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1                                          The stereociliary actin-bundling protein espin was targeted
2 r cells and strial marginal cells but not to stereociliary ankle links or pillar cells, which nonspec
3 FL-) whirlin in photoreceptors and hair cell stereociliary bases is important for the USH type 2 prot
4 2+ extrusion by pumps, Ca2+ binding to fixed stereociliary buffers, and Ca2+ binding to mobile buffer
5                   The asymmetric location of stereociliary bundle (hair bundle) on the apical surface
6        Complex assembly likely occurs at the stereociliary bundle but not along the protein transport
7  the molecular mechanism underlying cochlear stereociliary bundle development and hearing loss pathog
8                Defects in this complex cause stereociliary bundle disorganization and hearing loss.
9 he ankle link region of the mechanosensitive stereociliary bundle in hair cells.
10 mmalian Phenotype Level 4 abnormal hair cell stereociliary bundle morphology and related phenotypes;
11 toxin-damaged utricles and the maturation of stereociliary bundle morphology.
12        Vestibular identity was based on: (1) stereociliary bundle morphology; (2) spacing of hair cel
13    The vertebrate hair cell is named for its stereociliary bundle or hair bundle that protrudes from
14 and guidance, hair follicle orientation, and stereociliary bundle orientation in inner ear sensory ha
15 es Frzb in regulating cochlear extension and stereociliary bundle orientation in vitro, and that Wnt5
16   By contrast, vinculin planar asymmetry and stereociliary bundle orientation were restored in Fz3(-/
17 e homology, disrupts neural tube closure and stereociliary bundle orientation, and shows genetic inte
18  regulates the development of unidirectional stereociliary bundle orientation.
19 ey must result from an active process in the stereociliary bundle suggested to underlie amplification
20 receptor potential are the deflection of the stereociliary bundle, and the subsequent flow of transdu
21 lanar cell polarity and morphogenesis of the stereociliary bundle, including bundle fragmentation or
22 n, several aspects of the development of the stereociliary bundle, including its elongation and orien
23 lar their mechanotransduction organelle, the stereociliary bundle, requires highly organized remodeli
24 is involved both in the morphogenesis of the stereociliary bundle, the sensory antenna of inner ear h
25 n mechanically sensitive ion channels in the stereociliary bundle.
26 evident in the polarized organization of the stereociliary bundle.
27 nesis of the auditory sensory epithelium and stereociliary bundle.
28 tops of the hair cell stereocilia within the stereociliary bundle.
29 ractions and is required for cohesion of the stereociliary bundle.
30 he ankle link complex (ALC) at the hair cell stereociliary bundle; however, little is known about the
31 air cells that fail to properly orient their stereociliary bundles along the mediolateral axis of the
32 ll projection phenotypes including hair cell stereociliary bundles and cilium assembly.
33 ially differentiated hair cells fail to form stereociliary bundles and degenerate by apoptosis in the
34 t mechanism that actively reorients auditory stereociliary bundles and reveals an unexpected role of
35  polarity is necessary for normal hearing as stereociliary bundles are only sensitive to vibrations i
36 ed of neuromasts, patches of hair cells with stereociliary bundles arranged with morphological mirror
37 require a coordinated alignment of hair cell stereociliary bundles as an essential element of mechano
38 showing that even severe dysmorphogenesis of stereociliary bundles can be corrected.
39 eated water-jet stimulation of the hair cell stereociliary bundles caused adaptation of the action po
40 yonic development, hair cells acquire apical stereociliary bundles for mechanosensation, basolateral
41 gnificant disruptions in the polarization of stereociliary bundles in mouse cochlea as a result of de
42                    The observed disorganized stereociliary bundles in the bpck inner ear and the conv
43             Otoliths, which are connected to stereociliary bundles in the inner ear, serve as inertia
44 ts for spontaneous oscillations of hair cell stereociliary bundles in the lower vertebrate inner ear.
45 nolabelling demonstrated TMC1 throughout the stereociliary bundles in wild type but not in Lhfpl5 mut
46 t mechanical deflections of their actin-rich stereociliary bundles into electrochemical signals.
47          OHCs transduce deflections of their stereociliary bundles into receptor potentials that driv
48   The morphogenesis and maintenance of these stereociliary bundles is a tightly regulated process req
49                    In the mammalian cochlea, stereociliary bundles located on mechanosensory hair cel
50 ration are mediated through the vibration of stereociliary bundles located on the lumenal surfaces of
51  tube closure, as well as the orientation of stereociliary bundles of sensory hair cells in the inner
52 cture of the cochlea that is attached to the stereociliary bundles of the outer hair cells (OHCs), el
53 in vertebrates is the uniform orientation of stereociliary bundles of the sensory hair cells in the m
54                            The structures of stereociliary bundles of trans-differentiated hair cells
55 ransduction, sound-induced vibrations of the stereociliary bundles on the sensory hair cells are conv
56 on, sound-evoked vibrations of the hair cell stereociliary bundles open mechanotransducer (MET) ion c
57 ), with oppositely oriented planar-polarized stereociliary bundles that detect motion in opposite dir
58 two groups of cells with oppositely oriented stereociliary bundles that meet at a line of polarity re
59 trical signals depends upon mechanosensitive stereociliary bundles that project from the apical surfa
60 ends on mechanosensitive ion channels in the stereociliary bundles that project from the apical surfa
61 itory hair cells requires highly specialized stereociliary bundles that project from their apical sur
62 ral conductances and synaptic properties yet stereociliary bundles were absent, or small and nonfunct
63  is the uniform orientation of the hair cell stereociliary bundles within the cochlea.
64 nctions in hair cell survival, maturation of stereociliary bundles, and auditory function.
65               The induced hair cells display stereociliary bundles, attract neuronal processes and ex
66 s the architecture and mechanosensitivity of stereociliary bundles, improves hearing thresholds, and
67 CP defects, including mis-oriented hair cell stereociliary bundles, in Bbs8 and Ift20 single mutants
68 ells showed normal development of individual stereociliary bundles, indicating that asymmetry was est
69 be, and misorientation of cochlear hair cell stereociliary bundles, indicative of defects in planar c
70 mbrane directly overlies the inner hair cell stereociliary bundles, these data provide the most accur
71 a role in the polarization of the developing stereociliary bundles.
72 air cells, including within their actin-rich stereociliary bundles.
73 eages of hair cells with oppositely oriented stereociliary bundles.
74  from the traumatic stimulation of hair cell stereociliary bundles.
75 olarized membrane protrusions reminiscent of stereociliary bundles.
76 lusters displaying hair cell-like cells with stereociliary bundles.
77 es in the shape and orientation of hair cell stereociliary bundles.
78 eased during the formation and maturation of stereociliary bundles.
79 , neural tube defects and disrupted cochlear stereociliary bundles.
80                 We also developed a model of stereociliary Ca2+ homeostasis that incorporates four re
81 rmally may be stimulated by the reduction in stereociliary Ca2+ when gating springs rupture and trans
82 r cells, where it is regulated by changes in stereociliary calcium.
83 ractions in the glycocalyx contribute to the stereociliary coherence that is essential for hearing.
84 RV1 signaling in vitro, are localized to the stereociliary compartments that overlap with AC6 distrib
85 d can elicit tonic OHC motility in mice with stereociliary defects that eliminate cochlear amplificat
86                                              Stereociliary development is unaffected in samba mice, b
87 ir-cell bundle in vivo is required to ensure stereociliary displacement similarity, increasing the sp
88                            The similarity of stereociliary displacements within a bundle regulates fu
89 e (nonuniform stimulation) caused dissimilar stereociliary displacements.
90  hair cell stereociliary tips participate in stereociliary elongation.
91 ncing identified a downregulation of several stereociliary genes in the Myo7a-deficient cochlea, indi
92 apillary electrophoresis, we showed that the stereociliary glycocalyx acts as a negatively charged po
93 es for the ALC in regulating inner hair cell stereociliary growth and differentiation as well as oute
94  isoforms are required for normal vestibular stereociliary growth, although they may play slightly di
95 , including the shape and orientation of the stereociliary hair bundle essential for sound detection.
96 the Trpml3 gene cause disorganization of the stereociliary hair bundle, structural aberrations in out
97          In the inner ear sensory epithelia, stereociliary hair bundles atop sensory hair cells are m
98 gulate the maturation and maintenance of the stereociliary hair bundles.
99 ouse embryos displayed disrupted polarity of stereociliary hair cells in the cochlea, a characteristi
100 mechanically sensitive ion channels in their stereociliary (hair) bundle.
101 uency region, rootlet length correlates with stereociliary height but between regions it changes disp
102  hair bundle cohesiveness and the absence of stereociliary imprints in the TM observed in these mice
103                                          The stereociliary link protein cadherin 23 (Cdh23) was targe
104                                         This stereociliary localization domain was absent in Va(J) he
105                          Here we refined the stereociliary localization of TRPML3 and investigated co
106 ressed in the vestibular organs, where their stereociliary localizations are similar to those of deve
107 e acoustic trauma-induced tip link damage or stereociliary loss by disrupting tip links or ablating t
108 HCs that express characteristic synaptic and stereociliary markers and survive to adulthood, although
109 e hair cell, a detachment of the apical, non-stereociliary membrane of the hair cell from the underly
110 density (UTLD), where CDH23 inserts into the stereociliary membrane.
111 achment between glycan components of apposed stereociliary membranes.
112  protein despite the presence of PIP2 in the stereociliary membranes.
113                                              Stereociliary morphological defects in USH2 mutant mice
114                           Hair-cell fate and stereociliary morphology and function were examined usin
115 fication, two mechanisms have been proposed: stereociliary motility and somatic motility.
116 lative squeezing but not the sliding mode of stereociliary motion.
117 ured the coherence and phase of the relative stereociliary motions with a dual-beam differential inte
118 g vesicular trafficking, cell migration, and stereociliary movements of hair cells.
119 two regions of hair cells (HC) with opposite stereociliary orientation.
120                  Two methods of blocking the stereociliary PMCA2 Ca(2+) pump both elicited scramblase
121 osis at P6 and argue for connections between stereociliary PMCA2 density, hair cell apoptosis, and de
122 ations do not impact on the cation selective stereociliary process or the endolymphatic potential, ou
123 ian vertebrates amplification is produced by stereociliary processes, related to the mechanotransduce
124 PDZ-binding site with the PDZ1 domain of the stereociliary protein harmonin, and potentially via a we
125                            We identify a new stereociliary protein, the I-BAR protein BAIAP2L2, which
126 found that BAIAP2L2 interacts with other key stereociliary proteins involved in normal hair bundle mo
127   Instead, the interaction of myosin-1c with stereociliary receptors depended on its calmodulin-bindi
128 d differentiation as well as outer hair cell stereociliary rigidity and organization during developme
129              Moreover, pejvakin localizes to stereociliary rootlets in hair cells and is required for
130 -VI in cuticular plates and association with stereociliary rootlets suggest that this isozyme partici
131 ly and indirectly, via microtubules, to some stereociliary rootlets.
132 the stereociliary taper, peaked in the lower stereociliary shaft, and declined progressively toward t
133 losed that radixin labeling commenced in the stereociliary taper, peaked in the lower stereociliary s
134 hat the cdh23 mutation may be harmful to the stereociliary tip link and cause the hair cell apoptosis
135 on: cadherin 23 (Cdh23), a candidate for the stereociliary tip link, and phosphatidylinositol 4,5-bis
136 pre-embedding EM immunogold microscopy, with stereociliary tip-link and subcuticular plate sites.
137           A unique coupling between HCN1 and stereociliary tip-link protein protocadherin 15 has been
138 Myo1c that mediates CaM-sensitive binding to stereociliary tips and to PIP2 immobilized on a solid su
139                          Binding of Myo1c to stereociliary tips of cochlear and vestibular hair cells
140 ied multiprotein complex associated with the stereociliary tips of hair bundles.
141 L- and C-terminal (C-) whirlins in hair cell stereociliary tips participate in stereociliary elongati
142 filaments, myosin-Ibeta is found mostly near stereociliary tips, myosin-VI is largely absent, and myo
143 ously that Myo1c interacts with molecules at stereociliary tips, the site of transduction, through se
144 f radial links and an increase in tenting of stereociliary tips.
145 l membrane attachment crowns are seen at the stereociliary tips.
146 maller number of channels transported to the stereociliary tips; this may stem from impaired TMC1 bin

 
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