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1 ceptive afferents to prevent recovery of the stretch reflex.
2 -reflex, the electrical analog of the spinal stretch reflex.
3 a strong peripheral amplifier of the spinal stretch reflex.
4 jor contribution to the phase advance of the stretch reflex.
5 H-reflex, an electrical analog of the spinal stretch reflex.
6 motoneuron connections and an absence of the stretch reflex.
7 SPs in motoneurons, and the disappearance of stretch reflexes.
8 e input favours relatively tonic and diffuse stretch reflexes.
9 limb position sensation (proprioception) and stretch reflexes.
10 to influence the gain of artificially evoked stretch reflexes.
11 e 'natural' inputs involved in transcortical stretch reflexes.
12 contribute to maintain or even increase the stretch reflex after nerve crush and by difference to ne
15 gests that twitches trigger the monosynaptic stretch reflex and, by doing so, contribute to its activ
16 This pattern cannot be produced by muscle stretch reflexes and can only arise from the anticipator
18 are likely to convey long-latency jaw-muscle stretch reflexes and may contribute to stiffness regulat
19 nerve transection, Ia afferent synapses and stretch reflexes are permanently lost, even after regene
20 he first demonstration that the long-latency stretch reflex can be modified by repeated, precisely ti
23 otoneurons (MNs), the central portion of the stretch reflex circuit, are highly specific, but the mec
29 ese findings are difficult to reconcile with stretch reflex control of the pendulum and are of partic
30 -reflex, the electrical analog of the spinal stretch reflex, creates a memory trace that includes cha
31 on, however, the extent to which unmodulated stretch reflexes disrupt voluntary movement, and whether
32 LUT1 synapses are not re-established and the stretch reflex does not recover; however, electrically e
33 velocity dependent, increase in muscle tonic stretch reflexes, due to the amplified reactivity of mot
35 s an electrical analogue of the monosynaptic stretch reflex, elicited by bypassing the muscle spindle
37 that reinnervated muscles failed to generate stretch reflexes, extending observations of areflexia to
39 y reduced when modulating velocity-dependent stretch reflex feedback (i) as per the commonly proposed
43 limb muscles disrupted the modulation of the stretch reflex for force generation and sensory feedback
47 icance Statement Spinal cord injury leads to stretch reflex hyperexcitability, which underlies the cl
50 s on locomotion, the phase-dependency of the stretch reflexes implies a dynamically fluctuating role
51 eparate studies, we examined recovery of the stretch reflex in decerebrate cats, and found that it re
52 trate a previously uncharacterized nonneural stretch reflex in gastric muscles and provide physiologi
55 such fundamental input is velocity-dependent stretch reflexes in lengthening muscles, which should be
61 l disparity in recovery between strEPSPs and stretch reflex led us to conclude that factors in additi
65 evels (1 microM) decreased the monosynaptic "stretch" reflex (MSR) amplitude in WT animals and increa
66 bthreshold corticospinal conditioning of the stretch reflex of biceps and quadriceps was abnormal in
68 loops appear to add flexibility to the human stretch reflex, once considered to be immutable, allowin
71 mechanisms, such as recurrent inhibition and stretch reflex, probably play a major role in the synerg
72 tap to Tri evoked its own homonymous phasic stretch reflex, providing neurophysiological evidence fo
73 udy was designed to estimate the fraction of stretch reflex recovery attributable to functional recov
74 anding, and they produced no evidence of any stretch reflex response in soleus, or gastrocnemius.
75 o how the nervous system centrally modulates stretch reflex responses.A common measure of H-reflex ga
76 -and-hold perturbations used to elicit tonic stretch reflexes revealed significantly prolonged EMG re
78 tryptamine) from enterochromaffin cells, and stretch reflexes that determine the site of origin and p
83 ted that rapid motor responses (i.e., muscle-stretch reflexes) to mechanical perturbations can be mod
84 stationarity the amplitude of biceps phasic stretch reflex varied <10% in the first six repeats of t
85 both methods the magnitude of biceps phasic stretch reflex varied linearly with tap force over the r
86 troke the threshold of the homonymous phasic stretch reflex was low, but it had a normal onset latenc
87 elements in the sensorimotor circuit of the stretch reflex were examined in both the PNS and CNS.
90 ent low thresholds for the homonymous phasic stretch reflex, which had abnormally short onset latenci
91 as calf muscles shortened in contrast to the stretch reflex whose amplitude decreases as muscle short
92 exes, such as the vestibulo-ocular reflex or stretch reflex, whose gains adapt in response to novel a
93 Paradoxical movements cannot be generated by stretch reflexes with constant intrafusal drive but migh