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1 as for example in the well-known case of the squid giant axon.
2 transport of peptide-conjugated beads in the squid giant axon.
3 yed rectifier K+ channel (SqK(v)1.1A) in the squid giant axon.
4 us in neurofilaments (NFs) isolated from the squid giant axon.
5 n on the Na+ activation gate were studied in squid giant axons.
6 ransport along microtubules in axoplasm from squid giant axons.
7 nd, more recently, in axoplasm isolated from squid giant axons.
8 n of this pathway in axoplasms isolated from squid giant axons.
9       The temperature difference between the squid giant axon (6.3 degrees C) and RGCs (37 degrees C)
10  regeneratively during the action potential (squid giant axon); a wasteful 85% enter during the falli
11 xley established that sodium currents in the squid giant axons activate after a delay, which is expla
12 ast axonal transport using axoplasm from the squid giant axon and suggest that axonal transport defec
13                                          The squid giant axon and synapse, for example, laid the foun
14                                              Squid giant axons are formed by giant fiber lobe (GFL) n
15 e membrane excitability and results from the squid giant axon as an experimental data base.
16 ley (HH) description of Na and K channels in squid giant axons as the basis of the calculations and f
17                In addition, working with the squid giant axon, Cole and Moore noted that strong hyper
18                                 In contrast, squid giant axons do not seal, even though injury-induce
19 , (1) we internally dialyzed isolated intact squid giant axons (GAs) and showed that elevation of int
20 or the ionic currents in the membrane of the squid giant axon has become the standard model for the e
21 the myosin motors that transport S-ER in the squid giant axon has been determined.
22          Organelles in the axoplasm from the squid giant axon move along exogenous actin filaments to
23      Axoplasmic organelles obtained from the squid giant axon move on actin filaments at an average v
24  modification of native cysteines present in squid giant axon Na channels with methanethiosulfonates.
25 lity assays performed with axoplasm from the squid giant axon showed a requirement for a Rab GTPase i
26 qKv1A compose the "delayed rectifier" in the squid giant axon system, but discrepancies regarding ina
27 ll sites) are edited more extensively in the squid giant axon than in its cell bodies.
28 he falling phase of action potentials in the squid giant axon, the diversity of voltage-gated potassi
29 nd Huxley model and for eliciting a spike in squid giant axons, the preparation for which the model w
30                     After injection into the squid giant axon, tubulin was transported in an anterogr
31           By applying rapid voltage steps to squid giant axons, we previously identified three compon
32 in to the bath saline induces the sealing of squid giant axons, whereas the addition of inhibitors of
33 t intracellular perfusion of voltage-clamped squid giant axons with a solution containing K+ and TEA+