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1 A PET-derived parameters with serum prostate-specific antigen.
2 c response among KD patients and controls to specific antigen.
3 t are associated with pre-operative prostate-specific antigen.
4 ed with different concentrations of prostate specific antigen.
5 S) for real-time and label-free detection of specific antigen.
6 ineered dendritic cells (tolDCs) loaded with specific antigens.
7 ogeneity since antibodies can only recognize specific antigens.
8 ood, which suggests that they may experience specific antigens.
9 r T cells become activated against recipient-specific antigens.
10 stimulated with both unspecific and pathogen-specific antigens.
11 ic antigen < 10 ng/mL or Gleason 6, prostate-specific antigen, 10 to 20 ng/mL), LDR brachytherapy alo
14 t host T cells properly primed against tumor-specific antigens after conventional treatment, which ca
15 gh affinity and specificity of mAb for tumor-specific antigens allow these vesicular antibodies (VAs)
16 ove specificity over measurement of prostate-specific antigen alone for detecting cancer with Gleason
22 d with the singlet oxygen in the presence of specific antigens and emitted anti-Stokes fluorescence w
23 ndings highlight an important class of tumor-specific antigens and have implications for targeting ge
26 umors is hampered by the lack of truly tumor-specific antigens and poor control over T cell activity.
27 is relevant for most MCL cases, although the specific antigens and the precise location of affinity m
28 thod to rapidly discover conserved, pathogen-specific antigens and their epitopes, and applied it to
29 significant decrease in both KLK3 (prostate-specific antigen ) and FOLH1 (prostate-specific membrane
30 nalysis, controlling for age, serum prostate specific antigen, and abnormal digital rectal examinatio
31 nce status, higher testosterone and prostate-specific antigen, and lower hemoglobin than white men.
32 ression-free survival (PFS); tumor, prostate-specific antigen, and pain response; pharmacokinetics; a
33 by the pathogen, host antibody responses to specific antigens, and peripheral lymphocyte population
35 ity of pCB-(DOPA)4-modified paper sensor for specific antigen-antibody detection was demonstrated via
37 e cancers after the introduction of prostate-specific antigen-based screening are now being observed
38 ociated antigen-4) possessing an outer tumor-specific antigen-binding site engineered to shield the i
39 and explored their relation to the prostate-specific antigen blood value, the bone scan index (BSI),
40 ging and unspecific fluctuations in prostate-specific antigen can hamper early diagnosis of castratio
43 t anti-GRP78 AutoAb levels parallel prostate-specific antigen concentrations in patient-derived serum
45 iochemical response as defined by a prostate-specific antigen decline >/= 50% from baseline to at lea
47 30), but not when also adjusted for prostate-specific antigen density (PSAD; HR, 1.85; 95% CI, 0.99 t
48 ccurrence and strong immunogenicity of tumor-specific antigens derived from shared frameshift mutatio
49 Randomisation was stratified by prostate-specific antigen doubling time and baseline use of a bon
50 ion-resistant prostate cancer and a prostate-specific antigen doubling time of 10 months or less.
51 ion-resistant prostate cancer and a prostate-specific antigen doubling time of up to 10 months were r
52 risk for metastatic disease (rapid prostate-specific antigen doubling time or velocity) but otherwis
53 late autoimmune diseases induced with tissue-specific antigens emulsified in adjuvant oils, such as c
54 ansmission SPR measurements of free prostate specific antigen (f-PSA), which is similar in size to BS
56 mous cell carcinoma antigen (SCCA) is a good specific antigen for cancer diagnosis specifically for s
57 formed in 36 subjects with a rising prostate-specific antigen for known (n = 15) or suspected (n = 21
59 e sequential cleavage of the precursor group-specific antigen (Gag) polyprotein by HIV-1 protease.
60 es covering the packaging signal (PS), group-specific antigen (gag), polymerase (pol), and envelope (
61 rade model and clinical parameters (prostate-specific antigen, Gleason score) for pathologic EPE pred
62 received no previous chemotherapy; prostate-specific antigen greater than 5 ng/mL; and a Karnofsky p
64 monstrated a sustained reduction in prostate-specific antigen in a patient with CRPC, and another stu
65 trate femtomolar-level detection of prostate-specific antigen in biological fluids, as well as reduce
66 ion of this technology in detecting Prostate Specific Antigen in clinically relevant levels (ng/ml),
68 ultaneous detection of antibody binding with specific antigens in arrays containing 96- and 384-spots
69 stochemically by antibodies that target cell-specific antigens in the cytosol or plasma membrane.
70 hat demonstrates for the first time that ASD-specific antigen-induced maternal autoantibodies produce
71 s the MRI-based EPE grading system (prostate-specific antigen, International Society of Urological Pa
72 ng carcinomas, subsequently releasing tumour-specific antigens into the tumour microenvironment highl
77 ebo (Arm P; n = 433), stratified by prostate-specific antigen (less than 50 ng/mL v 50 ng/mL or more)
78 ical failure was defined as a serum prostate-specific antigen level > 0.2 ng/mL that increased on 2 c
79 nd/or radiation therapy with rising prostate-specific antigen level (median, 2.27 ng/mL; range, 0.2-2
80 lobin level (r = -0.521, P < .001), prostate-specific antigen level (r = 0.556, P < .001), lactate de
81 n tDV and best percentage change in prostate-specific antigen level and circulating tumor cell count
82 gistic regression; correlation with prostate-specific antigen level and circulating tumor cell count
84 d Gleason scores of 7-10 and a mean prostate-specific antigen level of 7.8 mug/L (range, 5.4-10.6 mug
85 e cancer, particularly those with a prostate-specific antigen level of less than 10 ng/mL and Gleason
87 tate-specific antigen relapse (mean prostate-specific antigen level, 5 ng/mL; range, 0.25-294 ng/mL),
90 Hypofractionation, pretreatment prostate-specific antigen level, Gleason score, and clinical tumo
91 astatic findings were compared with prostate-specific antigen level, International Society of Urologi
92 and was associated with increasing prostate-specific antigen level, ISUP grade, and clinical stage.
95 tal examination results or elevated prostate-specific antigen levels (age groups: 41-50 years, 51-60
96 say was superior to the analysis of prostate-specific antigen levels (area under the curve: 0.94 vers
97 f circulating MDSCs correlates with prostate-specific antigen levels and metastasis in patients with
98 ineate the relationship between the prostate-specific antigen levels and the diffusion parameters as
99 of 140 patients with elevated serum prostate specific antigen levels and/or abnormal digital rectal e
100 by applying mathematical models to prostate-specific antigen levels as the representation of tumour
101 -2, localized prostate cancer, with prostate-specific antigen levels less than 50 ng/mL, and enrolled
102 = 16), after surgery and with serum prostate-specific antigen levels lower than 0.2 ng/mL (n = 13), a
107 te risk prostate cancer (Gleason 7, prostate-specific antigen < 10 ng/mL or Gleason 6, prostate-speci
108 = 7, clinical stage T1b to T3a, and prostate-specific antigen < 30 ng/mL were randomly allocated to n
110 g detection of high levels of serum prostate-specific antigen, many men are advised to have transrect
111 e, biopsy at 12-month intervals and prostate-specific antigen measurement and digital rectal examinat
113 Lake Biwa and speculated that a novel koayu-specific antigen might have been the cause of the condit
115 curred most often for patients with prostate-specific antigen of 0.5 to less than 2.0 ng/mL (81/147,
117 1gp49) which binds and cleaves B-band LPS (O-specific antigen, OSA) of Pseudomonas aeruginosa PAO1.
118 tors common across the trials (age, prostate-specific antigen, performance status, alkaline phosphata
119 id cocktail expressing HIV-1 subtype C group-specific antigen, polymerase, and envelope antigen genes
120 mbinant canarypox virus expressing HIV group-specific antigen, polymerase, and envelope genes) follow
122 matory response in sarcoidosis is induced by specific antigens, possibly including self-antigens, whi
123 target solid tumors without the necessity of specific antigen presentation on tumors are developed.
124 B7-CD28 co-stimulation and B7 expression by specific antigen-presenting cell (APC) types are require
126 owever, gaps in our knowledge as to dormancy-specific antigens prevent a relapse preventing vaccine d
130 igible men had an elevated level of prostate-specific antigen (PSA) (>0.2 ng/mL) and high-risk featur
131 All patients had a rising level of prostate-specific antigen (PSA) (range, 0.3-119.0 ng/mL; mean, 10
134 erapeutic response as determined by prostate-specific antigen (PSA) and alkaline phosphatase (ALP), a
137 high Gleason score, clinical stage, prostate-specific antigen (PSA) and extent of disease, as well as
138 ate MR imaging in men with elevated prostate-specific antigen (PSA) and negative transrectal ultrason
139 evaluated the relationship between prostate-specific antigen (PSA) and overall survival in the conte
140 s decorated with antibodies against prostate specific antigen (PSA) and prostate specific membrane an
141 mune complexes of the cancer marker prostate-specific antigen (PSA) are detected and counted by wide-
143 roxy-2'-deoxyguanosine (8-OHdG) and prostate specific antigen (PSA) as representatives for small mole
144 f prostate volume, uroflowmetry and prostate specific antigen (PSA) at one, 3 and 6 months and one ye
146 ned to utilize for the detection of prostate specific antigen (PSA) based on three different generati
148 post-prostatectomy surveillance of prostate specific antigen (PSA) can be achieved with a detection
149 ratification systems use presenting prostate-specific antigen (PSA) concentration, biopsy Gleason gra
151 ific quality of life, health worry, prostate-specific antigen (PSA) concern, and outlook on life.
152 Men who did not achieve >/= 30% prostate-specific antigen (PSA) decline by cycle 4 (C4) switched
154 ation-sensitive prostate cancer and prostate-specific antigen (PSA) doubling time (DT) of less than 1
155 sion criterion for this study was a prostate-specific antigen (PSA) doubling time of less than 12 mo
157 CT is increasingly used in men with prostate-specific antigen (PSA) failure after radical prostatecto
158 ions of histopathology, imaging, or prostate-specific antigen (PSA) follow up, defined as composite r
159 Surveillance was performed with prostate-specific antigen (PSA) follow-up every 3 months, with re
160 quantitatively precise analysis of prostate-specific antigen (PSA) in 10 min from merely one drop of
162 ndothelial growth factor (VEGF) and prostate-specific antigen (PSA) in human serum for early diagnosi
163 bel-free and real-time detection of prostate specific antigen (PSA) in human serum using silicon nano
170 mine the relationship between serum prostate-specific antigen (PSA) level categories (<5, 5-10, 10-20
171 on therapy (ADT), as reflected by a prostate-specific antigen (PSA) level higher than 4.0 ng/mL after
174 state cancer in 60% of cases with a prostate-specific antigen (PSA) level of >=0.4 to <0.5, 78% with
177 -free survival as determined by the prostate-specific antigen (PSA) level, clinical progression-free
178 tive biopsy findings with increased prostate-specific antigen (PSA) level, or (c) had a prior history
179 rence was correlated with patients' prostate-specific antigen (PSA) level, primary Gleason score, and
180 ent characteristics, including age, prostate-specific antigen (PSA) level, PSA density, race, digital
182 e blood of 72 asymptomatic men with Prostate-Specific Antigen (PSA) levels < 20 ng ml(-1), of whom 31
183 an imaging protocol based on rising prostate-specific antigen (PSA) levels (mean, 3.43 ng/mL; median,
184 an imaging protocol based on rising prostate-specific antigen (PSA) levels (mean:3.43 ng/mL, median:0
185 of 85%, which increased with higher prostate-specific antigen (PSA) levels (ng/mL): 50% (PSA < 0.5),
186 scan results (P < 0.001) and higher prostate-specific antigen (PSA) levels (P = 0.024) were associate
187 MA I&T PET/CT because of increasing prostate-specific antigen (PSA) levels after radical prostatectom
188 icant proportion of men with rising prostate-specific antigen (PSA) levels after radical prostatectom
189 t statistical model of longitudinal prostate-specific antigen (PSA) levels and risks for biopsy upgra
192 tients with postprostatectomy serum prostate-specific antigen (PSA) levels of at least 0.2 ng/mL and
193 state cancer and rapidly increasing prostate-specific antigen (PSA) levels while taking androgen-depr
194 survival of patients with different prostate-specific antigen (PSA) levels, Gleason scores, marital s
195 in Solid Tumors 1.1), a decrease in prostate-specific antigen (PSA) of 50% or more (PSA50) from basel
196 red eighteen patients with a median prostate-specific antigen (PSA) of 6.4 ng/mL (range, 2.2-158.4 ng
197 s in a selected patient cohort with prostate-specific antigen (PSA) persistence after salvage lymph n
198 tive radiotherapy, 51% (88/172) for prostate-specific antigen (PSA) persistence, and 36% (62/172) for
199 week 13 (PET2), and at the time of prostate-specific antigen (PSA) progression, standard radiographi
201 nued until radiologic, clinical, or prostate-specific antigen (PSA) progression; otherwise, treatment
204 investigators) and locally assessed prostate-specific antigen (PSA) response (>= 50% decrease from ba
207 13 circulating tumor cell (CTC) and prostate-specific antigen (PSA) response end points in five prosp
208 Primary objectives were: confirmed prostate-specific antigen (PSA) response rate (RR) and whether ET
210 GBq); afterward, safety lab tests, prostate-specific antigen (PSA) response, and clinical findings w
211 tive disease associated with better prostate-specific antigen (PSA) responses (100% vs. 54%, P = 0.03
213 on on enzalutamide with a continued prostate-specific antigen (PSA) rise after enzalutamide treatment
215 fication to identify those for whom prostate-specific antigen (PSA) testing is likely to be most valu
217 ood and Drug Administration) is the prostate specific antigen (PSA) that is detected by conventional
218 ngle molecules of the cancer marker prostate specific antigen (PSA) using photon-upconversion nanopar
221 n of recurrent disease at low serum prostate specific antigen (PSA) values below 0.5 ng/mL compared w
222 o standard-of-care imaging at serum prostate-specific antigen (PSA) values low enough to affect targe
223 rd, data were compared with current prostate-specific antigen (PSA) values, Gleason score (GS), and d
229 low rate, postvoid residual volume, prostate-specific antigen (PSA), and prostate volume were assesse
230 tients with renewed increase in the prostate-specific antigen (PSA), commonly referred to as biochemi
231 omes was constructed combining age, prostate-specific antigen (PSA), histological grade, biopsy core
232 ription of AR target genes, such as prostate-specific antigen (PSA), is also lowered in the HSP90 Thr
233 led for PSMA RLT were evaluated for prostate-specific antigen (PSA), lactate dehydrogenase (LDH), and
235 e implemented for glycoprofiling of prostate specific antigen (PSA), what can be applied for better p
236 ent-free survival (EFS), an earlier prostate-specific antigen (PSA)-based composite end point, may fu
237 individualised decision-making for prostate-specific antigen (PSA)-based screening in men aged 55-69
243 able for the primary end point of a prostate-specific antigen (PSA)50 response (PSA decline >=50% at
244 thin films as transducers to detect prostate specific antigens (PSA) in a physiological buffer soluti
245 of either biochemical progression (prostate-specific antigen [PSA] >=0.4 ng/mL and rising after comp
246 sk (clinical stage cT1 to cT2bN0M0, prostate-specific antigen [PSA] <=20 ng/mL, and Grade Group 1-2)
247 (T1 to 2a, Gleason score </= 6, and prostate-specific antigen [PSA] 10.1 to 20 ng/mL; T2b to 2c, Glea
248 formance of 4 candidate surrogates (prostate-specific antigen [PSA] failure, PSA nadir >0.5 ng/mL, PS
249 P; that is, persistently measurable prostate-specific antigen [PSA] values after robot-assisted lapar
250 al stage, T1c; Gleason score, </=6; prostate-specific antigen [PSA], <10 ng/mL; PSA density <0.15 ng/
251 intermediate-risk prostate cancer (prostate-specific antigen [PSA], <=15 ng/mL; Gleason score, <=3 +
252 nted biochemical recurrence (median prostate-specific antigen [PSA], 2.5 ng/mL; range, 0.21-35.5 ng/m
253 ucing alphabeta T cell clones with psoriasis-specific antigen receptors exist in clinically resolved
254 at delivery of liposomes containing both the specific antigen recognized by the mast cell-bound IgE a
256 ean age, 68 y; range, 44-87 y) with prostate-specific antigen relapse (mean prostate-specific antigen
258 ; 95% CI, 0.40 to 0.68; P<0.001), a prostate-specific antigen response occurred in 35.7% and 13.5% of
259 le response, mainly as defined by a prostate-specific antigen response of more than 50%, comparable t
260 xploratory analyses showed a higher prostate-specific antigen response rate with ipilimumab (23%) tha
261 es in progression-free survival and prostate-specific antigen response rates suggest antitumor activi
263 every 2 wk by laboratory tests, the prostate-specific antigen response was checked every 4 wk, and ot
264 -positive mCRPC had fewer confirmed prostate-specific antigen responses (0% to 11%) or soft tissue re
269 Studies demonstrate that use of prostate-specific antigen screening decreased significantly follo
272 the AR-responsive gene that encodes prostate specific antigen, shows the greatest variability in expr
273 esponse, whereas patient 1 achieved prostate-specific antigen stabilization after 3 therapy cycles.
277 cells displayed enhanced expression of stage-specific antigens that extravillous CTBs normally upregu
279 was shown to supress expression of prostate specific antigen, the cardinal clinical biomarker of pro
280 the type as well as level of expressed tumor-specific antigens, thereby presenting methods for select
281 of biomaterial scaffolds loaded with disease-specific antigens to identify and study rare, therapeuti
283 Re-activation and clonal expansion of tumor-specific antigen (TSA)-reactive T cells are critical to
284 low-abundance MHC I peptides including tumor-specific antigens (TSAs) and minor histocompatibility an
287 l) and tumor characteristics (serum prostate-specific antigen, tumor grade and clinical stage) and by
288 ns limited by the rarity of targetable tumor-specific antigens, tumor-mediated immune suppression, an
289 %) demonstrated a >= 50% decline in prostate-specific antigen; two (6%) of 36 with measurable disease
291 rrelations were found between serum prostate-specific antigen value and both PSMA-TV (r = 0.72, P < 0
292 The patients had a median prescan prostate-specific antigen value of 12.2 ng/mL (range, 1.2-81.6 ng
294 ts with prostate cancer BCR (median prostate-specific antigen value, 1.7 ng/mL; range, 0.05-202 ng/mL
296 0.74, respectively, P < 0.001) and prostate-specific antigen values (r = 0.57 and 0.54, respectively
297 ad undergone PSMA PET for CRPC, had prostate-specific antigen values of at least 1 ng/mL, and had und
299 erspective explore the detection of prostate-specific antigen which enables a comparison between stra
300 l outputs, the PT-Disk can quantify prostate specific antigen with limits of detection of 1.4-2.8 ng