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1 e and Necator americanus], and Strongyloides stercoralis).
2 (63/668) participants tested positive for S. stercoralis.
3 e immune responses in mice against larval S. stercoralis.
4 tures consistent with B. dermatitidis and S. stercoralis.
5 e candidate against infection with larval S. stercoralis.
6 se precursor from the nematode Strongyloides stercoralis.
7 the human-infective threadworm Strongyloides stercoralis.
8 the human-infective threadworm Strongyloides stercoralis.
9 s fuelleborni fuelleborni with Strongyloides stercoralis.
10 ticipants (63 of 668) tested positive for S. stercoralis.
11 cycle of the nematode parasite Strongyloides stercoralis.
12 nd targeted the human parasite Strongyloides stercoralis.
13 he Ss-riok-2 promoter in post free-living S. stercoralis.
14 nus), Trichuris trichiura, and Strongyloides stercoralis.
15 nd neutrophils to control infections with S. stercoralis.
16 encoding gene (Ss-riok-2) from Strongyloides stercoralis, a medically important parasitic nematode (O
17           Nonspecific cues for Strongyloides stercoralis, a nematode that infects humans and a few ot
18  nematode parasites, including Strongyloides stercoralis, Ancylostoma spp., and Necator americanus.
19 mixed pulmonary infection with Strongyloides stercoralis and Blastomyces dermatitidis.
20 nt mechanisms control male development in S. stercoralis and Caenorhabditis species.
21 L3i) of the parasitic nematode Strongyloides stercoralis and compared the results to Caenorhabditis e
22 The human-parasitic threadworm Strongyloides stercoralis and hookworm Ancylostoma ceylanicum have hig
23 olecular and cellular biological study of S. stercoralis and of parasitic nematodes generally, and pr
24 eurons of the human threadworm Strongyloides stercoralis and show that they display unique functional
25 ine signaling inhibits these behaviors in S. stercoralis and the human hookworm Ancylostoma ceylanicu
26 h for generating stable knockout lines in S. stercoralis and use this approach to show that Ss-gcy-9
27  were residents in an area not endemic for S stercoralis, and either were positive for S stercoralis
28 omparisons between C. elegans, Strongyloides stercoralis, and Haemonchus contortus.
29 Schistosoma mansoni, hookworm, Strongyloides stercoralis, and Mansonella perstans were the most preva
30 ricoides, Trichuris trichiura, Strongyloides stercoralis, and Necator americanus.
31 , including the human pathogen Strongyloides stercoralis, and their close relatives that are facultat
32 lity of eosinophils to present Strongyloides stercoralis antigen in naive and immunized mice.
33 ophils were exposed to soluble Strongyloides stercoralis antigens, and the expression of various surf
34                Infections with Strongyloides stercoralis are of considerable public health relevance.
35 s study provides a useful mouse model for S. stercoralis autoinfection and suggests a therapeutic str
36 e of the best evidence of transmission of S. stercoralis by renal transplantation.
37                                Strongyloides stercoralis causes chronic asymptomatic infections which
38 signature.' Strikingly, in comparisons of S. stercoralis clusters to C. elegans homologs with RNAi kn
39                      In the comparison of S. stercoralis clusters with stage-specific expression to C
40 lso provide functional classifications of S. stercoralis clusters.
41               Our aim was to determine if S. stercoralis co-infection influenced clinical presentatio
42                                           S. stercoralis co-infection may modulate the intracerebral
43          Our aim was to determine whether S. stercoralis coinfection influenced clinical presentation
44                                           S. stercoralis coinfection may modulate the intracerebral i
45                              For example, S. stercoralis collagen transcripts were abundant in L1 but
46 earance of infections, such as Strongyloides stercoralis, commonly found in the developing world but
47 histolytica, Balantidium coli, Strongyloides stercoralis, cytomegalovirus, and adenovirus.
48 solved the 3-dimensional structure of the S. stercoralis DAF-12 ligand-binding domain cocrystallized
49                              We show that S. stercoralis engages in repeated cycles of pushing, punct
50                              We show that S. stercoralis exhibits life-stage-specific behavioral pref
51 this by whole genome sequence analysis of S. stercoralis from sympatric human and dog populations in
52 nt in myeloperoxidase (MPO) infected with S. stercoralis had significantly decreased larval killing.
53       We find that human- and dog-derived S. stercoralis have genetically distinct nuclear genomes, b
54 f parasitic species, including Strongyloides stercoralis Here, we identified a parasite-specific coac
55                                Strongyloides stercoralis hyperinfection causes high mortality rates i
56                         A second antigen, S. stercoralis immunoreactive antigen (SsIR), was tested al
57  stercoralis, and either were positive for S stercoralis in faecal tests and on serology (any titre)
58  Protective immunity to larval Strongyloides stercoralis in mice has been shown to be dependent on an
59 ective adaptive immune response to larval S. stercoralis in mice.
60  protective immunity to larval Strongyloides stercoralis in mice.
61  protective immunity to larval Strongyloides stercoralis in mice.
62 te and adaptive immune response to larval S. stercoralis in mice.
63 s in the secondary response to Strongyloides stercoralis in mice.
64  of the secondary Th2 immune responses to S. stercoralis in mice.
65 omplement to kill the parasite Strongyloides stercoralis in vitro.
66  reduced the pathogenic iL3 population in S. stercoralis, indicating the potential use of DAF-12 liga
67 inical endpoints were performed in active S. stercoralis infected and uninfected groups.
68 gG antibody easily differentiated between S. stercoralis-infected and uninfected patients (P< .0001)
69 Tc9, Th17/Tc17, and Th22/Tc22 cells in 15 S. stercoralis-infected individuals and 10 uninfected indiv
70           Identification and treatment of S. stercoralis-infected individuals is required because imm
71 umbers of adult and first-stage larvae in S. stercoralis-infected NSG mice, no hyperinfection was obs
72 ) than in uninfected (n=3) and Strongyloides stercoralis-infected patients (n=4), and greater in pati
73 ficant immunoreactivity was observed with S. stercoralis-infected sera, but a small number of patient
74 or without (n = 25) coincident Strongyloides stercoralis infection (S. stercoralis-positive and S. st
75 ranslocation is a feature of asymptomatic S. stercoralis infection and is associated with an inflamma
76                   However, their roles in S. stercoralis infection are yet to be defined.
77 oportion of participants with clearance of S stercoralis infection at 12 months, which was assessed i
78 and with or without coexistent Strongyloides stercoralis infection before and after anthelmintic ther
79 antly reduced in participants with active S. stercoralis infection compared with uninfected participa
80  that individuals with LTB and coexistent S. stercoralis infection have significantly lower levels of
81  Th17/Tc17, Th2/Tc2, and Th9/Tc9 cells in S. stercoralis infection in 15 infected individuals stimula
82  provide insight into the pathogenesis of S. stercoralis infection in the immunocompromised populatio
83                                Strongyloides stercoralis infection is a neglected condition that plac
84                                Strongyloides stercoralis infection is associated with a significant m
85                                Strongyloides stercoralis infection is associated with diminished anti
86      Our data reveal that the presence of S. stercoralis infection is associated with lower frequenci
87                                     Thus, S. stercoralis infection is characterized by a parasite Ag-
88                                     Thus, S. stercoralis infection is characterized by alterations in
89                   Our data thus show that S. stercoralis infection is characterized by microbial tran
90                                Strongyloides stercoralis infection is characterized by the downmodula
91     It has long been suggested that human S. stercoralis infection may be a zoonosis from dogs.
92                  Donor-derived Strongyloides stercoralis infection occurs rarely after transplantatio
93      In addition, definitive treatment of S. stercoralis infection resulted in a significant reversal
94                 In addition, treatment of S. stercoralis infection significantly increased the Ag-spe
95 nths were significantly reduced in active S. stercoralis infection vs. uninfected participants (3.8%[
96                              In contrast, S. stercoralis infection was associated with higher frequen
97                                    Active S. stercoralis infection was significantly associated with
98                                    Active S. stercoralis infection was significantly associated with
99 is the drug of choice for the treatment of S stercoralis infection, but there is no definitive eviden
100 native to ivermectin for the treatment of S. stercoralis infection, given that only slight difference
101                    Following treatment of S. stercoralis infection, the elevated levels of microbial
102 atory response in asymptomatic Strongyloides stercoralis infection, we measured the plasma levels of
103 induction of particular T-cell subsets in S. stercoralis infection, which suggests the importance of
104 zed assay for the diagnosis of Strongyloides stercoralis infection.
105 erformed in groups with or without active S. stercoralis infection.
106 g stimulation compared with those without S. stercoralis infection.
107 thout (Ss-) seropositivity for Strongyloides stercoralis infection.
108 ing CD4(+) and CD8(+) T cell responses in S. stercoralis infection.
109 ry cytokines that are highly expressed in S. stercoralis infection.
110 in INF individuals following treatment of S. stercoralis infection.
111  immune response that characterizes human S. stercoralis infection.
112 sents a major advance in the diagnosis of S. stercoralis infection.
113  subsets along with cytokine responses in S. stercoralis infections, which suggest the importance of
114 us of the subjects and the development of S. stercoralis infections.
115 (8 mg) vs ivermectin (200 mug/kg) against S. stercoralis infections.
116 ected [INF]) or without (uninfected [UN]) S. stercoralis infections.
117         Protective immunity to Strongyloides stercoralis infective larvae in mice has been shown to b
118 gand-dependent reactivation of infectious S. stercoralis infective third-stage larvae, and that these
119         The parasitic nematode Strongyloides stercoralis infects an estimated 600 million people worl
120         A prevalent feature of Strongyloides stercoralis is a life-long and potentially lethal infect
121                                Strongyloides stercoralis is a soil-transmitted helminth organism that
122                                Strongyloides stercoralis is common among populations with high tuberc
123                                Strongyloides stercoralis is common amongst populations with high tube
124                                Strongyloides stercoralis is considered to be historically endemic in
125 ed immune function, direct development of S. stercoralis is favored, whereas, in individuals with les
126 ponses in human infection with Strongyloides stercoralis is not well defined.
127  Here we show that the infective larva of S. stercoralis is strongly attracted to an extract of mamma
128 dauer or nondauer stages, matches between S. stercoralis L1 and C. elegans nondauer-expressed genes d
129          Although a greater proportion of S. stercoralis L3i than L1 genes have homologs among the C.
130 as determined that eosinophils killed the S. stercoralis larvae in naive mice, whereas these cells we
131              Three antigens or genes from S. stercoralis larvae were identified as tropomyosin (Sstmy
132 r transplant patient revealed evidence of S. stercoralis larvae.
133           Thus, our data demonstrate that S. stercoralis (+)LTB(+) individuals are associated with a
134        Upon mycobacterial Ag stimulation, S. stercoralis (+)LTB(+) individuals exhibited significantl
135  baseline (in the absence of a stimulus), S. stercoralis (+)LTB(+) individuals exhibited significantl
136 nd CXCL10 were significantly increased in S. stercoralis (+)LTB(+) individuals.
137  in individuals with LTB with (Strongyloides stercoralis (+)LTB(+)) or without S. stercoralis (S. ste
138 CL2, CXCL9, and CXCL10 in comparison with S. stercoralis (-)LTB(+) and/or HC individuals.
139 L9, CXCL10, and CXCL11 in comparison with S. stercoralis (-)LTB(+) and/or HC individuals.
140 lis (+)LTB(+)) or without S. stercoralis (S. stercoralis (-)LTB(+)) infection and in individuals with
141  antigen-stimulated cytokine responses in S. stercoralis-LTB coinfection is reversible (for the most
142                           Analysis of the S. stercoralis mitochondrial genome reveals evidence of his
143  small bowel (Giardia lamblia, Strongyloides stercoralis, Mycobacterium avium-intracellulare complex,
144 terial-Ag stimulation than monocytes from S. stercoralis-negative individuals.
145 is infection (S. stercoralis-positive and S. stercoralis-negative respectively).
146 om patients with parasitologically proven S. stercoralis or filarial infections and from healthy, uni
147 perstans, Onchocerca volvulus, Strongyloides stercoralis, or Wuchereria bancrofti.
148 eated mice undergoing hyperinfection with S. stercoralis Overall, this study provides a useful mouse
149 as seen in those infected with Strongyloides stercoralis (P < 0.05) and when all helminth infections
150 dent Strongyloides stercoralis infection (S. stercoralis-positive and S. stercoralis-negative respect
151                     Finally, T cells from S. stercoralis-positive individuals exhibited significantly
152                            Monocytes from S. stercoralis-positive individuals had a reduced capacity
153 ice, which are eosinophil deficient, with S. stercoralis resulted in development of primary and secon
154 yloides stercoralis (+)LTB(+)) or without S. stercoralis (S. stercoralis (-)LTB(+)) infection and in
155             Immunoglobulin G (IgG) from a S. stercoralis-seropositive individual passively transferre
156            However, the genes controlling S. stercoralis sex determination and male development are u
157 anus, Trichuris trichiura, and Strongyloides stercoralis soil-transmitted helminths ("ANTS" parasites
158 es antigen and/or to a soluble extract of S. stercoralis somatic antigens ("crude antigen") using enz
159                  We found two clusters of S. stercoralis-specific genes encoding predicted transmembr
160 n a soil-transmitted helminth, Strongyloides stercoralis (Ss), and T2DM, we examined analytes associa
161                S. mansoni, N. americanus, S. stercoralis, T. trichiura, M. perstans and A. lumbricoid
162   CRISPR/Cas9-mediated mutagenesis of the S. stercoralis tax-4 gene abolishes iL3 attraction to a hos
163 r LAST ACT trials, underwent pretreatment S. stercoralis testing by serology, stool microscopy, and/o
164 17; NCT03100786), underwent pre-treatment S. stercoralis testing by serology, stool microscopy, and/o
165 bodies are both protective against larval S. stercoralis, they recognize different antigens and utili
166  in infected (INF) individuals with Fo in S. stercoralis-uninfected (UN) individuals.
167  and canine parasitic nematode Strongyloides stercoralis utilizes an XX/XO sex determination system,
168 s, and the intestinal parasite Strongyloides stercoralis was investigated in persons infected with on
169 he prevalence of antibodies to Strongyloides stercoralis was measured in 0-12-year-olds using a bead-
170      Based on these data, we suggest that S. stercoralis was originally a parasite of canids, that be
171 ive third-stage larvae (L3) of Strongyloides stercoralis was shown to be dependent on immunoglobulin
172                    Cure rate (CR) against S. stercoralis was the primary outcome.
173 tent mice are resistant to infection with S. stercoralis, we hypothesized that NSG mice, which have a
174 mical patterns of Ss-riok-2 expression in S. stercoralis, we observed expression patterns of a transg
175 ed from the infective stage of Strongyloides stercoralis were characterized.
176 ites Entamoeba histolytica and Strongyloides stercoralis were predictors of LBW despite their low pre
177 rom Caenorhabditis elegans and Strongyloides stercoralis, were distinct from the coelomate ones.
178 lopment of infective larvae of Strongyloides stercoralis, which may facilitate hyperinfection and, he

 
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