ライフサイエンスコーパス: splenocyte

1 ation in 5-wk-old pups (ex vivo assay of pup splenocytes).

2 eted population, but was also in sIgkappa(+) splenocytes.

3 d Th17, and Tc17 responses in immunized mice splenocytes.

4 human gamma interferon response in HIS mouse splenocytes.

5 normal as well as proteolipid protein-primed splenocytes.

6 mption of opsonized murine RBCs by double-KO splenocytes.

7 inflammation depends on alpha7nAChR-positive splenocytes.

8 fts survived an in vivo challenge with naive splenocytes.

9 ly reduced CD11b(+) monocyte counts in mouse splenocytes.

10 gamma-producing NK cells within L.m infected splenocytes.

11 creased TNF production in antigen-stimulated splenocytes.

12 psis-adapted human blood monocytes and mouse splenocytes.

13 the proliferation of alloantigen-stimulated splenocytes.

14 immunodeficiency mice receiving diabetogenic splenocytes.

15 by i.p. injection of class II-disparate bm12 splenocytes.

16 mised by immunosuppressive CD71(+) erythroid splenocytes.

17 d and separately cocultured with primary rat splenocytes.

18 red for efficient reactivation of MHV68 from splenocytes.

19 arable to those seen with stimulated UNG(-/-)splenocytes.

20 ), and interleukin-2 (IL-2) were detected in splenocytes.

21 ing and promoted T(H)2 cytokine responses in splenocytes.

22 er adoptive transfer of activated autologous splenocytes.

23 ocytes but did not show any effect on KCa3.1 splenocytes.

24 depletion was confirmed by flow cytometry of splenocytes.

25 NO) and the expression of iNOS in MBP-primed splenocytes.

26 cific Abs, and decreased CD19(+) survivin(+) splenocytes.

27 wed by adoptive transfer of human allogeneic splenocytes.

28 reduces basal AC activity in COS-7 cells and splenocytes.

29 ow inoculum was supplemented with donor-type splenocytes.

30 NFgamma-induced inflammatory response in the splenocytes.

31 atory gene transcription in isolated primary splenocytes.

32 (-/-) Tlr9 (-/-), Tlr7 (-/-), and Tlr9 (-/-) splenocytes.

33 monstrated increased IFN-gamma production in splenocytes.

34 (Cyclooxygenases-2 (COX-2) gene) in chicken splenocytes.

35 ung leukocytes and in cryptococcal Ag-pulsed splenocytes, 3) diminished IgE production in sera, and 4

36 age, which was associated with increases in splenocyte accumulation and B-cell activation but not wi

37 Splenocytes adoptively transferred from mice with HF, bu

38 ient, B cells plus B-cell-depleted wild-type splenocytes adoptively transferred into B-cell-deficient

39 ) cDNAs, the dominant clonotype generated in splenocytes after immunization of SJL mice, that differe

40 ymph nodes and spleen T-cell population, and splenocytes alloantigen responsiveness of graft recipien

41 Ablation of Cul3 in mature CD4(+) splenocytes also resulted in dramatically exaggerated Tf

42 polarization of wild type or CerS6-deficient splenocytes also revealed no defects in the development

43 Itfg2-deficient splenocytes also showed a defect in cell migration in vi

44 tection from the challenge tumor and durable splenocyte and serum reactivity to B16 cell antigens.

45 d the cytokine secretion of CD4+ T cells and splenocytes and altered the cellular profile in the lymp

46 Splenocytes and bone marrow cells were exposed to nicoti

47 Ex vivo studies using splenocytes and bone marrow-derived macrophages revealed

48 evels in supernatants from cultures of mouse splenocytes and dendritic cells, as well as from human g

49 Here, we employed allogeneic apoptotic splenocytes and discovered that the efferocytic receptor

50 Single cell RNA sequencing of splenocytes and experiments in genetically-immunodeficie

51 In vitro culture of splenocytes and flow-sorted dendritic cells and T cells

52 B cells from murine splenocytes and from blood samples of healthy donors wer

53 against Francisella tularensis using murine splenocytes and further demonstrated that the relative l

54 ells, real-time confocal microscopy of mouse splenocytes and human macrophages, as well as FRET-based

55 We show that in vitro both murine splenocytes and human PBMCs secrete CCL22 spontaneously

56 onfirmed through in vitro assays using mouse splenocytes and human peripheral blood mononuclear cells

57 hrough manual gating of two datasets (murine splenocytes and human whole blood) confirmed its accurac

58 f IL-17 and its promoting cytokines in total splenocytes and in both CD4 and CD8 T cells following im

59 control ovalbumin peptide (OVA(323-339)) to splenocytes and induced apoptosis in the conjugated cell

60 mice enhanced bone resorption by co-cultured splenocytes and induced interleukin-6, a molecule that p

61 increased production of IL-5 and IL-13 from splenocytes and liver mononuclear cells (MNCs) of infect

62 Splenocytes and lung lymph node cells from sensitized EP

63 ovalbumin-specific IgE, a reduced number of splenocytes and lymph node cells with a decreased number

64 It did not induce any toxicity to splenocytes and on hematopoiesis, induced protective cyt

65 MHV68 established and maintained latency in splenocytes and peritoneal cells but did not reactivate

66 shed T cell activation induced by allogeneic splenocytes and protected allogeneic MIN6 beta cells and

67 Purified B220(+) cells from PA splenocytes and purified CD4(+) cells from naive (NA) sp

68 Transplantation of splenocytes and serum exosomes from elafin-overexpressin

69 ation of ISGs in STING N153S fibroblasts and splenocytes and STING N154S SAVI patient fibroblasts.

70 HT2B receptors restored IL-17 secretion from splenocytes and Th17 cell differentiation in Tph1(-/-) m

71 re cocultured for 5 d with irradiated BALB/c splenocytes and then photodepleted (PD).

72 ast, all mice that received untreated primed splenocytes and third-party mice that received PD-treate

73 or no effect on the number of MoMLV-infected splenocytes and thymocytes.

74 ody levels, cytokine release by restimulated splenocytes) and local (infiltration of immune cells int

75 , flow cytometric analysis of thymocytes and splenocytes, and histologic/transcriptomic analyses of t

76 ergen-induced IL-13 and IL-4 production from splenocytes, and inflammatory cell infiltrations in the

77 Antibody levels, cytokine production by splenocytes, and splenic forkhead box P3 (FOXP3)(+) regu

78 ice, cecal ligation and puncture resulted in splenocyte apoptosis and significant lymphopenia after 3

79 d parasite burden and increased induction of splenocyte apoptosis.

80 cytotoxicity assays in which peptide-pulsed splenocytes are killed by CTL in the spleens of immunise

81 es and purified CD4(+) cells from naive (NA) splenocytes are the minimal requirements for the adoptiv

82 n was significantly increased in septic mice splenocytes as compared with shams.

83 uced IFN-gamma/TNF-alpha recall responses by splenocytes, as well as fewer IL-12p70-producing APCs.

84 Same-party mice that received PD-treated splenocytes at the time of transplant lived 100 d withou

85 edly suppressed type-2 cytokine secretion of splenocytes beyond the effect of AIT alone.

86 uppressed proliferation of wild-type C57B/6J splenocytes but did not show any effect on KCa3.1 spleno

87 MPER restimulated, H-2(d)-restricted primed splenocytes by class II-blocking Abs), and failed to neu

88 In vitro restimulation of splenocytes by myelin oligodendrocyte glycoprotein 35-55

89 of the TGF-beta gene, activation of STAT6 in splenocytes by NaB, recruitment of STAT6 to the TGF-beta

90 ion of NaB-induced expression of TGF-beta in splenocytes by small interfering RNA knockdown of STAT6

91 he R144A mutant), flow cytometry analysis of splenocytes by tetramer and intracellular cytokine stain

92 ed a significant impairment in thymocyte and splenocyte CD1d gene and protein expression.

93 Mice were injected with allogeneic splenocytes, CD4(+) T cells, or CD8(+) T cells and treat

94 vo fluorescence in the spleen, and decreased splenocyte cell death.

95 ads, particularly from Tlr7 (-/-) Tlr9 (-/-) splenocytes compared to levels in the WT.

96 une attack initiated by adoptive transfer of splenocytes compared with that of the same area before t

97 ed ITP, transfer of allogeneic MHC-immunized splenocytes completely prevented CD61-induced ITP develo

98 he spleen is a heterogeneous environment and splenocytes comprise multiple cell types.

99 ouse models were used to generate a diseased splenocyte conditioned media (D-SCM) containing immune c

100 tive transfer of VNS-conditioned alpha7nAChR splenocytes conferred protection to recipient mice subje

101 model indicate that administering apoptotic splenocytes conjugated with the immunodominant MPO pepti

102 The SHR splenocytes constitutively expressed more RORgammat than

103 addition, treatment of human PBMCs or mouse splenocytes containing destabilizing STAT3 mutations wit

104 compared to Th2 cytokines (IL-4 and IL-5) in splenocyte culture supernatants of the microneedle treat

105 In primary splenocyte culture, lipopolysaccharide increased TNF-alp

106 antileishmanial IL-17 cytokine production in splenocyte culture.

107 In addition, splenocytes cultured 24 days after RSD exhibited a prime

108 Splenocytes cultured from female mice exhibited fewer Tr

109 control animals, peanut recall responses in splenocyte cultures from nanoparticle-treated mice showe

110 ystem tissues, and in activated T cells from splenocyte cultures.

111 ELISA, as were cytokine recall responses in splenocyte cultures.

112 IL-2 and IFN-gamma cytokines in restimulated splenocyte cultures.

113 Allergen-specific antibodies, splenocyte cytokine production and splenic forkhead box

114 to L. monocytogenes through the promotion of splenocyte death, we examined the effect of C5aR1 on typ

115 the culture supernatant of stimulated mouse splenocytes decreased IFN-gamma concentration.

116 hylepoxyquinomicin, or transferring p50(-/-) splenocytes, decreased CXCR4 expression on CD8(+) T cell

117 Human cord blood (CB)-derived or mouse splenocyte-derived DCs were loaded with purified recombi

118 tive in Yop translocation into CHO cells and splenocyte-derived neutrophils and macrophages.

119 ated reduction in neonatal CD71(+) erythroid splenocytes did not alter murine neonatal survival to en

120 A and IFNgamma by cortactin-deficient and WT splenocytes did not differ, suggesting that the lack of

121 nd third-party mice that received PD-treated splenocytes died of lethal GVHD.

122 stimulated BDC-2.5.Ncf1(m1J) CD4 T cells and splenocytes displayed elevated synthesis of Th1 cytokine

123 Administration of apoptotic donor splenocytes effectively induces antigen-specific toleran

124 f cytokines from activated IELs but not from splenocytes ex vivo.

125 transfer experiments showed that transgenic splenocytes exhibited attenuated diabetogenic ability.

126 ulate the NKG2D receptor with tumor cells or splenocytes expressing Rae-1.

127 we performed a co-culture assay using mouse splenocytes (expressing PD-L1 receptor PD-1) and murine

128 capsule and adoptively transferred 5 x 10(6) splenocytes from 6-week-old nonobese diabetic mice.

129 When treated with 2-(1-adamantyl)-ethanol, splenocytes from 8 wk-infected BALB/c mice showed signif

130 Treatment with VIPR2 agonist or splenocytes from agonist-treated mice resulted in increa

131 nflammation in mice receiving OVA-sensitized splenocytes from AQP3(-/-) mice compared with wild-type

132 hed alloHCT using bone marrow (BM) cells and splenocytes from B6 (H-2) donor mice transplanted into B

133 ) (B6) mice were injected intravenously with splenocytes from B6.C.H-2 (H-2k) (bm1) or F1 (B6 x bm1)

134 Splenocytes from BALB/c mice immunized with a recombinan

135 Splenocytes from C57BL/6 J (B6) AID(-/-) mice were used

136 nfection with L. monocytogenes compared with splenocytes from C5aR2(+/+) mice in an NF-kappaB-depende

137 Purified naive splenocytes from C5aR2(-/-) mice produced significantly

138 ex vivo system for aneuploidy where primary splenocytes from Casp2(-/-) mice were exposed to anti-mi

139 FA-1 inhibition blocked IFN-gamma secretion, splenocytes from CD11a(-/-) mice did not respond to ISG1

140 10 million bone marrow cells and 20 million splenocytes from either syngeneic C57BL/6 or allogeneic

141 press the recall antibody response of murine splenocytes from FVIII knockout mice to FVIII in vitro a

142 Studies that used splenocytes from GPR120-deficient mice have confirmed th

143 Splenocytes from HLA-DQ8 transgenic mice given TIMP-GLIA

144 nked immunosorbent spot [ELISPOT] assays) by splenocytes from IKEPLUS-immunized C57BL/6J mice, we ide

145 portantly, results from adoptive transfer of splenocytes from immunized animals in a Parkinson's dise

146 Furthermore, splenocytes from immunized H(1-4)RKO mice, compared with

147 In addition, splenocytes from infected Cd14(-/-) mice exhibited incre

148 Flow cytometric analysis of splenocytes from infected mice revealed that cellular ex

149 accumulation was significantly reduced when splenocytes from mice deficient in NK cells by genetic a

150 Splenocytes from mice vaccinated with chi10057(pYA5199)

151 uman promonocytes during inflammation and in splenocytes from mice with sepsis.

152 Leishmania antigen-stimulated splenocytes from miR155KO mice produced significantly lo

153 e, and IL-6 induction by 17beta-estradiol in splenocytes from naive female mice (p<0.05) suggested th

154 duced markedly lower IFN-gamma production in splenocytes from naive mice.

155 port adoptively transferred T cells or whole splenocytes from NOD-PerIg mice expectedly induce T1D in

156 specifically CD4+T helper cells, compared to splenocytes from non-irradiated mice.

157 Moreover, polyclonal splenocytes from nonobese diabetic (NOD) mice transplace

158 y, a significantly decreased T1D transfer by splenocytes from prediabetic NOD donors was observed in

159 IFN-gamma ELISpot analysis of splenocytes from rAAV-K-treated mice indicated reduced r

160 ed more IL-6 ex vivo in response to LPS than splenocytes from sham mice.

161 erferon gamma (IFN-gamma) recall response of splenocytes from T. cruzi-infected mice confirmed that 1

162 Additionally, in vitro stimulation of splenocytes from these regulatory T cell-depleted mice r

163 Adoptive transfer of splenocytes from ultrasound-treated (but not sham) mice

164 Splenocytes from VACV-WR-infected mice were assayed with

165 ere reconstituted with nonregulatory CD25(-) splenocytes from wild-type (WT) or Tlr9(-/-) mice, AKI w

166 Splenocytes from wild-type mice infected i.v. produced s

167 Splenocytes from wild-type or LAIR-1(-/-) mice were stim

168 flammatory cytokine production by stimulated splenocytes, from female triple transgenic (3xTg-AD) mic

169 blot analysis revealed that Notch2HCS mutant splenocytes had increased phospho-Akt and phospho-Jun N-

170 esence of collagen, whereas LAIR-1-deficient splenocytes had no attenuation.

171 had been preincubated with noradrenaline or splenocytes harvested from stressed mice.

172 in the lungs at later disease stages as were splenocyte IL12/23p40 and IFN-gamma levels following ex

173 epletion of maternal Foxp3(+) cells from pup splenocytes illustrated a substantial role for lactation

174 allogeneic stimulation in vitro, allogeneic splenocyte immunization in vivo, and allogeneic heart tr

175 mediates development and immune function via splenocyte immunohistochemistry analysis in association

176 /-) splenocytes were comparable to wild type splenocytes in proliferation responses, alloreactivity,

177 IL-23 in DCs abolished IL-17 production from splenocytes in response to Ag challenge.

178 ncreased Th1 effector cytokine production by splenocytes in response to myelin oligodendrocyte gp35-5

179 ed expression of the CD161 surface marker on splenocytes in SHRs and normotensive control Wistar-Kyot

180 e expression of proinflammation cytokines by splenocytes in vitro Moreover, in vivo, AgTRIO decreased

181 17) production by vaccine antigen-stimulated splenocytes in vitro.

182 Immunization with allogeneic splenocytes in vivo resulted in increased alloreactivity

183 flammation induced by injection of activated splenocytes increased Deaf1-Var1 and Srsf10, but not Ptb

184 l blood mononuclear cells (PBMCs), and mouse splenocytes incubated without or with chaperone protein

185 ed an increase in nuclear 5-LO expression in splenocytes, indicating enzyme activation after GVHD.

186 ivo culture of cervical lymph node cells and splenocytes, indicating that in allergic mice SD favors

187 n the induction of inflammatory responses in splenocytes induced by the exosomes released from infect

188 and posttransplant (donor) bone marrow, and splenocyte infusion followed by posttransplantation cycl

189 xed with IL-23 in primary cultures of murine splenocytes inhibits IL-23-mediated immune signaling.

190 Isolated splenocytes, intestinal lymphocytes, and genital lymphoc

191 A single transfusion of F1 splenocytes into B6 mice without any additional immune m

192 iated ITP was initiated by transfer of their splenocytes into severe combined immunodeficiency (SCID)

193 experiments showed that MALT1 deficiency in splenocytes is sufficient for EAE resistance.

194 However, after immune activation splenocytes isolated from Anp32b KO mice showed a strong

195 the proliferation and cytokine responses of splenocytes isolated from Met e 1-sensitized Balb/c mice

196 However, splenocytes isolated from mice 24 hours after ultrasound

197 t CD11b(+) cells was not limited to neonatal splenocytes; it also occurred with adult and neonatal bo

198 To address these questions we revisited the splenocyte killing assay, using CTL specific for an epit

199 Moreover, silencing IRF8 ex vivo in splenocytes lead to a profound downregulation of IRF8 pr

200 with esxL-expressing M. smegmatis and mouse splenocytes led to down-regulation of IL-2, a key cytoki

201 llowing adoptive transfer of CerS6-deficient splenocytes maybe related to their ability to migrate an

202 Here we show that F4/80(+)/CD11b(low) splenocytes mediate the resistance to DNA-damaging chemo

203 We observed that the splenocyte-mediated apoptosis of cancer cells during co-

204 ministered MPO- and OVA-conjugated apoptotic splenocytes (MPO-Sps and OVA-Sps, respectively) to mice

205 reduction of pristane-dependent induction of splenocyte number and weight.

206 try using peripheral blood, lymph nodes, and splenocytes obtained from dogs undergoing graft-versus-h

207 Splenocytes of control-diet-fed whey-sensitized donors t

208 Finally, in splenocytes of DNA.HTI-vaccinated mice, costimulation of

209 in-10 production that were not evident among splenocytes of infected mice in which Treg cells were no

210 Augmented PGE2 production was also found in splenocytes of infected mice, and administration of EP2

211 esponse was noted from cytokines secreted by splenocytes of mice immunized with CpG 1018 and alum.

212 nced CD4(+) and CD8(+) T cell populations in splenocytes of mice.

213 Splenocytes of S. aureus-infected mice lost most of thei

214 presence of immunosuppressive mechanisms in splenocytes of S. aureus-infected mice that inhibited th

215 nd IL-13 by CD4(+) T cells isolated from the splenocytes of these mice strongly suggests that the TCR

216 n in interleukin-17 by in vitro-restimulated splenocytes of TNFR1-deficient mice.

217 Thus, splenocytes of ultrasound-treated mice are capable of mo

218 e present at higher frequencies among CD4(+) splenocytes of vitamin A deficient vs. sufficient mice.

219 We examined the impact of CD71(+) erythroid splenocytes on murine neonatal mortality to endotoxin ch

220 nhibiting CXCR4 in AA mice, using CXCR4(-/-) splenocytes or AMD3100, significantly reduced BM infiltr

221 ncubating brain-derived PrP(Sc) with primary splenocytes or cultured macrophage RAW 264.7 cells.

222 EMMPRIN expression in cultures of mouse splenocytes or human PBMCs was elevated upon polyclonal

223 to reactivate from either latently infected splenocytes or PECs.

224 Using splenocytes or purified CD4(+) cells that were sufficien

225 ular LVS growth, we found that IL-6 KO total splenocytes or purified T cells were slightly defective

226 In contrast to splenocytes, peripheral blood leukocytes (PBLs) represen

227 ased with age, reaching more than 30% of the splenocyte population at 38 weeks.

228 Splenocytes (presumably splenic monocytes and dendritic

229 l improvements to ERT in mice: (a) decreased splenocyte proliferation after in vitro GALNS stimulatio

230 s, but not rapid desensitization, suppressed splenocyte proliferation and production of IL-4, IL-5, a

231 Splenocyte proliferation and TH1 cytokine transcription

232 chroism measurement, in vitro digestion, and splenocyte proliferation assays using synthetic Cyp c 1-

233 rPfMSP2 formed fibrils, which induced splenocyte proliferation in an antigen receptor-independ

234 The capacity of DC to stimulate allogenic splenocyte proliferation was also enhanced by GUWE treat

235 L-17 triggered a pro-inflammatory state; and splenocyte proliferation was elevated in response to ocu

236 s both chronic and acute exposure suppressed splenocyte proliferation, although viral replication rat

237 rsed the immunosuppressive effect of MSCs on splenocyte proliferation.

238 lar responses by ELISA, basophil activation, splenocyte proliferations, and intragastric allergen cha

239 d the complement receptor 3 (CR3), on murine splenocytes, purified B cells, and human neutrophils.

240 Primary splenocytes readily aggregated and formed osteoclast-lik

241 lerogenic properties of allogeneic apoptotic splenocytes require MerTK transmission of intracellular

242 h the levels of cell death in thymocytes and splenocytes, respectively, as measured by flow cytometry

243 and numbers of IL-17-producing CD4(+) mouse splenocytes, respectively.

244 E formation, as indicated by splenectomy and splenocyte restoration experiments.

245 ECP-treated autologous splenocytes resulted in immune tolerance in the host, in

246 Increasing doses of splenocytes resulted in increased incidence of GVHD in R

247 Analysis of splenocytes revealed alterations in T cell profiles that

248 brain-infiltrating T lymphocytes, and CD3(+)splenocytes (SCs) of EAE mic, and found that global RISC

249 Resident splenocytes show higher interferon-gamma and tumor necro

250 Mass cytometry analyses of splenocytes showed a significantly reduced level of infl

251 omplex (MHC) class I- and class II-deficient splenocytes showed our previously published chlamydia-sp

252 coculture system, but cultures with T-bet-KO splenocyte supernatants contained less IFN-gamma and inc

253 d, or male mice; undetectable IL-6 levels in splenocyte supernatants from ovariectomized and male mic

254 (IL-4) and IL-10 were also lower in miR155KO splenocyte supernatants than in WT mice.

255 ed CD4 + Foxp3+ CD25- and CD8 + Foxp3+ CD25- splenocyte T-cell populations compared to controls.

256 onal work showed that cytokine production by splenocytes taken post mortem from patients who died of

257 Finally, by blocking RKIP in wild-type SIRS splenocytes, the IFN-gamma response by CD8(+) Vbeta3(+)

258 CD161a(+)/CD68(+) macrophages in SHR-derived splenocytes, their renal infiltration, and premature hyp

259 ptive transfer of enriched CD71(+) erythroid splenocytes to CD71(+)-reduced animals did not reduce ba

260 cytokine production, and abilities of primed splenocytes to control intracellular LVS growth, we foun

261 s tolerance in a fully mismatched allogeneic splenocyte transfer model in mice.

262 onoclonal antibody (mAb) plus donor-specific splenocyte transfusion (DST) induces alloantigen-specifi

263 de immune or tolerant (donor-specific BALB/c splenocyte transfusion -/+ anti-CD40L monoclonal antibod

264 BALB/c donor-specific splenocyte transfusion and anti-CD40L monoclonal antibod

265 Murine splenocytes treated with TAT-Runx1.d190 showed an increa

266 that preemptive infusion of apoptotic donor splenocytes treated with the chemical cross-linker ethyl

267 transfer or diminution of CD71(+) erythroid splenocytes under these experimental conditions suggests

268 y AID and its removal by UNG2 glycosylase in splenocytes undergoing maturation and in B cell cancers.

269 erformed global gene expression profiling of splenocytes using high throughput microarray technology.

270 FSE staining, and the expression of GRAIL in splenocytes was measured by immunohistochemistry, real-t

271 MHV68 reactivation from latent splenocytes was not altered in the absence of the vUNG.

272 ction in immunosuppressive CD71(+) erythroid splenocytes, was likely responsible for the reported enh

273 Investigating primary mouse splenocytes, we could demonstrate that MALT1-induced MYC

274 l epithelial cells, macrophages, and primary splenocytes, we demonstrate K. pneumoniae 51-5 upregulat

275 s previously identified by the evaluation of splenocytes were also found to be differentially express

276 A were transferred into naive mice and their splenocytes were co-cultured with fresh OVA-loaded DCs.

277 etion of alloimmune responses, donor C57BL/6 splenocytes were cocultured for 5 d with irradiated BALB

278 The AID(-/-) splenocytes were comparable to wild type splenocytes in

279 reated BALB/c mice (tolerant Treg cell), and splenocytes were cotransferred into islet transplanted n

280 and NK (natural killer) cell populations in splenocytes were elevated in case of vaccinated mice.

281 PD-treated splenocytes were infused into lethally irradiated BALB/c

282 Splenocytes were isolated and subjected to flow cytometr

283 L-5 and IL-10, secreted by terpenoid-treated splenocytes were measured using the ELISA method.

284 pheral cytokines from autoantigen-stimulated splenocytes were measured, and central nervous system in

285 tramers, C57BL/6 mice sensitized with BALB/c splenocytes were shown to harbor H-2K(d)-specific IgG(+)

286 L/6 CD61 KO (CD61(-)/H-2(b)) mice, and their splenocytes were transferred into severe combined immuno

287 cells exhibited enhanced proliferation when splenocytes were transferred into WT recipient mice.

288 When nondepleted splenocytes were transferred to induce antibody-mediated

289 In contrast, when B-cell-depleted splenocytes were transferred to induce T-cell-mediated I

290 he combination provided full protection when splenocytes were transferred.

291 and less Th2 cytokine production in exposed splenocyte, were evident in the glycated protein treated

292 eoclastogenesis when cocultured with primary splenocytes, whereas ABCs slightly but significantly pro

293 Adoptive transfer of CerS6-deficient splenocytes, which have significantly decreased levels o

294 ished migration toward S1P in the Pparg(C/-) splenocytes, which impeded lymphocyte egression.

295 a dramatic reduction in all major subsets of splenocytes, which was associated with elevated caspase-

296 Stimulation of Fng tKO splenocytes with anti-CD3/CD28 beads or LPS gave reduced

297 Incubation of dendritic cells or splenocytes with BMP-4 did not affect lipopolysaccharide

298 nd chemokine (C-X-C motif) ligand, CXCL10 by splenocytes with no discernable effect on central nervou

299 A*02:01, and HLA-B*07:02q11 transgenic mouse splenocytes with peptides demonstrated predicted genetic

300 dditional murine-encoded Ag, expressed by B6 splenocytes, with sufficient avidity to induce a TLR-ind