ライフサイエンスコーパス: subdural

1 hile, Brain computed tomography demonstrated subdural abscess in right parietal area.

2 Using subdural and depth recordings from epileptic patients, w

3 ence of diffuse cerebral oedema, presence of subdural and extradural hematoma; however in isolation t

4 A sensor was tested subdural and in vitro, simulating a supine infant with a

5 morrhage (intraparenchymal, subarachnoid, or subdural, and cerebral microbleed [CMB]).Twenty-six pati

6 Although complications such as intracystic, subdural, and extradural hematomas are well known after

7 parenchymal, intraventricular, subarachnoid, subdural, and/or epidural hemorrhage) and segmentations

8 n death was induced by sudden inflation of a subdural balloon catheter with continuous monitoring of

9 Brain death was induced by inflation of a subdural balloon in ten mongrel dogs weighing 23 to 30 k

10 participants in the open-shunt group having subdural bleeding (12% vs. 2%) and positional headaches

11 s were skull fractures (36% of cases), acute subdural bleeding (72%) and retinal haemorrhages (71%);

12 ic stroke/systemic embolic event/epidural or subdural bleeding; 4: noncerebral International Society

13 correlated with the increasing volume of the subdural blood clot (sham: 9+/-3 mm3; 200 microl: 81+/-1

14 ter and again at 2 h after completion of the subdural blood infusion.

15 ureus infective endocarditis, with suspected subdural brain hemorrhage, disseminated intravascular co

16 e (ICP) was monitored in all patients with a subdural catheter (Camino Systems, San Diego, CA) for up

17 g studies revealed a large mixed-attenuation subdural collection in the right frontal region with pro

18 aminations showed no white matter changes or subdural collections.

19 ionally in four participants - recorded from subdural cortical electrodes.

20 sies, 12 intracranial cyst evaluations, four subdural drainages, and five transsphenoidal pituitary r

21 e and interictal spike frequency measures on subdural ECoG recording may both be useful in predicting

22 High density scalp EEG and subdural ECoG recordings provide an opportunity to map t

23 Subdural ECoG signals were recorded while each patient v

24 We generated normative brain atlases, using subdural EEG signals from 8251 nonepileptic electrode si

25 ured local cortical activity using arrays of subdural electrocorticographic (ECoG) electrodes in huma

26 Subdural electrocorticographic (ECoG) recordings in pati

27 human functional brain mapping, we recorded subdural electrocorticographic (ECoG) signals in five cl

28 wed by 2-stage epilepsy surgery with chronic subdural electrocorticographic monitoring, and were seiz

29 quency measures obtained from extraoperative subdural electrocorticography (ECoG) recording could pre

30 sequently recorded cortical physiology using subdural electrocorticography during a spatial-attention

31 tentials from hand sensorimotor cortex using subdural electrocorticography during a visually cued, in

32 ation surgery, we use the novel technique of subdural electrocorticography in combination with subtha

33 Here, we use subdural electrocorticography to sample both normal-appe

34 To address this, we utilized subdural electrocorticography to study cortical oscillat

35 The aim was to compare the outcomes of subdural electrode (SDE) implantations versus stereotact

36 We do so using invasive subdural electrode arrays from a population of 16 patien

37 imeslicing), in a subject in whom indwelling subdural electrode arrays had been placed for clinical p

38 with motor movement across 22 subjects with subdural electrode arrays placed for identification of s

39 graphy (ECoG) signals measured directly from subdural electrode arrays that were implanted in patient

40 It can help guide subdural electrode implantation and narrow the search fo

41 Thirteen patients had undergone subdural electrode implantation for the surgical managem

42 After subdural electrode implantation, we used DCS to localize

43 propofol-anaesthetized juvenile swine using subdural electrode strips (electrocorticography) and int

44 preading depolarizations were monitored with subdural electrode strips and regional cerebral blood fl

45 corticography [duration: 54 h (34, 66)] from subdural electrode strips was analysed over Days 0-3 aft

46 electrocorticographic recordings obtained by subdural electrode-strip monitoring during intensive car

47 ECoG from 63 subdural electrodes (500 Hz/channel) chronically implant

48 observed across all sites and for depth and subdural electrodes (P < 0.001 and P < 0.001, respective

49 cy oscillations were seen in recordings from subdural electrodes adjacent to the microelectrode array

50 implanted with bilateral VC/VS DBS leads and subdural electrodes adjacent to the orbitofrontal cortex

51 s by stimulating a part of the brain through subdural electrodes and recording the cortical evoked po

52 These patients had chronic implantation of subdural electrodes covering part of the lateral and med

53 tex in neurosurgical patients implanted with subdural electrodes during viewing of face subcategories

54 Subdural electrodes for electrocorticography were implan

55 42 years) underwent invasive monitoring with subdural electrodes for epilepsy surgery.

56 EEG studies from subdural electrodes have demonstrated a face-specific ev

57 ecorded high gamma (62-100 Hz) activity from subdural electrodes implanted for seizure monitoring.

58 ctrical stimulation of chronically implanted subdural electrodes in 34 patients (mean age, 12.2 years

59 cortex (M1), using electocorticography from subdural electrodes in four patients while they performe

60 re recorded from human temporal cortex using subdural electrodes in order to investigate in greater a

61 face perception in a patient implanted with subdural electrodes in the right inferior temporal lobe.

62 using electrocorticographic recordings from subdural electrodes over frontal and temporal cortices.

63 ed brain-computer interface that consists of subdural electrodes placed over the motor cortex and a t

64 bilateral CM thalamic macroelectrodes and M1 subdural electrodes that were connected to two neurostim

65 ctrodes, and the precise localization of the subdural electrodes was defined by MRI co-registration.

66 The exact location of the subdural electrodes was determined on high-resolution th

67 nd verb generation in 11 humans in whom 1210 subdural electrodes were implanted.

68 Subdural electrodes were placed over the prefrontal cort

69 lated visual cortex in humans implanted with subdural electrodes while recording from other brain sit

70 ored by invasive electrocorticography (ECoG; subdural electrodes) and noninvasive scalp EEG during in

71 ified with intracranial recordings (depth or subdural electrodes).

72 s performed by electrical stimulation of the subdural electrodes, and the precise localization of the

73 trial stimulation, using a temporary grid of subdural electrodes.

74 rect cortical stimulation (DCS) of implanted subdural electrodes.

75 Invasive subdural electroencephalogram monitoring is valuable and

76 efore, we aimed to evaluate whether invasive subdural electroencephalogram recording leads to earlier

77 on that a greater proportion of patients had subdural empyema and hemiparesis in 2011-2013.

78 ge subtypes (subarachnoid, intraventricular, subdural, epidural, and intraparenchymal hemorrhage) typ

79 as isolated deep (no insular, subarachnoid, subdural extension) or lobar.

80 occurred by an increase in a combination of subdural, extradural, and subarachnoid bleeding with asp

81 %), two intraventricular masses (0.05%), two subdural fluid collections (0.05%), and two other tumors

82 magnetic resonance imaging findings include subdural fluid collections, enhancement of the pachymeni

83 ing of the brain, pituitary enlargement, and subdural fluid collections.

84 19.8, 95%CI 9.4-30.2; p = 0.001), and recent subdural grid implantation (beta = 42.8, 95%CI 11.8-73.8

85 gies if the desired signals are local, while subdural grids and strips sample more gray matter if the

86 edically intractable epilepsy implanted with subdural grids.

87 as was underlying traumatic brain injury or subdural haematoma (4.4 [1.4-14.0]), a Glasgow Coma Scal

88 according to treatment preference for acute subdural haematoma (acute surgical evacuation or initial

89 ncytopenia [n=2], bone marrow failure [n=1], subdural haematoma [n=1], and intracranial haemorrhage [

90 TBI, of whom 1407 (31%) presented with acute subdural haematoma and were included in our study.

91 Chronic subdural haematoma causes serious morbidity and mortalit

92 ients aged 18 years and older with a chronic subdural haematoma for burr-hole drainage were assessed

93 Therefore, in a patient with acute subdural haematoma for whom a neurosurgeon sees no clear

94 ]), and evacuation of a supratentorial acute subdural haematoma in the very high HDI tier (155 [47%])

95 stablished, acute surgery in traumatic acute subdural haematoma is based on low-grade evidence.

96 a drain after burr-hole drainage of chronic subdural haematoma is safe and associated with reduced r

97 show that treatment for patients with acute subdural haematoma with similar characteristics differed

98 rological disorders who presented with acute subdural haematoma within 24 h of traumatic brain injury

99 The results indicate that following acute subdural haematoma, a rapid cellular redistribution of a

100 ors, if any, are associated with presence of subdural haematoma.

101 sone for patients with a symptomatic chronic subdural haematoma.

102 2 h or 4 h following production of an acute subdural haematoma.

103 ly, or a procedure for drainage of a chronic subdural haematoma.

104 but at the expense of adverse events such as subdural haematoma.

105 Trial of dexamethasone for chronic subdural haematoma.

106 ring initial conservative treatment in acute subdural haematoma.

107 ient in the 1.5% twice daily group developed subdural haematoma; one patient in the 1.5% once daily g

108 Subdural haematomas are thought to be uncommon in babies

109 Subdural haematomas occurred in two patients.

110 onates; to study the natural history of such subdural haematomas; and to ascertain which obstetric fa

111 a [n=4], gastrointestinal haemorrhage [n=1], subdural haemorrhage [n=1], or mesenteric vessel thrombo

112 Presence of unilateral and bilateral subdural haemorrhage is not necessarily indicative of ex

113 All babies with subdural haemorrhage were assessed clinically but no int

114 tis, epidural haemorrhage, humerus fracture, subdural haemorrhage, and tibia fracture [all n=1, 3%]).

115 lso to have a skull fracture, a thin film of subdural haemorrhage, but lack extracranial injury.

116 s in symptomatic neonates and babies in whom subdural haemorrhages are detected fortuitously.

117 We aimed to establish the frequency of subdural haemorrhages in asymptomatic term neonates; to

118 They tend to have larger subdural haemorrhages, and where traumatic axonal injury

119 Nine babies had subdural haemorrhages: three were normal vaginal deliver

120 in >/=2% of patients were hematuria (2%) and subdural hematoma (2%).

121 ere 128 subarachnoid hemorrhage (33.4%), 134 subdural hematoma (35.0%), and 121 intraparenchymal hemo

122 ming surgery for most patients with an acute subdural hematoma (ASDH) and traumatic brain injury (TBI

123 cus (SE) are frequent complications of acute subdural hematoma (aSDH) associated with increased morbi

124 agonist, BAY X3702, in a rat model of acute subdural hematoma (ASDH).

125 Background Chronic subdural hematoma (cSDH) is a common neurosurgical condi

126 A) embolization (MMAE) treatment for chronic subdural hematoma (CSDH) is limited.

127 perioperative phase of treatment for chronic subdural hematoma (cSDH) may reduce recurrence rates but

128 recurrence rate in the evacuation of chronic subdural hematoma (cSDH) needs further study.

129 roposed as a potential treatment for chronic subdural hematoma (CSDH).

130 anisms behind the pathophysiology of chronic subdural hematoma (CSDH).

131 Among 10010 patients with subdural hematoma (mean age, 69.2 years; 3462 women [34.

132 lowing events: recurrent or residual chronic subdural hematoma (measuring >10 mm) at 180 days; reoper

133 y (positive LR, 3.4 [95% CI, 1.8-6.4]), or a subdural hematoma (positive LR, 3.2 [95% CI, 2.6-3.8]) i

134 The rat subdural hematoma (SDH) model produces a zone of ischemi

135 In that series, one patient died of subdural hematoma 380 days after implant.

136 tient experienced worsening of a preexisting subdural hematoma after USCDT and therapeutic anticoagul

137 TS: The SECA (Surgical Evacuation of Chronic Subdural Hematoma and Aspirin) trial was an investigator

138 ceiving apixaban who developed a spontaneous subdural hematoma and declining mental status that impro

139 There were 6 cases of subdural hematoma and intracranial injury reported in fo

140 a trial that involved patients with chronic subdural hematoma and that was stopped early, dexamethas

141 , status epilepticus in the acute phase, and subdural hematoma at presentation.

142 ticularly among those >80 yrs of age (36% of subdural hematoma cohort), in lower income patients, in

143 Neurosurgical intervention for subdural hematoma decreased from 41% in 1998 to 31% in 2

144 Subdural hematoma evacuation was associated with decreas

145 Contusion, subarachnoid hemorrhage, and/or subdural hematoma features were associated with incomple

146 The increased incidence of subdural hematoma from 2000 to 2015 appears to be associ

147 bably or definitely related to treatment): 1 subdural hematoma grade 4, 1 anemia grade 3, 1 thrombocy

148 Incidence of subdural hematoma has been reported to be increasing.

149 The prevalence and total cost for subdural hematoma has increased significantly in the las

150 thasone on outcomes in patients with chronic subdural hematoma has not been well studied.

151 nts receiving standard treatment for chronic subdural hematoma have a high risk of treatment failure.

152 y assigned symptomatic patients with chronic subdural hematoma in a 1:1 ratio to a 19-day tapering co

153 bdural hematoma risk and determine trends in subdural hematoma incidence and antithrombotic drug use

154 Subdural hematoma incidence and antithrombotic drug use

155 The overall subdural hematoma incidence rate increased from 10.9 per

156 dural hematoma with antithrombotic drug use, subdural hematoma incidence rate, and annual prevalence

157 Hospitalizations for subdural hematoma increased from 59,373 (30 per 100,000

158 The prevalence of subdural hematoma increased with age (p < .001), particu

159 Chronic subdural hematoma is a common neurologic disorder that i

160 Subdural hematoma is a common type of intracranial hemor

161 Health resource consumption for subdural hematoma is increasing without clear evidence t

162 gical evacuation in the treatment of chronic subdural hematoma is unclear.

163 Subdural hematoma occurred in 18% of HI (5% of TP), with

164 Subdural hematoma occurred in 8 patients (2 in the core

165 of various ages, particularly rib fractures, subdural hematoma of the brain, and retinal hemorrhages.

166 rge disposition, length of stay, and cost of subdural hematoma over time.

167 tients aged 20 to 89 years with a first-ever subdural hematoma principal discharge diagnosis from 200

168 tion between use of antithrombotic drugs and subdural hematoma risk and determine trends in subdural

169 ritical care unit with an acute nontraumatic subdural hematoma that required emergent surgical evacua

170 y assigned patients with symptomatic chronic subdural hematoma to undergo middle meningeal artery emb

171 f subdural hematoma; and the highest odds of subdural hematoma was associated with combined use of a

172 The risk of subdural hematoma was highest when a VKA was used concur

173 ients undergoing surgery for traumatic acute subdural hematoma were randomly assigned to undergo cran

174 Among patients with traumatic acute subdural hematoma who underwent craniotomy or decompress

175 atients with symptomatic subacute or chronic subdural hematoma with an indication for surgical evacua

176 atients with symptomatic subacute or chronic subdural hematoma with an indication for surgical evacua

177 Association of subdural hematoma with antithrombotic drug use, subdural

178 rmatory cranial CT scan revealed a worsening subdural hematoma with midline shift, a single dose of f

179 echanical fall with head trauma resulting in subdural hematoma with no associated neurological defici

180 s contusion, subarachnoid hemorrhage, and/or subdural hematoma, 3.23 [95% CI 1.59-6.58]).

181 (43%, 26/60); central pontine myelinolysis, subdural hematoma, acute infarcts, and Aspergillus brain

182 Among patients with symptomatic chronic subdural hematoma, adjunctive middle meningeal artery em

183 in injury, primary intracerebral hemorrhage, subdural hematoma, brain tumor, central nervous system i

184 Among adults with symptomatic chronic subdural hematoma, most of whom had undergone surgery to

185 ciousness or amnesia for more than 24 hours, subdural hematoma, or brain contusion).

186 for later seizures were brain contusion with subdural hematoma, skull fracture, loss of consciousness

187 eal artery embolization in the management of subdural hematoma.

188 8-4.03]) were associated with higher risk of subdural hematoma.

189 A CT scan of his head revealed a subdural hematoma.

190 lled adult patients with symptomatic chronic subdural hematoma.

191 s old, 66% were male patients, and 62.6% had subdural hematoma; admission Glasgow Coma Scale score wa

192 drug use was associated with higher risk of subdural hematoma; and the highest odds of subdural hema

193 : contusion, subarachnoid hemorrhage, and/or subdural hematoma; intraventricular and/or petechial hem

194 ognostic factors following surgery for acute subdural hematomas (ASDHs) in England and Wales over a 2

195 Subdural hematomas (SDH) can induce ischemia and neurona

196 Subacute and chronic subdural hematomas are common and frequently recur after

197 Traumatic acute subdural hematomas frequently warrant surgical evacuatio

198 morrhagic contusions or underlying evacuated subdural hematomas was studied.

199 ors of failure of MMAE treatment for chronic subdural hematomas were identified, with small diameter

200 djacent to cerebral contusions or underlying subdural hematomas, even brief periods of hyperventilati

201 may shorten detection time for epidural and subdural hematomas, increase sensitivity (especially for

202 Major discrepancies were four subdural hematomas, one pneumocephalus, one hemorrhagic

203 llowed by intracerebral hemorrhage (8%), and subdural hemorrhage (4%).

204 (OR, 2.17; 95% CI, 1.40-3.38; P < .001), and subdural hemorrhage (OR, 2.05; 95% CI, 1.05-3.98; P = .0

205 Background Subdural hemorrhage (SDH) is thought to have a benign co

206 c subtypes of ICH (eg, 69.2% [74 of 107] for subdural hemorrhage and 77.4% [24 of 31] for acute subar

207 AHT was defined as 1 or more subdural hemorrhage and a positive multidisciplinary eva

208 ot differ between asymptomatic neonates with subdural hemorrhage and control neonates.

209 ury, only one (1%) of 70 children had spinal subdural hemorrhage at presentation; this patient had di

210 icance of the proportion of the spinal canal subdural hemorrhage in abusive head trauma versus that i

211 The comparison of incidences of spinal subdural hemorrhage in abusive head trauma versus those

212 21 years were tabulated for histopathology: subdural hemorrhage in the optic nerve sheath, intrascle

213 Spinal canal subdural hemorrhage was present in more than 60% of chil

214 he brain showed intraventricular hemorrhage, subdural hemorrhage, or intraparenchymal white matter mi

215 bidity Index, TBI sustained from a low fall, subdural hemorrhage, subarachnoid hemorrhage, higher Inj

216 rticularly cardioembolic stroke and possibly subdural hemorrhage, with sensitivity analyses showing s

217 maging, and 24 (63%) of 38 had thoracolumbar subdural hemorrhage.

218 are unit stay) and clinical characteristics (subdural hemorrhages and retinal hemorrhages) were compa

219 atients with intracerebral, subarachnoid, or subdural hemorrhages who had at least 1 follow-up image

220 t are characteristic of abusive head trauma--subdural hemorrhages, optic nerve sheath hemorrhages, an

221 We implanted a subdural, high-density, multielectrode array over the ar

222 As a conclusion, spontaneous subdural hygroma is a rare complication of the arachnoid

223 Subdural infusion of CA-I in rats induced cerebral vascu

224 tempted to mimic the actions of glutamate by subdural infusion of the selective glutamate receptor ag

225 Initiation of EAE or subdural injection of IL-1beta induces a similar cytokin

226 implanted with penetrating depth or surface subdural intracranial electrodes, heard auditory recordi

227 Our findings provide proof-of-principle that subdural intraspinal pressure at the injury site can be

228 Perivascular, subdural meningeal and choroid plexus macrophages are no

229 crophages (BAMs) residing in the dura mater, subdural meninges and choroid plexus consisted of distin

230 t leukocytes in CNS parenchyma, pia-enriched subdural meninges, dura mater, choroid plexus and cerebr

231 east the same or higher compare to effect of subdural motor or combined pre-motor and motor cortex st

232 16, 62%), followed by subpial (n = 4, 15%), subdural (n = 4, 15%), and parenchymal (n = 2, 8%) hemor

233 xtraction had a significantly higher rate of subdural or cerebral hemorrhage (odds ratio, 2.7; 95 per

234 dence interval, 1.8 to 3.4), but the rate of subdural or cerebral hemorrhage associated with vacuum e

235 heart failure, chest pain, other angina, and subdural or extradural haemorrhage.

236 ve patients with refractory epilepsy in whom subdural or intracerebral electrodes were implanted for

237 ither spontaneous intracerebral, spontaneous subdural, or postoperative.

238 ent ICH, including any new intraparenchymal, subdural, or subarachnoid hemorrhage after initiation of

239 udy demonstrate that the long-term effect of subdural pre-motor cortex stimulation is at least the sa

240 obes were inserted to simultaneously monitor subdural pressure below the injury and extradural pressu

241 pressure at the injury site was higher than subdural pressure below the injury or extradural pressur

242 brospinal fluid (CSF) dynamics, intracranial subdural pressure recordings were taken from sub-adult a

243 k of high frequency oscillations in adjacent subdural recording sites, despite the presence of a stro

244 When applied to the broader subdural recording, this measure consistently predicted

245 Here we describe subdural recordings from epileptic patients learning to

246 nature of MTL-PFC interactions, we examined subdural recordings from MTL and PFC in 21 neurosurgical

247 ing depolarizations were first identified in subdural recordings, and EEG was then examined visually

248 recordings in conjunction with intracranial subdural recordings, we asked whether fine duration disc

249 array allowed us to record both epidural and subdural responses at stimulation currents that are well

250 methods, such as intraventricular catheter, subdural screw, epidural sensor, lumbar puncture, are as

251 ction of 100 or 200 microl of blood into the subdural space (SDH) or into the caudate nucleus (ICH) o

252 isation to receive a drain inserted into the subdural space and 107 to no drain after evacuation.

253 The injection of blood into the subdural space or into the brain parenchyma induced bloo

254 n of 400 microl of autologous blood into the subdural space.

255 extending to cortex/insula, subarachnoid, or subdural spaces.

256 Insertion of subdural spinal pressure probe.

257 The electrode group was implanted with a subdural strip electrode providing up to 7 days of real-

258 ble, small molecules can diffuse through the subdural/subarachnoid space into the underlying neocorte

259 tal high frequency activity at the cortical (subdural) surface.

260 Seventeen neonates with ICHs (16 subdural, two subarachnoid, and six parenchymal hemorrha