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1 otocol may decrease the rates of devastating spinal cord ischemia.
2 sion with subsequent reperfusion to generate spinal cord ischemia.
3  arteries are effective for the treatment of spinal cord ischemia.
4 on systemic blood pressure in a rat model of spinal cord ischemia.
5 onstrated to be neuroprotective in brain and spinal cord ischemia.
6  microl/day for 7 days prior to induction of spinal cord ischemia.
7                                              Spinal cord ischemia after thoracoabdominal aortic inter
8                           The progression of spinal cord ischemia after thoracoabdominal aortic inter
9 orta for 60 minutes results in high rates of spinal cord ischemia, an effect mitigated by limiting ba
10      Rabbits were subjected to 30 minutes of spinal cord ischemia and reperfusion (1 hour) with or wi
11 azoxide improves neurological function after spinal cord ischemia and reperfusion by diminishing leve
12              The mechanism of the antecedent spinal cord ischemia and reperfusion injury (IR) remains
13                                              Spinal cord ischemia and reperfusion injury occurs in 2
14 otection protocol could decrease the risk of spinal cord ischemia and/or paraplegia and consists of p
15  a critical determinant of outcome following spinal cord ischemia, and controlled peri-operative bloo
16 y matter involvement similar to that seen in spinal cord ischemia, and three (16%) had isolated white
17                             Risk factors for spinal cord ischemia are aneurysm extent, open surgical
18        Strategies for mitigating the risk of spinal cord ischemia are discussed, drawing on evidence
19 y early detection and immediate treatment of spinal cord ischemia before it evolves to infarction.
20                           Early detection of spinal cord ischemia by intraoperative neurophysiologic
21                   There have been 6 cases of spinal cord ischemia during pancreas transplantation sin
22 es the potential catastrophic consequence of spinal cord ischemia from an unexpectedly prolonged aort
23 ctors and interventions to prevent and treat spinal cord ischemia has the potential to prevent spinal
24 ive events associated with increased IM were spinal cord ischemia (HR = 9.46; 95% CI: 3.98-22.47; P <
25 atory complications in 45 patients (22%) and spinal cord ischemia in 22 (11%), of whom 10 (45%) fully
26 ings, there is approximately a 1:440 risk of spinal cord ischemia in pancreas transplantation.
27 im of this study was to quantify the risk of spinal cord ischemia in pancreatic transplantation.
28                             Risk factors for spinal cord ischemia include extensive aortic repair, pr
29  are consistent with the idea that transient spinal cord ischemia induces the formation of a death-in
30                              Paraplegia from spinal cord ischemia is a devastating complication of th
31 id receptor function, in a rabbit reversible spinal cord ischemia model (RSCIM).
32 istered intrathecally (i.t.) in a reversible spinal cord ischemia model (RSCIM).
33                                    Permanent spinal cord ischemia occurred in 4.5% and stroke in 3%.
34 Neurologic events included stroke (5.0%) and spinal cord ischemia (permanent 1.7%, temporary 7.9%).
35                                              Spinal cord ischemia remains an important complication o
36                          We hypothesize that spinal cord ischemia-reperfusion injury after aortic occ
37                     The elusive mechanism of spinal cord ischemia-reperfusion injury has delayed the
38 unction, DNA damage-repair, and apoptosis in spinal cord ischemia-reperfusion injury.
39 ntials (MEPs) monitoring can promptly detect spinal cord ischemia (SCI) from aortic clamping during o
40 ed paralysis with histopathologic changes of spinal cord ischemia (SCI) in 4/8 (50%), with no SCI in
41                                              Spinal cord ischemia (SCI) is a rare but devastating con
42  has demonstrated low risks of mortality and spinal cord ischemia (SCI), but few large series have be
43 NGF) to reduce functional deficits following spinal cord ischemia was investigated.