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1 jury site but also rostral and caudal to the spinal injury.
2 al motor score from baseline to 1 year after spinal injury.
3 h by corticospinal tract (CST) neurons after spinal injury.
4 out S1 CST involvement in recovery following spinal injury.
5 etour in restoration of motor function after spinal injury.
6 orelimb movements, and can be regained after spinal injury.
7 r function is important in rehabilitation of spinal injury.
8 sponsible for a lack of neurogenesis after a spinal injury.
9 uitable as a therapeutic approach to chronic spinal injury.
10 h volunteers and in patients with incomplete spinal injury.
11 tion in multiple motor systems after chronic spinal injury.
12 es influencing functional recovery following spinal injury.
13 ency in patients with, for example, cervical spinal injury.
14  plasticity, which may foster recovery after spinal injury.
15 ependent response of the spinal cord after a spinal injury.
16 tical representation of tactile events after spinal injury.
17  transmit neural signals to bypass a chronic spinal injury.
18 ing performance, particularly after neonatal spinal injury.
19 ving transgenic mice over several days after spinal injury.
20 ders such as respiratory insufficiency after spinal injury.
21 osed as a strategy to improve movement after spinal injury.
22  to improve walking ability in patients with spinal injury.
23 equired preconditions established by chronic spinal injury.
24  incontinence, depending on the level of the spinal injury.
25 sponses of diverse supraspinal cell types to spinal injury.
26  factors restricting axonal growth following spinal injury.
27 and improved breathing in rats with cervical spinal injuries.
28  of cAMP, however, is useless as therapy for spinal injuries.
29 lation will be useful for treatment of human spinal injuries.
30  rats (Rattus norvegicus) receiving thoracic spinal injury 3 d to 8 months earlier and recorded 1 d a
31 se noradrenergic receptors proliferate after spinal injury and descending noradrenergic pathways cont
32 ated with respiratory insufficiency, such as spinal injury and motor neuron disease.
33 ll number of investigations done on cervical spinal injury and tetraplegia, and the differences in le
34 ents were stratified into early (<24 h after spinal injury) and late ( 24 h after spinal injury) deco
35 ents were stratified into early (<24 h after spinal injury) and late (>=24 h after spinal injury) dec
36 eurocan, which increase in the vicinity of a spinal injury, and aggrecan, which decreases, into the l
37 , measure population-specific sensitivity to spinal injury, and test the relationships between region
38 inal sympathetic reflexes is increased after spinal injury, and these reflexes may result in life-thr
39                                         Most spinal injuries are incomplete, leaving spared neural pa
40 injury, pelvic fracture, femur fracture, and spinal injury as a result of their trauma were included
41 apeutics to treat traumatic brain injury and spinal injury as well as neuronal and cardiac ischemic e
42 ects was positively correlated with American Spinal Injuries Association (ASIA) grade: the smallest a
43         In two of two patients with American Spinal Injuries Association grade C traumatic spinal cor
44 evere traumatic spinal cord injury (American Spinal Injuries Association grades A-C).
45 matic spinal cord injuries (TSCIs), American spinal injuries association Impairment Scale (AIS) grade
46          NfL levels correlated with American Spinal Injury Association (ASIA) motor score at baseline
47  numeric rating scale (NRS) scores, American Spinal Injury Association (ASIA) motor score, and Nurick
48 rological outcomes were assessed by American Spinal Injury Association (ASIA), or International Stand
49 with relevant motor impairment (ie, American Spinal Injury Association [ASIA] impairment scale [AIS]
50 ation of Spinal Cord Injury and the American Spinal Injury Association Impairment Scale (AIS).
51 ith traumatic spinal cord injuries, American spinal injury association Impairment Scale A-C.
52 ination with gross motor scoring by American Spinal Injury Association Impairment Scale and Inflammat
53 inal cord injury (cervical level 4, American Spinal Injury Association Impairment Scale category A).
54 em, stratified in blocks of four by American Spinal Injury Association Impairment Scale grade and rec
55  children with cerebral AVF and the American Spinal Injury Association impairment scale in children w
56 ts were change in motor and sensory American Spinal Injury Association Impairment Scale scores from b
57 nt had cervical injuries, 53.4% had American Spinal Injury Association injury severity A (complete in
58 cluding Brief Pain Inventory, ASIA (American Spinal Injury Association) impairment scale, SF-36 Short
59  after spinal injury) and late (>=24 h after spinal injury) decompression groups.
60 h after spinal injury) and late ( 24 h after spinal injury) decompression groups.
61                                              Spinal injury disrupts connections between the brain and
62                                  In opossums spinal injury early in development results in spontaneou
63 , after the bladder has been disconnected by spinal injury from the pontine micturition centre, vanil
64 together, these results indicate that lumbar spinal injuries have more severe consequences on hindlim
65 is responsible for 6 to 19% and 9% to 14% of spinal injuries in children and adults, respectively.
66                                              Spinal injuries in children remain a challenge despite s
67                                              Spinal injuries in many vertebrates can result in partia
68               We suggest that after neonatal spinal injury in the rat, the transplant-mediated reesta
69 ations in non-regenerating species.Following spinal injury in zebrafish, non-neural cells establish a
70                       Surprisingly, thoracic spinal injury induced only modest changes in gene expres
71  a regenerative peripheral but not a central spinal injury induces an increase in calcium, which acti
72   Thus, the muted response of CST neurons to spinal injury is linked to the injury's distal location,
73 challenge for enhancing regeneration after a spinal injury is the limited detection of distant injuri
74 e conditions, such as sleep apnoea, cervical spinal injury or amyotrophic lateral sclerosis.
75 ep apnea, or respiratory insufficiency after spinal injury or during neurodegenerative diseases.
76 plasticity and improves motor function after spinal injury or during neuromuscular disease.
77 ed (e.g., sleep apnea, apnea of prematurity, spinal injury, or motor neuron disease).
78        This represents a model that analyzes spinal injury recovery by combining a closed-loop neurom
79                                    Brain and spinal injury reduce mobility and often impair sensorimo
80 a putative role for miR-21 in the microglial spinal injury response.
81              These findings demonstrate that spinal injury results in increased taurine concentration
82 aditionally referred to as a glial scar, the spinal injury scar in fact comprises multiple cellular a
83 sition and phenotypic characteristics of the spinal injury scar, the oversimplification of defining t
84 icant increase in phosphorylated ERK1 at the spinal injury site after in vivo ChABC treatment, indica
85 lone, propelling axon growth well beyond the spinal injury site.
86 to sensorimotor cortex dysfunction above the spinal injury site.
87 t axon processes were able to grow through a spinal injury site.
88             The motor sequelae of incomplete spinal injury, such as central cord syndrome and 'crucia
89 best promote recovery.SIGNIFICANCE STATEMENT Spinal injuries that remove sensation from the hand, can
90                            Seven weeks after spinal injury, the Basso-Beattie Bresnahan locomotor sco
91 etic nerve activity is maintained after high spinal injury through circuits that remain in question.
92 d late decompression surgery at 1 year after spinal injury; total motor scores improved by 23.7 point
93 compression surgery (n=1020) at 1 year after spinal injury; total motor scores improved by 23.7 point
94 ed a general rehabilitation ward, a regional spinal injuries unit and stroke rehabilitation ward.
95 cruited from the patient lists of a tertiary spinal injury unit and a Specialist Continence Service.
96 y, whereas one patient with low suspicion of spinal injury was partially extended.
97                 Outcome data at 1 year after spinal injury were available for 1031 patients (66.6%).