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1 e monkeys were anisometropic, and three were strabismic.
2 r those who were amblyopic (24.3 +/- 6.6) or strabismic (34.0 +/- 9.8).
3 ach individual eye in 19 adults (7 esotropic strabismics, 6 anisometropes and 6 controls).
4 ithin striate cortex (area V1) of normal and strabismic, adult macaque monkeys.
5 estionnaire (NEI VFQ-25) was performed in 42 strabismic adults over the age of 50 years at a single i
6                                      In nine strabismic amblyopes (mean age, 32 years), the results c
7 in normal subjects, anisometropic amblyopes, strabismic amblyopes and non-amblyopic esotropes.
8                     Using the amblyopic eye, strabismic amblyopes counted inaccurately, markedly unde
9 ern of responses to normal-vision observers, strabismic amblyopes exhibited substantially reduced IM
10                    The results indicate that strabismic amblyopes have mechanisms for binocular summa
11                 Rather, counting deficits in strabismic amblyopes reflected a higher-level limitation
12 nance in visual perception also increases in strabismic amblyopes that have their vision to high spat
13 gnification is normal in the foveal field of strabismic amblyopes.
14 y occlusion group had a higher proportion of strabismic amblyopia (P = .007).
15 re newly diagnosed with anisometropic and/or strabismic amblyopia and had not undergone previous trea
16               Abnormal visual development in strabismic amblyopia drastically affects visual percepti
17 yopia, 15 amblyopic eyes of 15 patients with strabismic amblyopia due to esotropia, 12 amblyopic eyes
18 cular approach to treating anisometropic and strabismic amblyopia has recently been advocated.
19 ld be a potential test to detect and monitor strabismic amblyopia in young children.
20                                              Strabismic amblyopia is typically associated with severa
21                       Binocular summation in strabismic amblyopia is typically reported as being abse
22                                              Strabismic amblyopia significantly reduced excitatory co
23         The treatment effect was greater for strabismic amblyopia than for combined-mechanism amblyop
24 nificantly lower in anisometropic amblyopia, strabismic amblyopia, and deprivation amblyopia compared
25 cantly lower in the anisometropic amblyopia, strabismic amblyopia, and deprivation amblyopia groups (
26 ion are affected in anisometropic amblyopia, strabismic amblyopia, and deprivation amblyopia, and OCT
27  anisometropic amblyopes, in 6 patients with strabismic amblyopia, and in 4 patients with combined an
28  part to distorted skull development causing strabismic amblyopia, and OPG) were difficult to treat a
29 nown to be severely disrupted in adults with strabismic amblyopia, could be a potential test to detec
30 metropic amblyopia, anisometropic amblyopia, strabismic amblyopia, deprivation amblyopia) with age- a
31                  Our model fits show that in strabismic amblyopia, the excitatory contribution to bin
32 eractions disrupts binocular interactions in strabismic amblyopia.
33 sual outcomes significantly in patients with strabismic amblyopia.
34  and 71% positive predictive value (PPV) for strabismic amblyopia.
35 rating acuity may underestimate the depth of strabismic amblyopia.
36 n 4 patients with combined anisometropic and strabismic amblyopia.
37                                        Sixty strabismic and 80 control participants were prospectivel
38 ight strabismic, two anisometropic, and four strabismic and anisometropic amblyopes (mean age, 8.5 +/
39 the visual cortical hierarchy of humans with strabismic and anisometropic amblyopia in both sexes, us
40 ging to show that training human adults with strabismic and anisometropic amblyopia with dichoptic at
41                   Optical treatment alone of strabismic and combined-mechanism amblyopia results in c
42  were aetiology of amblyopia (anisometropic, strabismic and combined-mechanism amblyopia), treatment
43       Differences in gene expression between strabismic and normal human EOMs point to a relevant con
44 mmetry in ODC width and area were similar in strabismic and normal monkeys.
45 yes for the three less severe amblyopes (two strabismic and one anisometropic).
46 he three more severely affected animals (one strabismic and two anisometropic), the optimal spatial f
47     Comparison of SOA population activity in strabismic animals and normal monkeys (described in the
48  and neural sensitivities are altered in the strabismic animals compared with the normal animals.
49                                          The strabismic animals had early-onset natural esotropia (th
50 minor effect on the vergence behavior of the strabismic animals.
51 ours defined by Gabor patches is abnormal in strabismic, but not in anisometropic, amblyopia.
52 rally strabismic monkey and in a monkey made strabismic by early postnatal alternating monocular occl
53 eural responses in nonamblyopic monkeys made strabismic by surgery at the age of 10-60 d.
54                       Although our data from strabismic cats do not differ markedly from those report
55 of callosal neurons in the striate cortex of strabismic cats to that in normally reared cats.
56                                           In strabismic cats, we observed a dramatic shift in the ocu
57                                    Fifty-one strabismic children and 130 normal controls ages 3 to 17
58 would consider prescribing spectacles to non-strabismic children and determine the diopter value requ
59          Overall, non-amblyopic horizontally strabismic children demonstrated comparable VRF outcomes
60                     The injury prevalence in strabismic children was 30% in a study of >10 million pa
61                                          For strabismic children, hyperacuity and grating acuity were
62 n may inform about causes and effects of the strabismic condition in humans.
63 n of the peripheral oculomotor system to the strabismic condition.
64 n among esotropic patients compared with non-strabismic controls.
65 e deficiency of binocular connections in the strabismic cortex was evident qualitatively as a "skip"
66 usion is a realistic goal in the majority of strabismic CP children.
67                                              Strabismic (cross-eyed) humans and animals show an imbal
68  always improves, if not permanently curing, strabismic deviations.
69 who underwent extraocular muscle surgery for strabismic diplopia after treatment with teprotumumab at
70          Most patients requiring surgery for strabismic diplopia following teprotumumab achieve good
71 FS) represent a subset of monogenic isolated strabismic disorders that are characterized by restricti
72 s significantly different between normal and strabismic EOM.
73 es that were significantly down-regulated in strabismic EOMs; none was significantly upregulated.
74                                              Strabismic extraocular muscles (EOMs) differ from normal
75 Application of a prismatic correction to the strabismic eye in order to achieve bifoveal stimulation
76 rmal age-matched infants, V1 neurons in both strabismic groups exhibited reductions in sensitivity to
77 e stimulation in anisometropic compared with strabismic individuals.
78 atomical markers in V1 of two experimentally strabismic Macaca nemestrina monkeys.
79                            The CO results in strabismic macaque suggest that the nasal ODC bias promo
80     In all, 27% of anisometropic and >65% of strabismic/mixed amblyopes had nystagmus.
81    Regression was only seen in patients with strabismic/mixed amblyopia particularly those with FMNS.
82 revealed by CO staining of V1 in a naturally strabismic monkey and in a monkey made strabismic by ear
83          As do humans with large strabismus, strabismic monkey display disconjugate saccadic eye move
84                                          One strabismic monkey had infantile-onset, small-angle esotr
85                                    The other strabismic monkey had naturally occurring, large-angle (
86 ctive error was also small in the normal and strabismic monkeys (approximately 1 D to 2 D) as long as
87 ed Repair and 0.49 +/- 0.17 in the naturally strabismic monkeys (P < 0.01).
88                 Delayed Repair and naturally strabismic monkeys also had motion VEP asymmetries of eq
89 e accommodation in awake-behaving normal and strabismic monkeys and describe properties of photorefra
90 ors recorded binocular eye movements in four strabismic monkeys and one unaffected monkey.
91  contrast, both Delayed Repair and naturally strabismic monkeys had asymmetric motion VEP responses:
92 t an overabundance of neurons in V1 or V2 of strabismic monkeys preferring the temporal-to-nasal dire
93 rsuit adaptation during monocular viewing in strabismic monkeys with exotropia.
94                  In layers 2/3 and 4B of the strabismic monkeys, patchy projections and boutons termi
95                           In both normal and strabismic monkeys, small misalignment of the photorefra
96             Specific challenges posed by the strabismic monkeys, such as possible misalignment of the
97                                       In all strabismic monkeys, the sensitivity of V1 units to inter
98 accommodation in awake, behaving normal, and strabismic monkeys.
99 ong orthotropic or naturally or artificially strabismic monkeys.
100 onal bias was significantly reduced in these strabismic monkeys.
101 strabismus were surprisingly small in infant strabismic monkeys.
102                           In both normal and strabismic monkeys: (1) CO-blob compartment neurons show
103 ts were different across monkeys tested (two strabismic, one normal) but were similar for each monkey
104 dths of the ODCs having input from eyes with strabismic or anisometropic amblyopia were reduced in pr
105 the sole initial treatment for children with strabismic or combined-mechanism amblyopia before initia
106 .16] years; 11 boys and 9 girls) and 20 with strabismic or mixed amblyopia (mean [SD] age, 4.90 [1.36
107 to < 9 years with anisometropic, small-angle strabismic or mixed-mechanism amblyopia were randomized
108    Among children treated for anisometropic, strabismic, or combined mechanism amblyopia, there is a
109 years of age with anisometropic, small-angle strabismic, or mixed-mechanism amblyopia were initially
110 to < 9 years with anisometropic, small-angle strabismic, or mixed-mechanism amblyopia were randomized
111                                              Strabismic owl monkeys also showed ocular dominance colu
112                                 In contrast, strabismic participants demonstrated an increased correl
113             Moreover, both anisometropic and strabismic participants showed elevated OD responses acr
114 mblyopic eye, in anisometropic compared with strabismic participants.
115 tudinal health database that was queried for strabismic patients aged <19 years.
116 visual deficits impacting quality of life in strabismic patients and may represent a novel measure by
117 roof of concept study, especially useful for strabismic patients and/or those with reduced or null st
118                                              Strabismic patients demonstrate subnormal BiS and even b
119                               For 1.25% LCA, strabismic patients had a mean BiS score less than 1, in
120                                         Most strabismic patients had a monocular suppression or alter
121                                              Strabismic patients performed the same or worse on nearl
122                                Additionally, strabismic patients reported significantly worse ocular
123                          Physicians treating strabismic patients should recognize these quality-of-li
124      Mean BiS was significantly lower in the strabismic patients than controls for LCA (2.5% and 1.25
125                 A total of 130 postoperative strabismic patients were studied.
126                         This may explain why strabismic patients who are not diplopic close 1 eye in
127  strabismus surgery were compared with those strabismic patients who did not undergo surgery for inju
128                                    Pediatric strabismic patients who underwent strabismus surgery bet
129 reoacuity and bifoveal fusion in a cohort of strabismic patients with variable binocular sensory func
130                          The dominant eye of strabismic patients without anisometropia retained suppr
131 may be a factor in decreasing injury risk in strabismic patients, particularly in exotropia.
132 etely fixed in an infraducted (downward) and strabismic position, and markedly limited and aberrant r
133 ctural basis for lack of binocular fusion in strabismic primates, we investigated intrinsic horizonta
134 d differential expression between normal and strabismic samples, with an adjusted P value of </= 0.05
135 CISS score of 31.6 +/- 9.0 for non-amblyopic/strabismic students having near vision poorer than 0.1 L
136 ion for the amblyopic eye in one early-onset strabismic subject with binocular suppression, indicatin
137 mpared in seven orthotropic subjects and six strabismic subjects (four with primary microstrabismus a
138                                      All the strabismic subjects demonstrated normal retinal correspo
139 suggest that the eye movement asymmetries in strabismic subjects do not result from similar asymmetri
140 ate that V1 neurons do not appear to reflect strabismic suppression and therefore the elimination of
141 out anisometropia, consistent with classical strabismic suppression of the nondominant eye.
142                        Animals were rendered strabismic surgically at 8-10 days of age and were allow
143 ely small proportion of cortical neurons; in strabismics, the influence of the two eyes was more near
144                                        Eight strabismic, two anisometropic, and four strabismic and a
145 normal monkeys revealed that the deficits in strabismic V1 were due mainly to a loss of binocular con
146 ar ocular dominance columns if they are made strabismic within a few weeks of birth.

 
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