ライフサイエンスコーパス: stellate cell

1 and molecular layer interneurons (basket and stellate cells).

2 in the VCN, a projection neuron termed the D-stellate cell.

3 inergic cells in the VCN distinct from the D-stellate cell.

4 n of fibrosis-related genes in primary human stellate cells.

5 ular reaction, which were instead encased by stellate cells.

6 yclase (NO-GC), are expressed in somata of T-stellate cells.

7 fibroblasts, dermal fibroblasts and hepatic stellate cells.

8 ession suppresses MICA expression in hepatic stellate cells.

9 ic diseases and the activation of pancreatic stellate cells.

10 area, but over ten-fold more numerous than D-stellate cells.

11 y phenotype in pancreatic normal, cancer and stellate cells.

12 ctivates fibrosis-related markers in hepatic stellate cells.

13 ell as primary human hepatocytes and hepatic stellate cells.

14 s marker activation in primary human hepatic stellate cells.

15 (+) -dependent bile acid uptake mechanism in stellate cells.

16 only signalling in acinar cells but also in stellate cells.

17 n the overall bile acid uptake in pancreatic stellate cells.

18 ramidal cells were superimposed on scattered stellate cells.

19 asic effect on EPSC amplitudes recorded from stellate cells.

20 n mice, human hepatocytes, and human hepatic stellate cells.

21 osmotically obliged water flows through the stellate cells.

22 revealed a significant reduction in hepatic stellate cells.

23 in modulating the information processing of stellate cells.

24 R-induced AP threshold plasticity in AISs of stellate cells.

25 patocyte growth factor production by hepatic stellate cells.

26 lates the neuronal intrinsic excitability of stellate cells.

27 flammatory gene expression within pancreatic stellate cells.

28 vitamin A storage, resembled that of hepatic stellate cells.

29 t how dopamine modulates the function of MEC stellate cells.

30 lesions more likely contained predominantly stellate cells (6/14 [43%] vs 0/14 [0%]; P = .05) and fi

31 In stellate cells, a voltage-dependent increase in membrane

32 y liver disease activity scores, and hepatic stellate cell activation (alpha-smooth muscle actin) com

33 ic alphaSMA expression, a marker for hepatic stellate cell activation (r = -0.31, P < 0.02).

34 cided with alterations in markers of hepatic stellate cell activation and extracellular matrix remode

35 enefitted liver fibrosis via altered hepatic stellate cell activation and extracellular matrix remode

36 he activity of the PAR2 pepducin on cultured stellate cell activation and hepatocyte reactive oxygen

37 he dKO mice had similar levels of markers of stellate cell activation and matrix remodeling as Ppara(

38 nalicular formation and is needed to prevent stellate cell activation by modulating TGF-beta.

39 ally, LPI promoted the initiation of hepatic stellate cell activation by stimulating GPR55 and activa

40 usoidal endothelial cell capillarization and stellate cell activation demonstrates allograft injury i

41 C1QTNF2 expression is reduced during hepatic stellate cell activation in culture and in a mouse model

42 Vitamin A loss is accompanied by stellate cell activation in hepatic tissue.

43 osis and suppressed expression of markers of stellate cell activation in livers of mice fed a diet ri

44 Finally, MSDC-0602 directly reduced hepatic stellate cell activation in vitro, and MSDC-0602 treatme

45 ent or hepatocyte MPC2 deletion also limited stellate cell activation indirectly by affecting secreti

46 FR reduced hepatic stellate cell activation markers (transforming growth fa

47 LOX inhibition attenuated hepatic stellate cell activation markers and promoted F4/80-posi

48 rentiation into polarized MPhis that mediate stellate cell activation via TGF-beta.

49 Hepatic stellate cell activation was detected by immunofluoresce

50 r sinusoidal endothelial cell activation and stellate cell activation was increased in patients with

51 tion of hepatic progenitor cell response and stellate cell activation, and normalization of liver enz

52 lting in hyperammonemia, evidence of hepatic stellate cell activation, and progressive fibrosis.

53 The activated macrophages caused stellate cell activation, leading to liver injury, by a

54 l protein adducts accompanied by evidence of stellate cell activation, matrix remodeling, and fibrosi

55 iated with hepatic progenitor cell features, stellate cell activation, NOTCH signaling, and an aggres

56 Inhibiting IRE1alpha blocked stellate cell activation, which then decreased prolifera

57 iferation, senescence, fibrosis, and hepatic stellate cell activation, which were reduced in Hdc(-/-)

58 substrate stiffening resulted in attenuated stellate cell activation, with reduced YAP/TAZ nuclear s

59 tivation, prothrombin activation and hepatic stellate cell activation.

60 models of monocyte/macrophage and/or hepatic stellate cell activation.

61 vated monocytes/macrophages promoted hepatic stellate cell activation.

62 amine the relationship between retinoids and stellate cell activation.

63 owever, the molecular mechanisms for hepatic stellate-cell activation by HCV-infected hepatocytes are

64 Furthermore, during licking, stellate cell activity was anisotropic: the coordination

65 se progression, and an increase in activated stellate cells after BDL in mice lacking iRhom2 (Rhbdf2(

66 y, is trapped in the basal infoldings of the stellate cells after kinin diuretic peptide stimulation,

67 Activated hepatic stellate cell (aHSC)-mediated liver fibrosis is essentia

68 Activated hepatic stellate cells (aHSCs) orchestrate scarring during liver

69 myofibroblastic phenotype (activated hepatic stellate cell; aHSCs) expressing smooth muscle alpha-act

70 Addition of hepatic stellate cells allowed generation of myeloid-derived sup

71 etanercept reduced the presence of activated stellate cells and alleviated liver fibrosis after BDL.

72 Smooth-muscle actin expression by stellate cells and CD34 expression by liver sinusoidal e

73 in-1 (IL-1) receptor-dependent activation of stellate cells and endothelial cells, resulting in the t

74 secretory phenotype of quiescent pancreatic stellate cells and established an immunosuppressive mili

75 iculum (ER) stress in the cross-talk between stellate cells and HCC cells.

76 is an emerging new target expressed on liver stellate cells and hepatocytes that regulates the respon

77 ro experiments were performed in rat hepatic stellate cells and hepatocytes.

78 ression (LTD) between cortical layer 4 spiny stellate cells and layer 2/3 pyramidal cells requires th

79 aller fraction of NPs accumulated in hepatic stellate cells and liver sinusoidal endothelial cells, s

80 ls under normoxia and hypoxia, human hepatic stellate cells and LX2 cells, and xenograft tumors forme

81 e involvement of GATAe in the maintenance of stellate cells and migration of renal and nephritic stem

82 ct the tumor microenvironment by stimulating stellate cells and myeloid suppressors with TMZ-CD40L an

83 ractors for galectin-3 in live human hepatic stellate cells and peripheral blood mononuclear cells.

84 ting combined with RNA sequencing to isolate stellate cells and PMCs, and we identified determinants

85 xtracellular matrix and numbers of activated stellate cells and portal fibroblasts.

86 events, including the activation of hepatic stellate cells and regulation of immune responses.

87 othelial cell types as well as early hepatic stellate cells and reveal distinct spatiotemporal distri

88 We found that sGC is expressed in hepatic stellate cells and stellate-derived myofibroblasts, but

89 r escalated by bradykinin-induced signals in stellate cells and thus killing of stellate cells by bil

90 ECs (Liver Endothelial Cells), HSCs (Hepatic Stellate Cells) and/or myofibroblasts to mimic in vivo f

91 ssociation between macrophages and activated stellate cells, and a new potential role of tuft cells i

92 es in the majority of pyramidal cells, spiny stellate cells, and interneurons within the extrastriate

93 fibrogenic activation of attenuated hepatic stellate cells, and limits fibrosis reversal.

94 es, biliary epithelial cells, Kupffer cells, stellate cells, and liver sinusoidal endothelial cells e

95 They were activated by auditory nerve and T-stellate cells, and made local inhibitory synaptic conta

96 dothelin-induced vasoconstriction by hepatic stellate cells, and not platelet accumulation or coagula

97 etwork that includes macrophages, pancreatic stellate cells, and prominent cytokines that are present

98 a subset of TGF-beta target genes in hepatic stellate cells, and the cooperation between the JAK1-STA

99 reconstructions showed that NPY neurons are stellate cells, and the dendrites of NPY neurons in the

100 of interconnections could account for how T-stellate cells are able to encode spectral peaks over a

101 g of spectral peaks.SIGNIFICANCE STATEMENT T-stellate cells are interconnected through synapses that

102 Hepatic stellate cells are key players in the progression of HCC

103 Parenchymal stellate cells are the primary contributors to fibrosis

104 cinar cells but the effects of bile acids on stellate cells are unexplored.

105 ted excitatory synaptic responses, which, in stellate cells, are largely extrasynaptic, without a cha

106 arget of rapamycin, likely targeting hepatic stellate cells because differentiation and activation of

107 Barrel cortex layer IV spiny stellate cells (bSCs) are the primary recipients of asce

108 Nitric oxide (NO) donors evoked EPSCs in T-stellate cells but not in the other types of principal c

109 pamine release increases the AP threshold of stellate cells by activating D2Rs.

110 ignals in stellate cells and thus killing of stellate cells by bile acids might have important implic

111 Early postnatally, layer-2 pyramidal but not stellate-cells co-localized with doublecortin - a marker

112 In contrast, lack of stellate cell communication increases the regularity of

113 more dramatic Ca(2+) signals and necrosis in stellate cells compared to the adjacent acinar cells in

114 ectional and polysynaptic, indicating that T-stellate cells connect in networks.

115 arily potentiated interconnections between T-stellate cells could enhance the gain of auditory nerve

116 on of miR-200b in cholangiocytes and hepatic stellate cells decreased the expression of miR-200b, ang

117 Hepatocyte and stellate cell deletion of liver fatty acid binding prote

118 The alignment of local collaterals and T-stellate cell dendrites within the isofrequency lamina s

119 flammation accompanied with elevated hepatic stellate cell-derived TnC and Toll-like receptor 4 expre

120 ivation of the autophagic pathway in hepatic stellate cells during Brucella infection could have an i

121 The tonotopic array of T-stellate cells enhances the encoding of spectral peaks r

122 In vitro primary mouse hepatic stellate cells exhibited iRhom2-dependent shedding of th

123 rikingly, electrophysiological recordings in stellate cells from these PV-Cre/NL123 cKO mice revealed

124 Compared with stellate cells, Gli1(+) PMCs expressed a different subse

125 ), which is secreted by activated pancreatic stellate cells, has important functions in chronic pancr

126 s that the Kupffer cell niche is composed of stellate cells, hepatocytes, and endothelial cells that

127 poptosis resistance in primary human hepatic stellate cells (hHSC).

128 anscriptome of rat PMFs, compared to hepatic stellate cell HSC-derived myofibroblasts in culture, ide

129 n of obeticholic acid (OCA) prevents hepatic stellate cell (HSC) activation and fibrogenesis.

130 yrosine kinases, which contribute to hepatic stellate cell (HSC) activation and liver fibrosis.

131 Hepatic stellate cell (HSC) activation and transforming growth f

132 Hepatic stellate cell (HSC) activation on liver injury facilitat

133 ternal stiffness is known to promote hepatic stellate cell (HSC) activation through mechanotransducti

134 We investigated mechanisms of hepatic stellate cell (HSC) activation, which contributes to liv

135 ould partly account for reduction of hepatic stellate cell (HSC) activation.

136 pathways included overexpression of hepatic stellate cell (HSC) activators such as fibronectin 1 (FN

137 on, LPC biliary differentiation, and hepatic stellate cell (HSC) chemotaxis.

138 onsequences of hepatocyte-macrophage-hepatic stellate cell (HSC) crosstalk.

139 beta-4 (TB4) involved in regulating hepatic stellate cell (HSC) functions remain unclear.

140 zebrafish liver in vivo and in human hepatic stellate cell (HSC) lines in culture activates fibrotic

141 ssion in the parenchymal hepatocyte, hepatic stellate cell (HSC), and the inflammatory compartments i

142 type 2 (CCR2) is expressed by active hepatic stellate cells (HSC) and is a key monocyte recruitment s

143 g the crosstalk of hepatocytes (HC), hepatic stellate cells (HSC) and liver sinusoidal endothelial ce

144 Hepatic stellate cells (HSC) are the major cellular contributors

145 iorates liver fibrosis by inhibiting hepatic stellate cells (HSC), and loss of miR-200a is associated

146 nse (UPR) both promote activation of hepatic stellate cells (HSC), however the link between the two s

147 incipally regulated by activation of hepatic stellate cells (HSC).

148 MLH) comprising primary macrophages, hepatic stellate cells (HSC, LX-2), and hepatocytes (Huh-7), per

149 (FAK) plays a key role in promoting hepatic stellate cells (HSCs) activation in vitro and liver fibr

150 cipally expressed in quiescent mouse hepatic stellate cells (HSCs) and directly suppressed production

151 nti-fibrotic effects of neratinib in hepatic stellate cells (HSCs) and in vivo models of CCl(4)-induc

152 ulate transcription, is expressed by hepatic stellate cells (HSCs) and is required for development of

153 liver fibrosis remission by killing hepatic stellate cells (HSCs) and producing interferon (IFN)-gam

154 Hepatic stellate cells (HSCs) are key players in the development

155 (SM alpha-actin) is up-regulated in hepatic stellate cells (HSCs) as they transition to myofibroblas

156 hesis that the NLRP3 inflammasome in hepatic stellate cells (HSCs) can directly regulate their activa

157 Activation of hepatic stellate cells (HSCs) contributes to the development of

158 kinase receptor, is up-regulated in hepatic stellate cells (HSCs) during chronic liver injury.

159 Effects of LPS on fibrogenic hepatic stellate cells (HSCs) from WT and TLR4-KO mice were asse

160 Hepatic stellate cells (HSCs) have been identified as the main f

161 Activation of hepatic stellate cells (HSCs) in response to injury is a key ste

162 Hepatic stellate cells (HSCs) induce hepatic inflammation and im

163 Hepatic stellate cells (HSCs) inhibit T cells, a process that co

164 Fbeta induces the differentiation of hepatic stellate cells (HSCs) into tumor-promoting myofibroblast

165 Activation of hepatic stellate cells (HSCs) is a critical step in the developm

166 Rho kinase activity in hepatic stellate cells (HSCs) is associated with activation, tra

167 erating cholangiocytes and activated hepatic stellate cells (HSCs) participate in the promotion of li

168 Hepatic stellate cells (HSCs) play a major role increasing IHVR

169 Hepatic stellate cells (HSCs) play critical roles in liver fibro

170 FXR activation in hepatic stellate cells (HSCs) reduces liver fibrosis (LF).

171 Finally, hepatic stellate cells (HSCs) serve as a hub of intrahepatic sig

172 e demonstrated previously that mouse hepatic stellate cells (HSCs) suppress T cells via programmed de

173 rix proteins, such as collagen I, by hepatic stellate cells (HSCs) that culminates in cirrhosis.

174 hese secreted cytokines may activate hepatic stellate cells (HSCs) toward fibrosis.

175 Hepatic stellate cells (HSCs) were recently identified as liver-

176 increased LPA levels, activation of hepatic stellate cells (HSCs), and amplification of profibrotic

177 vity was detected in cholangiocytes, hepatic stellate cells (HSCs), and hepatocytes.

178 enic microenvironment, activation of hepatic stellate cells (HSCs), and progression of biliary fibros

179 liver disease is mostly displayed in hepatic stellate cells (HSCs), causing fibrosis/cirrhosis, and i

180 In cultured hepatic stellate cells (HSCs), disrupting Hedgehog signaling blo

181 nt with activation and senescence of hepatic stellate cells (HSCs), exhibiting a senescence-associate

182 coholic steatohepatitis (NASH) using hepatic stellate cells (HSCs), hepatocytes, and mouse models of

183 Activated hepatic stellate cells (HSCs), liver sinusoidal endothelial cell

184 directly targeting Gli3 in activated hepatic stellate cells (HSCs), reduces expression of Gli3 and pr

185 en promotes mechanical quiescence in hepatic stellate cells (HSCs), stromal fibroblast-like cells who

186 transcriptome signature of activated hepatic stellate cells (HSCs), the primary collagen-secreting ce

187 Hepatic stellate cells (HSCs), the primary mediators of fibrosis

188 tion (BDL) and in cultured activated hepatic stellate cells (HSCs), we show that OPN, besides being o

189 sm of this resistance by focusing on hepatic stellate cells (HSCs), which are known to regulate Treg

190 or beta (TGFbeta) potently activates hepatic stellate cells (HSCs), which promotes production and sec

191 cells (LSECs) promote quiescence of hepatic stellate cells (HSCs).

192 liferation and activation of resting hepatic stellate cells (HSCs).

193 fibrosis is marked by activation of hepatic stellate cells (HSCs).

194 elial neoplasia, or PDAC, as well as hepatic stellate cells (HSCs).

195 luding laminin (Ln)-332, produced by hepatic stellate cells (HSCs).

196 in cell lines of cholangiocytes and hepatic stellate cells (HSCs).

197 ing in enterocytes, hepatocytes, and hepatic stellate cells (HSCs).

198 from relay neurons in the thalamus to spiny stellate cells in layer 4 of the primary visual cortex (

199 cellular electrophysiological properties of stellate cells in layer II of MEC change systematically

200 Stellate cells in layer II of the mEC project to the hip

201 s in acinar cells, had only minor effects on stellate cells in lobules.

202 Recordings from pairs of T-stellate cells in mice of both sexes revealed that firin

203 cells (NFkappaB) in hepatocytes and hepatic stellate cells in monoculture; however, they do not acco

204 uring pancreatic cancer cells and pancreatic stellate cells in multiple ratios to mimic variable tumo

205 Voltage clamp recordings from mEC stellate cells in rat brain slices showed that GTx inhib

206 tor function at parallel fibre synapses onto stellate cells in the cerebellum using whole-cell patch-

207 Stellate cells in the medial entorhinal cortex (MEC) are

208 duced fibrosis and the numbers of pancreatic stellate cells in the tumor stroma and altered the types

209 ly, RSCs do not replenish principal cells or stellate cells in the upper tubules.

210 T-stellate cells in the ventral cochlear nucleus (VCN) for

211 e major cerebellar neuroligin isoforms, from stellate cells in triple NL123 conditional knock-out mic

212 pathophysiological effects of bile acids on stellate cells in two experimental models: ex vivo (mous

213 This compound decreases the activation of stellate cells in vitro and in vivo, by reducing the lev

214 on of miR-200b in cholangiocytes and hepatic stellate cells in vitro, we evaluated angiogenesis and f

215 D3 expression, whereas endothelial cells and stellate cells induced LXR-alpha via a synergistic NOTCH

216 Cells showed decreased fibrogenesis, hepatic stellate cell infiltration, Kupffer cells and inflammato

217 t recruit neutrophils and convert pancreatic stellate cells into cancer-associated fibroblasts (CAFs)

218 pairment in inhibitory synaptic responses in stellate cells lacking NL123 despite a nearly complete s

219 olate induced cytosolic Ca(2+) elevations in stellate cells, larger than those elicited simultaneousl

220 lls (iPS-HPCs) and human iPS-derived hepatic stellate cell-like cells (iPS-HSCs).

221 , but not in co-culture with a human hepatic stellate cell line (LX-2) overexpressing LHX2.

222 involving the TRAIL receptors in the hepatic stellate cell line, LX2.

223 ulation; coculture of hepatocyte and hepatic stellate cell lines significantly increased expression o

224 fibrogenic program in hepatocyte and hepatic stellate cell lines through ROS, NFkappaB, and TGFbeta1

225 Treatment of hepatic stellate cells (liver cells responsible for fibrosis) wi

226 tumor stroma had fewer activated pancreatic stellate cells, lower levels of periostin, and alteratio

227 med in vitro studies on immortalized hepatic stellate cells (LX-2).

228 f hepatocyte markers, oval cell markers, and stellate cell markers.

229 ate cultured fibroblasts and primary hepatic stellate cells (myofibroblast precursors in the liver) i

230 decreases neuronal excitability in layer II stellate cells of medial entorhinal cortex.

231 esponses in medial entorhinal cortical (MEC) stellate cells of rats, which express strong sub-thresho

232 but not under hyperpolarization, of layer II stellate cells of the MEC.

233 , mutation or RNAi-mediated knockdown in the stellate cells of the tubule of TAR2 (tyrR, CG7431) resu

234 ral cell types, including pancreatic acinar, stellate cells (PaSCs) and immune cells, SOCE is mediate

235 is likely that these neurons, here termed L-stellate cells, play a significant role in frequency-spe

236 tate transaminase, alanine transaminase, and stellate cell proliferation by up to 50-100%.

237 Recent findings implicate pancreatic stellate cells (PSC) as prominent mediators of inflammat

238 Conditioned media from pancreatic stellate cells (PSC), as well as from other fibroblasts,

239 ber 17 (SLC22A17) in human pancreatic cancer stellate cells (PSC), key mediators of the PDAC stroma.

240 oplasia, which is orchestrated by pancreatic stellate cells (PSCs) and accounts for the majority of t

241 Activation of pancreatic stellate cells (PSCs) and consequent development of dens

242 rix proteoglycan overexpressed by pancreatic stellate cells (PSCs) and pancreatic ductal adenocarcino

243 Pancreatic stellate cells (PSCs) are key mediators in the productio

244 Normal pancreatic stellate cells (PSCs) are regarded as quiescent, only to

245 Persistent activation of pancreatic stellate cells (PSCs) can perturb the biomechanical homo

246 Pancreatic stellate cells (PSCs) differentiate into cancer-associat

247 Differentiation of pancreatic stellate cells (PSCs) into myofibroblasts is inhibited b

248 activation of myofibroblast-like pancreatic stellate cells (PSCs) plays a predominant role in the fo

249 hen PC cells are co-cultured with pancreatic stellate cells (PSCs) they are significantly more resist

250 play a passive role in activating pancreatic stellate cells (PSCs) via recruitment of immune cells th

251 er of myofibroblasts or activated pancreatic stellate cells (PSCs)).

252 ng tumor-associated platelets and pancreatic stellate cells (PSCs), the two major players in the TME,

253 nalling between pancreatic acinar (PACs) and stellate cells (PSCs).

254 or olive glycinergic synapse, and the basket/stellate cell-Purkinje GABAergic synapse in the cerebell

255 Contrary to the stellate cells, pyramidal cells show weaker temporal cod

256 During liver injury, quiescent hepatic stellate cells (qHSCs) transdifferentiate into prolifera

257 parate putative pyramidal cells and putative stellate cells recorded extracellularly in layer II of t

258 re killed by oncolysis, whereas infection of stellate cells reduced factors involved in stroma format

259 brosis, attenuated the activation of hepatic stellate cells, reduced frequencies of Th9, Th17 and Th1

260 ver, bile acid-elicited signalling events in stellate cells remain unexplored.

261 ut not putative dentate gyrus/CA3-projecting stellate cells, represented speed prospectively.

262 Yet by recording from axons of cerebellar stellate cell (SC) interneurons, we show that AP width v

263 ffer (KC), sinusoidal endothelial (LSEC) and stellate cells (SC) are major cellular components of the

264 s and the synaptic integrative properties of stellate cells (SCs) in the medial entorhinal cortex.

265 s (BCs) in the mouse cochlear nucleus with T-stellate cells (SCs), which do have normal overshooting

266 tigate Kv2 channel functions in mEC layer II stellate cells (SCs).

267 Primary hepatic stellate cell-seeded hydrogels stiffened in situ at late

268 ctin mRNA), whereas EX increased the hepatic stellate cell senescence marker CCN1 (P < 0.01 vs. O-SED

269 ell types show phase precession but putative stellate cells show steeper slopes of phase precession a

270 ffects of neuroligin deletions on cerebellar stellate cell synapses by electrophysiology in acute sli

271 t cell synapses, but not at distal dendritic stellate cell synapses.

272 In addition, KCs activate hepatic stellate cells that are involved in liver fibrosis.

273 put image-based screen using primary hepatic stellate cells that identified the antifungal drug itrac

274 is a circulating factor produced by hepatic stellate cells that plays a critical role in vascular qu

275 active component) directly activate hepatic stellate cells, the fibrogenic cell in the liver, and dr

276 lize to opposite plasma membranes, and small stellate cells, the site of the chloride shunt conductan

277 we found that HCC-cells activate IREalpha in stellate cells, thereby contributing to their activation

278 ctly inhibiting the activation of pancreatic stellate cells, thereby reducing the deposition of extra

279 anism by which D2Rs modulate AP threshold of stellate cells through T-type Ca(2+) channels in MEC, in

280 iber (PF) to PC synapses and DSI at putative Stellate cell to PC synapses.

281 ivation of the autophagic pathway in hepatic stellate cells to create a microenvironment that promote

282 trol that could account for the ability of T-stellate cells to enhance the encoding of spectral peaks

283 aliciguat acted directly on isolated hepatic stellate cells to inhibit fibrotic and inflammatory sign

284 AIL pathway can mediate apoptosis of hepatic stellate cells to promote the resolution of liver fibros

285 proaches in mice, we compare how the lack of stellate cell versus basket cell GABAergic neurotransmis

286 DNA damage, whereas proliferation of hepatic stellate cells was stimulated by KCa3.1 inhibition.

287 By co-culturing HCC-cells with stellate cells, we found that HCC-cells activate IREalph

288 and as ammonia is known to activate hepatic stellate cells, we hypothesized that ammonia may be invo

289 howed that macrophages and activated hepatic stellate cells were the main cell types expressing PTX3

290 2 emerged around birth while reelin-positive stellate-cells were scattered throughout development.

291 The inflammatory cells activate hepatic stellate cells, which are the major source of myofibrobl

292 e can induce prolonged activation of hepatic stellate cells, which may result in liver fibrosis.

293 ADAM17-mediated shedding of TNFRs in hepatic stellate cells, which reduces TNFR signaling and liver f

294 d treatment caused necrosis predominantly in stellate cells, which was abolished by removal of extrac

295 and decreased the intrinsic excitability of stellate cells, which was caused by shifting rightward t

296 Astrocytes are stellate cells whose appearance can resemble a pointed s

297 models have shown that targeting pancreatic stellate cells with all-trans-retinoic-acid (ATRA) repro

298 VIP neurons are glutamatergic stellate cells with sustained firing patterns.

299 We previously demonstrated that pancreatic stellate cells within pancreatic ductal adenocarcinoma (

300 Mechanistically, PTX3 mediated the hepatic stellate cell wound-healing response.