ライフサイエンスコーパス: stenoses

1 the deep femoral artery (one thrombosis, 13 stenoses).

2 ree of six shunts and moderate in two (focal stenoses).

3 asurements were completed in 28 patients (28 stenoses).

4 stin insufficiency causes recurrent vascular stenoses.

5 city for predicting functionally significant stenoses.

6 provided, presenting only focal intermediate stenoses.

7 is used to treat significant atherosclerotic stenoses.

8 detecting functionally significant coronary stenoses.

9 city for predicting functionally significant stenoses.

10 for the pressure-only assessment of coronary stenoses.

11 edicting the true significance of individual stenoses.

12 and coronary CTA 1-6 d after PCS of culprit stenoses.

13 FFR was </=0.80 in 28 (45.9%) stenoses.

14 k between radiation and location of coronary stenoses.

15 identifying hemodynamically severe coronary stenoses.

16 tenoses and identifying more unstable milder stenoses.

17 , Massachusetts) in treating coronary artery stenoses.

18 severe saphenous vein aorto-coronary bypass stenoses.

19 integrating the functional component of the stenoses.

20 essel coronary artery disease, and left main stenoses.

21 xcellent vessel conspicuity and depiction of stenoses.

22 ol for the functional assessment of coronary stenoses.

23 There were 292 patients with 613 matched stenoses.

24 ngiography and DSA for detection of arterial stenoses.

25 erial image quality and presence of arterial stenoses.

26 ctive value for exclusion of coronary artery stenoses.

27 separately for extracranial and intracranial stenoses.

28 valuated vascular image quality and arterial stenoses.

29 management of select benign tracheobronchial stenoses.

30 urate assessment of the presence of coronary stenoses.

31 ptomatic hemodynamically significant carotid stenoses.

32 with grafts free of significant (> or =75%) stenoses.

33 This applies to native and artificial stenoses.

34 5 treated lesions were 88 occlusions and 417 stenoses.

35 compared with post-procedural duplex-defined stenoses.

36 ) were correlated with the number of complex stenoses.

37 rs of the number of angiographically complex stenoses.

38 6% for the detection of significant coronary stenoses.

39 patients with focal, native coronary artery stenoses.

40 ion of moderate and severe epicardial artery stenoses.

41 ion scan were both abnormal, 70% had >or=50% stenoses.

42 que in patients without significant coronary stenoses.

43 coronary arteries with only mild or moderate stenoses.

44 had hemodynamically significant renal artery stenoses.

45 hemodynamically significant accessory artery stenoses.

46 ree patients had hemodynamically significant stenoses.

47 cation matched arteries with the most severe stenoses.

48 discriminate malignant from nonmalignant CBD stenoses.

49 s, although the reverse held true for severe stenoses.

50 scriminating between different severities of stenoses.

51 patients with severe (>/=70%) single-vessel stenoses.

52 mine the functional significance of coronary stenoses.

53 , was the reference for defining significant stenoses.

54 with SIHD and angiographically intermediate stenoses.

55 PCI had revascularisation of all identified stenoses.

56 calcium score or obstructive coronary artery stenoses.

57 ate the hemodynamic significance of coronary stenoses.

58 ermediate or borderline significant coronary stenoses: (1) pressure wire-derived coronary fractional

59 METHODS AND In 1382 coronary stenoses (1104 patients), percent diameter stenosis by v

60 Particularly, in 38 stenoses (19%) with FFR values in the 0.81 to 0.85 range

61 D stenoses than controls or nonmalignant CBD stenoses (2.41 x 10(15) vs 1.60 x 10(14) nanoparticles/L

62 impaired intestinal transit comprised tumor stenoses 22% (11/50), infections 18% (9/50), post-operat

63 ks, 10 symptomatic marginal ulcers, 5 stomal stenoses, 3 bowel obstructions, 26 incisional hernias (n

64 At milder degrees of stenoses (50-69%), presence of collaterals was associate

65 n an additional 9 patients with intermediate stenoses (53+/-7%), 14 fractional flow reserve (FFR) mea

66 these two techniques of measuring CFR in 25 stenoses (6 vessels) artificially created by inflating s

67 stenoses (92%) and eight of 13 with coronary stenoses (62%) solely in the left anterior descending ar

68 Seventy-five stenoses (67 patients) underwent paired flow velocity as

69 patients without significant coronary artery stenoses (76 +/- 37 ms vs. 38 +/- 23 ms, p < 0.001).

70 ivity for detection of all individual vessel stenoses (78% vs. 58%, p < 0.001) and patients overall (

71 ICA confirmed 81% of calcific stenoses, 91% of mixed, and 67% of soft plaques.

72 eterization revealing 12 of 13 with coronary stenoses (92%) and eight of 13 with coronary stenoses (6

73 the per-stenosis analysis were 97% (29 of 30 stenoses), 96% (23 of 24 stenoses), 97% (29 of 30 stenos

74 ses), 96% (23 of 24 stenoses), 97% (29 of 30 stenoses), 96% (23 of 24 stenoses), and 96% (52 of 54 st

75 were 97% (29 of 30 stenoses), 96% (23 of 24 stenoses), 97% (29 of 30 stenoses), 96% (23 of 24 stenos

76 ding pressure by stenting of severe coronary stenoses, a proportional increase in vessel diameter is

77 was significantly smaller than DSQCA in mild stenoses, although the reverse held true for severe sten

78 condition with a common theme of multifocal stenoses and aneurysms in large arteries, accompanied by

79 Data for 50-69% stenoses and combinations of non-invasive tests were spa

80 in Eln(-)(/)(-) mice are also tortuous with stenoses and dilations.

81 between angiographic assessment of coronary stenoses and fractional flow reserve (FFR) is weak.

82 demonstrating a protective role with severe stenoses and identifying more unstable milder stenoses.

83 up 2: 85 patients with intermediate coronary stenoses and no AS treated with percutaneous coronary in

84 A total of 543 preoperative stenoses and occlusions were quantified and followed.

85 specificity were calculated for detection of stenoses and occlusions, as well as for confidence level

86 n images in the evaluation of central venous stenoses and occlusions.

87 on which coronary arteries have significant stenoses and on patient management.

88 In four arteries with severe stenoses and one occlusion (mean, 86%; range, 75%-100%),

89 uation of the ischemic potential of coronary stenoses and the expected benefit from revascularization

90 ment in multiple focal intracranial arterial stenoses and two demonstrated worsening.

91 ses), 97% (29 of 30 stenoses), 96% (23 of 24 stenoses), and 96% (52 of 54 stenoses), respectively.

92 nce of coronary calcification, morphology of stenoses, and anatomic characteristics, are under geneti

93 lates in vivo geometries, such as aneurysms, stenoses, and bifurcations, and supports endothelial cel

94 ved outcomes in small vessels, long coronary stenoses, and possibly saphenous vein graft intervention

95 uality of vessel definition, the severity of stenoses, and the presence of collateral vessels.

96 angiography for detection of coronary artery stenoses appears promising enough to warrant pursuit of

97 iagnosis of malignant common bile duct (CBD) stenoses are complex and lack accuracy.

98 ificant undergo PCI than when nonsignificant stenoses are treated.

99 identifying hemodynamically severe coronary stenoses as determined by fractional flow reserve (FFR).

100 at in patients with functionally significant stenoses, as determined by measurement of fractional flo

101 h coincided with cases revealing significant stenoses at CT coronary angiography.

102 ween FFR and CFVR occurred in 31% and 37% of stenoses at the 0.75, and 0.80 FFR cut-off value, respec

103 val, 12 swine underwent surgical creation of stenoses at the left common carotid, right renal, and le

104 For detection of >/=70% stenoses based on angiographic criteria, a fully automat

105 mic flow velocity was observed when treating stenoses below physiological cut points; treating stenos

106 There were no significant PV stenoses, but adverse events included 1 episode of cardi

107 the functional significance of intermediate stenoses, but also have inherent limitations.

108 care for treatment of native coronary artery stenoses, but optimum treatment strategies for bare meta

109 infarcts are generated by non-flow-limiting stenoses, but PCI is solely focused on treating flow-lim

110 om PCI was being considered, we assessed all stenoses by measuring FFR.

111 ion of hemodynamically significant (>or=50%) stenoses by using various image postprocessing methods,

112 th left main or ostial right coronary artery stenoses, bypass graft stenoses, chronic total occlusion

113 ion of plaques that may not produce critical stenoses causes many acute coronary syndromes (ACS).

114 nd the number and localization of functional stenoses changed in 32%.

115 right coronary artery stenoses, bypass graft stenoses, chronic total occlusions, planned two-stent bi

116 CI, hyperemic flow velocity is diminished in stenoses classed as physiologically significant compared

117 mic flow velocity increases 6-fold more when stenoses classed as physiologically significant undergo

118 higher in those with severe coronary artery stenoses compared to those with no vessel disease.

119 in patients with intermediate single-vessel stenoses, complex bifurcation and ostial branch stenoses

120 In 22 patients without significant coronary stenoses, contrast-enhanced MDCT (0.75-mm collimation, 4

121 ing and hyperemic flow velocity after PCI in stenoses defined physiologically by fractional flow rese

122 chemia may facilitate evaluation of moderate stenoses, designation of the culprit lesion, and predict

123 extracranial internal carotid artery (eICA) stenoses, detectable via submandibular Doppler sonograph

124 Long-term problems include coronary stenoses, distortion of the pulmonary arteries, dilatati

125 y enlargement) has expanded attention beyond stenoses evident by angiography to encompass the biology

126 Across all stenoses, extent of collaterals was a predictor for subs

127 addition to revealing 15 central vasculature stenoses, ferumoxytol-enhanced MR angiography resulted i

128 w was higher than iFR flow in nonsignificant stenoses (FFR >0.75; mean FFR flow, 42.3+/-22.8 cm/s ver

129 artery disease and functionally significant stenoses, FFR-guided PCI plus the best available medical

130 In significant stenoses, flow velocity over the resting wave-free perio

131 e extent and complexity of residual coronary stenoses following percutaneous coronary intervention (P

132 ssure and flow velocity were measured in 216 stenoses from 186 patients with coronary disease.

133 iating fixed muscle hypertrophy and fibrotic stenoses from acute transmural inflammatory stenoses in

134 rams at 2 centers had each of their coronary stenoses graded serially by using 6 thresholds (grade 0

135 ced MR angiography for detection of arterial stenoses greater than 50% were 94% and 98% for reader 1

136 rator characteristic curve for prediction of stenoses > or = 70% by the MF method was 0.92 +/- 0.04 v

137 All coronary stenoses > or =30% diameter reduction (n =531 in 322 pat

138 m 96% survival for 1 stenosis > or =70% or 2 stenoses > or =50% (p = 0.013) to 85% survival for > or

139 with 3-vessel disease and noninfarct-related stenoses >/=90%, and in this subgroup, there was a nonsi

140 Patients with complex coronary stenoses had a significantly (P<0.001) higher PAPP-A/pro

141 Patients in whom all stenoses had an FFR of more than 0.80 received medical t

142 Patients in whom all stenoses had an FFR of more than 0.80 were entered into

143 nically dilate obstructive coronary arterial stenoses has vastly improved our approach to managing pa

144 g functionally significant coronary arterial stenoses; however, larger studies are required to determ

145 ngiography (one stenosis in 13 patients, two stenoses in 15 patients, and three stenoses in four pati

146 We studied 157 intermediate coronary stenoses in 157 patients, evaluated by FFR and CFVR betw

147 A total of 213 intermediate coronary stenoses in 184 patients were enrolled.

148 using the intracoronary pressure wire in 38 stenoses in 34 patients with significant coronary stenos

149 ents, two stenoses in 15 patients, and three stenoses in four patients).

150 onclusion Venous elastic recoil after PTA of stenoses in hemodialysis access circuits is common, but

151 Vessel diameters, frequency, and severity of stenoses in IVUS-imaged and nonimaged coronary arteries

152 ther PCI should be performed immediately for stenoses in nonculprit arteries is controversial.

153 ng can identify severe, unsuspected coronary stenoses in patients who had prior mediastinal irradiati

154 stenoses from acute transmural inflammatory stenoses in patients with Crohn's disease (CD) scheduled

155 R by CMR differentiates moderate from severe stenoses in patients with known or suspected CAD.

156 in therapy slows the progression of coronary stenoses in proportion to average low-density lipoprotei

157 cterize the accuracy of assessing individual stenoses in serial disease using invasive FFR pullback a

158 eoretically limited in evaluating individual stenoses in serially diseased vessels.

159 mmed stenosis score (p = 0.002), integrating stenoses in series, was the best predictor of MFR(region

160 CT coronary angiography revealed significant stenoses in seven of 17 cases (41%).

161 perform FFR in angiographically intermediate stenoses in the absence of stress testing or in the pres

162 vessel coronary artery disease that includes stenoses in the proximal left anterior descending artery

163 raphy as multifocal if there were at least 2 stenoses in the same arterial segment; otherwise, they w

164 heritabilities were identified for proximal stenoses, in particular, left main CAD (h2=0.49+/-0.12;

165 velocities, which were associated with worse stenoses (incidence risk ratio [IRR] = 5.1, P </= .0001

166 ate the treatment decisions for intermediate stenoses, indicative of a worrisome disconnect between r

167 sured distal to the stenosis; in part 2 (118 stenoses), intracoronary pressure alone was measured.

168 In part 1 (39 stenoses), intracoronary pressure and flow velocity were

169 mporary use in longer, more complex coronary stenoses is lacking.

170 e (FFR) measurement of intermediate coronary stenoses is recommended by guidelines when demonstration

171 The clinical significance of small-vessel stenoses is therefore questionable.

172 ies, and the prognostic significance of such stenoses is uncertain.

173 ndex of the hemodynamic severity of coronary stenoses, is derived from invasive measurements and requ

174 re no instances of graft vascular thromboses/stenoses/leaks (0%).

175 ortion of patients with significant coronary stenoses, left ventricular systolic dysfunction, and dea

176 ement was significantly more pronounced with stenoses located in proximal and middle as compared with

177 nicity, fistula age, fistula type, number of stenoses, maximal angioplastic balloon diameter used, an

178 dentification of hemodynamically significant stenoses may be confounded by coronary remodeling.

179 Patients with severe bilateral carotid stenoses may be predisposed to ICH, particularly if ther

180 One hundred ninety-seven patients with 257 stenoses (mean diameter stenosis 48%) were studied.

181 For 70 to 99% stenoses, more extensive collaterals diminished risk of

182 noses, complex bifurcation and ostial branch stenoses, multivessel coronary artery disease, and left

183 ly occluded (n = 6) or compromised by ostial stenoses (n = 5).

184 In the case of bifurcation stenoses, new evidence suggests that opening a stenotic

185 DISCOVER-FLOW (Diagnosis of Ischemia-Causing Stenoses Obtained Via Noninvasive Fractional Flow Reserv

186 -related complications including thromboses, stenoses, occlusions, and aneurysms.

187 TIPS was more often associated with shunt stenoses/occlusions, recurrent hemorrhage, shunt revisio

188 dicated whenever hemodynamically significant stenoses occur.

189 d disengaged [FFR(dis)]) in 202 intermediate stenoses of 173 patients.

190 Stenoses of 50% or greater were detected with accuracy,

191 e CT angiography for detecting or ruling out stenoses of 50% or more according to conventional angiog

192 Stenoses of 50% or more were considered obstructive.

193 d fashion on 287 of 569 baseline angiograms (stenoses of 50-99% and adequate collateral views) in the

194 ngiography and duplex US accurately depicted stenoses of 70% or more compared with those depicted at

195 s had coexistent hemodynamically significant stenoses of accessory and main renal arteries.

196 ts with hemodynamically significant isolated stenoses of accessory renal arteries were calculated.

197 women [mean age, 71 years]) with 55 coronary stenoses of at least 50% underwent coronary CT angiograp

198 re of generalized arteriopathy presenting as stenoses of elastic arteries and hypertension.

199 mputed tomography angiography, DSA) revealed stenoses of its main branches, indicating Takayasu arter

200 Stenoses of the coronary arteries were graded from 0 to

201 gly utilized for hemodynamically significant stenoses of the extracranial carotid artery.

202 The percent stenoses of the veins draining each independent lung wer

203 However, the impact of incidental stenoses on future cardiac events remains unknown.

204 then compare this to the effect of coronary stenoses on the coronary microcirculation.

205 phy depicted 28 abnormalities in the CIA (27 stenoses, one dissection), 185 in the EIA (17 thromboses

206 section), 185 in the EIA (17 thromboses, 167 stenoses, one dissection), one in the common femoral art

207 ed by the presence of occlusive/subocclusive stenoses or FFR measurements </= 0.80 in vessels >2mm.

208 y and popliteal artery, and six patients had stenoses or occlusions below the popliteal artery.

209 lization techniques and angioplasty to treat stenoses or occlusions in 16 patients: 10 patients had h

210 s in 16 patients: 10 patients had high-grade stenoses or occlusions longer than 5 cm in the superfici

211 pecificity (0.56) in the detection of venous stenoses or occlusions.

212 cessfully completed without incurring venous stenoses or phrenic injury.

213 were also analyzed for significant coronary stenoses (over 50% luminal narrowing) by segment, by art

214 o 0.78) to identify functionally significant stenoses (p = 0.04).

215 cluded were patients with stent occlusion or stenoses, peripheral arterial disease (ABI <1.0), sympto

216 CIA and EIA stenoses predominantly involved the distal and proximal

217 I in noninfarct coronary arteries with major stenoses (preventive PCI) is unknown.

218 th multiple arteries, localized renal artery stenoses produced focal elevations of R2*, suggesting ar

219 of the distribution of coronary vessels and stenoses provided a measure of myocardial jeopardy that

220 tive (r(2) = 0.52; p < 0.001) and artificial stenoses (r(2) = 0.54; p < 0.05), although the pressure-

221 neys downstream of high-grade renal arterial stenoses, R2* was elevated at baseline, but fell after f

222 f 48 patients were enrolled: 27 patients (40 stenoses) randomly assigned to the 1-minute group and 21

223 ve that the presence of endothelial cells in stenoses reduces platelet adhesion but increases sickle

224 Overall, the stenoses regressed from 79% at baseline (interquartile r

225 Before randomisation, stenoses requiring PCI were identified on the angiogram.

226 , 96% (23 of 24 stenoses), and 96% (52 of 54 stenoses), respectively.

227 normal, luminal irregularities, and moderate stenoses, respectively, which were confirmed by catheter

228 Upon hip flexion, 23 CIA and 116 EIA stenoses showed kinking (mean amplitude, 76 degrees +/-

229 substantial number of coronary arteries with stenoses showing an FFR>0.80 present disturbed hemodynam

230 I in noninfarct coronary arteries with major stenoses significantly reduced the risk of adverse cardi

231 However, CTA assessment of coronary stenoses tends toward overestimation, and even among CTA

232 terminant of the natural history of coronary stenoses than anatomy (DS).

233 ile samples from patients with malignant CBD stenoses than controls or nonmalignant CBD stenoses (2.4

234 rease in systolic VI was noted with coronary stenoses that resulted in progressive increases in the s

235 cal success was achieved in the treatment of stenoses that were resistant to high-pressure angioplast

236 y artery disease, we assessed the FFR in all stenoses that were visible on angiography.

237 evolve from mild-to-moderate coronary artery stenoses, that patients who experience a fatal coronary

238 us coronary intervention of complex coronary stenoses, their use appears to be reasonably cost-effect

239 uminal angioplasty of dialysis access venous stenoses, there was no significant difference in postint

240 s of pressure wire drift are enough to cause stenoses to change classification.

241 re chronically instrumented with LAD and LCX stenoses to produce viable dysfunctional myocardium and

242 nature (anatomic or functional) of coronary stenoses to the perfused myocardium supplied by the targ

243 ly assigned to the 1-minute group and 21 (36 stenoses) to the 3-minute group.

244 Retrospective review of 175 cervical carotid stenoses treated with elective CAS from April 2001 to Fe

245 nts with severe AS and intermediate coronary stenoses treated with transcatheter aortic valve implant

246 Three stenoses (two mild, one severe) were identified at SSFP

247 ses in 34 patients with significant coronary stenoses undergoing percutaneous intervention.

248 Seventeen patients with severe carotid stenoses underwent FDG-PET imaging 3 h after FDG adminis

249 Thirty-two subjects with carotid stenoses underwent noninvasive imaging with FDG positron

250 A total of 283 patients (310 coronary stenoses) underwent coronary angiography with FFR using

251 with SIHD and angiographically intermediate stenoses, use of FFR has slowly risen, and was associate

252 Mean MRA index of number and severity of stenoses was 0.84 +/- 0.68 (normal 0), % wall volume 74

253 sitivity for detecting more than 50% luminal stenoses was 89%; specificity, 65%; positive predictive

254 sitivity for detecting more than 70% luminal stenoses was 94%; specificity, 67%; positive predictive

255 angiography to depict substantial (>/= 50%) stenoses was assessed by using quantitative coronary ang

256 n defects was only 43+/-31%, and 68% of such stenoses were <50%.

257 The percentage stenoses were 21% +/- 22% versus 19% +/- 15% (not statis

258 MACE) at 2 years in 607 patients in whom all stenoses were assessed by FFR and who were treated with

259 Four hundred forty-seven stenoses were assessed with FFR, iFR, and whole-cycle Pd

260 A total of 157 stenoses were assessed.

261 ents were stratified based on calcium score; stenoses were classified by type and by coronary segment

262 1 coronaries (78 patients) with intermediate stenoses were classified in 4 FFR and coronary flow rese

263 Post-procedural angiographic stenoses were compared with post-procedural duplex-defin

264 Relevant stenoses were correctly identified with SSFP MR angiogra

265 Variable stenoses were created in the left main coronary arteries

266 meter were assessed while graded, controlled stenoses were created in the stented segment by progress

267 When graded stenoses were created, a decrease in diameter of 18 +/-

268 Arterial stenoses were detected with contrast-enhanced MR angiogr

269 All deep femoral artery stenoses were diaphragm-like and involved the lateral ci

270 those with evidence of >10% coronary artery stenoses were divided into 2 groups, with either stable

271 The stenoses were divided into 4 groups according to FFR and

272 st 2001, 1058 patients with complex coronary stenoses were enrolled in the SIRIUS trial and randomize

273 Four to five intermediate-severity stenoses were generated in the left anterior descending

274 Stenoses were identified in 23 (68%) of 34 ankles, fibro

275 Hemodynamically significant (>50%) stenoses were identified in 88% (89 of 101) of patients;

276 Stenoses were labeled as severe if FFR </=0.80.

277 On subsequent angiograms, stenoses were observed in 16.2% (21/130) of nonimaged ar

278 tients with abnormal TCD and/or intracranial stenoses were placed on a transfusion program.

279 Overall, 35 coronary artery stenoses were present.

280 rformed in three steps: (a)Coronary arterial stenoses were scored for severity and reader confidence

281 No coronary ostial stenoses were seen.

282 nformation required) and also recorded which stenoses were significant.

283 EIA stenoses were significantly longer in women (P < .003).

284 All ureteric stenoses were treated by surgical reconstruction.

285 Stenoses were treated with primary stenting obtaining fa

286 ctive values for the presence of significant stenoses were: by segment (n = 935), 86%, 95%, 66%, and

287 R/CFVR discordance is mainly attributable to stenoses where CFVR is abnormal.

288 termine their accuracy, especially at 50-69% stenoses where the balance of risk and benefit for carot

289 ssessing functional significance of coronary stenoses, which is more accurate than resting indices an

290 MRA detected moderate to severe anastomotic stenoses, which were confirmed at catheter angiography a

291 lenge in patients with intracranial vascular stenoses who are potential candidates for bypass surgery

292 tenotic > or = 70% aorto-ostial renal artery stenoses, who underwent implantation of a balloon-expand

293 atherosclerosis, but only 39% had 1 or more stenoses with >or=50% narrowing.

294 iscriminated malignant from nonmalignant CBD stenoses with 100% accuracy.

295 malignant vs patients with nonmalignant CBD stenoses with 63.3% diagnostic accuracy.

296 evaluated the data sets for the presence of stenoses with diameter reduction of 50% or more, by usin

297 ses below physiological cut points; treating stenoses with fractional flow reserve </=0.80 gained Del

298 The model is used to compute shear rates in stenoses with growing boundaries.

299 the hemodynamic benefit of stenting coronary stenoses with instantaneous wave-free ratio values <0.74

300 Sixty-one stenoses with intermediate angiographic severity were st