ライフサイエンスコーパス: starburst

1 disk, rather than in a merger-driven nuclear starburst.

2 ral gas reservoir that is fueling a vigorous starburst.

3 years of cosmic history in a short, extreme starburst.

4 star-formation rates in protogalaxies and in starbursts.

5 low illumination including glare, halos, and starbursts.

6 that can be observed as luminous quasars and starbursts.

7 tidal dwarfs(7,14,15) and intragroup-medium starbursts(16), as well as widespread intergalactic shoc

8 (two orders of magnitude higher than extreme starbursts(5)) and star-formation rates of 200 solar mas

9 ve glare (84%/36%), blurry vision (68%/22%), starbursts (66%/28%), hazy vision (63%/18%), snowballs (

10 pes, Rbfox2 was colocalized with cholinergic starburst ACs, NPY (neuropeptide Y)- and EBF1 (early B-c

11 on of H(2) is not co-spatial with the buried starburst activity and originates outside the obscured r

12 d-state ortho-H(2)O(1(10)-1(01)) line due to starburst activity in the dusty galaxy HFLS3 results in

13 ative corneal curvature is a risk factor for starbursts after laser-assisted in situ keratomileusis (

14 he retina, individual dendritic sectors of a starburst amacrine cell (SAC) are preferentially activat

15 ieving radially symmetric arborization of On starburst amacrine cell (SAC) dendrites and normal SAC s

16 mate retina, direction selectivity arises in starburst amacrine cell (SAC) dendrites, which provide s

17 ymmetric GABAergic inhibition exerted by the starburst amacrine cell (SAC), a cholinergic and GABAerg

18 ring of a single inhibitory interneuron, the starburst amacrine cell (SAC), with each DSGC subtype pr

19 inputs and neurotransmitter release sites on starburst amacrine cell dendrites: the excitatory input

20 s) directs laminar segregation of OFF and ON starburst amacrine cell dendritic scaffolds, which thems

21 In contrast, the Starburst amacrine cell has GABA(B) receptors in a subpo

22 Aside from rare counterexamples (e.g. the starburst amacrine cell in retina), neurons are polarize

23 The starburst amacrine cell in the mouse retina presents an

24 ABA release from the presynaptic interneuron starburst amacrine cell in the mouse retina.

25 These features of the starburst amacrine cell network suggest that starburst c

26 two different strains of mice that differ in starburst amacrine cell number.

27 spontaneous cholinergic activity or reducing starburst amacrine cell numbers prevents invasion of end

28 ltransferase-IR) but in less than 50% of the starburst amacrine cell somata.

29 tute a novel cholinergic, non-GABAergic, non-starburst amacrine cell type described for the first tim

30 In fact, the starburst amacrine cell was the only amacrine cell type

31 etina, this study reports that the displaced starburst amacrine cell, a unique cholinergic interneuro

32 scribed of these pairs is the very elaborate starburst amacrine cell, A5, which stains regularly in t

33 ing, ON-OFF polyaxonal amacrine cell and the starburst amacrine cell, all of which show direction sel

34 n the dendrites of an interneuron, i.e., the starburst amacrine cell, and that these responses are hi

35 neurons, that a presynaptic interneuron, the starburst amacrine cell, delivers direct inhibition to d

36 for generating direction selectivity in the starburst amacrine cell.

37 rlying direction-selective GABA release from starburst amacrine cells

38 ; and dendrites of orthotopic- and displaced-starburst amacrine cells (identified by choline acetyltr

39 ccur first in the retina in the dendrites of starburst amacrine cells (interneurons presynaptic to th

40 earch of clues, here we reconstruct Off-type starburst amacrine cells (SACs) and bipolar cells (BCs)

41 rupts self-avoidance of dendrites in retinal starburst amacrine cells (SACs) and cerebellar Purkinje

42 BA release during null-direction motion from starburst amacrine cells (SACs) and DS acetylcholine and

43 he mouse retina to show connectivity between starburst amacrine cells (SACs) and their known synaptic

44 Starburst amacrine cells (SACs) are an essential compone

45 nisms driving input-output transformation in starburst amacrine cells (SACs) are not fully understood

46 the mammalian retina, inhibitory inputs onto starburst amacrine cells (SACs) are required for robust

47 Starburst amacrine cells (SACs) are thought to mediate t

48 in the ganglion cell layer (GCL) and ON-type starburst amacrine cells (SACs) during early postnatal d

49 erations in the retina, and loss of Slit2 in starburst amacrine cells (SACs) enhances oDSGC firing pr

50 Pharmacologically isolated starburst amacrine cells (SACs) in perinatal rabbit reti

51 The dendrites of starburst amacrine cells (SACs) in the mammalian retina

52 processing of visual information by retinal starburst amacrine cells (SACs) involves transforming ex

53 y selective wiring of inhibitory inputs from starburst amacrine cells (SACs) onto four subtypes of ON

54 Starburst amacrine cells (SACs) process complex visual s

55 recordings revealed that distal processes of starburst amacrine cells (SACs) received largely excitat

56 that local inhibition arising from GABAergic starburst amacrine cells (SACs) strongly contributes to

57 try in the inhibitory neurotransmission from starburst amacrine cells (SACs) to direction selective g

58 mission of acetylcholine (ACh) and GABA from starburst amacrine cells (SACs) to direction-selective g

59 lective ganglion cells (DSGCs) and GABAergic starburst amacrine cells (SACs), and the SACs then provi

60 In this study, we used retinal starburst amacrine cells (SACs), critical components of

61 y connected cholinergic interneurons, called starburst amacrine cells (SACs), generates spontaneous r

62 neonatal rodents, retinal waves initiate in starburst amacrine cells (SACs), propagating across reti

63 Here, we focused on one such pair, starburst amacrine cells (SACs), to explore how closely

64 ne receptors (beta2-nAChRs) selectively from starburst amacrine cells (SACs), we show that mutual exc

65 to be initiated by the spontaneous firing of Starburst Amacrine Cells (SACs), whose dense, recurrent

66 ecorded in retinal ganglion cells (RGCs) and starburst amacrine cells (SACs).

67 tually modulated cholinergic excitation from starburst amacrine cells (SACs).

68 ll-side inhibition, which is provided by OFF starburst amacrine cells (SACs).

69 of the mature inner retina are the so-called starburst amacrine cells (SACs).

70 Directional GABA release from starburst amacrine cells (SBACs) is critical for generat

71 established that the inhibition arises from starburst amacrine cells (SBACs) located on the null sid

72 tric GABA release from the dendritic tips of starburst amacrine cells (SBACs).

73 at glycinergic cells inhibit the cholinergic Starburst amacrine cells and are in turn inhibited by GA

74 rgic connections between direction-selective starburst amacrine cells and downstream ganglion cells i

75 mosaics by two retinal interneuron subtypes, starburst amacrine cells and horizontal cells.

76 Cholinergic "retinal waves" are initiated in starburst amacrine cells and propagate to retinal gangli

77 ch costratify near the processes of both the starburst amacrine cells and the ON-OFF directionally se

78 Starburst amacrine cells and their synaptic partners, ON

79 sitive bipolar cell provides input to the ON-starburst amacrine cells and/or the ON-plexus of the ON-

80 We also confirmed that starburst amacrine cells are presynaptic to ganglion cel

81 lls in the ganglion cell layer revealed that starburst amacrine cells are the most KA-responsive cell

82 allowed imaging spontaneous calcium waves in starburst amacrine cells during development, and light-e

83 the DAPI-3 cell, we have also shown that the starburst amacrine cells exhibit no immunoreactivity for

84 Thus, starburst amacrine cells form the first identified netwo

85 Starburst amacrine cells have a synaptic asymmetry that

86 57BL/6J (B6) strain, although the mosaics of starburst amacrine cells in both strains are comparably

87 Starburst amacrine cells in the macaque retina were stud

88 udy has compared the dendritic morphology of starburst amacrine cells in two different strains of mic

89 that acetylcholine release from presynaptic starburst amacrine cells is crucial for its generation.

90 blocked by TTX, indicating that release from starburst amacrine cells is independent of sodium action

91 The light response of starburst amacrine cells is initiated by glutamate relea

92 ude that the predominant excitatory input to starburst amacrine cells is mediated by AMPA receptors.

93 A prominent role of starburst amacrine cells is the generation of directiona

94 erned by direction-selective inhibition from starburst amacrine cells occurring during stimulus movem

95 tive release of gamma-aminobutyric acid from starburst amacrine cells onto direction-selective gangli

96 The network of starburst amacrine cells plays a fundamental role in the

97 Starburst amacrine cells release two classical neurotran

98 olution localization of GABA(B) receptors on starburst amacrine cells shows that they are discretely

99 (A) strain contains about one-quarter fewer starburst amacrine cells than does the C57BL/6J (B6) str

100 sitize rapidly, enhancing the sensitivity of starburst amacrine cells to moving or other rapidly chan

101 idence is found for inhibitory synapses from starburst amacrine cells to the ON-OFF DS GC.

102 In the INL, presumed starburst amacrine cells were homogenous in appearance a

103 Labeling the processes of starburst amacrine cells with antibodies against choline

104 f a mutually inhibitory relationship between starburst amacrine cells with overlapping dendrites.

105 re dictated by recurrent connectivity within starburst amacrine cells, and retinal ganglion cells act

106 SV2C was present in starburst amacrine cells, other conventional synapses in

107 e of the neurotransmitter acetylcholine from starburst amacrine cells, the effect of DCG-IV on ON-OFF

108 te in the dendrites of GABAergic/cholinergic starburst amacrine cells, where it is first observed.

109 An important issue is how the starburst amacrine cells, which are known to provide a m

110 ass radial direction selective excitation to starburst amacrine cells, which contributes to their dir

111 For example, starburst amacrine cells, which lack syt 2 in adult reti

112 ing RGC loss in the Ndufs4 KO is the loss of starburst amacrine cells, which may be an important targ

113 dritic field, is dependent on the density of starburst amacrine cells.

114 RGCs by inhibiting transmitter release from starburst amacrine cells.

115 te receptor (mGluR) subtype are expressed in starburst amacrine cells.

116 f GABA(B) receptors on cholinergic/GABAergic starburst amacrine cells.

117 nd SV2C being important for the functions of starburst amacrine cells.

118 rret retina through immunotoxin depletion of starburst amacrine cells.

119 ly belonged to several different cholinergic starburst amacrine cells.

120 tly more distally to the processes of the ON-starburst amacrine cells.

121 e of the two mirror-symmetric populations of starburst amacrine cells.

122 ic, nicotinic acetylcholine (ACh) input from starburst amacrine cells.

123 rd GABAergic/cholinergic signals mediated by starburst amacrine cells.

124 production (by 30%) of RGCs, and absence of starburst amacrine cells.

125 bpopulation of W3 ganglion cells, but not to starburst amacrine cells.

126 y be generated by a mechanism independent of starburst amacrine cells.

127 ic acid (GABA) release from the dendrites of starburst amacrine cells.

128 Regular spacing was also observed for the starburst amacrine cells.

129 natal stages and adult retinas, the presumed starburst amacrine ChAT-IR cells formed two mirror-like

130 re transformed by the GABAergic/cholinergic "starburst" amacrine cells (SACs), which are critical com

131 In the retina, the beautifully symmetrical 'starburst' amacrine cells interact with each other in a

132 mate inputs across the dendrites of retinal 'starburst' amacrine cells is one of the several mechanis

133 The membrane excitability of cholinergic (starburst) amacrine cells was studied in the rabbit reti

134 h-clamp recordings from pairs of neighboring starburst and ganglion cells show that the rhythmic acti

135 ay limit uncorrected visual acuity and cause starbursts and glare at night.

136 son) showed a correlation with postoperative starbursts and pupil size and a decrease in starbursts w

137 gh redshift these have only been observed in starbursts and quasars(11,12).

138 rative dysphotopsia introduces glare, halos, starbursts and shadows in a small number of patients.

139 tifacts manifest themselves as glare, halos, starbursts and shadows.

140 ved that they were formed in intense nuclear starbursts and that they ultimately grew into the most m

141 This ancestral starburst appears similar to those being found by submil

142 eparation of 19 kiloparsecs, the two massive starbursts are about to merge and form a passive ellipti

143 ct, GNz7q, associated with a dust-enshrouded starburst at a redshift of 7.1899 +/- 0.0005.

144 ganglion cell that does not stratify in the starburst band, this suggests that its GABA-dependent di

145 ynapses from a type of amacrine cell termed 'starburst' because of its regularly spaced, evenly radia

146 , but so far there has been no evidence that starbursts can propel substantial quantities of cold mol

147 te to the electrical isolation of individual starburst cell dendrites, a property thought essential f

148 o make these receptors active comes from the Starburst cell itself, making them autoreceptors.

149 ohistochemistry revealed their expression in starburst cell somata and dendrites.

150 cell, which then feeds back and inhibits the Starburst cell.

151 We report that the directional responses of starburst-cell dendrites and DS ganglion cells are highl

152 distribution of the two cotransporters along starburst-cell dendrites mediates direction selectivity.

153 evidence that the cholinergic outputs of the starburst cells affect the responses of the ganglion cel

154 tivity and that the directional responses of starburst cells and DS ganglion cells are exquisitely se

155 stead of being direction discriminators, the starburst cells appear to potentiate generically the res

156 The processes of starburst cells are connected asymmetrically to directio

157 ll recording, Zheng et al. now show that the starburst cells are mutually excitatory during early dev

158 h maturation, the nicotinic synapses between starburst cells dramatically diminished, whereas the GAB

159 lamp recordings were made from 110 displaced starburst cells in a thin retina] slice preparation of r

160 We labelled starburst cells in living retinas, then killed them by t

161 ase, we performed whole-cell recordings from starburst cells in mouse retina.

162 al nicotinic and GABAergic synapses between, starburst cells in the perinatal rabbit retina.

163 The same was true of starburst cells injected intracellularly with Neurobioti

164 ion cells show that the rhythmic activity in starburst cells is closely correlated with that in gangl

165 the directional discrimination in which the starburst cells participate, namely, that their choliner

166 Moreover, we discovered that macaque starburst cells possess a strong, non-GABAergic, antagon

167 Starburst cells project inhibition laterally ahead of a

168 starburst amacrine cell network suggest that starburst cells regulate their dendritic overlap to ensu

169 After vision begins, starburst cells release GABA in a prolonged and Ca2+-dep

170 cking the K-Cl cotransporter resulted in the starburst cells responding equally to light moving in op

171 erstand the intrinsic membrane properties of starburst cells responsible for direction-selective GABA

172 endrites and KCC2 on the distal dendrites of starburst cells results in a GABA-evoked depolarization

173 Ablating starburst cells revealed no asymmetric contribution to t

174 fter eye opening (P10), such that all of the starburst cells tested before eye opening had conspicuou

175 We show that this transition allows the starburst cells to use their neurotransmitters for two c

176 We report that displaced starburst cells undergo a dramatic transition from spiki

177 Our results suggest that starburst cells use action potentials transiently during

178 er, in contrast to ganglion cells, displaced starburst cells usually do not generate spontaneous soma

179 The Kv3-like current in starburst cells was absent in Kv3.1-Kv3.2 knock-out mice

180 e bipolar cells follow the pattern of the ON-starburst cells' processes.

181 sistently hyperpolarized and depolarized the starburst cells, respectively, and greatly reduced or el

182 olinergic and the GABAergic synapses between starburst cells.

183 immunolabeling favoring proximal regions of starburst cells.

184 glion cells in a manner opposite that of the starburst cells.

185 he bundles formed by the processes of the ON-starburst cells.

186 er an alternative concept of the function of starburst cells.

187 at S15 and included not only the presumptive starburst cholinergic amacrine cells but also a populati

188 om the differential connectivity patterns of starburst cholinergic and GABAergic synapses to ganglion

189 ed a high level of synaptic intricacy in the starburst circuit and suggested differential, yet synerg

190 A 'maximum starburst' converts the gas into stars at a rate more th

191 ngton black-hole accretion disk at the dusty starburst core.

192 contrast sensitivity without glare, halos or starbursts, defocus curves, optical scatter, retinal poi

193 l (i.t.) injection of 131I-labeled boronated starburst dendrimer (BSD) or BSD-EGF.

194 boronated precision macromolecule [boronated starburst dendrimer (BSD)] was chemically linked to EGF

195 bacteria using an optical sensor based on a starburst dendrimer film containing a lipophilic fluorop

196 A fourth generation starburst dendrimer was boronated and linked to EGF usin

197 Our results indicate that Starburst dendrimers can be effective carriers for the i

198 These studies demonstrate that Starburst dendrimers can transfect a wide variety of cel

199 onal release of gamma-aminobutyric acid from starburst dendrites and that the asymmetric distribution

200 ion cells remains remarkably unaffected when starburst dendrites are rendered non-directional, using

201 the NKCC2 and KCC2 cotransporters located on starburst dendrites consistently hyperpolarized and depo

202 dynamics of bipolar release onto ON- and OFF-starburst dendrites in the murine retina.

203 measure the input kinetics across individual starburst dendrites.

204 of the GABA reversal potential in different starburst dendritic compartments indicate that the GABA

205 Individual sectors of the starburst dendritic field are directionally selective by

206 robably approximately 3, indicating that the starburst-driven wind limits the star-formation activity

207 ymptoms (double images, glare, halos, and/or starbursts), dry eye symptoms, participant satisfaction

208 l symptoms (double images, glare, halos, and starbursts), dry eye symptoms, satisfaction with vision,

209 These galaxies transitioned rapidly between starburst episodes and phases of suppressed star formati

210 ure enough to form the most massive, intense starbursts existed at least as early as 880 million year

211 at resemble spherical baskets, cuboid cages, starbursts, flowers, scaffolds, fences, and frameworks,

212 The galaxy experienced a short starburst followed by rapid quenching; its stellar mass

213 cluster appears to be experiencing a massive starburst (formation of around 740 solar masses a year),

214 g of plate-like rectangles and fan-shaped or starburst forms.

215 g far (more than 10 kiloparsecs) outside the starburst galaxies (which have radii of less than 1 kilo

216 Consequently, starburst galaxies are ideal for studying the interplay

217 z > 4, indicating that the fraction of dusty starburst galaxies at high redshifts is greater than pre

218 Starburst galaxies at low redshifts, however, generally

219 bsorption lines in the spectra of six lensed starburst galaxies at redshifts near 2.5.

220 Starburst galaxies at the peak of cosmic star formation

221 Recent observations have revealed that starburst galaxies can drive molecular gas outflows thro

222 ormation within the cores of highly luminous starburst galaxies in the distant Universe.

223 tions indicate additional contributions from starburst galaxies or heavily filtered quasar radiation.

224 onized by the discovery that luminous, dusty starburst galaxies were 1,000 times more abundant in the

225 early epochs(2-4), most of them are extreme starburst galaxies with star-formation rates exceeding 1

226 are limited to a rare population of extreme starburst galaxies(8-12) and companions of rare quasars(

227 population of luminous, high-redshift, dusty starburst galaxies.

228 ntial masses and alter the evolution of post-starburst galaxies.

229 ocess, especially in the central regions of 'starburst' galaxies where star formation is vigorous.

230 Young clusters in 'starbursting' galaxies in the local and distant Universe

231 we report observations of NGC 253, a nearby starburst galaxy (distance approximately 3.4 megaparsecs

232 mpact (effective radius 100 parsecs) massive starburst galaxy at redshift 0.7, which is known to driv

233 resolution of 700 parsecs) of ALESS 073.1, a starburst galaxy at redshift [Formula: see text] when th

234 re we report the identification of a massive starburst galaxy at z = 6.34 through a submillimetre col

235 Here we report that the nearby dwarf starburst galaxy Henize 2-10 (refs 5 and 6) contains a c

236 outside the galactic plane of the archetypal starburst galaxy M82 (refs 4 and 5), but so far there ha

237 solution x-ray observations of the prototype starburst galaxy Messier 82 (M82) obtained with the adva

238 ocalized to a 20-square-arcmin region of the starburst galaxy NGC 253, located about 3.5 million pars

239 Henize 2-10 is a dwarf starburst galaxy previously reported to have a central m

240 times more mass than the cold gas in a post-starburst galaxy wind.

241 NGC 4449 is a nearby Magellanic irregular starburst galaxy with a B-band absolute magnitude of -18

242 icrowave background at z = 6.34 in a massive starburst galaxy, corresponding to a lookback time of 12

243 with significantly more reduction in halos, starbursts, glare, and rings and spider webs, but less i

244 out the bothersomeness of 7 visual symptoms: starburst, halo, glare, hazy vision, blurred vision, dou

245 Starbursts, halos, and glare were the most frequently ra

246 redshift (z) distribution of dusty, massive starbursts has long been suspected to be biased low in z

247 hile positive dysphotopsia (glare, halos and starbursts) has been largely attributed to edge effects

248 Winds from compact starbursts have been observed to flow to distances somew

249 porting any visual experience (glare, halos, starbursts, hazy vision, blurred vision, distortion, dou

250 following 14 symptoms: glare, blurry vision, starbursts, hazy vision, snowballs, halos, floaters, dou

251 sities and star-formation rates than extreme starbursts, in line with typical star-forming galaxies a

252 In addition, starburst inhibition is itself directionally selective:

253 , indicating a coordinated conversion of the starburst network excitability from an early hyperexcita

254 During early development, the starburst network mediates recurrent excitation and spon

255 arising from distinct specializations of the starburst network.

256 essing amacrine cells are distinct from AII, starburst, nGnG, vGluT3-positive, VIP-positive, and dopa

257 gham model in which GABA(B) receptors are on starburst, not glycinergic amacrine cells.

258 Halos, glare, and starbursts occurred in 52%, 29%, and 24% of subjects, re

259 g individual amacrine cell circuits like the starburst or A17 circuit have demonstrated that single t

260 f 138) for a pigmented lesion with a typical starburst pattern seen via dermatoscopy.

261 e total number of photons emitted during the starburst phase is sufficient to ionize intergalactic me

262 intly to turbulence and gravity, extends the starburst phase of a galaxy instead of quenching it.

263 Starburst polyamidoamine (PAMAM) dendrimers are a new ty

264 Starburst polyamidoamine dendrimers are a new class of s

265 t synthesized a novel resin monomer, 9-armed starburst polyurethane acrylate (NPUA), via the grafting

266 unknown how soon after the Big Bang massive starburst progenitors exist.

267 ce of gaps in the neutral gas enveloping the starburst region.

268 to expanding molecular shells located in the starburst region.

269 screen was ejected from the galaxy during a starburst several 10(8) years earlier and has subsequent

270 Expanding molecular shells observed in starburst systems such as NGC 253 and M 82 may facilitat

271 thway for in situ spheroid formation through starbursts that is probably preferentially triggered by

272 quasars emerging from heavily dust-obscured starbursts that then transition to unobscured luminous q

273 cript autonomously bioluminescent reporters (STARBURSTs) that link the five genes of a fungal biolumi

274 vy elements through intense, dust-enshrouded starbursts--that is, increased rates of star formation--

275 If the emission is from dust heated by starbursts, then the majority of stars in spheroids were

276 facilitate the transfer of information from starburst to ganglion cell dendrites.

277 of glutamate is processed differentially by starbursts via AMPA receptors and DSGCs via NMDA recepto

278 ense gas, an essential feature of an immense starburst, which contributes, together with the active g

279 s observation, we synthesized six GC-boosted STARBURSTs, which produced robust luminescence in both t

280 t have formed at early epochs in spectacular starbursts, which should be luminous phenomena when obse

281 starbursts and pupil size and a decrease in starbursts with wavefront-guided treatments compared wit