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1 the presynaptic terminal of the adult squid stellate ganglion.
2 ere analyzed following PRV injections in the stellate ganglion.
3 days, PRV was injected into the ipsilateral stellate ganglion.
4 ic collaterals connecting neurons within the stellate ganglion.
5 sine hydroxylase-positive nerves in the left stellate ganglion 1 week after cessation of LL-VNS were
6 fter RTX administration, increases in GP and stellate ganglion activity and blood pressure during apn
7 her oxygen desaturation, a tonic increase in stellate ganglion activity and blood pressure ensued.
9 after unilateral anaesthetic blockade of the stellate ganglion, alone or in combination with selectiv
10 recordings from bilateral vagal nerves, left stellate ganglion, and anterior right GP were obtained b
11 hibition of P2X3R located in the sympathetic stellate ganglion as a promising novel therapeutic for s
12 erlie the anti-arrhythmic effects of partial stellate ganglion block in the setting of chronic MI.
13 low-level vagus nerve stimulation [LL-VNS], stellate ganglion block, baroreceptor stimulation, spina
16 ushes and night awakenings were treated with stellate-ganglion block at the anterolateral aspect of t
17 lot study to investigate our hypothesis that stellate-ganglion block can be a safe and effective trea
19 re were no adverse events resulting from the stellate-ganglion block, although patients had temporary
23 ather than fluoroscopy, to perform bilateral stellate ganglion blocks at the patient's bedside in the
25 into either the superior cervical ganglion, stellate ganglion, celiac ganglion, or adrenal gland of
26 ional sympathetic postganglionic cell in the stellate ganglion complex exhibited NOS immunoreactivity
27 e safety and effectiveness of CT-guided left stellate ganglion cryoneurolysis (SGC) in the treatment
33 t efferent cardiac sympathetic branch of the stellate ganglion in Zucker diabetic fatty rats revealed
34 anaesthetic block of the sympathetic nerves (stellate ganglion) innervating the contralateral (restin
35 Significant neural remodeling of the left stellate ganglion is evident 1 week after cessation of c
36 eft ventricle free wall (LVFW), and the left stellate ganglion (LSG) at several time points (30 minut
38 threshold electrical stimulation to the left stellate ganglion (LSG) in six normal dogs for 41 +/- 9
39 ession of either TcMS that targeted the left stellate ganglion (n = 14) or sham stimulation (n = 12).
40 low-level vagus nerve stimulation suppresses stellate ganglion nerve activities and reduces the incid
41 nuous (24/7) simultaneous monitoring of left stellate ganglion nerve activity (SGNA), vagal nerve act
42 nsic cardiac nerve activity (ECNA; including stellate ganglion nerve activity and vagal nerve activit
43 y were preceded by or coactivated with ECNA (stellate ganglion nerve activity or vagal nerve activity
44 des were activated alone without concomitant stellate ganglion nerve activity or vagal nerve activity
46 at 20 Hz for 1 week and then monitored left stellate ganglion nerve activity, left vagal nerve activ
47 transmitter for continuous recording of left stellate ganglion nerve activity, vagal nerve activities
49 e trunk evoked fast synaptic transmission in stellate ganglion neurons with low and high jitter laten
51 tigated the role of P2X3R in the sympathetic stellate ganglion of Wistar and spontaneously hypertensi
54 82% of sympathetic neurons isolated from the stellate ganglion predominantly express Na(V)1.7 mRNA.
55 hat noradrenergic sympathetic neurons in the stellate ganglion (SG) have excitatory cholinergic colla
56 remodeling occurs within the neurons of the stellate ganglion (SG) in the setting of heart failure (
58 nnervating the heart primarily reside in the stellate ganglion (SG), alongside neurons innervating ot
60 rhythm and nerve activity (NA) from the left stellate ganglion (SNA), left cardiac vagus (VNA), and a
62 Effects of electrically stimulating the left stellate ganglion to activate cardiopulmonary sympatheti
63 refractory to drug therapy treated with left stellate ganglion transcutaneous magnetic stimulation (T
64 0.9%, at the right or left (randomized 1:1) stellate ganglion was performed by a board-certified ane
65 erals from postganglionic neurons within the stellate ganglion, we knocked out choline acetyltransfer
66 dynamics in the embryonic mouse sympathetic stellate ganglion, where neuroblasts continue to prolife
67 nctionally different targets: adrenal gland, stellate ganglion which regulates the heart, or celiac g
68 cific primers detected the transcript in the stellate ganglion, which contains the cell bodies that g