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1 tients had either inherited prion disease or sporadic Creutzfeldt-Jakob disease.
2 ensitivity of 80 to 90% for the diagnosis of sporadic Creutzfeldt-Jakob disease.
3 enerally symmetric pattern of involvement in sporadic Creutzfeldt-Jakob disease.
4 ) diffusivities in the total white matter in sporadic Creutzfeldt-Jakob disease.
5 wed by the correct histological diagnosis of sporadic Creutzfeldt-Jakob disease.
6 xist in the most common human prion disease, sporadic Creutzfeldt-Jakob disease.
7 erentiating autoimmune encephalopathies from sporadic Creutzfeldt-Jakob disease.
8 incorporated into the diagnostic criteria of sporadic Creutzfeldt-Jakob disease.
9 rebellar abnormalities that closely resemble sporadic Creutzfeldt-Jakob disease.
10 es and some have met diagnostic criteria for sporadic Creutzfeldt-Jakob disease.
11 ted individuals resembled those of classical sporadic Creutzfeldt-Jakob disease.
12 (Sc) exists both between and within cases of sporadic Creutzfeldt-Jakob disease.
13 olecular basis for a novel classification of sporadic Creutzfeldt-Jakob disease.
14 blot and immunohistochemistry in 19 cases of sporadic Creutzfeldt-Jakob disease.
15 y using the standard cut-offs often used for sporadic Creutzfeldt-Jakob disease.
16 hs for the confident pre-mortem diagnosis of sporadic Creutzfeldt-Jakob disease.
17  the brain (gene: STX6) are risk factors for sporadic Creutzfeldt-Jakob disease.
18 ting observed in experimental, acquired, and sporadic Creutzfeldt-Jakob diseases.
19 zfeldt-Jakob disease (15 of 15 with definite sporadic Creutzfeldt-Jakob disease, 13 of 14 with probab
20 is detectable at low levels in some cases of sporadic Creutzfeldt-Jakob disease and conversely, that
21 ble spongiform encephalopathies (variant and sporadic Creutzfeldt-Jakob disease and genetic forms of
22  forms found in all of the major subtypes of sporadic Creutzfeldt-Jakob disease and in a case of iatr
23                     Twenty-six patients with sporadic Creutzfeldt-Jakob disease and nine age- and gen
24 ples from patients with and patients without sporadic Creutzfeldt-Jakob disease and tested them using
25 zfeldt-Jakob disease, 13 of 14 with probable sporadic Creutzfeldt-Jakob disease, and 2 of 2 with inhe
26 al PrP aggregates derived from patients with sporadic Creutzfeldt-Jakob disease are taken up and degr
27 the prion, in brain samples from humans with sporadic Creutzfeldt-Jakob disease, as well as in rodent
28 rogeneous and may be clinically mistaken for sporadic Creutzfeldt-Jakob disease because of a negative
29 of abnormal prion protein that characterizes sporadic Creutzfeldt-Jakob disease can be found in certa
30 ting on PrP(Sc) aggregates extracted from 60 sporadic Creutzfeldt-Jakob disease (CJD) and 6 variant C
31    Fourth, protease-resistant PrPSc cores in sporadic Creutzfeldt-Jakob disease (CJD) and Gerstmann-S
32 that prions from the brains of patients with sporadic Creutzfeldt-Jakob disease (CJD) bind to very lo
33 prion amyloid seeding assay for diagnosis of sporadic Creutzfeldt-Jakob disease (CJD) but several pri
34 al and molecular data from a large number of sporadic Creutzfeldt-Jakob disease (CJD) cases.
35                                              Sporadic Creutzfeldt-Jakob disease (CJD) is the most pre
36 ted elk and deer with those in subjects with sporadic Creutzfeldt-Jakob disease (CJD), as well as CJD
37 s has transformed the diagnostic approach to sporadic Creutzfeldt-Jakob disease (CJD), facilitating e
38 -QuIC proved 100% sensitive and specific for sporadic Creutzfeldt-Jakob disease (CJD), iatrogenic and
39 els of the most common human prion disorder, sporadic Creutzfeldt-Jakob disease (CJD), in which prion
40 and amniotic fluid, in a pregnant woman with sporadic Creutzfeldt-Jakob disease (CJD).
41  these were classified as either familial or sporadic Creutzfeldt-Jakob disease (CJD); there was no c
42  tensor imaging to study the white matter in sporadic Creutzfeldt-Jakob disease compared to healthy c
43 acterize the phenotypic spectrum of the rare sporadic Creutzfeldt-Jakob disease cortical subtype (sCJ
44                                           In sporadic Creutzfeldt-Jakob disease, disease-associated P
45   EEG has long been used in the diagnosis of sporadic Creutzfeldt-Jakob disease; however, the charact
46 eases cohorts (FTLD, ALS, Alzheimer disease, sporadic Creutzfeldt-Jakob disease, Huntington disease-l
47  phenotype resembling those causing MM1-type sporadic Creutzfeldt-Jakob disease in humans.
48                        Definite diagnosis of sporadic Creutzfeldt-Jakob disease in living patients re
49 ings from olfactory epithelium in diagnosing sporadic Creutzfeldt-Jakob disease in living patients.
50        Updated criteria for the diagnosis of sporadic Creutzfeldt-Jakob disease, including applicatio
51                                              Sporadic Creutzfeldt-Jakob disease is a fatal neurodegen
52                                              Sporadic Creutzfeldt-Jakob disease is considered primari
53                                              Sporadic Creutzfeldt-Jakob disease is the most common of
54 o distinct disease phenotypes, identified as sporadic Creutzfeldt-Jakob disease (M/M2 sCJD) and spora
55 ity within the white matter of patients with sporadic Creutzfeldt-Jakob disease, mainly in the left h
56 ase (n = 59) and compare these with cases of sporadic Creutzfeldt-Jakob disease (n = 170) in the Unit
57                     Its appearance can mimic sporadic Creutzfeldt-Jakob disease on MRI and should be
58 iseases can occur as an idiopathic disorder (sporadic Creutzfeldt-Jakob disease) or can be acquired,
59 eas extracts from the brains of familial and sporadic Creutzfeldt-Jakob disease patients produced the
60 fmeister analysis also improved detection of sporadic Creutzfeldt-Jakob disease prions in human nasal
61             Cumulatively, the data show that sporadic Creutzfeldt-Jakob disease PrP(Sc) is not a sing
62  of PRNP thought to confer susceptibility to sporadic Creutzfeldt-Jakob disease (rs1029273), all pati
63 list prion clinic who were suspected to have sporadic Creutzfeldt-Jakob disease (sCJD) and yet were f
64  Current cerebrospinal fluid (CSF) tests for sporadic Creutzfeldt-Jakob disease (sCJD) are based on t
65                                              Sporadic Creutzfeldt-Jakob disease (sCJD) comprises seve
66 oradic fatal insomnia (sFI) and a subtype of sporadic Creutzfeldt-Jakob disease (sCJD) identified as
67 e-adapted strains, and different subtypes of sporadic Creutzfeldt-Jakob disease (sCJD) in humans.
68                                              Sporadic Creutzfeldt-Jakob disease (sCJD) is a rapidly l
69                                              Sporadic Creutzfeldt-Jakob disease (sCJD) is a rare, rap
70                                    A case of sporadic Creutzfeldt-Jakob disease (sCJD) is described i
71 unting for 85% of human prion disease cases, sporadic Creutzfeldt-Jakob disease (sCJD) is the prevale
72                  Phenotypic heterogeneity in sporadic Creutzfeldt-Jakob disease (sCJD) is well docume
73 e CSF RT-QuIC differentiated 94% of cases of sporadic Creutzfeldt-Jakob disease (sCJD) MM1 from the s
74 hological studies confirmed the diagnosis of sporadic Creutzfeldt-Jakob disease (sCJD) MM1 subtype.
75                                              Sporadic Creutzfeldt-Jakob disease (sCJD) presents as a
76 activate both hamster Sc237 prions and human sporadic Creutzfeldt-Jakob disease (sCJD) prions.
77    We report a detailed study of a cohort of sporadic Creutzfeldt-Jakob disease (sCJD) VV1-2 type-mix
78 in misfolding, the most common subtype being sporadic Creutzfeldt-Jakob disease (sCJD), a rapidly pro
79 sfolding, with the most common subtype being sporadic Creutzfeldt-Jakob disease (sCJD), a rapidly pro
80 ly distinct subtypes have been identified in sporadic Creutzfeldt-Jakob disease (sCJD), based on the
81 cts from brains of two patients, who died of sporadic Creutzfeldt-Jakob disease (sCJD), contained eit
82                                        Human sporadic Creutzfeldt-Jakob disease (sCJD), endemic sheep
83 ans on bound PrP species present in control, sporadic Creutzfeldt-Jakob disease (sCJD), or variant CJ
84 served in the heart tissue of a patient with sporadic Creutzfeldt-Jakob Disease (sCJD), the most comm
85 ibodies to neuronal antigens misdiagnosed as sporadic Creutzfeldt-Jakob disease (sCJD).
86 horless PrP species has been also claimed in sporadic Creutzfeldt-Jakob disease (sCJD).
87 egenerative disorders, the most common being sporadic Creutzfeldt-Jakob disease (sCJD).
88 l and ethnic differences in individuals with sporadic Creutzfeldt-Jakob disease (sCJD).
89 are susceptible to prions from patients with sporadic Creutzfeldt-Jakob disease (sCJD).
90 th the most common phenotype in humans being sporadic Creutzfeldt-Jakob disease (sCJD).
91 free human PrP (TgGlyc-), with isolates from sporadic Creutzfeldt-Jakob disease subtype MM2 (sCJDMM2)
92  mean diffusivity within the white matter in sporadic Creutzfeldt-Jakob disease, suggesting possible
93                           Cases diagnosed as sporadic Creutzfeldt-Jakob disease with atypical neuropa