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1 RYGB, 12 of 26 patients had mild to moderate steatorrhea.
2 ve insufficient lipase to abolish pancreatic steatorrhea.
3 e test is an inaccurate marker of pancreatic steatorrhea.
4 ight the difficulty in abolishing pancreatic steatorrhea.
5 w fat diet, the Slc10a2-/- mice did not have steatorrhea.
6  low fat diet, the Clps-/- mice did not have steatorrhea.
7 ieved, one of which was discarded because of steatorrhea.
8 IU of porcine lipase (18 g) nearly abolished steatorrhea.
9 hould be evaluated in humans with pancreatic steatorrhea.
10 steatorrhea and may abolish human pancreatic steatorrhea.
11 th high-fat meals corrects canine pancreatic steatorrhea.
12 ated with rapid transit and mild to moderate steatorrhea.
13 ess dietary fat that can produce hyperphagic steatorrhea.
14 pain was 75%, jaundice 25%, weight loss 42%, steatorrhea 37.5%, diabetes 37.5%, and history of pencre
15 dosing is common for treatment of pancreatic steatorrhea; 90 000 United States Pharmacopeia units of
16 ile dogs exhibited stunted postnatal growth, steatorrhea, abdominal distension and a wiry coat.
17  infected with T3SA+ but not T3SA- developed steatorrhea and bilirubinemia.
18                                              Steatorrhea and hyperoxaluria were common in obese patie
19                                              Steatorrhea and hyperoxaluria were defined as fecal fat
20                The classic sprue syndrome of steatorrhea and malnutrition coupled with multiple defic
21 l lipase and high-fat meals abolishes canine steatorrhea and may abolish human pancreatic steatorrhea
22 an cause false-positive results on tests for steatorrhea and may therefore lead to an erroneous diagn
23            After RYGB, 24 of 26 patients had steatorrhea and urine oxalate excretion averaged 69 mg/d
24     Hyperoxaluria after RYGB correlated with steatorrhea and was presumably caused by excess fatty ac
25 intestines, abdominal pain, excessive feces, steatorrhea, and malnutrition.
26  gene (Slc10a2) lead to congenital diarrhea, steatorrhea, and reduced plasma cholesterol levels.
27 oms other than the typical ones of diarrhea, steatorrhea, and weight loss.
28 oscopic colitis; 23 patients with pancreatic steatorrhea; and 7 patients with treated celiac sprue wh
29 ncreased to levels observed in patients with steatorrhea caused by the malabsorption syndrome.
30                          Because post-bypass steatorrhea caused little increase in fecal water excret
31 e at risk of vitamin A deficiency because of steatorrhea, despite pancreatic enzyme replacement.
32  as common as is the classic presentation of steatorrhea, diarrhea, and weight loss.
33 in fecal water excretion, most patients with steatorrhea did not consider themselves to have diarrhea
34           Chow-fed, Mttp-IKO mice manifested steatorrhea, growth arrest, and decreased cholesterol ab
35 andard meal, powder bacterial lipase reduced steatorrhea in a dose-dependent manner (P = 0.03), and 1
36 isorder associated with congenital diarrhea, steatorrhea, interruption of the enterohepatic circulati
37                                              Steatorrhea may be seen in more severe cases.
38  and 300,000 IU of the liquid form decreased steatorrhea more than powder bacterial lipase (P = 0.017
39            Liquid bacterial lipase decreases steatorrhea more than powder, and 300,000 IU of powder b
40 requent symptom in untreated celiac disease, steatorrhea occurred in only one-fifth of patients.
41 e F508del gene variant begin in infancy with steatorrhea, poor weight gain, and respiratory symptoms
42                                For orlistat, steatorrhea produces the principal gastrointestinal side
43                                   Correcting steatorrhea requires 75 times more porcine than bacteria
44 ansit carcinoid diarrhea in association with steatorrhea results in impairment of gastric emptying.
45 ting to this study were microscopic colitis, steatorrhea secondary to exocrine pancreatic insufficien
46 labsorption, magnitudes of hyperoxaluria and steatorrhea should correlate.
47 aintain their weight gain in the presence of steatorrhea, the Clps-/- mice had hyperphagia on a high
48            To create an animal model of PEI, steatorrhea was induced by embolization of the exocrine
49 lations of bacterial lipase and diets affect steatorrhea was tested in pancreatic-insufficient dogs.