ライフサイエンスコーパス: subacute

1 eir occurrence as acute (</=24 h after PCI), subacute (1 day to 30 days), and late (30 days to 1 year

2 port on the use of acute (1 hour, n = 7) and subacute (1 week, n = 6) resting first-pass perfusion an

3 The rates of subacute (1-30 days) ST were similar with bivalirudin an

4 CVEs were classified as acute (</=24 hours), subacute (1-30 days), or late (>30 days).

5 tion and cognitive function at various acute/subacute (1-7 day post-injury) and chronic (14-60 days p

6 he early (5 weeks) increase in edema and the subacute (10 weeks) increase in fibrosis (r=0.90; P<0.00

7 lesions were detected on pmMRI (chronic: 25; subacute: 16; acute: 30; and peracute: 53).

8 ionts, we investigated acute (7 to 10 days), subacute (2 to 3 weeks), and chronic (4 to 6 weeks) phas

9 Limited data for treatment of subacute (2 to 6 weeks after onset) type B aortic dissec

10 antation intervals: acute (</=30 days; n=7), subacute (31 to 90 days; n=23), chronic (91 to 300 days;

11 (>30 days; HR, 0.48; 95% CI, 0.24-0.96) and subacute (4 hours-30 days; HR, 0.60; 95% CI, 0.39-0.93)

12 Subacute (48 hours) exposure to bifenthrin commencing 2

13 radiography of radioligands was performed at subacute (5-6 d) and chronic (40-42 d) time points after

14 or cells and two treatment schedules, either subacute (7 days) or early chronic (21 days) neural stem

15 .2%-77.5%]) even when symptoms were acute or subacute (89.0% [95% CI, 81.0%-93.9%]).

16 itis) or by prominent barrier abnormalities (subacute allergic contact dermatitis, atopic dermatitis)

17 ration in the lung parenchyma in response to subacute alveolar hypoxia.

18 leus accumbens resting-state activity in the subacute and chronic back pain patients revealed loss of

19 e predictors and patient stratifiers in both subacute and chronic clinical trials.

20 riable clinical manifestations indicative of subacute and chronic disease.

21 C) disaster has been associated with several subacute and chronic health effects, but whether excess

22 Importantly, subacute and chronic HP may mimic several interstitial l

23 i-implantitis was in most cases a mixture of subacute and chronic inflammation dominated by plasma ce

24 ention had a sustained effect on troublesome subacute and chronic low-back pain at a low cost to the

25 d cine MR images allows for the diagnosis of subacute and chronic MI with high accuracy.

26 re analysis (TA) allows for the diagnosis of subacute and chronic myocardial infarction (MI) on nonco

27 Subacute and chronic myocardial infarction could be dete

28 Effect sizes for subacute and chronic neurobehavioral endpoints in other

29 al changes (P-Tau and GFAP induction) in the subacute and chronic phase as well as development of chr

30 on of leukocytes and macrophages, but in the subacute and chronic phases of injury the glial scar is

31 Sixty-three patients with subacute and chronic rotary knee joint trauma of either

32 revealed increased anxiety-like behavior at subacute and chronic time-points, respectively.

33 al cells are sources of viral persistence in subacute and chronic ZIKV infection, and if infection of

34 ing the acute disease, which resolved at the subacute and convalescent periods in patients with no co

35 Patients with subacute and Discoid Lupus Erythematous had elevated Int

36 history of infantile liver involvement, and subacute and remitting course simulating multiple sclero

37 Chronic, subacute, and acute infarction cases correlated excellen

38 3-T pmMRI visualizes chronic, subacute, and acute myocardial infarction in situ.

39 Medical treatment of uncomplicated acute, subacute, and chronic type B aortic dissection is manage

40 oth imaging modalities at each stage (acute, subacute, and chronic) and it is important that physicia

41 ods on 8 pediatric KD patients at the acute, subacute, and convalescent time points.

42 ecular pathways that contribute to early and subacute axonal degeneration after stroke.

43 In a longitudinal brain imaging study, subacute back pain (SBP) patients were followed over the

44 n activity for back pain in the early, acute/subacute back pain group is limited to regions involved

45 hed in time, whereas in the persistent acute/subacute back pain group, activity diminished in acute p

46 In the recovered acute/subacute back pain group, brain activity diminished in t

47 istory of back pain for 1 year (early, acute/subacute back pain group, n = 94), to subjects who have

48 We tracked brain properties in subacute back pain patients longitudinally for 3 years a

49 At baseline, subacute back pain patients showed altered local nucleus

50 Specifically, subacute back pain patients who are at risk for developi

51 In a subset of subacute back pain patients, we followed brain activity

52 m persistent bacteraemia in association with subacute bacterial endocarditis and shunt nephritis.

53 or children with attention deficit disorder, subacute bacterial endocarditis prophylaxis and follow-u

54 denial of valve replacement in patients with subacute bacterial endocarditis, and posthumous donation

55 reports of vanishing bile duct syndrome(3), subacute biliary injury and immune cholangitis.(4).

56 teins as those recognized by sera from acute/subacute, blood culture-positive brucellosis patients bu

57 The results suggest that in a subacute canine infarct model, the LBs are a source of a

58 atients with a PA delay were discharged to a subacute care facility compared with an outpatient setti

59 multiple variable regression, discharge to a subacute care facility was associated with an increased

60 itted infection etiologies in this community subacute care setting.

61 and to have had prolonged stays in-acute and subacute care settings during their final year of life.

62 transfer of undocumented patients in need of subacute care to their country of origin.

63 chy areas of air trapping, are seen in acute/subacute cases, whereas reticular opacities, volume loss

64 the differential diagnosis of patients with subacute cerebellar ataxia of unknown cause.

65 The patient presented with subacute cerebellar syndrome and myoclonus several days

66 and traction bronchiectasis superimposed on subacute changes are observed in chronic cases.

67 ly and invasively, lesions developing in the subacute-chronic phase can be managed with medical treat

68 c human neural stem cell line CTX0E03 in the subacute-chronic recovery phase of stroke and potential

69 ute self-limited necrotizing arteritis (NA), subacute/chronic (SA/C) vasculitis, and luminal myofibro

70 Our results reveal that subacute/chronic axon loss induced by vincristine occurs

71 e form and by Th1 and likely Th17 T cells in subacute/chronic cases.

72 r dysfunction, as well as imaging during the subacute/chronic phase of COVID-19.

73 to the injured spinal cord parenchyma at the subacute/chronic phase.

74 uced peripheral neuropathy, is the result of subacute/chronic processes that may be regulated differe

75 abnormalities are observed in the acute and subacute/chronic stages.

76 e industrial exposure have been implemented, subacute cobalt-related cardiomyopathy had become rare.

77 vated intracranial pressure in children with subacute conditions, such as craniosynostosis or tumor,

78 emperature >38 degrees C [>100.4 degrees F], subacute course, large effusion or tamponade, and failur

79 ents with lupus erythematosus (LE) including subacute cutaneous lupus erythematosus (SCLE) and discoi

80 rapeutic agents have been reported to induce subacute cutaneous lupus erythematosus (SCLE).

81 Evaluation of discoid and subacute cutaneous lupus erythematosus lesions showed si

82 c valve implantation, the rates of acute and subacute CVEs were 2.7% and 2.4%, respectively.

83 2.76; 95% CI, 1.11-6.83) was a predictor of subacute CVEs.

84 Conclusions Doxorubicin causes a subacute decrease in cardiac mass in both mice and human

85 Subacute decrease of hemoglobin deposition was also obse

86 largely was attributable to a lower risk of subacute definite stent thrombosis: 0.1% versus 0.6% (ra

87 ed blood-brain barrier (BBB) was involved in subacute diaschisis.

88 rom nodular centrilobular opacities in acute/subacute disease to increased reticular markings and hon

89 Classical AAG is a subacute disorder associated with antibodies against gan

90 Relative to TAA without dissection, acute or subacute dissection (OR=2.9; 95% CI, 1.6-5.3), but not c

91 hronic dissection and 109 (10%) had acute or subacute dissection.

92 ngin (MFG) for the treatment of experimental subacute disseminated candidiasis.

93 Mice were put on the subacute dosing regimen of 1-methyl-4-phenyl-1,2,3,6-tet

94 The clinical presentation was subacute during a period of weeks in 13 patients (38%).

95 g; CCC of 0.995 and 95% CI: 0.993, 0.997 for subacute DW imaging).

96 Step 2 (n=7) examined the subacute effects of PFA on block durability, phrenic ner

97 , and most patients had repeated episodes of subacute encephalopathy with motor regression, irritabil

98 minent clinical manifestations are headache, subacute encephalopathy, optic papillitis, inflammatory

99 METHODS AND We used subacute endotoxemia in the rodent macrophage-to-feces R

100 4 levels increase after experimental stroke, subacute EphA4 inhibition followed by environmental enri

101 (4.4%), including 28 acute events (0.9%), 49 subacute events (1.6%), 32 late events (1.0%), and 33 ve

102 al fibrillation determined a higher risk for subacute events.

103 In contrast, untrained subacutes exhibited spontaneous improvements in luminanc

104 Subacute exposure (19 d after initiating ascent to Evere

105 Collectively, these data indicate that subacute exposure to DiNP alters colon morphology and ph

106 Thus, this study tested the hypothesis that subacute exposure to DiNP dysregulates cellular, endocri

107 elper T-cell-type inflammatory response, and subacute fibrosis were recognized.

108 with the ail mutant resulted in an atypical, subacute form of bubonic plague associated with extensiv

109 ithin the medullary cavity consistent with a subacute form of hematogenous osteomyelitis termed a Bro

110 ly, e2 significantly increased risk of acute/subacute gross haemorrhage in the presence of CAA.

111 e patients have prolonged stays in acute and subacute health care settings and appear to have limited

112 Subacute, hematogenous, and extrapulmonary disease tends

113 inhibition (P = 0.021, n = 5/group) without subacute hematologic toxicity (n = 3/group).

114 Differences in the subacute hemodynamic effects of amlodipine and lisinopri

115 ly healthy 37-year-old female presented with subacute hepatitis, in the context of a previous admissi

116 HIT antibodies indicates that patients with subacute HIT undergoing repeated TPE before heparin reex

117 ts who have serologically-confirmed acute or subacute HIT; for this situation, a negative platelet ac

118 rial serum samples in a patient with recent (subacute) HIT who underwent serial TPE precardiac surger

119 (nGD), a mouse model (4L;C*), an analogue to subacute human nGD, was investigated for global profiles

120 rgical lung biopsy that were consistent with subacute hypersensitivity pneumonitis (and IgG positive)

121 We suggest that during subacute hypoxia, mitochondria might be protected from o

122 en; mean (SD) age, 50.05 (18.88) years) with subacute (ie, 1 month) spinal cord injury (25 patients w

123 ed into 3 phases: the acute viral phase, the subacute immune phase, and the chronic cardiac remodelin

124 The early increase in edema and subacute increase in fibrosis are strongly linked and ar

125 phalopathy, metachromatic leukodystrophy and subacute infarct.

126 (VSMC) were injected into the border zone of subacute infarcted syngeneic Fischer rat hearts and comp

127 d a high percentage of macrophages linked to subacute infarcts, reactive astrocytes, and damaged bloo

128 Subacute infection was more frequent (median, 4 weeks [r

129 Streptococcus oralis is a leading cause of subacute infective endocarditis.

130 expansion in the liver and can precipitate a subacute inflammatory hepatitis with stellate cell activ

131 etwork over-stimulation that may explain the subacute inflammatory presentation.

132 ntribute to endogenous protection during the subacute injury phase.

133 Ten patients (group A) with acute and subacute ischemic stroke underwent perfusion-weighted (P

134 to clinical adverse events in patients with subacute ischemic stroke, although there was a higher in

135 ular, patients with BVS had a higher risk of subacute, late, and very late ST, whereas the risk of ac

136 Of note, BVS had a higher risk of subacute, late, and very late ST, whereas the risk of TL

137 r parietal cortex in patients suffering from subacute left spatial neglect, in a randomized, double-b

138 ting neutrophils in 25 patients with ALF and subacute liver failure (SALF).

139 stablished, the study of the pathogenesis of subacute liver failure with ascites complication is hamp

140 l tool for investigating the pathogenesis of subacute liver failure with ascites complication.

141 nt study aimed at providing a mouse model of subacute liver failure with ascites complication.

142 me of the features observed in patients with subacute liver failure, especially ascites.

143 ailure is divided into hyperacute, acute and subacute liver failure.

144 Ascites is a common complication of subacute liver failure.

145 ary optic neuropathy (LHON) is a syndrome of subacute loss of central vision associated with mutation

146 on 1: Given that most patients with acute or subacute low back pain improve over time regardless of t

147 ate lumbar imaging in patients with acute or subacute low-back pain and without features suggesting a

148 Results are most applicable to acute or subacute low-back pain assessed in primary-care settings

149 has the potential to help reliably quantify subacute microinfarction.

150 market when it was linked to an epidemic of subacute myelo-optico-neuropathy (SMON).

151 estoring heart function after both acute and subacute myocardial infarction in rats.

152 Necrotizing Myopathy is a poorly studied subacute myopathy triggered by toxic, viral, or autoimmu

153 For participants with acute and subacute neck pain, SMT was more effective than medicati

154 to guide the choice of therapy for acute and subacute neck pain.

155 Leigh syndrome (LS) is a subacute necrotizing encephalomyelopathy with gliosis in

156 ng normal liver function and Leigh syndrome (subacute necrotizing encephalomyelopathy) seen in associ

157 nts without neglect, the group including all subacute neglect patients had decreased fractional aniso

158 ell tolerated, without any signs of acute or subacute nephrotoxicity during a mean follow-up of nearl

159 rial disease is Leigh syndrome, an episodic, subacute neurodegeneration that can lead to death within

160 receptor was examined in vivo in a model of subacute neuroinflammation induced by administration of

161 an with erosive rheumatoid arthritis in whom subacute neurologic and psychiatric symptoms developed a

162 All patients but one presented with subacute neurological deterioration in infancy or childh

163 nts with this disorder present with acute or subacute neurological deterioration, with MRI showing on

164 All patients had manifested acute or subacute neurological symptoms; the brain MRIs indicatin

165 Between November, 2006, and May, 2008, a subacute neurological syndrome affected workers from two

166 ed as a possible cause of diffuse unilateral subacute neuroretinitis in humans.

167 al signs and diagnosis of diffuse unilateral subacute neuroretinitis in its early stage, followed by

168 with early- or late-stage diffuse unilateral subacute neuroretinitis were included.

169 e teenage years and is often associated with subacute onset and marked muscle inflammation.

170 The patient developed subacute onset of bilateral blindness following his 44th

171 Predictors of clinical response included subacute onset of CA (odds ratio [OR], 0.50; 95% CI, 0.2

172 a-myalgia syndrome (EMS) is characterized by subacute onset of myalgias and peripheral eosinophilia,

173 rolizumab or nivolumab), 10 (2.9%) developed subacute onset of neurological complications.

174 p of disorders characterised by the acute-to-subacute onset of painful sensory and motor deficits tha

175 Patients present with the subacute onset of severe heart failure, which is accompa

176 In patients with CA and GAD65-Abs, subacute onset of symptoms and prompt immunotherapy are

177 ody encephalitis, it usually has an acute or subacute onset, and antipsychotic treatment may be ineff

178 gth dependence, motor predominance, acute or subacute onset, and prominent autonomic involvement shou

179 Atypical neuropathy features, such as acute/subacute onset, asymmetry, and/or motor predominant sign

180 ted at age 7 years with cerebellar ataxia of subacute onset.

181 Four patients had a subacute-onset and sensory ataxia.

182 bulatory visits were primarily for acute- or subacute-onset diplopia.

183 atients presented with difficult-to-diagnose subacute or chronic conditions.

184 2) or small (enhanced volume </=20%, n = 48) subacute or chronic ischemic scars were included.

185 aken in England, 701 adults with troublesome subacute or chronic low-back pain were recruited from 56

186 elpful in evaluating ongoing symptoms in the subacute or chronic setting.

187 ng G-CSF in patients with hyperacute, acute, subacute or chronic stroke, and asked Investigators to s

188 emisphere, or bilateral; (2) recovery stage: subacute or chronic stroke; (3) stimulation timing: tDCS

189 canalization used in the treatment of acute, subacute or even chronic occlusions or stenosis of perip

190 HP may present as acute, subacute, or chronic clinical forms but with frequent ov

191 tient population includes adults with acute, subacute, or chronic low back pain.

192 No adverse acute, subacute, or long-term consequences from the Experimenta

193 separate groups of dogs with pacing-induced subacute overt HF complicated by renal dysfunction.

194 superior longitudinal fasciculus II/III for subacute patients and in its caudal portion for chronic

195 81.5% (22 of 27 patients) in the context of subacute pelvic pain for readers 1, 2, and 3, respective

196 All patients with acute or subacute pelvic pain who were undergoing MR examination

197 ients who have an adnexal mass with acute or subacute pelvic pain.

198 Reperfusion was assessed using subacute perfusion MRI coregistered to baseline imaging.

199 ination abilities are often preserved inside subacute, perimetrically-defined blind fields, but they

200 ive phase of axonal injury occurs during the subacute period and damages axons that survive the initi

201 premovement IHI was normal during the acute/subacute period and only became abnormal at the chronic

202 e of the MIS-C and KD groups, and during the subacute period in the MIS-C group (interval 5.2 +/- 3 d

203 r how PMO, a phenomenon limited to the acute/subacute period of MI, drives adverse remodeling in chro

204 the initial insult, most patients undergo a subacute period with intense plasticity and rapid functi

205 During the subacute period, LVEF returned to normal (median from 54

206 ties of the post-stroke visual system in the subacute period, nor do we know if these properties can

207 tive testing for neglect twice, first at the subacute phase (<3 months after onset) and then at the c

208 levels while GGA1 levels are restored in the subacute phase (7 d) after injury.

209 ), the ADBR was greater than the ADIR in the subacute phase (LSMD: -0.39%; 95% CI: -0.58% to -0.20%;

210 7 days, and 10 days) assessing the acute and subacute phase after injury.

211 y occluded infarct-related artery during the subacute phase after myocardial infarction compared with

212 and without severe inducible ischemia in the subacute phase after myocardial infarction.

213 ous-scaly as the condition progresses to the subacute phase and papular-hyperkeratotic in the chronic

214 , the effect of IRL-1620 is not known in the subacute phase of cerebral ischemia, where development o

215 d BACE1 solely depends on GGA3 levels in the subacute phase of injury.

216 and in vivo regulation of miRNAs during the subacute phase of stroke has not yet been proposed.

217 ect of a neuromodulatory intervention in the subacute phase of stroke.

218 nying motor abnormalities at least up to the subacute phase, advances this cortical hemorrhage model

219 terial soft tissue injury is greatest in the subacute phase, and least in the chronic phase, suggesti

220 or evaluating infarct growth and size in the subacute phase.

221 We sequenced tissue samples from acute and subacute phases (2 days and 7 days after injury) and sys

222 raction between MA and mTBI during acute and subacute phases after injury.

223 We conclude that in the acute and subacute phases of disease, KD patients do not exhibit s

224 the bifurcation point at both the acute and subacute phases.

225 n presented to the emergency department with subacute poorly localized abdominal pain that was increa

226 S-mediated BBB dysfunction during this acute/subacute post-blast interval (24-71 hours after the last

227 This negative regulation was mediated by subacute postseizure increases in mammalian target of ra

228 Subacute potentially catastrophic illnesses in three div

229 Patients in ICUs are susceptible to subacute potentially catastrophic illnesses such as resp

230 Acute/subacute presentations accounted for the majority of pri

231 The natural history of LHON is not a subacute process, as previously believed, but progresses

232 , newer understandings suggest that acute or subacute processes are involved, and with proper diagnos

233 However, subacute recovery was attained six times faster; it also

234 After experimental stroke, various subacute rehabilitation paradigms improve recovery.

235 The subacute response exhibited granulation tissue with earl

236 7-year-old Tunisian woman who presented with subacute right upper quadrant pain and a large multi-loc

237 ectivity (FC) and ROI-to-ROI connectivity in subacute right-hemisphere stroke patients.

238 we prospectively included 60 patients with a subacute right-hemispheric stroke.

239 degeneration of perturbed axons in acute and subacute rodent axon degeneration models.

240 ntrast to its remarkable effect on acute and subacute SCI.

241 Subacute sclerosing panencephalitis (SSPE) is a fatal co

242 In some cases, MeV persistence and subacute sclerosing panencephalitis (SSPE) occur even in

243 measles virus infection of the brain causes subacute sclerosing panencephalitis (SSPE), a progressiv

244 essive fatal neurodegenerative complication, subacute sclerosing panencephalitis (SSPE), occurs durin

245 Years later, children are presenting with subacute sclerosing panencephalitis (SSPE).

246 s the in vivo response to acute (sepsis) and subacute (skin) inflammatory challenge.

247 In patients with subacute spinal cord injury (SCI), the motor system unde

248 uncovered struts and underexpansion in acute/subacute ST and neoatherosclerosis and uncovered struts

249 and out-of-hospital ST groups indicated that subacute ST had the highest mortality.

250 T was uncovered struts (66.7% of cases); for subacute ST, the most common dominant finding was uncove

251 ussive blast injury (n = 50) in the acute to subacute stage and combat-deployed control individuals (

252 ncy and CR2-fH treatment is sustained in the subacute stage of infarct development, adding to the cli

253 al hemorrhage (ICH) at hyperacute, acute and subacute stages by comparing with susceptibility weighte

254 singly viewed as a new therapeutic target in subacute stages of brain infarction.

255 RI could detect ICH at hyperacute, acute and subacute stages.

256 intensities of ICH at hyperacute, acute and subacute stages.

257 r for detecting ICH at hyperacute, acute and subacute stages.

258 ntly decreases after stroke in the acute and subacute stages.

259 flammation can be better differentiated in a subacute state.

260 death (26 [3.5%] vs 18 [1.7%]; P = .01), and subacute stent thrombosis (8 [1.1%] vs 3 [0.3%]; P = .03

261 alences of successful reperfusion, acute and subacute stent thrombosis, 30-day death or combined endp

262 independent predictors of reduced acute and subacute stent thrombosis, respectively.

263 1), with nonsignificantly different rates of subacute stent thrombosis.

264 Forty patients with subacute stroke (3-14 days after neurological symptom on

265 Here, for the first time, a large sample of subacute stroke patients (N = 667) was assessed without

266 otocols devised to enhance motor recovery in subacute stroke patients have failed to show benefits wi

267 nt (BOLD) data in detecting hypoperfusion of subacute stroke patients through comparison with dynamic

268 Twenty-four hospitalized subacute stroke patients were randomly assigned to 2 int

269 ve to DSC-PWI for detecting hypoperfusion in subacute stroke patients who had obvious MRA lesions wit

270 Twenty-eight hospitalized subacute stroke patients with severe motor deficits were

271 ficantly better motor functional outcomes in subacute stroke patients with severe motor impairments.

272 ed in a randomized controlled pilot study in subacute stroke patients.

273 part of the weekly rehabilitation program in subacute stroke patients.

274 tidepressants may provide a novel target for subacute stroke therapy.

275 cy medical services; acute stroke treatment; subacute stroke treatment and secondary prevention; reha

276 cular ischemic events (acute stroke, n = 20; subacute stroke, n = 2; chronic stroke, n = 3; transient

277 ubsequent MTX without recurrence of acute or subacute symptoms.

278 es (n = 16), acute ataxia (n = 4), and other subacute syndromes (transverse myelitis [n = 1], opsoclo

279 traumatic cervical spinal cord lesions in 24 subacute tetraplegic patients.

280 thin hours of ingestion; collective n = 53), subacute (three active doses over 4 weeks; n = 28), and

281 These results indicate subacute to chronic Alzheimer-like alterations after CCI

282 of the work presented here was to develop a subacute to chronic caprine model of melioidosis and to

283 Most of these diseases have a subacute to chronic course and they can become recalcitr

284 op systemic acute infections that can become subacute to chronic infections.

285 d be restricted to patients with progressive subacute to chronic neurological symptoms due to congest

286 ecovery by promoting brain plasticity in the subacute to chronic phase after ischaemic stroke.

287 spontaneous partial recovery of functions in subacute to chronic phases.

288 e to the diffusion tensor imaging changes in subacute to chronic traumatic brain injury.

289 echniques, particularly in the assessment of subacute to early chronic stroke evolution.

290 anisms of recovery from DoC in the acute and subacute-to-chronic stages, and we discuss recent progre

291 ) and 85.1% (23 of 27 patients) in acute and subacute torsion, respectively.

292 Subacute toxicity at 8 wk and chronic toxicity at 1 y af

293 se anticonvulsant studies, being nontoxic in subacute toxicity studies.

294 In subacute-toxicity of LNCs (1 and 10 mg/kg body weight) r

295 Subacute training stops this degradation, and is far mor

296 Only the subacute transplant of neural stem/precursor cells enhan

297 e imaging (MRI) protocol to 14 patients with subacute traumatic SCI at baseline, 2 months, 6 months,

298 Finally, spontaneous visual improvements in subacutes were restricted to luminance detection; discri

299 Subacute zinc deficiency significantly increases systemi

300 We hypothesized that subacute zinc deficiency would amplify immune responses