refoil family factor 2 (TFF2), also known as spasmolytic peptide, is a low-molecular-weight protein t
7 estrogen-inducable gene pS2 (BCEI) and human spasmolytic polypeptide (hSP/SML1).
8                       The trefoil pancreatic spasmolytic polypeptide (PSP) was tested for growth acti
9                            Recombinant human spasmolytic polypeptide (rHSP) or rat intestinal trefoil
10  trefoil auto- and cross-induction, and both spasmolytic polypeptide (SP) and ITF stimulation of gast
11 r (ITF, 59 residues), pS2 (60 residues), and spasmolytic polypeptide (SP, 106 residues) form a small
12          Both intestinal metaplasia (IM) and spasmolytic polypeptide (TFF2/SP) expressing metaplasia
13                      Trefoil factor family 2/spasmolytic polypeptide and intrinsic factor dual-immuno
14 adenocarcinoma are intestinal metaplasia and spasmolytic polypeptide expressing metaplasia (SPEM), de
15 broblasts, loss of parietal and chief cells, spasmolytic polypeptide expressing metaplasia, and dyspl
16 c marker of mouse gastric corpus metaplasia (spasmolytic polypeptide expressing metaplasia, SPEM).
17  family (pS2, intestinal trefoil factor, and spasmolytic polypeptide) are produced in the mammalian g
18 e described a trefoil factor family 2 (TFF2; spasmolytic polypeptide) expressing metaplasia (SPEM) as
19 refoil factor family 2 (TFF2), also known as spasmolytic polypeptide, is a member of the trefoil fami
20 ntified cellular and molecular mechanisms in spasmolytic polypeptide-expressing (pseudopyloric) metap
21               Intestinal metaplasia (IM) and spasmolytic polypeptide-expressing metaplasia (SPEM) are
22 develop spontaneous gastric inflammation and spasmolytic polypeptide-expressing metaplasia (SPEM) but
23 ers of wound-healing; these cells are called spasmolytic polypeptide-expressing metaplasia (SPEM) cel
24 enosis to re-enter the cell cycle and become spasmolytic polypeptide-expressing metaplasia (SPEM) cel
25                                              Spasmolytic polypeptide-expressing metaplasia (SPEM) dev
26  differentiation factor that correlates with spasmolytic polypeptide-expressing metaplasia (SPEM) in
27 ve gastritis and, ultimately, intestinalized spasmolytic polypeptide-expressing metaplasia (SPEM) was
28 s gastric inflammation, oxyntic atrophy, and spasmolytic polypeptide-expressing metaplasia (SPEM), a
29                                    To induce spasmolytic polypeptide-expressing metaplasia (SPEM), am
30 velops in the context of parietal cell loss, spasmolytic polypeptide-expressing metaplasia (SPEM), an
31 hy in the gastric corpus, a process known as spasmolytic polypeptide-expressing metaplasia (SPEM), in
32 emergence of a type of metaplasia designated spasmolytic polypeptide-expressing metaplasia (SPEM).
33 MBT1, were validated as specific markers for spasmolytic polypeptide-expressing metaplasia and intest
34 lasia can harbor both actively proliferating spasmolytic polypeptide-expressing metaplasia cells and
35 hief cells are recognized as mucus-secreting spasmolytic polypeptide-expressing metaplasia cells.
36 ker), YFP (lineage marker), and GSII lectin (spasmolytic polypeptide-expressing metaplasia marker) we
37                                 Induction of spasmolytic polypeptide-expressing metaplasia with DMP-7
38 lete intestinal metaplasia and proliferative spasmolytic polypeptide-expressing metaplasia) may be at
39 yntic atrophy, mucous neck cell hyperplasia, spasmolytic polypeptide-expressing metaplasia, dysplasia
40 ucous cell lineage that stained positive for spasmolytic polypeptide.
41 with DMP-777 leads to the rapid emergence of spasmolytic polypeptide/trefoil factor family 2 (TFF2)-e
42  substances also have antibacterial and anti-spasmolytic properties.
43 ly for centuries as a memory aid, analgesic, spasmolytic, vasorelaxant and antihypertensive, with rec