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1                       However, we found that spinothalamic ablation in humans, whilst profoundly impa
2 entation of selectively nociceptive lamina I spinothalamic activity.
3                        Thus, the overlapping spinothalamic and cerebellar inputs may provide a substr
4 rns and corresponding nerves, atrophy of the spinothalamic and spinocerebellar tracts and posterior c
5                     The question whether the spinothalamic and spinoreticular fibres cross the cord t
6 is then transmitted to brain regions via the spinothalamic and thalamocortical pathways.
7 n C7 and over one-fourth of those in L4 were spinothalamic, and at each level some projected to both
8 dial SPFp receives unique inputs from lumbar spinothalamic cells and brain regions involved in proces
9 d the significance of a population of lumbar spinothalamic cells for male sexual behavior in rats.
10 hese results suggest that this population of spinothalamic cells plays a pivotal role in generation o
11 lamina I projection cells per side, and that spinothalamic cells therefore make up approximately 42%
12 ounted for only on the assumption that these spinothalamic fibres are crossing the cord transversely.
13                                          The spinothalamic input was directed mostly to the ventral p
14 lls were distinguished from other classes of spinothalamic lamina I neurones by their peripheral inpu
15 racteristics of warming-specific lumbosacral spinothalamic lamina I neurones.
16                           We have shown that spinothalamic lamina I neurons are infrequent in rat lum
17                                Virtually all spinothalamic lamina I neurons at both levels were label
18                                              Spinothalamic lamina I neurons differed from those label
19 p of their collaterals that results in every spinothalamic neurone receiving an input from several do
20 olecystokinin as a marker for this subset of spinothalamic neurons and Fos-immunoreactivity as a mark
21  PAG and LPb, to determine the proportion of spinothalamic neurons at lumbar and cervical levels that
22                                              Spinothalamic neurons have generally been associated wit
23 mina I and lamina III/IV NK1r-immunoreactive spinothalamic neurons in cervical and lumbar segments co
24  investigate the involvement of these lumbar spinothalamic neurons in conveying copulation-related in
25  into the thalamus to estimate the number of spinothalamic neurons in each of these two populations,
26 This pathway originates from a population of spinothalamic neurons in the lumbar spinal cord containi
27 esults demonstrated that activation of these spinothalamic neurons is triggered by stimuli associated
28            The numbers of spinomedullary and spinothalamic neurons on the left side were comparable,
29 r results suggest that there are 90 lamina I spinothalamic neurons per side in C7 and 15 in L4 and th
30 demonstrate that a specific subpopulation of spinothalamic neurons signals information associated wit
31                   Second, spinomedullary and spinothalamic neurons were compared in retrograde double
32                              Moreover, these spinothalamic neurons were not activated by vaginocervic
33 urons with collaterals to the medulla and 2) spinothalamic neurons with collaterals to the midbrain.
34 mation conveyed by both these populations of spinothalamic neurons.
35  more ventrally located within lamina I than spinothalamic neurons.
36 ly, the recent identification of a candidate spinothalamic pathway involved in relay of ejaculation-s
37 ding the Neurologic Pain Signature (NPS) and spinothalamic pathway regions, with strong support for n
38 d others has demonstrated the existence of a spinothalamic pathway that is a candidate to relay infor
39 thalamic and cortical organoids to model the spinothalamic pathway.
40                               Intact crossed spinothalamic pathways are unable to support the normal
41 ately 1%, indicating that spinomedullary and spinothalamic pathways arise from separate subpopulation
42 th other unmyelinated afferents, in lamina I-spinothalamic pathways.
43 nd that this projection is distinct from the spinothalamic projection.
44 dings preclude privileged C-tactile-lamina I-spinothalamic projections and imply integrated hedonic a
45 risingly, placebo increased activity in some spinothalamic regions for unconditioned mechanical pain.
46  (spectral phase-coherence) between the main spinothalamic sensory area (posterior insula) and 12 oth
47                             Classically, the spinothalamic (ST) system has been viewed as the major p
48 us results in monkeys show that mid-cervical spinothalamic (STT) neurons are activated by groups II a
49 ion of rat lumbar laminae VII and X putative spinothalamic (STT) neurons that co-contain cholecystoki
50 otopically organized lamina I trigemino- and spinothalamic terminations in a cytoarchitectonically di
51                  In addition, the patches of spinothalamic terminations intermingled and partly overl
52 al spinal cord to inhibit activity of lumbar spinothalamic tract (SST) cells and dorsal horn (DH) cel
53 ed neuron, indicating the termination of the spinothalamic tract (STT) axon.
54 oposed to contribute to the sensitization of spinothalamic tract (STT) cells and hyperalgesia.
55 ted NR1 subunits (pNR1) are expressed in the spinothalamic tract (STT) cells and immunohistochemistry
56                             The responses of spinothalamic tract (STT) cells were recorded before and
57                                          The spinothalamic tract (STT) is an integral pathway in the
58                                  Nociceptive spinothalamic tract (STT) neurones in lamina I of the lu
59                     Central sensitization of spinothalamic tract (STT) neurons in anesthetized monkey
60 ogy and distribution of retrogradely labeled spinothalamic tract (STT) neurons in lamina I (the margi
61 c pain, central sensitization of dorsal horn spinothalamic tract (STT) neurons is a major underlying
62 mine the effects of central sensitization of spinothalamic tract (STT) neurons produced by intraderma
63 perior sagittal sinus (SSS) can excite C1-C2 spinothalamic tract (STT) neurons receiving thoracic vis
64                                              Spinothalamic tract (STT) neurons respond to itch-produc
65                 We found a class of lamina I spinothalamic tract (STT) neurons selectively excited by
66     The distribution of retrogradely labeled spinothalamic tract (STT) neurons was analyzed in macaqu
67     The distribution of retrogradely labeled spinothalamic tract (STT) neurons was analyzed in monkey
68                                     Lamina I spinothalamic tract (STT) neurons were identified by ret
69                          Fifty-seven primate spinothalamic tract (STT) neurons were identified using
70 by disruption of the DC pathway, but not the spinothalamic tract (STT).
71  in the pain-signaling pathway to the brain [spinothalamic tract (STT)] that respond only to painful
72 ing peripheral nociceptive sensation via the spinothalamic tract and brainstem nuclei to the thalamus
73 ll tissues caudal to the neck eliminates the spinothalamic tract and the transmission of somatosensor
74                                Excitation of spinothalamic tract cells in the upper thoracic and lowe
75 la and then descend to excite upper cervical spinothalamic tract cells.
76             Our results demonstrate that the spinothalamic tract contains mutually exclusive populati
77 07, P = 0.0440, R2 = 0.20) and cervical cord spinothalamic tract fractional anisotropy associated wit
78  width of ventral tissue bridges-a proxy for spinothalamic tract function-at 1 month post-spinal cord
79 tic or demyelinating lesions centered on the spinothalamic tract in rats.
80 ntact animals, electrical stimulation of the spinothalamic tract induces increases in thalamic CCL21
81 st that GRPR+ neurons are different from the spinothalamic tract neurons that have been the focus of
82                 We examined the responses of spinothalamic tract neurons to histaminergic and, for th
83 pinocervical, postsynaptic dorsal column and spinothalamic tract neurons was used to simulate the pop
84                                          The spinothalamic tract projects to the medial and lateral t
85 ified itch-specific neuronal pathways in the spinothalamic tract that are distinct from pain pathways
86 ensory neuronal circuits of nociception (the spinothalamic tract) and proprioception (the dorsal spin
87 s in C6 and 17% of those in L5 belong to the spinothalamic tract, and these apparently project exclus
88                   This review focuses on the spinothalamic tract, but other pathways are excited as w
89 n of the sensory component of pain along the spinothalamic tract, or stereotactic cingulotomy (n = 7)
90 s in each of these populations belong to the spinothalamic tract, which conveys nociceptive informati
91 he DC at T10, but not by interruption of the spinothalamic tract.
92 influenced by interfering with the ascending spinothalamic tract.
93 hose in the cervical cord also belong to the spinothalamic tract.
94 linical outcome groups in the left and right spinothalamic tracts (p = 0.003 and 0.020) and MTRh (p =
95  prion protein in the substantia gelatinosa, spinothalamic tracts, posterior columns and nuclei and i
96 icroscope to examine lamina I trigemino- and spinothalamic (TSTT) terminations in the posterior part
97                 PHA-L-labeled trigemino- and spinothalamic (TSTT) terminations were identified immuno