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1 ferred for the treatment of non-disseminated strongyloidiasis.
2 nnovative serodiagnosis method for the human strongyloidiasis.
3 ved drug efficacies against trichuriasis and strongyloidiasis.
4 f malabsorption from severe gastrointestinal strongyloidiasis.
5 use of DAF-12 ligands to treat disseminated strongyloidiasis.
6 tate hyperinfection and, hence, disseminated strongyloidiasis.
7 l in diagnostic and epidemiologic studies of strongyloidiasis.
8 were common among hospitalized patients with strongyloidiasis.
9 re present in 41.3% of hospitalizations with strongyloidiasis.
10 sed to identify risk factors associated with strongyloidiasis.
11 c approaches for treating parasitism such as Strongyloidiasis.
12 low median income were also associated with strongyloidiasis.
13 n RR of mortality and regional prevalence of strongyloidiasis.
14 e dose for the treatment of non-disseminated strongyloidiasis.
18 sian analyses using prevalence estimation of strongyloidiasis and onchocerciasis as two relevant exam
21 did a systematic review and meta-analysis of strongyloidiasis and schistosomiasis prevalence among mi
22 hmaniasis, toxoplasmosis, cryptosporidiosis, strongyloidiasis, and filariasis) as well as travelers'
24 urveillance data, especially data evaluating strongyloidiasis associated with hospitalization, are la
27 ther patient also had complete resolution of strongyloidiasis, but required a course of parenteral iv
30 tified 6931 hospitalizations associated with strongyloidiasis during the study period (11.8 per milli
32 and HIV/AIDS were reported from all regions, strongyloidiasis from most regions, and chronic hepatiti
34 Identification of reliable biomarkers for strongyloidiasis in immunosuppressed patients is critica
36 es - ascariasis, trichuriasis, hookworm, and strongyloidiasis - in addition to the intestinal and liv
37 ar epidemiological surveillance for zoonotic strongyloidiasis is confounded by a genus-specific TaqMa
38 ding visceral leishmaniasis, Chagas disease, strongyloidiasis, malaria, schistosomiasis, histoplasmos
39 ween the results of groups with low and high strongyloidiasis prevalence (chi21 = 4.79; P = .03).
40 r trials (33%) took place in regions of high strongyloidiasis prevalence and 8 (67%) trials took plac
41 ivermectin trials in regions of high vs low strongyloidiasis prevalence and correlation coefficient
42 alysis of 12 trials including 3901 patients, strongyloidiasis prevalence was found to interact with t
43 ls that took place in areas of high regional strongyloidiasis prevalence were associated with a signi
45 als that took place in areas of low regional strongyloidiasis prevalence were not associated with a s
48 sis (TB), hepatitis B, hepatitis C, malaria, strongyloidiasis, schistosomiasis, other intestinal para
51 rent but limited drug of choice for treating strongyloidiasis, the combinatorial effects of the two d
52 This study suggests that, in SOT patients, strongyloidiasis triggers both eosinophilia and eosinoph
53 ction in serum samples from individuals with strongyloidiasis using a sandwich enzyme-linked immunoso
54 a population-based retrospective analysis on strongyloidiasis using the National Inpatient Sample fro
57 s expressing Th1, Th2, and Th17 cytokines in strongyloidiasis, we compared the frequency (Fo) of thes