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1 ation to look for alternative causes such as stress fracture.
2 he gold standard for definitive diagnosis of stress fractures.
3 uptions of the alveolar walls, suggestive of stress fractures.
4 rains, and reflex sympathetic dystrophy; and stress fractures.
5 o volunteers, the changes resembled those of stress fractures.
6 ation of tissue following experimental ulnar stress fracture and assessed the impact of loss of TrkA
7 s differed in runners with history of tibial stress fractures and current Achilles tendinopathy (p <
16 s to be a risk factor for the development of stress fracture, but this difference may be secondary to
18 nce imaging may be required to differentiate stress fractures from other processes such as malignant
19 biomechanics can have an etiological role in stress fracture, gait re-training may be an important fe
22 tact forces, the risk of hip pain, falls and stress fractures might be greater during stair descent t
27 rate an essential role of TrkA signaling for stress fracture repair and implicate skeletal sensory ne
30 al diagnosis of exertional leg pain includes stress fractures, stress reaction, periostitis, claudica
31 isposed to different sites and mechanisms of stress fractures than their skeletally mature counterpar
32 individuals at high risk for lower extremity stress fracture when beginning a rigorous physical train
33 and treatment strategies of lower extremity stress fractures, while highlighting new research relate