ライフサイエンスコーパス: streptomycin

1 sensitivity of mycobacteria to rifampin and streptomycin.

2 t substantially affecting the sensitivity to streptomycin.

3 and high-level resistance to gentamicin and streptomycin.

4 red in a subset of mutants when treated with streptomycin.

5 ance to isoniazid, rifampicin, ofloxacin and streptomycin.

6 hich confers resistance to spectinomycin and streptomycin.

7 follicle growth in serum-free median without streptomycin.

8 ly, this mutation also confers resistance to streptomycin.

9 veats associated with the common antibiotic, streptomycin.

10 henicol, sulphamethoxazole, trimethoprim and streptomycin.

11 istance to the antibiotics spectinomycin and streptomycin.

12 retched cells that were exposed to 40 microM streptomycin.

13 tRNA genes (such as mutA) or by exposure to streptomycin.

14 monly used on plants are oxytetracycline and streptomycin.

15 se conformational changes in the presence of streptomycin.

16 istance to the antibiotics spectinomycin and streptomycin.

17 confers resistance to both spectinomycin and streptomycin.

18 thoxazole, trimethoprim, chloramphenicol and streptomycin.

19 uberculosis drugs, isoniazid, rifampicin and streptomycin.

20 ns of S. typhimurium after administration of streptomycin.

21 Resistances to isoniazid (15.5%) and streptomycin (13.4%) were about twice as high as resista

22 oral rifampicin 10 mg/kg plus intramuscular streptomycin 15 mg/kg once daily for 8 weeks (RS8) is hi

23 atG S315T [isoniazid]; gidB 130 bp deletion [streptomycin]; 1957 [95% highest posterior density (HPD)

24 205 bp), pncA (pyrazinamide; 579 bp); rpsL (streptomycin; 196 bp), and embB (ethambutol; 185 bp).

25 ants with significantly higher resistance to streptomycin; (2) the exposure to pesticides (in mug/L)

26 y sul1 (for sulfonamide; 44.16%), aadA1 (for streptomycin; 33.50%), ereA (for erythromycin; 27.41%),

27 ) were resistant to ampicillin, 649 (58%) to streptomycin, 402 (36%) to trimethoprim-sulfamethoxazole

28 st commonly used were sulfonamides (75%) and streptomycin (54%).

29 stance most often to sulfamethoxazole (57%), streptomycin (56%), tetracycline (56%), ampicillin (52%)

30 Here we demonstrate that streptomycin, a bactericidal aminoglycoside that increas

31 Streptomycin, a blocker of SACs, had no effect upon the

32 In contrast, streptomycin, a nonspecific inhibitor of cationic MSCs,

33 mals received a 5 d course of treatment with streptomycin, a vestibulotoxic aminoglycoside.

34 ting hybrid RNA can then be immobilized on a streptomycin affinity matrix.

35 to the matrix, subsequent elution with free streptomycin allows efficient recovery of specific ribon

36 onses, as compared with animals treated with streptomycin alone.

37 Treatment with streptomycin altered the SCFAs in the cecum, significant

38 The aminoglycosides streptomycin, amikacin, and kanamycin and the cyclic pol

39 results for isoniazid, rifampin, ethambutol, streptomycin, amikacin, kanamycin, capreomycin, ofloxaci

40 Here, we show that exposure to streptomycin, an antibiotic known to promote mistranslat

41 SP204(1-1) was selected for resistance to streptomycin and 5-fluoro-2-deoxyuridine to distinguish

42 se of Escherichia coli cells to penicillin G-streptomycin and cefazolin.

43 Other inhibitors of MSC such as streptomycin and GsTMx-4 also suppressed the expression

44 n products that increased resistance against streptomycin and kanamycin.

45 y, the rate of tRNA movement is increased by streptomycin and neomycin, decreased by tetracycline, an

46 eak strain acquired resistance to isoniazid, streptomycin and rifampicin by around 1973, indicating c

47 riplex, and differences in susceptibility to streptomycin and rifampin between this strain and M. tri

48 ial community, including metabolic capacity, streptomycin and salicylic acid synthesis, and nitrifica

49 adenylation on 3''- and 9-hydroxyl groups of streptomycin and spectinomycin, respectively.

50 complexed with the antibiotics paromomycin, streptomycin and spectinomycin, which interfere with dec

51 analyses of an aggregated proteome caused by streptomycin and suggests that cellular defense against

52 f isolates that expressed resistance only to streptomycin and sulfamethoxazole (the StSu phenotype; 8

53 plasmid, encoding resistance to gentamicin, streptomycin and sulfonamides.

54 ptomycin, the Apt leaves the CS and binds to streptomycin and the Arch-shape structure is disassemble

55 Prenatal exposure to streptomycin and TMP-SMX was associated with select birt

56 antibiotics (erythromycin, tetracycline, and streptomycin) and whiB7 null mutants (Streptomyces and M

57 for strains resistant to both isoniazid and streptomycin, and 0.88 (95% CI, 0.53-1.47) for streptomy

58 se was associated with higher mortality than streptomycin, and aminoglycoside use was linked to highe

59 rimethoprim-sulfamethoxazole, sulfisoxazole, streptomycin, and furazolidone increased from 7 (8%) of

60 h vancomycin or a combination of penicillin, streptomycin, and gentamicin (PSG) and then inoculated t

61 matched the purported source for ethambutol, streptomycin, and pyrazinamide.

62 ncing libraries, resistance to tetracycline, streptomycin, and sulfonamide/trimethoprim was assigned

63 resistance to the error-inducing antibiotic streptomycin, and the ribosomes from many such streptomy

64 roduces both Stx1a and Stx2a was virulent in streptomycin- and ciprofloxacin-treated mice and that mi

65 ometry metabolomic features from a cohort of streptomycin- and rifampicin-resistant mutants grown in

66 ntly in white adipocytes (browning), whereas streptomycin antagonized TRPM8-mediated calcium entry, d

67 In contrast, the conversion of the streptomycin aptamer into functional riboswitches appear

68 In the presence of streptomycin, aptamer binds to its target and FAM-labele

69 In the absence of streptomycin, aptamer/FAM-labeled complementary strand d

70 by applying the approach to tetracycline and streptomycin aptamers.

71 In addition, pyranmycins, like streptomycin, are bacteriocidal while isoniazid (INH) is

72 c aptasensors were designed for detection of streptomycin based on aqueous gold nanoparticles (AuNPs)

73 ped for sensitive and selective detection of streptomycin, based on exonuclease I (Exo I), compliment

74 Mice treated with oral streptomycin before oral administration of MRSA develope

75 anged the electrochemical gradient such that streptomycin better accessed the cytoplasm.

76 The aminoglycosides paromomycin and streptomycin bind to the decoding center and induce rela

77 The streptomycin-binding aptamer, termed 'StreptoTag,' is em

78 of M. tuberculosis-infected A549 cells with streptomycin, but not cycloheximide, demonstrated a sign

79 In fura 2-loaded myocytes, HOE 642 and streptomycin, but not L-NAME, ablated the stretch-induce

80 mice treated perinatally with vancomycin or streptomycin by repeated intranasal administration of Sa

81 Our application to aptamers for streptomycin, chloramphenicol, neomycin B and ATP identi

82 ty over other antibiotics such as kanamycin, streptomycin, ciprofloxacin, and tetracycline.

83 A-site oligonucleotide, and paromomycin and streptomycin complexed to the 30S subunit was used as a

84 ntrast, mice whose water contained 5 g/liter streptomycin consistently became colonized at high level

85 A genetic system based on rpsL-streptomycin counter selection was developed to further

86 dentified mutations in 16S rRNA conferring a streptomycin dependence phenotype and from these derived

87 The structural basis for the streptomycin dependence phenotype of ribosomal protein S

88 Spontaneous suppressors of streptomycin dependence were also readily isolated.

89 ding ribosomal protein S4), which suppressed streptomycin dependence, were able to partially restore

90 isolated spontaneous streptomycin-resistant, streptomycin-dependent and streptomycin-pseudo-dependent

91 resistant, streptomycin-pseudo-dependent and streptomycin-dependent mutants described here is expecte

92 The streptomycin-dependent rpsL(P90K) mutant also showed sig

93 Streptomycin did not inhibit A-site cleavage in rpsL mut

94 Animals treated with streptomycin displayed vestibular system impairment as m

95 The aminoglycoside antibiotic streptomycin disrupts decoding by binding close to the s

96 These data reveal how streptomycin disrupts the recognition of cognate anticod

97 chanosensitive channel blocker gadolinium or streptomycin dramatically increased longitudinal conduct

98 %) accurate results for isoniazid, rifampin, streptomycin, ethambutol, amikacin, kanamycin, capreomyc

99 /109 (16%) pregnant women following prenatal streptomycin exposure.

100 this study, we showed that 4- to 6-week-old streptomycin-fed C57BL/6 mice were susceptible to intest

101 cide vector carrying wild-type rpsL and used streptomycin for counterselection.

102 ary metabolites (penicillin, cephalosporins, streptomycin, fosfomycin, gramicidin S, rapamycin, indol

103 The discovery of Streptomyces-produced streptomycin founded the age of tuberculosis therapy.

104 channels with the aminoglycoside antibiotic streptomycin from onset of disease in the mdx mouse mode

105 s such as chloramphenicol, tetracycline, and streptomycin gather predominantly at the cell poles and

106 Prompt treatment with streptomycin, gentamicin, doxycycline, or ciprofloxacin

107 on Candida growth in optisol-gentamicin and streptomycin (GS) with and without antifungal supplement

108 current systemic administration of zVAD with streptomycin had both significantly greater hair cell su

109 that confers resistance to spectinomycin and streptomycin, has been considered critical for recovery

110 ette confers resistance to spectinomycin and streptomycin in both B. burgdorferi and Escherichia coli

111 response in the presence of the SAC blocker streptomycin in both muscle (80 microm) and myocytes (40

112 l aptasensor was successfully used to detect streptomycin in milk and serum with LODs of 14.1 and 15.

113 aptasensors were successfully used to detect streptomycin in milk and serum.

114 be very effective for low-level detection of streptomycin in milk samples.

115 search Council (1948) study of the effect of streptomycin in the treatment of tuberculosis.

116 However, mice given streptomycin in their drinking water developed long-term

117 l distortion of 16S ribosomal RNA induced by streptomycin, including the crucial bases A1492 and A149

118 Using experimental evolution of streptomycin-independent pseudorevertants, we identified

119 d tobramycin, as compared to paromomycin and streptomycin, indicates differences in the efficacy of t

120 n the study of this antibiotic, a mysterious streptomycin-induced potassium efflux preceding any decr

121 only necessary for the previously described streptomycin-induced potassium efflux, but also directly

122 ate that the aminoglycosides paromomycin and streptomycin inhibit A-site cleavage of stop codons duri

123 y for 8 weeks (RS8) is highly effective, but streptomycin injections are painful and potentially harm

124 The antibiotic streptomycin is widely used in the treatment of microbia

125 biotic susceptibility testing with rifampin, streptomycin, isoniazid, and ethambutol were compared to

126 nce or borderline resistance to at least one streptomycin-isoniazid-rifampin-ethambutol drug or PZA.

127 eginning with sulfanilamide, penicillin, and streptomycin, led to additional strategies for managing

128 ver, substitutions that confer resistance to streptomycin likely represent a very distinct class of a

129 ), rrs (aminoglycosides), rpsL, rrs and giB (streptomycin) loci.

130 Specifically, fidaxomicin and streptomycin markedly altered hepatic bile acid signalin

131 Transfer of the streptomycin marker was independent of the H. pylori com

132 um produced C. jejuni progeny containing the streptomycin marker.

133 Mice received 2 mg of streptomycin/ml of drinking water and 1,500 U of penicil

134 For kanamycin and streptomycin, molecular DST significantly outperformed p

135 ), gentamicin (n = 345), plazomicin (n = 0), streptomycin (n = 285), tobramycin (n = 43), chloramphen

136 ere, we used a toeprinting assay to how that streptomycin, neomycin, kanamycin, gentamycin, and hygro

137 etection for ofloxacin, chloramphenicol, and streptomycin of 0.3, 0.12, and 0.2 ng mL(-1), respective

138 ation, as transient selective suppression by streptomycin of bacterial growth in the intestine delaye

139 -ray crystallography to define the impact of streptomycin on the decoding site of the Thermus thermop

140 hree antibiotics--neomycin, polymyxin B, and streptomycin--on diabetes development.

141 Treatment with aminoglycosides such as streptomycin or gentamicin is effective when initiated e

142 Early treatment and prophylaxis with streptomycin or gentamicin or the tetracycline or fluoro

143 suppress mainly gram-positive (fidaxomicin, streptomycin) or gram-negative (ceftriaxone) bacteria on

144 contained aadA, which confers resistance to streptomycin, or aadA and dhfr, which confer resistance

145 ltured in media supplemented with penicillin-streptomycin (PenStrep) or vehicle control.

146 methylthiolation is not a determinant of the streptomycin phenotype.

147 Experiments with streptomycin-pretreated caspase-1-deficient mice suggest

148 n acute inflammatory reaction in the ceca of streptomycin-pretreated mice that involves T-cell-depend

149 inflammation within 48 h after infection of streptomycin-pretreated mice, and the presence of the vi

150 n acute inflammatory reaction in the ceca of streptomycin-pretreated mice.

151 flammation caused by serotype Typhimurium in streptomycin-pretreated mice.

152 colonization of the ileum and caecum in the streptomycin-pretreated mouse model of colitis.

153 the bovine ligated ileal loop model and the streptomycin-pretreated mouse model, suggesting that thi

154 to elicit acute intestinal inflammation in a streptomycin-pretreated mouse model.

155 ating these inflammatory responses using the streptomycin-pretreated mouse model.

156 ot elicit marked inflammatory changes in the streptomycin-pretreated mouse model.

157 arly after serotype Typhimurium infection of streptomycin-pretreated wild-type mice regardless of fla

158 C57BL/6 mice were pretreated with streptomycin prior to receiving MET-1 or control, then g

159 -butyrolactone A-factor and consequently for streptomycin production in Streptomyces griseus).

160 to actII-ORF4 that is the final regulator of streptomycin production in Streptomyces griseus.

161 related, and suggests that the regulation of streptomycin production, which serves an important parad

162 at perinatal exposure to vancomycin, but not streptomycin, profoundly alters gut microbiota and enhan

163 is of ribosomes from streptomycin-resistant, streptomycin-pseudo-dependent and streptomycin-dependent

164 omycin-resistant, streptomycin-dependent and streptomycin-pseudo-dependent mutants of the thermophili

165 A good co-relation was observed between the streptomycin recoveries measured through the developed b

166 e, the miscoding antibiotics paromomycin and streptomycin rescue the defects in tRNA selection with t

167 an ultrasensitive biosensor for detection of streptomycin residues in milk samples using flow injecti

168 neous mutants by the flanking, cotransformed streptomycin resistance and Pst I markers.

169 We propose a novel mechanism of streptomycin resistance by which A1408G influences confo

170 onitor the transfer, a chromosomally encoded streptomycin resistance cassette prearranged by a specif

171 Whilst nine putative streptomycin resistance conferring markers in gid (8) an

172 rain 5051, was constructed by insertion of a streptomycin resistance gene cassette.

173 iazid, kanamycin, ofloxacin, rifampicin, and streptomycin resistance in Mycobacterium tuberculosis, a

174 ated that mutations conferring isoniazid and streptomycin resistance in this clone were acquired 50 y

175 allele of oppD interrupted by kanamycin and streptomycin resistance markers.

176 ance mutation is flanked on the 5' side by a streptomycin resistance mutation and on the 3' side by a

177 markers and heteroplasmic for the unselected streptomycin resistance trait.

178 he results for ethambutol, pyrazinamide, and streptomycin resistance were more variable.

179 cin resistance), strA and strB (which encode streptomycin resistance), class B tetA (which encodes te

180 ctor DNA and selection for spectinomycin and streptomycin resistance, respectively.

181 sistance and 96% when testing for high-level streptomycin resistance.

182 and a 1-kb class I integron bearing aadA for streptomycin resistance.

183 However, the most common vehicles of streptomycin-resistance genes in human and plant pathoge

184 A fraction of streptomycin-resistance genes in plant-associated bacter

185 TB activity against rifampin, isoniazid, and streptomycin resistant Mtb strains.

186 osomes isolated from strains that are either streptomycin resistant or dependent for growth do not sh

187 e virG(icsA) deletion was constructed from a streptomycin-resistant (Str(r)) mutant of 1617 by a filt

188 mutations could be evolved from slow-growing streptomycin-resistant mutants.

189 A spontaneous streptomycin-resistant rpsL mutant of F. johnsoniae was

190 We propose that tmRNA.SmpB binds to streptomycin-resistant rpsL ribosomes less efficiently,

191 f rifampin-resistant S. gordonii DLl than of streptomycin-resistant S. gordonii SK12 from the hearts

192 , all animals were challenged with 5 x 10(6) streptomycin-resistant S. mutans strain SJ-r organisms.

193 reptomycin, and the ribosomes from many such streptomycin-resistant S12 mutants display decreased lev

194 A novel streptomycin-resistant strain of NTHi was developed, and

195 xytetracycline is rare, but the emergence of streptomycin-resistant strains of Erwinia amylovora, Pse

196 Whereas streptomycin-resistant strains were more commonly found

197 reptomycin, and 0.88 (95% CI, 0.53-1.47) for streptomycin-resistant strains.

198 ion along the entire intestinal tract by the streptomycin-resistant V. parahaemolyticus.

199 site cleavage in rpsL mutants, which express streptomycin-resistant variants of ribosomal protein S12

200 NA sequencing studies with low-fitness rpsL (streptomycin-resistant) mutants of E. coli with and with

201 We have isolated spontaneous streptomycin-resistant, streptomycin-dependent and strep

202 crystallographic analysis of ribosomes from streptomycin-resistant, streptomycin-pseudo-dependent an

203 had high-level resistance to gentamicin and streptomycin, respectively, including 10% that were resi

204 that treatment with the ototoxic antibiotic streptomycin results in a nearly complete elimination of

205 tion of wild-type rpsL on a plasmid restored streptomycin sensitivity, demonstrating that wild-type r

206 against isoniazid (INH), rifampin (RMP), and streptomycin (SM) resistant Mtb strains.

207 ultaneous, sensitive and visual detection of streptomycin (SM), tetracycline (TC), and penicillin G (

208 gs was associated with four-drug resistance (streptomycin, spectinomycin, sulfisoxazole, and tetracyc

209 Excellent recoveries were obtained from the streptomycin spiked milk samples in the range 98-99.33%,

210 onsistent with kinetic data, we observe that streptomycin stabilizes the near-cognate anticodon stem-

211 hat of rifampicin against the nonreplicating streptomycin-starved M. tuberculosis 18b-Lux strain, and

212 Ferrets treated with streptomycin (Stm) had higher counts of E. coli O157:H7

213 ned for selective and sensitive detection of streptomycin (STR) based on Exonuclease III (Exo III), S

214 rifampin, isoniazid (INH), pyrazinamide, or streptomycin (STR), 4 were resistant to STR and INH, and

215 n specific Asp-->Gly misreading, we asked if streptomycin (Str), an aminoglycoside antibiotic known t

216 Streptomycin (STR), isoniazid (INH), rifampin (RIF), eth

217 For the second-line drug streptomycin (STR), overall concordance between the agar

218 the activatable Stx2d are more virulent in a streptomycin (str)-treated mouse model of infection [let

219 dfrA12, and dfrA15], amoxicillin [bla(TEM)], streptomycin [strA-strB], chloramphenicol [cat-1], and e

220 Upon treatment with streptomycin, substantial changes in the microbial compo

221 ond was resistance to ampicillin, kanamycin, streptomycin, sulfamethoxazole, and tetracycline (the Am

222 s resistance to ampicillin, chloramphenicol, streptomycin, sulfamethoxazole, and tetracycline (the Am

223 istinct resistance to ampicillin, kanamycin, streptomycin, sulfamethoxazole, and tetracycline and to

224 acycline and to ampicillin, chloramphenicol, streptomycin, sulfamethoxazole, and tetracycline, respec

225 re resistant to ampicillin, chloramphenicol, streptomycin, sulfamethoxazole, and tetracycline.

226 g resistance to ampicillin, chloramphenicol, streptomycin, sulfamethoxazole-sulfisoxazole, and tetrac

227 uginosa as an agent conferring resistance to streptomycin, sulfanilamide, gentamicin, and carbenicill

228 re resistant to ampicillin, chloramphenicol, streptomycin, sulfisoxazole, and tetracycline.

229 ampicillin, chloramphenicol, nalidixic acid, streptomycin, sulfisoxazole, tetracycline, and trimethop

230 nce to 5 drugs (ampicillin, chloramphenicol, streptomycin, sulfonamides, and tetracycline), has emerg

231 s resistance to ampicillin, chloramphenicol, streptomycin, sulfonamides, and tetracycline.

232 percent of the strains showed resistance to streptomycin, sulfonamides, or tetracycline only; all th

233 Levels of agreement for streptomycin tested at 2.0 and 6.0 micro g/ml were 77 an

234 complex antibiotics from nature: penicillin, streptomycin, tetracycline, and erythromycin, among othe

235 e critical concentrations for ethambutol and streptomycin that accurately detect susceptibility to th

236 n of ribosome inhibitors (chloramphenicol or streptomycin) that indirectly constrain transcription ra

237 In the absence of streptomycin the gate remains closed.

238 Upon addition of streptomycin, the Apt leaves the CS and binds to strepto

239 antimicrobials, including crystal violet and streptomycin (this phenotype could also be complemented)

240 ized model in which mice are pretreated with streptomycin to result in gut-restricted acute enteritis

241 which were also treated with an antibiotic (streptomycin) to knockdown the gut microbiota.

242 pregnancy (amikacin, gentamicin, plazomicin, streptomycin, tobramycin, chloramphenicol, doxycycline,

243 We demonstrated that streptomycin-treated adult C57BL/6 mice experienced prol

244 ns and in gastrointestinal colonization of a streptomycin-treated adult mouse.

245 detes dominated the intestinal microbiota of streptomycin-treated animals, while vancomycin promoted

246 an O157:H7 strain, is known to colonize the streptomycin-treated CD-1 mouse intestine by growing in

247 ds (SCFAs) present in the ileum and cecum of streptomycin-treated mice and untreated controls.

248 42 initially grew in the large intestines of streptomycin-treated mice but then failed to colonize (<

249 Neither conventional nor streptomycin-treated mice developed clinical signs or hi

250 Here we report that to colonize streptomycin-treated mice E. coli HS consumes six of the

251 ts exclusively to colonize the intestines of streptomycin-treated mice when it is the only E. coli st

252 Infection of streptomycin-treated mice with Salmonella was characteri

253 oth bacterial genetic backgrounds was fed to streptomycin-treated mice, together with the respective

254 Using streptomycin-treated mice, we were able to establish sub

255 creased spleen colonization by Salmonella in streptomycin-treated mice, with more of the pat mutant r

256 In contrast, no colonization occurred in non-streptomycin-treated mice.

257 ve EAEC virulence factors to colonization of streptomycin-treated mice.

258 c conditions in cecal contents of saline- or streptomycin-treated mice.

259 Previously we reported that the streptomycin-treated mouse intestine selected for two di

260 Previously, we reported that the streptomycin-treated mouse intestine selected nonmotile

261 rain E. coli EDL933 colonizes a niche in the streptomycin-treated mouse intestine that is distinct fr

262 strain MG1655, following passage through the streptomycin-treated mouse intestine, that has colonizat

263 bacterial biofilms in the mucus layer of the streptomycin-treated mouse intestine.

264 t they appear to use different sugars in the streptomycin-treated mouse intestine.

265 d as being important for colonization of the streptomycin-treated mouse large intestine by Escherichi

266 de that E. coli is the only bacterium in the streptomycin-treated mouse large intestine that respires

267 , there was no defect in colonization of the streptomycin-treated mouse model by the gadX mutant in c

268 Studies with the streptomycin-treated mouse model have produced important

269 te, and examined the effects in vivo using a streptomycin-treated mouse model of colonization.

270 pathogens, and murine models, including the streptomycin-treated mouse model of infection, are frequ

271 ne the role of respiratory metabolism in the streptomycin-treated mouse model of intestinal colonizat

272 Stx2d1-producing mutant was attenuated in a streptomycin-treated mouse model of STEC infection.

273 In the streptomycin-treated mouse model, more animals infected

274 In a streptomycin-treated mouse model, parenteral administrat

275 By using the streptomycin-treated mouse model, we found that cataboli

276 By using the streptomycin-treated mouse model, we found that Taconic

277 erobic respiration in colonization using the streptomycin-treated mouse model.

278 suppressed aggregated protein formation upon streptomycin treatment and increased aminoglycoside resi

279 Here we show that streptomycin treatment depleted commensal, butyrate-prod

280 However, streptomycin treatment did not show positive effects in

281 pansion were also observed in the absence of streptomycin treatment in genetically resistant mice but

282 Prenatal neomycin, polymyxin B, and streptomycin treatment protected NOD mice from diabetes

283 ock regulon was induced only by puromycin or streptomycin treatment, which lead to truncation or mist

284 in the mouse and the changes that occur upon streptomycin treatment.

285 ificantly increased hair cell survival after streptomycin treatment.

286 vancomycin, but increased dramatically after streptomycin treatment.

287 The streptomycin trial in the United Kingdom in the 1940s in

288 s to ampicillin, erythromycin, tetracycline, streptomycin, trimethoprim-sulfamethoxazole, chloramphen

289 e demonstrations in clinical trials that (1) streptomycin was effective; (2) combination of drugs pre

290 Monoclonal antibody specific to streptomycin was immobilized on to the thiol modified go

291 ptibility testing results for ethambutol and streptomycin was poor.

292 The binding of streptomycin was too weak to assess.

293 , conferring resistance to spectinomycin and streptomycin, was associated with an OXA-23 carbapenemas

294 mportantly, a single dose of one antibiotic, streptomycin, was sufficient to reduce CVB3 shedding and

295 d, rifampicin, ethambutol, pyrazinamide, and streptomycin), we demonstrate that this new approach is

296 nate, immune response because the effects of streptomycin were not observed during the early phases o

297 s to be highly specific to spectinomycin and streptomycin, while the acetyltransferase domain shows a

298 namic range (0.3-300 ng/mL) was obtained for streptomycin with a good linearity in the range 0.3-10 n

299 aptasensor exhibited high selectivity toward streptomycin with a limit of detection (LOD) as low as 1

300 asensors showed excellent selectivity toward streptomycin with limit of detections as low as 73.1 and