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1 particularly Tourette's disorder and autism spectrum disorder.
2 ations of patients with neuromyelitis optica spectrum disorder.
3 variants in TMPRSS9 are related to an autism spectrum disorder.
4 on-deficit/hyperactivity disorder and autism spectrum disorder.
5 disorders including schizophrenia and autism spectrum disorder.
6 indings compatible with neuromyelitis optica spectrum disorder.
7 also to some degree with findings in autism spectrum disorder.
8 icated in intellectual disability and autism spectrum disorder.
9 on deficit hyperactivity disorder and autism spectrum disorder.
10 Pten mutations are associated with autism spectrum disorder.
11 eatures among individuals with fetal alcohol spectrum disorder.
12 neurodevelopmental disorders such as autism spectrum disorder.
13 ntial risk factors and biomarkers for autism spectrum disorder.
14 sociability in three mouse models of autism spectrum disorder.
15 nnectivity in individuals with fetal alcohol spectrum disorder.
16 find a significant relationship with Autism Spectrum Disorder.
17 of participants, including some with autism spectrum disorder.
18 date therapeutics in a mouse model of autism spectrum disorder.
19 57 subjects have a diagnosis of Autism Spectrum Disorder.
20 ifferences between men and women with autism spectrum disorder.
21 is associated with both epilepsy and autism spectrum disorders.
22 al deficits typically associated with autism spectrum disorders.
23 ication is a common characteristic in autism spectrum disorders.
24 of glutamate and liability for schizophrenia spectrum disorders.
25 ine dynamics, Parkinson's disease and autism spectrum disorders.
26 expected, specific for neuromyelitis optica spectrum disorders.
27 potential target for the treatment of autism spectrum disorders.
28 erm health conditions, such as fetal alcohol spectrum disorders.
29 us in a postnatal rat model of fetal alcohol spectrum disorders.
30 may serve as risk factors for schizophrenia spectrum disorders.
31 clinical outcomes in patients with psychosis spectrum disorders.
32 r neurodevelopmental issues including autism spectrum disorders.
33 ing youth as well as in those with psychosis spectrum disorders.
34 : Despite the rarity of neuromyelitis optica spectrum disorder, a relative abundance of preventive tr
36 d pairs con- or discordant for schizophrenia spectrum disorders, along with healthy control pairs (MZ
37 ns of videogame play in 39 youth with autism spectrum disorder and 23 age-matched youth with typical
38 s 56.4% (95% CI 55.4-57.4) for schizophrenia spectrum disorder and 49.7% (95% CI 48.1-51.3) for bipol
39 or childhood-onset disorders, such as autism spectrum disorder and attention-deficit/hyperactivity di
41 wo related neuropsychiatric disorders-autism spectrum disorder and bipolar disorder-to show that it i
42 of de novo variants in patients with autism spectrum disorder and intellectual disability or develop
43 atedly identified as risk factors for autism spectrum disorder and intellectual disability, but it ha
45 brain tissue in Alzheimer's disease, autism spectrum disorder and multiple sclerosis is significantl
52 st conceptualized as a mitochondrial disease spectrum disorder and should be routinely included in ge
53 tained CSF in 32 patients with schizophrenia spectrum disorders and 32 healthy volunteers and periphe
54 Twenty-seven patients with schizophrenia spectrum disorders and 36 healthy control subjects compl
55 ree subjects (43 with depression and anxiety spectrum disorders and 40 healthy controls) completed se
57 n Shank3 are strongly associated with autism spectrum disorders and neural circuit changes in several
59 ADHD traits occur in individuals with autism spectrum disorder, and autism spectrum disorder traits i
60 ental delay, intellectual disability, autism spectrum disorder, and behavioral abnormalities as well
65 ctual disability/developmental delay, autism spectrum disorder, and multiple medical comorbidities.
67 ng control youths, 194 youths with psychosis spectrum disorders, and 246 youths with other psychopath
68 ntion-deficit hyperactivity disorder, autism spectrum disorder, anxiety disorders, seizures and epile
71 ral nervous system or associated with autism spectrum disorder, as well as 773 genes under unusually
72 ment and behavior in 96 children with autism spectrum disorder (ASD) [45 with neurodevelopmental regr
75 tigated neuroradiological findings in autism spectrum disorder (ASD) and attention-deficit hyperactiv
76 s and neurogenetic syndromes, such as autism spectrum disorder (ASD) and Dravet, Fragile X, Prader-Wi
78 link between DIP2A gene variations in autism spectrum disorder (ASD) and highlight the contribution o
80 urodevelopmental disorders, including autism spectrum disorder (ASD) and intellectual disability (ID)
81 e been identified in individuals with autism spectrum disorder (ASD) and intellectual disability; how
82 oted consistently in individuals with autism spectrum disorder (ASD) and their families, including th
83 from 1,902 individuals affected with autism spectrum disorder (ASD) and their unaffected siblings in
84 for genes implicated in non-syndromic autism spectrum disorder (ASD) and were differentially expresse
85 of approximately 8,000 children with autism spectrum disorder (ASD) and/or attention deficit hyperac
89 on (BM) and its putative anomalies in autism spectrum disorder (ASD) are not completely clarified.
90 ciality in individuals diagnosed with autism spectrum disorder (ASD) are standardly explained in term
91 rain have been previously reported in autism spectrum disorder (ASD) but these alterations are hetero
93 ural suppression in young adults with autism spectrum disorder (ASD) compared to neurotypical control
97 GNIFICANCE STATEMENT Individuals with autism spectrum disorder (ASD) exhibit atypical attentional beh
99 hesized link between gut bacteria and autism spectrum disorder (ASD) has been explored through animal
103 f dysregulation of gene expression in autism spectrum disorder (ASD) have been reported, the role of
105 vo histological postmortem studies of autism spectrum disorder (ASD) have shown gray matter microstru
106 s been hypothesized to play a role in autism spectrum disorder (ASD) in an attempt to explain some of
118 eatment of ASD.SIGNIFICANCE STATEMENT Autism spectrum disorder (ASD) is a neurodevelopmental psychiat
135 s with etiological heterogeneity like Autism Spectrum Disorder (ASD) often pose a challenge for tradi
138 ize their diagnoses, individuals with autism spectrum disorder (ASD) show enhanced performance in sim
139 association between macrocephaly and autism spectrum disorder (ASD) suggests that the mechanisms und
141 re features in schizophrenia (SZ) and autism spectrum disorder (ASD) that have been linked to atypica
143 the largest exome sequencing study of autism spectrum disorder (ASD) to date (n = 35,584 total sample
144 l patients had autistic features, and autism spectrum disorder (ASD) was diagnosed in 5 of 11 (45%).
145 particular challenges for youths with autism spectrum disorder (ASD) who often struggle to behave in
146 in PTEN account for ~10% of cases of autism spectrum disorder (ASD) with coincident macrocephaly.
147 families containing individuals with autism spectrum disorder (ASD)(2,3) and population control indi
148 , is the leading monogenetic cause of autism spectrum disorder (ASD), a neurodevelopmental condition
150 ssociated with X-linked macrocephaly, autism spectrum disorder (ASD), and intellectual disability.
151 notypes in individuals diagnosed with autism spectrum disorder (ASD), and may potentially contribute
152 eficit hyperactivity disorder (ADHD), autism spectrum disorder (ASD), and obsessive-compulsive disord
153 ently disrupted in male subjects with autism spectrum disorder (ASD), but the functional consequences
154 y known about sleep in the context of autism spectrum disorder (ASD), including ASD-related condition
155 t neuropsychiatric diseases including Autism Spectrum Disorder (ASD), intellectual disabilities and P
156 ations of 16p11.2 are associated with autism spectrum disorder (ASD), intellectual disability (ID), a
158 cts of oxytocin on social deficits in autism spectrum disorder (ASD), no large-scale study has been c
160 among the most dangerous concerns in autism spectrum disorder (ASD), often requiring detailed and te
161 Variants (CNVs) confer high risk for Autism Spectrum Disorder (ASD), schizophrenia (SZ), and Attenti
162 are prone to developing macrocephaly, autism spectrum disorder (ASD), seizures and intellectual disab
163 ronmental risk factor contributing to autism spectrum disorder (ASD), which compromises fetal brain d
164 eurons are increasingly used to model Autism Spectrum Disorder (ASD), which is clinically and genetic
204 ies contributes to the development of autism spectrum disorders (ASD) has been entertained for over a
208 ion, is one of the possible causes of autism spectrum disorders (ASD) predominantly in patients with
210 ilitating symptom in individuals with autism spectrum disorders (ASD), but the neural basis underlyin
211 negative translation regulators cause autism spectrum disorders (ASD), which predominantly affect mal
212 ptoms following oxytocin treatment in autism spectrum disorders (ASD), while others point to endogeno
214 e presented to adult individuals with autism spectrum disorders (ASD; n = 24, mean age 23 years, 8 fe
215 response was altered in children with autism spectrum disorders (ASDs) and other neurological disorde
216 dependent ASDs.SIGNIFICANCE STATEMENT Autism spectrum disorders (ASDs) are linked to abnormal dendrit
217 of genetic biomarkers associated with autism spectrum disorders (ASDs) could improve recurrence predi
220 ajor challenge facing the genetics of autism spectrum disorders (ASDs) is the large and growing numbe
221 tal defects.SIGNIFICANCE STATEMENT In autism spectrum disorders (ASDs), prenatal infection or materna
223 dysfunction has been demonstrated in autism spectrum disorders (ASDs); however, the circuits underly
224 nteraction (SI) are a core symptom of autism spectrum disorders (ASDs); however, treatments for socia
226 sychiatric disorders [e.g., increased autism spectrum disorder, attention deficit hyperactivity disor
227 rocephaly, and psychiatric disorders (autism spectrum disorders/attention deficit hyperactivity disor
228 clinical phase 2 study in adults with autism spectrum disorder balovaptan demonstrated improvements i
229 eficit hyperactivity disorder (ADHD), autism spectrum disorder, bipolar disorder, major depressive di
230 y of psychiatric disorders, including autism spectrum disorder, bipolar disorder, major depressive di
232 ognized for its involvement in fetal alcohol spectrum disorders, but the lack of clear understanding
235 proportion of individuals with schizophrenia spectrum disorders (F20-29) who received any F20-29 diag
236 tional alcohol exposure causes fetal alcohol spectrum disorder (FASD) and is a prominent cause of neu
237 utes to the high prevalence of fetal alcohol spectrum disorder (FASD) is binge-like consumption of al
238 ng prenatal development causes fetal alcohol spectrum disorder (FASD), the most frequent preventable
242 on In participants with neuromyelitis optica spectrum disorders, focal areas of spinal cord atrophy a
244 peated administration of oxytocin for autism spectrum disorder have led researchers to hypothesize th
245 urodevelopmental disorders, including autism spectrum disorder, have complex polygenic etiologies.
246 tes of social behavior in adults with autism spectrum disorder in an exploratory proof-of-mechanism s
249 a behavioral phenotype suggestive of autism spectrum disorder in the xCT(-/-) mice; in tests sensiti
250 d randomised controlled trials (RCTs) in FTD spectrum disorders in order to identify limitations and
251 nd mood disorders, schizophrenia, and autism spectrum disorder, in which they have been identified.
253 red cognition is a hallmark of schizophrenia spectrum disorders, including schizotypal personality di
254 urodevelopmental disorders, including autism spectrum disorder, intellectual disability, and epilepsy
255 ntion-deficit/hyperactivity disorder, autism spectrum disorder, intelligence, and educational attainm
260 hough the prevalence of neuromyelitis optica spectrum disorder is limited to around 1-2 people per 10
263 hallenge in developing treatments for autism spectrum disorders is the heterogeneity of the condition
264 lirubin encephalopathy (ABE) and kernicterus spectrum disorder (KSD) are common newborn health condit
265 ygdala connectivity in youths with psychosis spectrum disorders may help us better understand the neu
267 N = 696), bipolar disorder (N = 211), autism spectrum disorder (N = 126), or major depressive disorde
268 turb-Seq, to functionally evaluate 35 autism spectrum disorder/neurodevelopmental delay (ASD/ND) de n
269 H17) drive pathology in neuromyelitis optica spectrum disorder (NMOSD) and in TH17-induced experiment
270 (AQP4-Ab)-seronegative neuromyelitis optica spectrum disorder (NMOSD) and related syndromes (optic n
271 %, AQP4-IgG-seropositive neuromyelitis optic spectrum disorder (NMOSD) in 3%, infectious type in 2%,
273 elapse in patients with neuromyelitis optica spectrum disorder (NMOSD) when added to immunosuppressan
279 correlated with liability for schizophrenia spectrum disorders (phenotypic correlation, 0.16, [0.02-
280 A pre-emptive intervention for the autism spectrum disorder prodrome had no immediate treatment ef
281 essive disorder, depressive symptoms, autism spectrum disorder, psychosis, and alcohol use disorder c
282 mosaicism quantification can stratify autism spectrum disorders recurrence risk due to de novo mutati
283 tegorical approach to defining schizophrenia spectrum disorders requires meeting established criteria
284 tive, and quantitative measurement of autism spectrum disorder's core symptoms are lacking, making it
285 d neurodegenerative disorders such as autism spectrum disorder, schizophrenia, and Huntington's disea
286 from RNA-sequencing studies including autism spectrum disorder, schizophrenia, and Zika virus induced
287 cohort were intellectual disability, autism spectrum disorder, seizures, variable brain malformation
288 schizophrenia, bipolar disorder, and autism spectrum disorder share similar white matter microstruct
289 ), myelitis (13 [11%]), neuromyelitis optica spectrum disorders (six [5%]), and other disorders (nine
291 ultiple sclerosis (e.g. neuromyelitis optica spectrum disorders, Susac syndrome), thus making the dia
292 o immediate treatment effect on early autism spectrum disorder symptoms, the quality of parent-child
293 s multiple sclerosis or neuromyelitis optica spectrum disorder; systemic diseases such as systemic lu
294 umab) for patients with neuromyelitis optica spectrum disorder that all showed a benefit in preventin
295 ffects treatment approaches in posttraumatic spectrum disorders, their etiology remains poorly unders
297 ls with autism spectrum disorder, and autism spectrum disorder traits in individuals with ADHD, the n
298 l of synaptic dysfunction relevant to autism spectrum disorders, were highly replicable across three
299 chondrial protein, are implicated in ALS-FTD spectrum disorders, which are pathologically characteriz
300 ion to heterogenous disorders such as autism spectrum disorder will require individual-subject analys