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1 ce with AMD3100 (plerixafor) and its role in stem cell mobilization.
2 on of HSCs into the hepatic parenchyma after stem cell mobilization.
3 inical trials evaluating novel approaches to stem cell mobilization.
4 f relapse, toxicity, mechanism of action, or stem cell mobilization.
5 Fifty patients were enrolled and underwent stem cell mobilization.
6 00-mediated and G-CSF-mediated hematopoietic stem cell mobilization.
7 eutrophil chemoattraction, angiogenesis, and stem cell mobilization.
8 100), a clinical candidate for hematopoietic stem cell mobilization.
9 f PPM1H as a potential inhibition target for stem cell mobilization.
10 gnaling networks that underlie hematopoietic stem cell mobilization.
11 for HIV infection and plerixafor (CXCR4) for stem-cell mobilization.
14 c malignancies, and problems associated with stem cell mobilization after lenalidomide treatment.
15 diabetes-associated defect of hematopoietic stem cell mobilization after stimulation with granulocyt
17 ICE chemotherapy every 21 days, followed by stem cell mobilization and collection, and then 1 cycle
18 rtaken to evaluate the effect of hemopoietic stem cell mobilization and harvesting on HIV-1 replicati
20 ng haematopoietic stem cells, haematopoietic stem cell mobilization and lineage determination and B c
21 own of PHD and FIH synergistically increases stem cell mobilization and myocardial angiogenesis, lead
22 viously unknown signaling pathway regulating stem cell mobilization and provide a new pharmacological
23 F nanodiscs had greater activity in inducing stem cell mobilization and recruitment to the site of in
24 he care of patients with diabetes undergoing stem cell mobilization and transplantation and for the v
27 ypothesize that neuropathy of the BM affects stem cell mobilization and vascular recovery after ische
30 progression-free survival, overall survival, stem cell mobilization, and neutrophil and platelet engr
32 tors and small molecule CXCR4 antagonist for stem- cell mobilization, and in vivo experimental transp
33 ed that the adverse effects of pharmacologic stem cell mobilization are primarily mediated by the con
35 p better define the complicated mechanism of stem cell mobilization by G-CSF and point to a wide role
36 visual impairment was reported one day after stem cell mobilization by granulocyte-colony stimulating
38 or a range of clinical conditions, including stem cell mobilization, cancer prognosis and treatment,
40 s for venous thromboembolism prophylaxis and stem cell mobilization failure associated with lenalidom
42 o, to receive two additional RVD cycles plus stem-cell mobilization, followed by either five addition
44 drug,Me6TREN, may have broad applications in stem-cell mobilization for cancer and in regenerative me
45 he US Food and Drug Administration (FDA) for stem cell mobilization in cancer and administered for th
47 sion, autonomic neuropathy in the BM impairs stem cell mobilization in diabetes with dysregulation of
50 develop renal insufficiency while undergoing stem cell mobilization in preparation for an autologous
51 proliferation and facilitates hematopoietic stem cell mobilization in vivo, while the stromal-derive
54 of 2 platelet transfusions was required for stem cell mobilization, intravenous catheter insertion,
55 o ICE chemotherapy did not negatively affect stem cell mobilization or collection or engraftment, sim
56 single injection of both agents resulted in stem cell mobilization peaking within 15 min that was eq
60 onstrate in mice that endogenous bone marrow stem cell mobilization, produced by a pharmacologic comb
61 cing its myeloprotective or peripheral blood stem cell mobilization properties, which can be used to
62 use of granulocytosis-inducing hematopoietic stem cell mobilization protocols for the prevention or t
63 In conclusion, plerixafor results in rapid stem cell mobilization regardless of route of administra
65 ing T cells are efficiently mobilized during stem cell mobilization (SCM) and hypothesized that mobil
66 eptors, 5-HT(2B), is involved in bone marrow stem cell mobilization that participates in the developm
67 poraneous with filgrastim administration for stem cell mobilization, the patient's slowly progressive
68 f CXCR4 antagonists is an efficient means of stem cell mobilization, this fails to evoke the immunomo
69 f IGF-1 transgene expression induced massive stem cell mobilization via SDF-1alpha signaling and culm
72 cause anti-CD49d also supports hematopoietic stem cell mobilization, we sought to determine the thera
73 patients who had been primed with VP-16 for stem cell mobilization were at a 12.3-fold increased ris
74 time of ASCT, disease status, and method of stem-cell mobilization, were then analyzed with respect
75 portant therapeutic target for hematopoietic stem cell mobilization, which enhances the success of au