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1 ce with AMD3100 (plerixafor) and its role in stem cell mobilization.
2 on of HSCs into the hepatic parenchyma after stem cell mobilization.
3 inical trials evaluating novel approaches to stem cell mobilization.
4 f relapse, toxicity, mechanism of action, or stem cell mobilization.
5   Fifty patients were enrolled and underwent stem cell mobilization.
6 00-mediated and G-CSF-mediated hematopoietic stem cell mobilization.
7 eutrophil chemoattraction, angiogenesis, and stem cell mobilization.
8 100), a clinical candidate for hematopoietic stem cell mobilization.
9 f PPM1H as a potential inhibition target for stem cell mobilization.
10 gnaling networks that underlie hematopoietic stem cell mobilization.
11 for HIV infection and plerixafor (CXCR4) for stem-cell mobilization.
12            Rituximab was administered during stem-cell mobilization (1 day before chemotherapy at 375
13 dary end points were: overall response rate, stem-cell mobilization activity, and toxicity.
14 c malignancies, and problems associated with stem cell mobilization after lenalidomide treatment.
15  diabetes-associated defect of hematopoietic stem cell mobilization after stimulation with granulocyt
16                  Plerixafor, a hematopoietic stem cell mobilization agent, increases the peripheral b
17  ICE chemotherapy every 21 days, followed by stem cell mobilization and collection, and then 1 cycle
18 rtaken to evaluate the effect of hemopoietic stem cell mobilization and harvesting on HIV-1 replicati
19 ory process overall as well as hematopoietic stem cell mobilization and homing.
20 ng haematopoietic stem cells, haematopoietic stem cell mobilization and lineage determination and B c
21 own of PHD and FIH synergistically increases stem cell mobilization and myocardial angiogenesis, lead
22 viously unknown signaling pathway regulating stem cell mobilization and provide a new pharmacological
23 F nanodiscs had greater activity in inducing stem cell mobilization and recruitment to the site of in
24 he care of patients with diabetes undergoing stem cell mobilization and transplantation and for the v
25 in murine and rhesus monkey peripheral blood stem cell mobilization and transplantation models.
26 cell levels could lead to better methods for stem cell mobilization and transplantation.
27 ypothesize that neuropathy of the BM affects stem cell mobilization and vascular recovery after ische
28                         The impact of VDD on stem-cell mobilization and collection also was evaluated
29                                           As stem-cell mobilization and in vitro culture techniques h
30 progression-free survival, overall survival, stem cell mobilization, and neutrophil and platelet engr
31 emphases on patient selection, approaches to stem cell mobilization, and peri-SCT management.
32 tors and small molecule CXCR4 antagonist for stem- cell mobilization, and in vivo experimental transp
33 ed that the adverse effects of pharmacologic stem cell mobilization are primarily mediated by the con
34                       All patients underwent stem cell mobilization before 1:1 randomization to immun
35 p better define the complicated mechanism of stem cell mobilization by G-CSF and point to a wide role
36 visual impairment was reported one day after stem cell mobilization by granulocyte-colony stimulating
37      We confirmed that Treg depletion during stem cell mobilization can mitigate constraints on tumor
38 or a range of clinical conditions, including stem cell mobilization, cancer prognosis and treatment,
39                                    Augmented stem cell mobilization could also be demonstrated in mic
40 s for venous thromboembolism prophylaxis and stem cell mobilization failure associated with lenalidom
41                                Is peripheral stem cell mobilization followed by autologous stem cell
42 o, to receive two additional RVD cycles plus stem-cell mobilization, followed by either five addition
43 d the development of therapies to facilitate stem cell mobilization for clinical purposes.
44 drug,Me6TREN, may have broad applications in stem-cell mobilization for cancer and in regenerative me
45 he US Food and Drug Administration (FDA) for stem cell mobilization in cancer and administered for th
46        AMD3100 was recently FDA-approved for stem cell mobilization in combination with granulocyte-c
47 sion, autonomic neuropathy in the BM impairs stem cell mobilization in diabetes with dysregulation of
48       However, the safety and feasibility of stem cell mobilization in individuals with sickle cell t
49                           Optimal methods of stem cell mobilization in multiple myeloma are undefined
50 develop renal insufficiency while undergoing stem cell mobilization in preparation for an autologous
51  proliferation and facilitates hematopoietic stem cell mobilization in vivo, while the stromal-derive
52 ors and induces hematopoietic progenitor and stem cell mobilization in vivo.
53                    The STEM-AMI OUTCOME CMR (Stem Cells Mobilization in Acute Myocardial Infarction O
54  of 2 platelet transfusions was required for stem cell mobilization, intravenous catheter insertion,
55 o ICE chemotherapy did not negatively affect stem cell mobilization or collection or engraftment, sim
56  single injection of both agents resulted in stem cell mobilization peaking within 15 min that was eq
57 uding one who required intubation during his stem cell mobilization period.
58          Pretransplantation R did not affect stem-cell mobilization, post-transplantation early compl
59         Here, we demonstrate that endogenous stem cell mobilization produced by AMD3100 and low-dose
60 onstrate in mice that endogenous bone marrow stem cell mobilization, produced by a pharmacologic comb
61 cing its myeloprotective or peripheral blood stem cell mobilization properties, which can be used to
62 use of granulocytosis-inducing hematopoietic stem cell mobilization protocols for the prevention or t
63   In conclusion, plerixafor results in rapid stem cell mobilization regardless of route of administra
64                         We developed a rapid stem cell mobilization regimen utilizing a unique CXCR2
65 ing T cells are efficiently mobilized during stem cell mobilization (SCM) and hypothesized that mobil
66 eptors, 5-HT(2B), is involved in bone marrow stem cell mobilization that participates in the developm
67 poraneous with filgrastim administration for stem cell mobilization, the patient's slowly progressive
68 f CXCR4 antagonists is an efficient means of stem cell mobilization, this fails to evoke the immunomo
69 f IGF-1 transgene expression induced massive stem cell mobilization via SDF-1alpha signaling and culm
70                                              Stem cell mobilization was attempted in 88 patients and
71        Toxicity was largely hematologic, and stem cell mobilization was successful in 43 of 45 patien
72 cause anti-CD49d also supports hematopoietic stem cell mobilization, we sought to determine the thera
73  patients who had been primed with VP-16 for stem cell mobilization were at a 12.3-fold increased ris
74  time of ASCT, disease status, and method of stem-cell mobilization, were then analyzed with respect
75 portant therapeutic target for hematopoietic stem cell mobilization, which enhances the success of au
76                                 In contrast, stem cell mobilization with the CXCR4 antagonist AMD3100