ライフサイエンスコーパス: stellate

1 kinin caused more extensive necrosis in both stellate and acinar cells than TLC-S alone.

2 We found that both stellate and basket cells engaged in synchronized waves

3 Molecular layer interneurons (MLIs, stellate and basket cells) of the cerebellar cortex are

4 alpha-Syn preformed fibrils (PFFs) into the stellate and celiac ganglia induces spreading of alpha-S

5 or colliculus or cochlear nucleus to label T-stellate and D-stellate neurons, respectively.

6 nct populations of multipolar cells exist: T-stellate and D-stellate neurons, thought to project to i

7 fully TARPed receptors typical of cerebellar stellate and Purkinje cells.

8 sure appears to conserve the distribution of stellate and pyramidal cells, periodic arrangement of ca

9 cortex (S1), excitatory neurons were mostly stellate and SOM(+) interneurons were non-Martinotti.

10 n the SGS and exhibit narrow field vertical, stellate, and horizontal dendritic morphologies, while G

11 ed that hepatocytes, sinusoidal endothelial, stellate, and liver-specific immune cells were released

12 cement, while those added labially achieve a stellate appearance.

13 into two distinct cell types, pyramidal and stellate, based on morphology, immunoreactivity, and fun

14 Activated hepatic stellate cell (aHSC)-mediated liver fibrosis is essentia

15 n of obeticholic acid (OCA) prevents hepatic stellate cell (HSC) activation and fibrogenesis.

16 yrosine kinases, which contribute to hepatic stellate cell (HSC) activation and liver fibrosis.

17 Hepatic stellate cell (HSC) activation and transforming growth f

18 ternal stiffness is known to promote hepatic stellate cell (HSC) activation through mechanotransducti

19 We investigated mechanisms of hepatic stellate cell (HSC) activation, which contributes to liv

20 ould partly account for reduction of hepatic stellate cell (HSC) activation.

21 pathways included overexpression of hepatic stellate cell (HSC) activators such as fibronectin 1 (FN

22 on, LPC biliary differentiation, and hepatic stellate cell (HSC) chemotaxis.

23 onsequences of hepatocyte-macrophage-hepatic stellate cell (HSC) crosstalk.

24 beta-4 (TB4) involved in regulating hepatic stellate cell (HSC) functions remain unclear.

25 zebrafish liver in vivo and in human hepatic stellate cell (HSC) lines in culture activates fibrotic

26 ssion in the parenchymal hepatocyte, hepatic stellate cell (HSC), and the inflammatory compartments i

27 cided with alterations in markers of hepatic stellate cell activation and extracellular matrix remode

28 he dKO mice had similar levels of markers of stellate cell activation and matrix remodeling as Ppara(

29 nalicular formation and is needed to prevent stellate cell activation by modulating TGF-beta.

30 ally, LPI promoted the initiation of hepatic stellate cell activation by stimulating GPR55 and activa

31 usoidal endothelial cell capillarization and stellate cell activation demonstrates allograft injury i

32 C1QTNF2 expression is reduced during hepatic stellate cell activation in culture and in a mouse model

33 osis and suppressed expression of markers of stellate cell activation in livers of mice fed a diet ri

34 Finally, MSDC-0602 directly reduced hepatic stellate cell activation in vitro, and MSDC-0602 treatme

35 ent or hepatocyte MPC2 deletion also limited stellate cell activation indirectly by affecting secreti

36 rentiation into polarized MPhis that mediate stellate cell activation via TGF-beta.

37 Hepatic stellate cell activation was detected by immunofluoresce

38 r sinusoidal endothelial cell activation and stellate cell activation was increased in patients with

39 tion of hepatic progenitor cell response and stellate cell activation, and normalization of liver enz

40 lting in hyperammonemia, evidence of hepatic stellate cell activation, and progressive fibrosis.

41 The activated macrophages caused stellate cell activation, leading to liver injury, by a

42 l protein adducts accompanied by evidence of stellate cell activation, matrix remodeling, and fibrosi

43 iated with hepatic progenitor cell features, stellate cell activation, NOTCH signaling, and an aggres

44 Inhibiting IRE1alpha blocked stellate cell activation, which then decreased prolifera

45 iferation, senescence, fibrosis, and hepatic stellate cell activation, which were reduced in Hdc(-/-)

46 tivation, prothrombin activation and hepatic stellate cell activation.

47 models of monocyte/macrophage and/or hepatic stellate cell activation.

48 vated monocytes/macrophages promoted hepatic stellate cell activation.

49 Furthermore, during licking, stellate cell activity was anisotropic: the coordination

50 In contrast, lack of stellate cell communication increases the regularity of

51 Hepatocyte and stellate cell deletion of liver fatty acid binding prote

52 The alignment of local collaterals and T-stellate cell dendrites within the isofrequency lamina s

53 , but not in co-culture with a human hepatic stellate cell line (LX-2) overexpressing LHX2.

54 involving the TRAIL receptors in the hepatic stellate cell line, LX2.

55 f hepatocyte markers, oval cell markers, and stellate cell markers.

56 iber (PF) to PC synapses and DSI at putative Stellate cell to PC synapses.

57 proaches in mice, we compare how the lack of stellate cell versus basket cell GABAergic neurotransmis

58 Mechanistically, PTX3 mediated the hepatic stellate cell wound-healing response.

59 flammation accompanied with elevated hepatic stellate cell-derived TnC and Toll-like receptor 4 expre

60 lls (iPS-HPCs) and human iPS-derived hepatic stellate cell-like cells (iPS-HSCs).

61 or olive glycinergic synapse, and the basket/stellate cell-Purkinje GABAergic synapse in the cerebell

62 in the VCN, a projection neuron termed the D-stellate cell.

63 inergic cells in the VCN distinct from the D-stellate cell.

64 myofibroblastic phenotype (activated hepatic stellate cell; aHSCs) expressing smooth muscle alpha-act

65 owever, the molecular mechanisms for hepatic stellate-cell activation by HCV-infected hepatocytes are

66 lesions more likely contained predominantly stellate cells (6/14 [43%] vs 0/14 [0%]; P = .05) and fi

67 Activated hepatic stellate cells (aHSCs) orchestrate scarring during liver

68 Barrel cortex layer IV spiny stellate cells (bSCs) are the primary recipients of asce

69 poptosis resistance in primary human hepatic stellate cells (hHSC).

70 type 2 (CCR2) is expressed by active hepatic stellate cells (HSC) and is a key monocyte recruitment s

71 g the crosstalk of hepatocytes (HC), hepatic stellate cells (HSC) and liver sinusoidal endothelial ce

72 Hepatic stellate cells (HSC) are the major cellular contributors

73 iorates liver fibrosis by inhibiting hepatic stellate cells (HSC), and loss of miR-200a is associated

74 nse (UPR) both promote activation of hepatic stellate cells (HSC), however the link between the two s

75 incipally regulated by activation of hepatic stellate cells (HSC).

76 MLH) comprising primary macrophages, hepatic stellate cells (HSC, LX-2), and hepatocytes (Huh-7), per

77 (FAK) plays a key role in promoting hepatic stellate cells (HSCs) activation in vitro and liver fibr

78 nti-fibrotic effects of neratinib in hepatic stellate cells (HSCs) and in vivo models of CCl(4)-induc

79 ulate transcription, is expressed by hepatic stellate cells (HSCs) and is required for development of

80 liver fibrosis remission by killing hepatic stellate cells (HSCs) and producing interferon (IFN)-gam

81 Hepatic stellate cells (HSCs) are key players in the development

82 (SM alpha-actin) is up-regulated in hepatic stellate cells (HSCs) as they transition to myofibroblas

83 hesis that the NLRP3 inflammasome in hepatic stellate cells (HSCs) can directly regulate their activa

84 Activation of hepatic stellate cells (HSCs) contributes to the development of

85 kinase receptor, is up-regulated in hepatic stellate cells (HSCs) during chronic liver injury.

86 Effects of LPS on fibrogenic hepatic stellate cells (HSCs) from WT and TLR4-KO mice were asse

87 Activation of hepatic stellate cells (HSCs) in response to injury is a key ste

88 Hepatic stellate cells (HSCs) induce hepatic inflammation and im

89 Fbeta induces the differentiation of hepatic stellate cells (HSCs) into tumor-promoting myofibroblast

90 Activation of hepatic stellate cells (HSCs) is a critical step in the developm

91 Rho kinase activity in hepatic stellate cells (HSCs) is associated with activation, tra

92 erating cholangiocytes and activated hepatic stellate cells (HSCs) participate in the promotion of li

93 Hepatic stellate cells (HSCs) play a major role increasing IHVR

94 Hepatic stellate cells (HSCs) play critical roles in liver fibro

95 FXR activation in hepatic stellate cells (HSCs) reduces liver fibrosis (LF).

96 Finally, hepatic stellate cells (HSCs) serve as a hub of intrahepatic sig

97 e demonstrated previously that mouse hepatic stellate cells (HSCs) suppress T cells via programmed de

98 rix proteins, such as collagen I, by hepatic stellate cells (HSCs) that culminates in cirrhosis.

99 hese secreted cytokines may activate hepatic stellate cells (HSCs) toward fibrosis.

100 Hepatic stellate cells (HSCs) were recently identified as liver-

101 increased LPA levels, activation of hepatic stellate cells (HSCs), and amplification of profibrotic

102 vity was detected in cholangiocytes, hepatic stellate cells (HSCs), and hepatocytes.

103 enic microenvironment, activation of hepatic stellate cells (HSCs), and progression of biliary fibros

104 liver disease is mostly displayed in hepatic stellate cells (HSCs), causing fibrosis/cirrhosis, and i

105 nt with activation and senescence of hepatic stellate cells (HSCs), exhibiting a senescence-associate

106 coholic steatohepatitis (NASH) using hepatic stellate cells (HSCs), hepatocytes, and mouse models of

107 Activated hepatic stellate cells (HSCs), liver sinusoidal endothelial cell

108 directly targeting Gli3 in activated hepatic stellate cells (HSCs), reduces expression of Gli3 and pr

109 en promotes mechanical quiescence in hepatic stellate cells (HSCs), stromal fibroblast-like cells who

110 transcriptome signature of activated hepatic stellate cells (HSCs), the primary collagen-secreting ce

111 Hepatic stellate cells (HSCs), the primary mediators of fibrosis

112 tion (BDL) and in cultured activated hepatic stellate cells (HSCs), we show that OPN, besides being o

113 or beta (TGFbeta) potently activates hepatic stellate cells (HSCs), which promotes production and sec

114 ing in enterocytes, hepatocytes, and hepatic stellate cells (HSCs).

115 cells (LSECs) promote quiescence of hepatic stellate cells (HSCs).

116 liferation and activation of resting hepatic stellate cells (HSCs).

117 fibrosis is marked by activation of hepatic stellate cells (HSCs).

118 in cell lines of cholangiocytes and hepatic stellate cells (HSCs).

119 Treatment of hepatic stellate cells (liver cells responsible for fibrosis) wi

120 med in vitro studies on immortalized hepatic stellate cells (LX-2).

121 ate cultured fibroblasts and primary hepatic stellate cells (myofibroblast precursors in the liver) i

122 ral cell types, including pancreatic acinar, stellate cells (PaSCs) and immune cells, SOCE is mediate

123 Recent findings implicate pancreatic stellate cells (PSC) as prominent mediators of inflammat

124 Conditioned media from pancreatic stellate cells (PSC), as well as from other fibroblasts,

125 ber 17 (SLC22A17) in human pancreatic cancer stellate cells (PSC), key mediators of the PDAC stroma.

126 Activation of pancreatic stellate cells (PSCs) and consequent development of dens

127 Pancreatic stellate cells (PSCs) are key mediators in the productio

128 Normal pancreatic stellate cells (PSCs) are regarded as quiescent, only to

129 Pancreatic stellate cells (PSCs) differentiate into cancer-associat

130 Differentiation of pancreatic stellate cells (PSCs) into myofibroblasts is inhibited b

131 activation of myofibroblast-like pancreatic stellate cells (PSCs) plays a predominant role in the fo

132 hen PC cells are co-cultured with pancreatic stellate cells (PSCs) they are significantly more resist

133 play a passive role in activating pancreatic stellate cells (PSCs) via recruitment of immune cells th

134 ng tumor-associated platelets and pancreatic stellate cells (PSCs), the two major players in the TME,

135 During liver injury, quiescent hepatic stellate cells (qHSCs) transdifferentiate into prolifera

136 s and the synaptic integrative properties of stellate cells (SCs) in the medial entorhinal cortex.

137 s (BCs) in the mouse cochlear nucleus with T-stellate cells (SCs), which do have normal overshooting

138 tigate Kv2 channel functions in mEC layer II stellate cells (SCs).

139 se progression, and an increase in activated stellate cells after BDL in mice lacking iRhom2 (Rhbdf2(

140 y, is trapped in the basal infoldings of the stellate cells after kinin diuretic peptide stimulation,

141 Addition of hepatic stellate cells allowed generation of myeloid-derived sup

142 etanercept reduced the presence of activated stellate cells and alleviated liver fibrosis after BDL.

143 Smooth-muscle actin expression by stellate cells and CD34 expression by liver sinusoidal e

144 in-1 (IL-1) receptor-dependent activation of stellate cells and endothelial cells, resulting in the t

145 secretory phenotype of quiescent pancreatic stellate cells and established an immunosuppressive mili

146 iculum (ER) stress in the cross-talk between stellate cells and HCC cells.

147 aller fraction of NPs accumulated in hepatic stellate cells and liver sinusoidal endothelial cells, s

148 ls under normoxia and hypoxia, human hepatic stellate cells and LX2 cells, and xenograft tumors forme

149 e involvement of GATAe in the maintenance of stellate cells and migration of renal and nephritic stem

150 ct the tumor microenvironment by stimulating stellate cells and myeloid suppressors with TMZ-CD40L an

151 ractors for galectin-3 in live human hepatic stellate cells and peripheral blood mononuclear cells.

152 ting combined with RNA sequencing to isolate stellate cells and PMCs, and we identified determinants

153 xtracellular matrix and numbers of activated stellate cells and portal fibroblasts.

154 events, including the activation of hepatic stellate cells and regulation of immune responses.

155 othelial cell types as well as early hepatic stellate cells and reveal distinct spatiotemporal distri

156 We found that sGC is expressed in hepatic stellate cells and stellate-derived myofibroblasts, but

157 of interconnections could account for how T-stellate cells are able to encode spectral peaks over a

158 g of spectral peaks.SIGNIFICANCE STATEMENT T-stellate cells are interconnected through synapses that

159 Hepatic stellate cells are key players in the progression of HCC

160 Parenchymal stellate cells are the primary contributors to fibrosis

161 arget of rapamycin, likely targeting hepatic stellate cells because differentiation and activation of

162 Nitric oxide (NO) donors evoked EPSCs in T-stellate cells but not in the other types of principal c

163 pamine release increases the AP threshold of stellate cells by activating D2Rs.

164 ectional and polysynaptic, indicating that T-stellate cells connect in networks.

165 arily potentiated interconnections between T-stellate cells could enhance the gain of auditory nerve

166 on of miR-200b in cholangiocytes and hepatic stellate cells decreased the expression of miR-200b, ang

167 ivation of the autophagic pathway in hepatic stellate cells during Brucella infection could have an i

168 The tonotopic array of T-stellate cells enhances the encoding of spectral peaks r

169 In vitro primary mouse hepatic stellate cells exhibited iRhom2-dependent shedding of th

170 from relay neurons in the thalamus to spiny stellate cells in layer 4 of the primary visual cortex (

171 Stellate cells in layer II of the mEC project to the hip

172 s in acinar cells, had only minor effects on stellate cells in lobules.

173 Recordings from pairs of T-stellate cells in mice of both sexes revealed that firin

174 uring pancreatic cancer cells and pancreatic stellate cells in multiple ratios to mimic variable tumo

175 Voltage clamp recordings from mEC stellate cells in rat brain slices showed that GTx inhib

176 Stellate cells in the medial entorhinal cortex (MEC) are

177 ly, RSCs do not replenish principal cells or stellate cells in the upper tubules.

178 T-stellate cells in the ventral cochlear nucleus (VCN) for

179 This compound decreases the activation of stellate cells in vitro and in vivo, by reducing the lev

180 on of miR-200b in cholangiocytes and hepatic stellate cells in vitro, we evaluated angiogenesis and f

181 D3 expression, whereas endothelial cells and stellate cells induced LXR-alpha via a synergistic NOTCH

182 t recruit neutrophils and convert pancreatic stellate cells into cancer-associated fibroblasts (CAFs)

183 decreases neuronal excitability in layer II stellate cells of medial entorhinal cortex.

184 but not under hyperpolarization, of layer II stellate cells of the MEC.

185 , mutation or RNAi-mediated knockdown in the stellate cells of the tubule of TAR2 (tyrR, CG7431) resu

186 parate putative pyramidal cells and putative stellate cells recorded extracellularly in layer II of t

187 re killed by oncolysis, whereas infection of stellate cells reduced factors involved in stroma format

188 ell types show phase precession but putative stellate cells show steeper slopes of phase precession a

189 In addition, KCs activate hepatic stellate cells that are involved in liver fibrosis.

190 put image-based screen using primary hepatic stellate cells that identified the antifungal drug itrac

191 is a circulating factor produced by hepatic stellate cells that plays a critical role in vascular qu

192 anism by which D2Rs modulate AP threshold of stellate cells through T-type Ca(2+) channels in MEC, in

193 ivation of the autophagic pathway in hepatic stellate cells to create a microenvironment that promote

194 trol that could account for the ability of T-stellate cells to enhance the encoding of spectral peaks

195 aliciguat acted directly on isolated hepatic stellate cells to inhibit fibrotic and inflammatory sign

196 AIL pathway can mediate apoptosis of hepatic stellate cells to promote the resolution of liver fibros

197 howed that macrophages and activated hepatic stellate cells were the main cell types expressing PTX3

198 Astrocytes are stellate cells whose appearance can resemble a pointed s

199 models have shown that targeting pancreatic stellate cells with all-trans-retinoic-acid (ATRA) repro

200 VIP neurons are glutamatergic stellate cells with sustained firing patterns.

201 We previously demonstrated that pancreatic stellate cells within pancreatic ductal adenocarcinoma (

202 ECs (Liver Endothelial Cells), HSCs (Hepatic Stellate Cells) and/or myofibroblasts to mimic in vivo f

203 and molecular layer interneurons (basket and stellate cells).

204 ssociation between macrophages and activated stellate cells, and a new potential role of tuft cells i

205 es in the majority of pyramidal cells, spiny stellate cells, and interneurons within the extrastriate

206 es, biliary epithelial cells, Kupffer cells, stellate cells, and liver sinusoidal endothelial cells e

207 They were activated by auditory nerve and T-stellate cells, and made local inhibitory synaptic conta

208 etwork that includes macrophages, pancreatic stellate cells, and prominent cytokines that are present

209 a subset of TGF-beta target genes in hepatic stellate cells, and the cooperation between the JAK1-STA

210 reconstructions showed that NPY neurons are stellate cells, and the dendrites of NPY neurons in the

211 Compared with stellate cells, Gli1(+) PMCs expressed a different subse

212 ), which is secreted by activated pancreatic stellate cells, has important functions in chronic pancr

213 s that the Kupffer cell niche is composed of stellate cells, hepatocytes, and endothelial cells that

214 is likely that these neurons, here termed L-stellate cells, play a significant role in frequency-spe

215 Contrary to the stellate cells, pyramidal cells show weaker temporal cod

216 ut not putative dentate gyrus/CA3-projecting stellate cells, represented speed prospectively.

217 active component) directly activate hepatic stellate cells, the fibrogenic cell in the liver, and dr

218 lize to opposite plasma membranes, and small stellate cells, the site of the chloride shunt conductan

219 we found that HCC-cells activate IREalpha in stellate cells, thereby contributing to their activation

220 ctly inhibiting the activation of pancreatic stellate cells, thereby reducing the deposition of extra

221 By co-culturing HCC-cells with stellate cells, we found that HCC-cells activate IREalph

222 and as ammonia is known to activate hepatic stellate cells, we hypothesized that ammonia may be invo

223 The inflammatory cells activate hepatic stellate cells, which are the major source of myofibrobl

224 e can induce prolonged activation of hepatic stellate cells, which may result in liver fibrosis.

225 ADAM17-mediated shedding of TNFRs in hepatic stellate cells, which reduces TNFR signaling and liver f

226 and decreased the intrinsic excitability of stellate cells, which was caused by shifting rightward t

227 ular reaction, which were instead encased by stellate cells.

228 lates the neuronal intrinsic excitability of stellate cells.

229 flammatory gene expression within pancreatic stellate cells.

230 vitamin A storage, resembled that of hepatic stellate cells.

231 t how dopamine modulates the function of MEC stellate cells.

232 n of fibrosis-related genes in primary human stellate cells.

233 yclase (NO-GC), are expressed in somata of T-stellate cells.

234 fibroblasts, dermal fibroblasts and hepatic stellate cells.

235 ession suppresses MICA expression in hepatic stellate cells.

236 area, but over ten-fold more numerous than D-stellate cells.

237 ic diseases and the activation of pancreatic stellate cells.

238 y phenotype in pancreatic normal, cancer and stellate cells.

239 ctivates fibrosis-related markers in hepatic stellate cells.

240 ell as primary human hepatocytes and hepatic stellate cells.

241 s marker activation in primary human hepatic stellate cells.

242 (+) -dependent bile acid uptake mechanism in stellate cells.

243 only signalling in acinar cells but also in stellate cells.

244 n mice, human hepatocytes, and human hepatic stellate cells.

245 revealed a significant reduction in hepatic stellate cells.

246 osmotically obliged water flows through the stellate cells.

247 in modulating the information processing of stellate cells.

248 R-induced AP threshold plasticity in AISs of stellate cells.

249 patocyte growth factor production by hepatic stellate cells.

250 s (OR 2.88, 95% CI:1.17-7.11, p = 0.022), or stellate cellular processes with no visible nuclei (OR 2

251 arger fraction of L4B input from M-dominated stellates compared with thin stripes, which project to a

252 ibution of L4B inputs from M-dominated spiny stellates compared with thin stripes.

253 or quantitative formation of the six-pointed stellated complex.

254 C is expressed in hepatic stellate cells and stellate-derived myofibroblasts, but not in hepatocytes.

255 However, it is unknown how pyramids and stellates distribute their outputs to the different V2 s

256 There was sympathetic hyperinnervation in stellate ganglia (p = 0.02) but not ventricles (p = 0.2)

257 ons in an intrinsic cardiac ganglion and the stellate ganglia, respectively, that project to the sino

258 Left stellate ganglion (LSG) hyperactivity promotes ischemia

259 rhythm and nerve activity (NA) from the left stellate ganglion (SNA), left cardiac vagus (VNA), and a

260 fter RTX administration, increases in GP and stellate ganglion activity and blood pressure during apn

261 her oxygen desaturation, a tonic increase in stellate ganglion activity and blood pressure ensued.

262 activity, followed by vagal bursts and tonic stellate ganglion firing.

263 refractory to drug therapy treated with left stellate ganglion transcutaneous magnetic stimulation (T

264 recordings from bilateral vagal nerves, left stellate ganglion, and anterior right GP were obtained b

265 ine of Drosophila melanogaster repression of Stellate genes by piRNAs generated from Supressor of Ste

266 erm cells in the testes, but in them harmful Stellate genes were derepressed due to the absence of Su

267 Co-cultures of human hepatoma and hepatic stellate (HSCs) cells were exposed to free fatty acids (

268 Poor outcomes were associated with stellate interconnected cellular processes with no visib

269 gins are selectively essential in cerebellar stellate interneurons for enabling the function of extra

270 Ethanol caused stellate lesions with patchy areas of normal myocardium,

271 Primary or immortalized human hepatic stellate (LX2) cells were exposed to exosomes derived fr

272 A series of stellated metallosupramolecular architectures have been

273 NPY neurons fire spontaneously, have a stellate morphology, and project to the auditory thalamu

274 thick stripes, but one type, the giant spiny-stellate neuron, resembling L4B neurons projecting to mo

275 ined the cellular properties of layer II mEC stellate neurons (mEC-SCs) in rTg4510 mice, a rodent mod

276 Our findings identify MeA stellate neurons as an important component in the respon

277 eonatal ventricular myocytes and sympathetic stellate neurons from normal (WKY) and pro-hypertensive

278 of the dendritic branches in layer IV spiny stellate neurons is reduced.

279 mine the effects of the mutation on layer II stellate neurons of the medial entorhinal cortex (mEC),

280 es were observed in MeA bipolar neurons, BLA stellate neurons or in lateral amygdala stellate neurons

281 This suggest that both T- and D-stellate neurons receive synaptic innervation from the M

282 e dendritic spine density of mGluR5 KO spiny stellate neurons was significantly higher than in wild-t

283 In layer 3C, spiny stellate neurons were distributed mainly in foveal repre

284 r cochlear nucleus to label T-stellate and D-stellate neurons, respectively.

285 of multipolar cells exist: T-stellate and D-stellate neurons, thought to project to inferior collicu

286 ructural remodeling of medial amygdala (MeA) stellate neurons.

287 BLA stellate neurons or in lateral amygdala stellate neurons.

288 elopment of functional connectivity in spiny stellate neurons.

289 ons, resulted in an unprecedented mixture of stellated octanuclear and dodecanuclear metallocages, wh

290 Stellate odontodes deposited directly on the bony plate

291 the Martinotti/pyramidal and non-Martinotti/stellate pairs, are used across the cortex as building b

292 had not previously understood, with abundant stellate projection neurons in the high-resolution fovea

293 the morphology of widefield amacrine cells (stellate, semilunar, and thorny amacrine cells).

294 Immature pericytes showed stellate shape and high proliferation, and mature pericy

295 genes by piRNAs generated from Supressor of Stellate (Su(Ste)) locus is required for male fertility,

296 revealed choline acetyltransferase (ChAT) in stellate sympathetic neurons and vesicular ACh transport

297 eceive a much larger contribution from spiny stellates than previously shown for V2 overall, indicati

298 sthetized cat to a detailed model of a spiny stellate V1 neuron, we found that output spike timing pr

299 ) information to downstream areas, and spiny stellates, which carry only M information.

300 parvocellular (P) visual signals, and spiny stellates, which carry only M signals.